In contrast to the opinion of numerous authors (e.g. R. Rudner, P. Kitcher, L. R. Graham, M. Dummett, N. Chomsky, R. Lewontin, etc.) it is argued here that the formation of opinion in science should be greatly insulated from political considerations. Special attention is devoted to the view that methodological standards for evaluation of scientific theories ought to vary according to the envisaged political uses of these theories.

This article is a synopsis of a triarchic theory of human intelligence. The theory comprises three subtheories: a contextual subtheory, which relates intelligence to the external world of the individual; a componential subtheory, which relates intelligence to the individual's internal world; and a two-facet subtheory, which relates intelligence to both the external and internal worlds. The contextual subtheory defines intelligent behavior in terms of purposive adaptation to, shaping of, and selection of real-world environments relevant to one's life. The normal course (...) of intelligent functioning in the everyday world entails adaptation to the environment; when the environment does not fit one's values, aptitudes, or interests, one may attempt to shape the environment so as to achieve a better person-environment fit; when shaping fails, an attempt may be made to select a new environment that provides a better fit. The two-facet subtheory further constrains this definition by regarding as most relevant to the demonstration of intelligence contextually intelligent behavior that involves either adaptation to novelty, automatization of information processing, or both. Efficacious automatization of processing allows allocation of additional resources to the processing of novelty in the environment; conversely, efficacious adaptation to novelty allows automatization to occur earlier in one's experience with new tasks and situations. The componential subtheory specifies the mental mechanisms responsible for the learning, planning, execution, and evaluation of intelligent behavior. Metacomponents of intelligence control one's information processing and enable one to monitor and later evaluate it; performance components execute the plans constructed by the metacomponents; knowledge-acquisition components selectively encode and combine new information and selectively compare new information to old so as to allow new information to be learned. (shrink)

Males are selectively afflicted with the neurodevelopmental and psychiatric disorders of childhood, a broad and virtually ubiquitous phenomenon that has not received proper attention in the biological study of sex differences. The previous literature has alluded to psychosocial differences, genetic factors and elements pertaining to male “complexity” and relative immaturity, but these are not deemed an adequate explanation for selective male affliction. The structure of sex differences in neurodevelopmental disorders is hypothesized to contain these elements: Males are more frequently afflicted, (...) females more severely; disorders arising in females are largely mediated by the genotype; in males, by a genotype by environment interaction; complications of pregnancy and delivery occur more frequently with male births; such complications are decisive and influence subsequent development. We hypothesize that there is something about the male fetus that evokes an inhospitable uterine environment. This “evocative principle” is hypothesized to relate to the relative antigenicity of the male fetus, which may induce a state of maternal immunoreactivity, leading either directly or indirectly to fetal damage. The immunoreactive theory thus constructed is borrowed from studies of sex ratios and is the only explanation consistent with negative parity effects in the occurrence of pregnancy complications and certain neurodevelopmental disorders. Although the theory is necessarily speculative, it is heuristic and hypotheses derived from it are proposed; some are confirmed in the existing literature and by the authors' research. (shrink)

City College of New York The hereditarian theory of race differences in IQ was briefly revived with the appearance of The Bell Curve but then quickly dismissed. The authors attempt a defense of it here, with an eye to conceptual and logical issues of special interests to philosophers, such as alleged infirmities in the heritability concept. At the same time, some relevant post-Bell Curve empirical data are introduced.

Philosophers of science widely believe that the hereditarian theory about racial differences in IQ is based on methodological mistakes and confusions involving the concept of heritability. I argue that this "received view" is wrong: methodological criticisms popular among philosophers are seriously misconceived, and the discussion in philosophy of science about these matters is largely disconnected from the real, empirically complex issues debated in science.

In this paper, I argue that epistemic injustice can result from transformative learning. Transformative learning causes a radical change in the structure of a student’s personal epistemic resources to bring them in line with the structure of a discipline’s shared epistemic resources. When those shared epistemic resources are biased, this transformation prevents students from retaining aspects of their personal epistemic resources which it is strongly in their interests (as well as in the interests of the broader epistemic community) to retain. (...) In these cases, transformative learning causes epistemic injustice in the form of inferential injustice. (shrink)

Genetic differences can lead to phenotypic differences either directly or indirectly (via causing differences in external environments, which then affect phenotype). This possibility of genetic effects being mediated by environmental influences is often used by scientists and philosophers to argue that heritability is not a very helpful causal or explanatory notion. In this paper it is shown that these criticisms are based on serious misconceptions about methods of behavior genetics.

Many psychometricians and behavioral geneticists believe that high heritability of IQ test scores within racial groups coupled with environmental hypotheses failing to account for the differences between the mean scores for groups lends plausibility to explanations of mean differences in terms of genetic factors. This two-component argument cannot be sustained when viewed in the light of the conceptual and methodological themes introduced in Taylor . These themes concern the difficulties of moving from the statistical analysis of variance of observed traits (...) to investigation of measurable genetic factors and measurable environmental factors. One such theme is that quantities estimated by an AOV of observed traits cannot be equated with measurable genetic or environmental factors involved in the development of those traits. Once this distinction is clear, the argument that environmental factors have failed to explain the differences lacks weight because it does not consider whether genetic factors have been more successful. This article exposes additional flaws in the lines of thinking associated with the two-component argument, with the distinction between passive, reactive, and active associations between genetic and environmental factors, and with the reciprocal causation models Dickens and Flynn propose in order to reconcile high estimates of heritability and large IQ test score gains between generations. Human heritability estimates are irrelevant in developing explanations of differences across groups or across generations. My critique is directed at opening up more conceptual space for deriving empirically validated models of developmental pathways whose components are heterogeneous and differ among individuals at any one time and over generations. (shrink)