In the present study we analyze if the risk concept of the hegemonic epidemiologychanges its nature in purportedly alternative currents as ecoepidemiology and socialepidemiology focused in multilevel analysis.We analyze the way this concept is distinguishedin every current and its relationship with other epidemiologic key notions as cause. We findthat the risk concept and the notion of cause remain relatively unchanged among the differentcurrents even when there is some theoretical discussion about the complexity of multilevelsystems and other explanations for the events. (...) Finally we discuss some consequences aboutthe appropriation of the risk concept in multiple interventions in the health field. We indicatethat the fragmented nature of the risk concept is problematic because it can make thesocial aspects of the disease considered only in a functional perspective. Alternatively to thatwe develop the vulnerability concept as a knowledge framed in a hermeneutical perspective.The vulnerability concept acts as a mediator knowledge between epidemiology and theinterventions in the health field. In the same way we point out some concerns from the anthropologicalfield about the simplification of social senses and omission of meanings aboutthe health of the communities. (shrink)

It is widely agreed that social factors are related to health outcomes: much research served to establish correlations between classes of social factors on the one hand and classes of disease on the other hand. However, why and how social factors are an active part in the aetiology of disease development is something that is gaining attention only recently in the health sciences and in the medical humanities. In this paper, we advance the view that, just as bio-markers help trace (...) the causal continuum from exposure to disease development at the biological level, socio-markers ought to be introduced and studied in order to trace the social continuum from exposure to disease development. We explain how socio-markers differ from social indicators and how they can be used in combination with bio-markers in order to reconstruct the mixed mechanisms of health and disease, namely mechanisms in which both biological and social factors have an active causal role. (shrink)