This book aims to show that recent developments in neuroscience permit a defense of free will. Through language, human beings can escape strict biological determinism.

According to the subset view of realization, a property realizes another if the causal powers of the latter are a subset of those of the former. Against this view, some authors (in particular, Kevin Morris and Paul Audi) have argued that it has an untenable consequence that realizing properties are less fundamental than the properties they realize, because the subset view characterizes realized properties as parts (subsets) of their realizers whereas it is generally true that a part is prior to (...) its whole. This paper defends the subset view from this “part-whole” objection, by arguing that if we compare individual powers of realizer and realizee with particular attention to their manifestation conditions, it turns out that each power of a realizee is grounded in some power of its realizer. This grounding relation between powers, I shall argue, allows subset theorists to explain why a realizer is more fundamental than its realizee, even while having the latter as a part. (shrink)

Strong notions of free will are closely connected to the possibility to do otherwise as well as to an agent’s ability to causally influence her environment via her decisions controlling her actions. In this paper we employ techniques from the causal modeling literature to investigate whether a notion of free will subscribing to one or both of these requirements is compatible with naturalistic views of the world such as non-reductive physicalism to the background of determinism and indeterminism. We argue that (...) from a causal modeler’s perspective the only possibility to get both requirements consists in subscribing to reductive physicalism and indeterminism. (shrink)

Jaegwon Kim’s views on mental causation and the exclusion argument are evaluated systematically. Particular attention is paid to different theories of causation. It is argued that the exclusion argument and its premises do not cohere well with any systematic view of causation.

In contemporary philosophy of science, the consensus view seems to be that scientific explanations describe mechanisms responsible for the phenomena to be explained. Two kinds of explanatory relevance figure in mechanistic accounts of explanation: causal and constitutive. Following prominent accounts, it seems natural to analyze both these relations in terms of systematic interventions into some factor X with respect to another factor Y. However, such interventions are tailored to uncover causal relations only. Construing the constitutive relationship between parts and wholes (...) in terms of interventions thus raises metaphysical, conceptual, and epistemological questions. We here review the barriers that intervention-based inquiry into mechanisms encounters and consider some solutions. (shrink)

John Campbell has claimed that the interventionist account of causation must be amended if it is to be applied to causation in psychology. The problem, he argues, is that it follows from the so-called ‘surgical’ constraint that intervening on psychological states requires the suspension of the agent’s rational autonomy. In this paper, I argue that the problem Campbell identifies is in fact an instance of a wider problem for interventionism, extending beyond psychology, which I call the problem of ‘abrupt transitions’. (...) I then defend a solution to the problem, which replaces the surgical constraint with a weaker constraint on interventions that nevertheless does all the work the surgical constraint was designed to do. I conclude by exploring some interesting consequences of this weaker constraint for causation in psychology. (shrink)

This paper discusses the role of levels and level-bound theoretical terms in neurobiological explanations under the presupposition of a regularity theory of constitution. After presenting the definitions for the constitution relation and the notion of a mechanistic level in the sense of the regularity theory, the paper develops a set of inference rules that allow to determine whether two mechanisms referred to by one or more accepted explanations belong to the same level, or to different levels. The rules are characterized (...) as underlying level distinctions in neuroscience research. (shrink)

Carl Craver’s mutual manipulability criterion aims to pick out all and only those components of a mechanism that are constitutively relevant with respect to a given phenomenon. In devising his criterion, Craver has made heavy use of the notion of an ideal intervention, which is a tool for illuminating causal concepts in causal models. The problem is that typical mechanistic models contain non-causal relations in addition to causal ones, which is why the standard concept of an ideal intervention is not (...) appropriate in that context. In this paper, I first show how top-down interventions in mechanistic models violate the conditions for ideal interventions. Drawing from recent developments in the causal exclusion literature, I then argue for extended interventionism better suited for the purposes of the new mechanist. Finally, I show why adopting such an extended account leads to the surprising consequence that an important subset of mechanistic interlevel relations comes out as causal. (shrink)

This paper is concerned with the content of the causal proportionality constraint. It investigates two general versions of the constraint, namely “horizontal” and “vertical” proportionality. Moreover, it discusses whether proportionality is considered an ontic or an epistemic, i.e. explanatory, constraint on causation in the context of some of the most prominent theories of causation. The following main claims are defended: (1) The horizontal (HP) and the vertical version (VP) of the proportionality constraint are logically independent. (2) HP is implied by (...) some prominent theories of causation, not by others. (3) None of the discussed popular theories of causation contradicts either HP or VP. (4) HP and VP are not ontic or epistemic principles as such; rather, whether they are ontic or epistemic depends on the theories chosen plus background assumptions about the existence of higher-level causes and their non-identity to lower-level ones. (shrink)

