Probabilistic phenomena are often perceived as being problematic targets for contrastive explanation. It is usually thought that the possibility of contrastive explanation hinges on whether or not the probabilistic behaviour is irreducibly indeterministic, and that the possible remaining contrastive explananda are token event probabilities or complete probability distributions over such token outcomes. This paper uses the invariance-under-interventions account of contrastive explanation to argue against both ideas. First, the problem of contrastive explanation also arises in cases in which the probabilistic behaviour (...) of the explanandum is due to unobserved causal heterogeneity. Second, it turns out that, in contrast to the case of pure indeterminism, the plausible contrastive explananda under causal heterogeneity are not token event probabilities, but population-level statistical facts. (shrink)

In a recent article in this journal, Federica Russo and Jon Williamson argue that an analysis of causality in terms of probabilistic relationships does not do justice to the use of mechanistic evidence to support causal claims. I will present Ronald Giere's theory of probabilistic causation, and show that it can account for the use of mechanistic evidence (both in the health sciences—on which Russo and Williamson focus—and elsewhere). I also review some other probabilistic theories of causation (of Suppes, Eells, (...) and Humphreys) and show that they cannot account for the use of mechanistic evidence. I argue that these theories are also inferior to Giere's theory in other respects. (shrink)

This paper outlines a general theory of efficient causation, a theory that deals in a unified way with traditional or deterministic, indeterministic, probabilistic, and other causal concepts. Theorists like Lewis, Salmon, and Suppes have attempted to broaden our causal perspective by reductively analysing causal notions in other terms. By contrast, the present theory rests in the first place on a non-reductive analysis of traditional causal concepts — into formal or structural components, on the one hand, and a physical or metaphysical (...) component, on the other. The analyzans is then generalised. The theory also affords a more general propensity notion than is standard, one that helps solve major problems facing propensity interpretations of probability. (shrink)

In a recent article (Sober 1982), I criticized an account of causation proposed by Giere (1979, 1980) by describing a series of examples concerning natural selection. Collier (1983) has criticized my criticisms, saying that I misapplied Giere's proposal and misconstrued the biology. More recently, Giere (1984) has defended his theory against my criticisms. Here I argue that my criticisms still stand.

The purpose of this essay is to investigate the properties of singular causal systems and their population manifestations, with special concern for the thesis of methodological individualism, which claims that there are no properties of social groups that cannot be adequately explained exclusively by reference to properties of individual members of those groups, i.e., at the level of individuals. Individuals, however, may be viewed as singular causal systems, i.e., as instantiations of (arrangements of) dispositional properties. From this perspective, methodological individualism (...) appears to be an ambiguous thesis: some properties of collections of (independent) systems of the same kind are reducible, but other properties of collections of (dependent) systems of the same kind are not. In cases of the first kind, therefore, methodological individualism is true, but trivial; while in cases of the second kind, it is significant, but false. Hence, if the arguments that follow are correct, at least some of the properties of social groups should qualify as emergent. (shrink)