Anosognosia for visual deficits—cases where significant visual deficits go unnoticed—challenges the view that our own conscious experiences are what we know best. We review these widespread and striking failures of awareness. Anosognosia can occur with total blindness, visual abnormalities induced by brain lesions, and eye diseases. We show that anosognosia for visual deficits is surprisingly widespread. Building on previous accounts, we introduce a framework showing how apparently disparate forms of anosognosia fit together. The central idea is that, to notice a (...) deficit, individuals need to form expectations about normal vision, compare expectations and visual input, and judge any mismatch at the metacognitive level. A failure in any of these three steps may lead to unawareness of visual deficits. (shrink)

Different reasons give rise to the question, what philosophy really is, and by tradition we know many answers. Plato’s answer can be found by examining his explicit statements about philosophy in his dialogues, or by analyzing his representation of Socrates – philosophy become fl esh. But an other way to fi nd an answer to the question lies in examining the things which – according to Plato – we cannot do without. There are three of them, namely the idea, logos (...) and aporia. These three taken together – the insight that we orient ourselves according to some unity in our different fi elds of life; that we cannot dismiss dialogue and debate; and questions outlive answers – paint a picture of philosophy as «unbehauptendes Denken»1 [undogmatic thought]. Such «unbehauptendes Denken» does not aim to insist on new claims against old ones, but instead seeks to analyze and to refl ect upon old views. (shrink)

We pose a foundational problem for those who claim that subjects are ontologically irreducible, but causally reducible (weak emergence). This problem is neuroscience’s notorious binding problem, which concerns how distributed neural areas produce unified mental objects (such as perceptions) and the unified subject that experiences them. Synchrony, synapses and other mechanisms cannot explain this. We argue that this problem seriously threatens popular claims that mental causality is reducible to neural causality. Weak emergence additionally raises evolutionary worries about how we’ve survived (...) the perils of nature. Our emergent subject hypothesis (ESH) avoids these shortcomings. Here, a singular, unified subject acts back on the neurons it emerges from and binds sensory features into unified mental objects. Serving as the mind’s controlling center, this subject is ontologically and causally irreducible (strong emergence). ESH draws on recent experimental evidence, including the motivation of a possible correlate (or “seat”) of the subject, which enhances its testability. (shrink)