Counterfactual conditionals have been appealed to in various ways to show how the mind can be causally efficacious. However, it has often been overestimated what the truth of certain counterfactuals actually indicates about causation. The paper first identifies four approaches that seem to commit precisely this mistake. The arguments discussed involve erroneous assumptions about the connection of counterfactual dependence and genuine causation, as well as a disregard of the requisite evaluation conditions of counterfactuals. In a second step, the paper uses (...) the insights of the foregoing analyses to formulate a set of counterfactuals-based conditions that are characterized as sufficient to establish singular causal claims. The paper concludes that there are ample reasons to believe that some mental events satisfy all these conditions with respect to certain further events and, hence, that mental events sometimes are causes. (shrink)

Supporters of the autonomy of higher-level causation (or explanation) often appeal to proportionality, arguing that higher-level causes are more proportional than their lower-level realizers. Recently, measures based on information theory and causal modeling have been proposed that allow one to shed new light on proportionality and the related notion of specificity. In this paper we apply ideas from this literature to the issue of higher vs. lower-level causation (and explanation). Surprisingly, proportionality turns out to be irrelevant for the question of (...) whether higher-level causes (or explanations) can be autonomous; specificity is a much more informative notion for this purpose. (shrink)

In this paper I reconstruct and evaluate the validity of two versions of causal exclusion arguments within the theory of causal Bayes nets. I argue that supervenience relations formally behave like causal relations. If this is correct, then it turns out that both versions of the exclusion argument are valid when assuming the causal Markov condition and the causal minimality condition. I also investigate some consequences for the recent discussion of causal exclusion arguments in the light of an interventionist theory (...) of causation such as Woodward's (2003) and discuss a possible objection to my causal Bayes net reconstruction. (shrink)

Strong notions of free will are closely connected to the possibility to do otherwise as well as to an agent’s ability to causally influence her environment via her decisions controlling her actions. In this paper we employ techniques from the causal modeling literature to investigate whether a notion of free will subscribing to one or both of these requirements is compatible with naturalistic views of the world such as non-reductive physicalism to the background of determinism and indeterminism. We argue that (...) from a causal modeler’s perspective the only possibility to get both requirements consists in subscribing to reductive physicalism and indeterminism. (shrink)

Part–whole causation (PWC) is the thesis that some causes are part of their effects. PWC has been objected to because of its incompatibility with the criterion that causes not be spatially included within their effects and the criterion that causes and effects are ontologically distinct in some sense. This paper serves to undermine the sufficiency of these ways of objecting to PWC by showing that for each criterion either cause-effect relationships need not satisfy it or part–whole relationships can. A case-study (...) of the collapse of Bridge 9340 is employed to facilitate the discussion. (shrink)

Part–whole causation is the thesis that some causes are part of their effects. PWC has been objected to because of its incompatibility with the criterion that causes not be spatially included within their effects and the criterion that causes and effects are ontologically distinct in some sense. This paper serves to undermine the sufficiency of these ways of objecting to PWC by showing that for each criterion either cause-effect relationships need not satisfy it or part–whole relationships can. A case-study of (...) the collapse of Bridge 9340 is employed to facilitate the discussion. (shrink)

The causal exclusion argument suggests that mental causes are excluded in favour of the underlying physical causes that do all the causal work. Recently, a debate has emerged concerning the possibility of avoiding this conclusion by adopting Woodward's interventionist theory of causation. Both proponents and opponents of the interventionist solution crucially rely on the notion of supervenience when formulating their positions. In this article, we consider the relation between interventionism and supervenience in detail and argue that importing supervenience relations into (...) the interventionist framework is deeply problematic. However, rather than reject interventionist solutions to exclusion wholesale, we wish to propose that the problem lies with the concept of supervenience. This would open the door for a moderate defence of the interventionist solution to the exclusion argument. (shrink)

The relationship between psychological states and the brain remains an unresolved issue in philosophy of psychology. One appealing solution that has been influential both in science and in philosophy is Dennett’s concept of the intentional stance, according to which beliefs and desires are real and objective phenomena, but not necessarily states of the brain. A fundamental shortcoming of this approach is that it does not seem to leave any causal role for beliefs and desires in influencing behavior. In this paper, (...) I show that intentional states ascribed from the intentional stance should be seen as real causes, develop this to an independently plausible ontological position, and present a response to the latest interventionist causal exclusion worries. (shrink)

