This paper attempts to elucidate three characteristics of causal relationships that are important in biological contexts. Stability has to do with whether a causal relationship continues to hold under changes in background conditions. Proportionality has to do with whether changes in the state of the cause “line up” in the right way with changes in the state of the effect and with whether the cause and effect are characterized in a way that contains irrelevant detail. Specificity is connected both to (...) David Lewis’ notion of “influence” and also with the extent to which a causal relation approximates to the ideal of one cause–one effect. Interrelations among these notions and their possible biological significance are also discussed. (shrink)

Price’s :157–176, 1991; 44:187–203, 1993 ; 2007, 2017) agency theory of causation has takes itself to provide a use-theory of our causal discourse. The theory’s aim is to describe the rules implicit to our linguistic behaviour when we describe things in causal terms. According to this theory, the rules governing our use of the concept of causation are based on our perspective as agents and our associated experiences of manipulating events. I argue that the observed relation between agency and our (...) concept of causation cannot exhaustively describe the conditions under which we enter into causal discourse. In particular, I demonstrate that the agency theory faces familiar problems with accounting for causal ascriptions to token cases. (shrink)

David Lewis championed a counterfactual account of causation, but counterfactual accounts have a notoriously difficult time handling cases of late preemption. These are cases in which we still count one event as the cause of another although the effect does not depend on the cause in the way taken to be necessary by the account. Lewis recognized these cases, but they have been shown to be problematic even for his final analysis of causation, the Influence Account. In this paper, I (...) show how these cases led to and remain a problem for Lewis’s Influence Account, and I give a new counterfactual account available to a Lewisian that can handle late preemption cases. What is crucial to my account is that a cause not merely has counterfactual influence over its effects, but that the cause has influence of the right kind. I conclude by arguing that even if problems remain with this account, they will not be problems of late preemption, and the problems stemming from other kinds of cases will be no worse for my account than for other counterfactual accounts. (shrink)

This paper is an investigation into the notion of degree of influence, and its application to the problem of pre-emption. In 'Causation as Influence', Lewis presented a new account of causation under determinism and some new observations on the problem of pre-emption. He claimed that, in cases of pre-emption, the pre-empting cause is much more of a cause than its pre-empted alternative; it has much more influence. I begin by trying to make sense of the notion of degree of influence. (...) Then I emend Lewis's approach to pre-emption in response to objections, compare it to Kvart's Sustainably Reducible Influence account, and finally conclude that all these accounts fail to solve the problem of pre-emption. (shrink)

In this paper, I will first clarify Lewis’s influence theory of causation by relying on his theory of events. And then I will consider Michael Strevens’s charge against the sufficiency of Lewis’s theory. My claim is that it is legitimate but does not pose as serious a problem for Lewis’s theory as Strevens thinks because Lewis can surmount it by limiting the scope of his theory to causation between concrete events. Michael Strevens raises an alleged counterexample to the necessity of (...) Lewis’s theory that, if successful, would have a very important advantage over other alleged counterexamples. But I will assert that it is simply mistaken. My defense of Lewis’s theory will shed interesting light on the relationship between Lewis’s theory and Salmon’s mark theory. (shrink)

The existence of non-local correlations between outcomes of measurements in quantum entangled systems strongly suggests that we are dealing with some form of causation here. An assessment of this conjecture in the context of the collapse interpretation of quantum mechanics is the primary goal of this paper. Following the counterfactual approach to causation, I argue that the details of the underlying causal mechanism which could explain the non-local correlations in entangled states strongly depend on the adopted semantics for counterfactuals. Several (...) relativistically-invariant interpretations of spatiotemporal counterfactual conditionals are discussed, and the corresponding causal stories describing interactions between parts of an entangled system are evaluated. It is observed that the most controversial feature of the postulated causal connections is not so much their non-local character as a peculiar type of circularity that affects them. (shrink)

Whether there exist causal relations between guns firing and people dying, between pedals pressed and cars accelerating, or between carbon dioxide emissions and global warming, is typically taken to be a mind-independent, objective, matter of fact. However, recent contributions to the literature on causation, in particular theories of contrastive causation and causal modelling, have undermined this central causal platitude by relativising causal facts to models or to interests. This thesis flies against the prevailing wind by arguing that we must pay (...) greater attention to which elements of our causal talk vary with context and which elements track genuine features of the world around us. I will argue that once these elements are teased apart we will be in a position to better understand some of the most persistent problems in the philosophy of causation: pre-emption cases, absence causation, failures of transitivity and overdetermination. The result is a naturalist account of causation, concordant with the contextual variability we find in our ordinary causal talk, and parsimonious with respect to the theoretical entities posited. (shrink)