La thérapie rationnelle des émotions est basée sur l’hypothèse qu’un trouble de la pensée conduit à des troubles du sentiment qui eux-mêmes conduisent à des troubles de comportement. Du point de vue thérapeutique, la stratégie consiste à corriger les sentiments et le comportement en modifiant le trouble de raisonnement. Cette forme très en vogue de psychothérapie des troubles émotionnels fournit une illustration intéressante des relations nomologiques intriquées qui peuvent exister entre les patrons relativement fixes d’états émotionnels, d’états comportementaux et d’états (...) sensoriels physiologiques. En tant que telle, elle fournit une source d’évidences plausible en faveur de l’hypothèse que les émotions sont une forme spécialisée de système nomologique ou une « espèce ». Le succès de la théorie rationnelle des émotions jette le doute sur le mantra philosophique actuel qui refuse aux émotions le statut d’espèce naturelle, car elle suggère précisément l’inverse, c’est-à-dire que les émotions sont bel et bien une forme d’« espèce naturelle ». Cet appui n’a pas été pris en considération dans le débat en cours au sujet du statut d’espèce naturelle de l’émotion. Il est particulièrement intéressant de noter la façon dont il nous force à repenser la notion d’espèce naturelle telle qu’elle est employée dans les débats philosophiques sur la question. D’autres formulations qui correspondent mieux aux pratiques des sciences concernées pourraient être nécessaires. -/- [English abstrast] Rational Emotive Therapy is based on the hypothesis that disordered thinking leads to disordered feeling, which then leads to disordered behavior. Therapeutically, the strategy it is to correct disordered feeling and behavior by changing the disordered thinking. This immensely popular form of psychotherapy for emotional disorders provides an interesting illustration of the intricate law-like relationships that can exist between relatively fixed patterns of emotional, behavioral, and physiological sensory states. As such, it provides a plausible source of evidence for the hypothesis that emotions form a specialized law-like system or ‘kind’. Evidence of the success of Rational Emotive Therapy casts doubt on the contemporary philosophical mantra that emotions do not form a natural kind. It suggests precisely the opposite, namely, that emotions do form a ‘natural kind’ of some sort. This evidence has not been considered in the ongoing debate over the natural kind status of emotion. Especially interesting is how it forces us to rethink the way in which the notion of a natural kind is usually deployed in philosophical debates in this area. Alternative formulations more in line with the practices of the relevant sciences may be needed. (shrink)

Emotion Review, Volume 14, Issue 1, Page 3-14, January 2022. Advocates for the psychological construction of emotion view themselves as articulating a non-essentialist alternative to basic emotion theory's essentialist notion of affect programs. Psychological constructionists have also argued that holding essentialist assumptions about emotions engenders misconceptions about the psychological constructionist viewpoint. If so, it is important to understand what psychological constructionists mean by “essentialism” and “non-essentialism.” To advance the debate, I take a deeper dive into non-essentialism, comparing the non-essentialist views (...) of the early empiricists with those of the psychological constructionists, focusing on the theories of James Russell and Lisa Barrett. Using Lakatos’ notion of scientific research programs, I also describe how Russell's and Barrett's views have evolved into different and potentially competing research programs under the psychological constructionist banner. (shrink)

Philosophical discussions regarding the status of emotion as a scientific domain usually get framed in terms of the question whether emotion is a natural kind. That approach to the issues is wrongheaded for two reasons. First, it has led to an intractable philosophical impasse that ultimately misconstrues the character of the relevant debate in emotion science. Second, and most important, it entirely ignores valence, a central feature of emotion experience, and probably the most promising criterion for demarcating emotion from cognition (...) and other related domains. An alternate philosophical hypothesis for addressing the issues is pro- posed. It is that emotion is a naturally occurring valenced phenomenon that is.. (shrink)

The position advanced in this paper is that the bedrock of emotional feelings is contained within the evolved emotional action apparatus of mammalian brains. This dual-aspect monism approach to brain–mind functions, which asserts that emotional feelings may reflect the neurodynamics of brain systems that generate instinctual emotional behaviors, saves us from various conceptual conundrums. In coarse form, primary process affective consciousness seems to be fundamentally an unconditional “gift of nature” rather than an acquired skill, even though those systems facilitate skill (...) acquisition via various felt reinforcements. Affective consciousness, being a comparatively intrinsic function of the brain, shared homologously by all mammalian species, should be the easiest variant of consciousness to study in animals. This is not to deny that some secondary processes cannot be evaluated in animals with sufficiently clever behavioral learning procedures, as with place-preference procedures and the analysis of changes in learned behaviors after one has induced re-valuation of incentives. Rather, the claim is that a direct neuroscientific study of primary process emotional/affective states is best achieved through the study of the intrinsic , albeit experientially refined, emotional action tendencies of other animals. In this view, core emotional feelings may reflect the neurodynamic attractor landscapes of a variety of extended trans-diencephalic, limbic emotional action systems—including SEEKING, FEAR, RAGE, LUST, CARE, PANIC, and PLAY. Through a study of these brain systems, the neural infrastructure of human and animal affective consciousness may be revealed. Emotional feelings are instantiated in large-scale neurodynamics that can be most effectively monitored via the ethological analysis of emotional action tendencies and the accompanying brain neurochemical/electrical changes. The intrinsic coherence of such emotional responses is demonstrated by the fact that they can be provoked by electrical and chemical stimulation of specific brain zones—effects that are affectively laden. For substantive progress in this emerging research arena, animal brain researchers need to discuss affective brain functions more openly. Secondary awareness processes, because of their more conditional, contextually situated nature, are more difficult to understand in any neuroscientific detail. In other words, the information-processing brain functions, critical for cognitive consciousness, are harder to study in other animals than the more homologous emotional/motivational affective state functions of the brain. (shrink)

Major depression (MD) is a highly heterogeneous diagnostic category. Diverse symptoms such as sad mood, anhedonia, and fatigue are routinely added to an unweighted sum-score, and cutoffs are used to distinguish between depressed participants and healthy controls. Researchers then investigate outcome variables like MD risk factors, biomarkers, and treatment response in such samples. These practices presuppose that (1) depression is a discrete condition, and that (2) symptoms are interchangeable indicators of this latent disorder. Here I review these two assumptions, elucidate (...) their historical roots, show how deeply engrained they are in psychological and psychiatric research, and document that they contrast with evidence. Depression is not a consistent syndrome with clearly demarcated boundaries, and depression symptoms are not interchangeable indicators of an underlying disorder. Current research practices lump individuals with very different problems into one category, which has contributed to the remarkably slow progress in key research domains such as the development of efficacious antidepressants or the identification of biomarkers for depression. The recently proposed network framework offers an alternative to the problematic assumptions. MD is not understood as a distinct condition, but as heterogeneous symptom cluster that substantially overlaps with other syndromes such as anxiety disorders. MD is not framed as an underlying disease with a number of equivalent indicators, but as a network of symptoms that have direct causal influence on each other: insomnia can cause fatigue which then triggers concentration and psychomotor problems. This approach offers new opportunities for constructing an empirically based classification system and has broad implications for future research. (shrink)