This paper argues that the interventionist account of causation faces a dilemma concerning macroscopic causation – i.e., causation by composite objects. Interventionism must either require interventions on a composite object to hold the behavior of its parts fixed, or allow such interventions to vary the behavior of those parts. The first option runs the risk of making wholes causally excluded by their parts, while the second runs the risk of mistakenly ascribing to wholes causal abilities that belong to their parts (...) only. Using as starting point Baumgartner’s well-known argument that interventionism leads to causal exclusion of multiply realized properties, I first show that a similar interventionist exclusion argument can be mounted against the causal efficacy of composite objects. I then show that Woodward’s (2015) updated interventionist account (explicitly designed to address exclusion worries) avoids this problem, but runs into an opposite issue of over-inclusion: it grants to composites causal abilities that belong to their parts only. Finally, I examine two other interventionist accounts designed to address Baumgartner’s argument, and show that when applied to composites, they too fall on one horn (exclusion) or the other (over-inclusion) of the dilemma. I conclude that the dilemma constitutes an open and difficult issue for interventionism. (shrink)

An important question for the causal modeling approach is how to integrate non‐causal dependence relations such as asymmetric supervenience into the approach. The most prominent proposal to that effect (due to Gebharter) is to treat those dependence relationships as formally analogous to causal relationships. We argue that this proposal neglects some crucial differences between causal and non‐causal dependencies, and that in the context of causal modeling non‐causal dependence relationships should be represented as mutual dependence relationships. We develop a new kind (...) of model – “hybrid models” ‐ based on this suggestion, and formulate a set of axioms for those models. Our formalism has important implications for Kim's exclusion problem: whereas Gebharter's framework vindicates Kim's causal exclusion objection against nonreductive physicalism, our framework has no such implication, and can help non‐reductive physicalists vindicate the efficacy of high‐level properties. A further benefit of our formalism is that it yields a natural and plausible way of thinking about interventions in multi‐level contexts. (shrink)

In this paper, we examine the causal framework within which integrated information theory of consciousness makes it claims. We argue that, in its current formulation, IIT is threatened by the causal exclusion problem. Some proponents of IIT have attempted to thwart the causal exclusion problem by arguing that IIT has the resources to demonstrate genuine causal emergence at macro scales. In contrast, we argue that their proposed solution to the problem is damagingly circular as a result of inter-defining information and (...) causation. As a solution, we propose that IIT should adopt the specific interventionist causal framework that we offer and show how IIT can harness this interventionist framework to avoid the causal exclusion problem. We demonstrate how our argument remains fully compatible with the methodology, empirical data, and conceptual aims of the theory. (shrink)

It has become a popular view among non-reductive physicalists that it is possible to devise empirical tests generating evidence for the causal efficacy of the mental, whereby the exclusion worries that have haunted the position of non-reductive physicalism for decades can be dissolved once and for all. This paper aims to show that these evidentialist hopes are vain. I argue that, if the mental is taken to supervene non-reductively on the physical, there cannot exist empirical evidence for its causal efficacy. (...) While causal structures without non-reductive supervenience relations can be conclusively identified in ideal discovery circumstances, it is impossible, in principle, to generate evidence that would favour models with mental causation over models without. Ascribing causal efficacy to the mental, for the non-reductive physicalist, is a modelling choice that must be made on the basis of metaphysical background theories or pragmatic maxims guiding the selection among empirically indistinguishable models. (shrink)

The first part of this paper argues that if Craver’s ([2007a], [2007b]) popular mutual manipulability account (MM) of mechanistic constitution is embedded within Woodward’s ([2003]) interventionist theory of causation--for which it is explicitly designed--it either undermines the mechanistic research paradigm by entailing that there do not exist relationships of constitutive relevance or it gives rise to the unwanted consequence that constitution is a form of causation. The second part shows how Woodward’s theory can be adapted in such a way that (...) (MM) neither undermines the mechanistic paradigm nor reduces constitution to causation. However, it turns out that this modified theoretical embedding of (MM) makes it impossible to produce empirical evidence for constitutive relations. The paper ends by suggesting an additional criterion, the fat-handedness criterion, which, when combined with (MM), generates indirect empirical evidence for constitutive relevance. (shrink)

