Richard Otte (1985) has recently criticized the resolution of Simpson's paradox given by Nancy Cartwright (1979). He argues that there are difficulties with the version of the theory of probabilistic causality that Cartwright has developed, and that there is a way in which Simpson's paradox can arise that Cartwright's theory cannot handle. And Otte develops his own theory of probabilistic causality. I defend Cartwright's solution, and I argue that there are difficulties with the theory of probabilistic causality that Otte proposes.

According to Nancy Cartwright, a causal law holds just when a certain probabilistic condition obtains in all test situations which in turn satisfy a set of background conditions. These background conditions are shown to be inconsistent and, on separate account, logically incoherent. I offer a corrective reformulation which also incorporates a strategy for problems like Hesslow's thrombosis case. I also show that Cartwright's recent argument for modifying the condition to appeal to singular causes fails.Proposed modifications of the theory's probabilistic condition (...) to handle effects with extreme probabilities (0 or 1) are found unsatisfactory. I propose a unified solution which also handles extreme causes. Undefined conditional probabilities give rise to three good, but non-equivalent, ways of formulating the theory. Various formulations appear in the literature. I give arguments to eliminate all but one candidate. Finally, I argue for a crucial new condition clause, and show how to extend the results beyond a simple probabilistic framework. (shrink)

The purpose of this essay is to investigate the properties of singular causal systems and their population manifestations, with special concern for the thesis of methodological individualism, which claims that there are no properties of social groups that cannot be adequately explained exclusively by reference to properties of individual members of those groups, i.e., at the level of individuals. Individuals, however, may be viewed as singular causal systems, i.e., as instantiations of (arrangements of) dispositional properties. From this perspective, methodological individualism (...) appears to be an ambiguous thesis: some properties of collections of (independent) systems of the same kind are reducible, but other properties of collections of (dependent) systems of the same kind are not. In cases of the first kind, therefore, methodological individualism is true, but trivial; while in cases of the second kind, it is significant, but false. Hence, if the arguments that follow are correct, at least some of the properties of social groups should qualify as emergent. (shrink)

In an earlier paper (Dupré 1984), I criticized a thesis sometimes defended by theorists of probabilistic causality, namely, that a probabilistic cause must raise the probability of its effect in every possible set of causally relevant background conditions (the "contextual unanimity thesis"). I also suggested that a more promising analysis of probabilistic causality might be sought in terms of statistical relevance in a fair sample. Ellery Eells (1987) has defended the contextual unanimity thesis against my objections, and also raised objections (...) of his own to my positive claims. In this paper I defend and amplify both my objections to the contextual unanimity thesis and my constructive suggestion. (shrink)

Philosophers of science, e.g. Cartwright (1979) and Skyrms (1980), have given accounts of general causation in probabilistic terms; not surprising since we often accept general causal sentences as true despite being aware of apparent counterinstances. Consider:(E1) Smoking is a cause of heart attacks.We accept (E1) as true though aware of smokers who never have a heart attack. Here I quickly review familiar problems in two existing probabilistic accounts, offer a new objection, and suggest the beginnings of an alternative account.The traditional (...) model of general causation maintains that causes, and only causes, raise the probability of the effect That is:(TM) F is a cause of G if and only if.Despite some intuitive appeal, the traditional model faces at least three basic objections. First, consider cases of epiphenomena in which there are properties F, G, and H such that F is a cause of G, F is a cause of H, but G is not a cause of H and H is not a cause of G. (shrink)

The unanimity theory is an account of property-level causation requiring that causes raise the probability of their effects in specified test situations. Richard Otte (1981) and others have presented counterexamples in which one property is probabilistically sufficient for at least one other property. Given the continuing discussion (e.g., Cartwright 1989; Cartwright and Dupre 1988; Eells 1988a,b), many apparently think that these problems are minor. By considering the impact of Otte's cases on recent versions of the theory, by raising several new (...) examples, and by criticizing natural replies, I argue that the problems for the unanimity theory are severe. (shrink)