Levels of organization are structures in nature, usually defined by part-whole relationships, with things at higher levels being composed of things at the next lower level. Typical levels of organization that one finds in the literature include the atomic, molecular, cellular, tissue, organ, organismal, group, population, community, ecosystem, landscape, and biosphere levels. References to levels of organization and related hierarchical depictions of nature are prominent in the life sciences and their philosophical study, and appear not only in introductory textbooks and (...) lectures, but also in cutting-edge research articles and reviews. In philosophy, perennial debates such as reduction, emergence, mechanistic explanation, interdisciplinary relations, natural selection, and many other topics, also rely substantially on the notion. -/- Yet, in spite of the ubiquity of the notion, levels of organization have received little explicit attention in biology or its philosophy. Usually they appear in the background as an implicit conceptual framework that is associated with vague intuitions. Attempts at providing general and broadly applicable definitions of levels of organization have not met wide acceptance. In recent years, several authors have put forward localized and minimalistic accounts of levels, and others have raised doubts about the usefulness of the notion as a whole. -/- There are many kinds of ‘levels’ that one may find in philosophy, science, and everyday life—the term is notoriously ambiguous. Besides levels of organization, there are levels of abstraction, realization, being, analysis, processing, theory, science, complexity, and many others. In this article, the focus will be on levels of organization and debates associated with them, and other kinds of levels will only be discussed when they are relevant to this main topic. (shrink)

In this dissertation, I develop and defend a model of causation that allows for dualist mental causation in worlds where the physical domain is physically complete. In Part I, I present the dualist ontology that will be assumed throughout the thesis and identify two challenges for models of mental causation within such an ontology: the exclusion worry and the common cause worry. I also argue that a proper response to these challenges requires a thoroughly lightweight account of causation, i.e. an (...) account that allows for causes to be metaphysically distinct from the phenomena that produce or physically necessitate their effects. In Part II, I critically evaluate contemporary responses to these challenges from the philosophical literature. In particular, I discuss List and Stoljar’s criticism of exclusion worries, Kroedel’s alternative dualist ontology, concerns about the notion of causal sufficiency, and Lowe’s models of dualist mental causation. I argue that none of these proposals provide independent motivation for a thoroughly lightweight account of causation and therefore leave room for improvement. In the first four chapters of Part III, I develop a thoroughly lightweight model of causation, which builds on interventionist approaches to causation. First, I explain how so-called ‘holding fixed’-requirements in standard interventionist accounts stand in the way of dualist mental causation. I then argue that interventionist accounts should impose a robustness condition on causal correlations and that, with this condition in place, the ‘holding fixed’-requirements can be weakened such that they do allow for dualist mental causation. I dub the interventionist model with such weakened ‘holding fixed’-requirements ‘insensitive interventionism’, argue that it can counter the exclusion worry as well as the common cause worry, and explain under which circumstances it would predict there to be dualist mental causation. Importantly, these circumstances might, for all we know, hold in the actual world. In the final three chapters of Part III, I defend insensitive interventionism against some objections. I consider the objection that causation must be productive, the objection that causes must physically necessitate their effects, and the objection that insensitive interventionism is too permissive. I respond by drawing from the literature on causation by absences and on the relation between causation and fundamental physics. Overall, insensitive interventionism performs as well as standard interventionist accounts. I conclude that insensitive interventionism is a credible model of causation. (shrink)

Jaegwon Kim famously challenged non-reductive materialism/physicalism, a popular stance in the philosophy of mind, with the so-called exclusion argument. The argument is alleged to show that if NRM is true, then mental properties cannot be causes. In recent years, there have been many reactions to the exclusion problem based on counterfactual accounts of causation. In particular, List and Menzies gave an interesting response based on a Lewis-style counterfactual theory of causation, with some modifications made to Lewis’s semantics of counterfactuals. In (...) this thesis, I first argue that their central thesis regarding the issue of exclusion can actually be established with Lewis’s original semantics, without modifications. I then clarify the real consequence of List and Menzies’ modification by showing that List and Menzies' modified counterfactual theory, but not Lewis’s original theory, can satisfy Zhong ’s requirement of causal autonomy. My analysis reveals that, despite Zhong ’s employment of an interventionist theory of causation, it is not any peculiarity with the interventionism that is essential for establishing the possibility of causal autonomy. On the contrary, I argue that the existing semantics for interventionist counterfactuals do not seem to serve Zhong’s purpose particularly well. These considerations suggest that once one decides to use a dependence notion of causation to counter the exclusion argument, a Lewis-style theory of causation is good enough. (shrink)

Explanations in psychiatry often integrate various factors relevant to psychopathology. Identifying genuine causes among them is theoretically and clinically important, but epistemically challenging. Woodward’s interventionism appears to provide a promising tool to achieve this. However, Woodward’s interventionism is too demanding to be applied to psychiatry. I thus introduce difference-making interventionism, which detects relevance in general rather than causation, to make interventionist reasoning viable in clinical practice. DMI mirrors the empirical reality of psychiatry even more closely than interventionism, but it needs (...) to be supplied with additional heuristics to disambiguate between causes and other difference-makers. To achieve this, I suggest employing heuristics based on multiple experiments, temporal order and scientific domain. (shrink)