This article describes abductions as special patterns of inference to the best explanation whose structure determines a particularly promising abductive conjecture and thus serves as an abductive search strategy. A classification of different patterns of abduction is provided which intends to be as complete as possible. An important distinction is that between selective abductions, which choose an optimal candidate from given multitude of possible explanations, and creative abductions, which introduce new theoretical models or concepts. While selective abduction has dominated the (...) literature, creative abductions are rarely discussed, although they are essential in science. The article introduces several kinds of creative abductions, such as theoretical model abduction, common cause abduction and statistical factor analysis, and illustrates them by various real case examples. It is suggested to demarcate scientifically fruitful abductions from purely speculative abductions by the criterion of causal unification. (shrink)

The existing philosophical views on what is the meaning of causality adequate to medicine are vastly divided. We approach this question and offer two arguments in favor of pluralism regarding concepts of causality. First, we analyze the three main types of research designs (randomized-controlled trials, observational epidemiology and laboratory research). We argue, using examples, that they allow for making causal conclusions that are best understood differently in each case (in agreement with a version of manipulationist, probabilistic and mechanistic definitions, respectively). (...) Second, we analyze clinical practice and argue that these manipulationist, probabilistic and mechanistic causal claims can be used as evidence for different therapeutic decisions. We differentiate among ‘predicting’ that does not change the relata of causal claims, (mechanistic) ‘interferences’, and ‘interventions’ in the strict sense that act on causes to change effects. The central conclusion is that causal claims agreeing with diverse concepts of causality can deliver evidence for different types of therapeutic decisions. (shrink)

This paper examines Patrick Suppes’ probabilistic theory of causality understood as a theory of causal inference, and draws some lessons for empirical economics and contemporary debates in the foundations of econometrics. It argues that a standard method of empirical economics, multiple regression, is inadequate for most but the simplest applications, that the Bayes’ nets approach, which can be understood as a generalisation of Suppes’ theory, constitutes a considerable improvement but is still subject to important limitations, and that the currently fashionable (...) ‘design-based approach’ suffers from the same flaws Suppes anticipated a long time ago. It then sketches an alternative in response, one that differs drastically from the formalisms Suppes endorsed but is consistent with his pragmatic general take on science. (shrink)

The characteristic difference between laws and accidental generalizations lies in our epistemic or inductive attitude towards them. This idea has taken various forms and dominated the discussion about lawlikeness in the last decades. Hence, ranking theory with its resources of formalizing defeasible reasoning or inductive schemes seems ideally suited to explicate the idea in a formal way. This is what the paper attempts to do. Thus it will turn out that a law is simply the deterministic analogue of a sequence (...) of independent, identically distributed random variables. This entails that de Finetti‘s representation theorems can be directly transformed into an account of confirmation of laws thus conceived. (shrink)

According to Nancy Cartwright, a causal law holds just when a certain probabilistic condition obtains in all test situations which in turn satisfy a set of background conditions. These background conditions are shown to be inconsistent and, on separate account, logically incoherent. I offer a corrective reformulation which also incorporates a strategy for problems like Hesslow's thrombosis case. I also show that Cartwright's recent argument for modifying the condition to appeal to singular causes fails.Proposed modifications of the theory's probabilistic condition (...) to handle effects with extreme probabilities (0 or 1) are found unsatisfactory. I propose a unified solution which also handles extreme causes. Undefined conditional probabilities give rise to three good, but non-equivalent, ways of formulating the theory. Various formulations appear in the literature. I give arguments to eliminate all but one candidate. Finally, I argue for a crucial new condition clause, and show how to extend the results beyond a simple probabilistic framework. (shrink)

In a recent article (Sober 1982), I criticized an account of causation proposed by Giere (1979, 1980) by describing a series of examples concerning natural selection. Collier (1983) has criticized my criticisms, saying that I misapplied Giere's proposal and misconstrued the biology. More recently, Giere (1984) has defended his theory against my criticisms. Here I argue that my criticisms still stand.

After clarifying the probabilistic conception of causality suggested by Good (1961-2), Suppes (1970), Cartwright (1979), and Skyrms (1980), we prove a sufficient condition for transitivity of causal chains. The bearing of these considerations on the units of selection problem in evolutionary theory and on the Newcomb paradox in decision theory is then discussed.

This paper outlines a general theory of efficient causation, a theory that deals in a unified way with traditional or deterministic, indeterministic, probabilistic, and other causal concepts. Theorists like Lewis, Salmon, and Suppes have attempted to broaden our causal perspective by reductively analysing causal notions in other terms. By contrast, the present theory rests in the first place on a non-reductive analysis of traditional causal concepts — into formal or structural components, on the one hand, and a physical or metaphysical (...) component, on the other. The analyzans is then generalised. The theory also affords a more general propensity notion than is standard, one that helps solve major problems facing propensity interpretations of probability. (shrink)

In this paper I wish to argue that counterfactual analyses of causation are inadequate. I believe the counterfactuals that are involved in counterfactual analyses of causation are often false, and thus the theories do not provide an adequate account of causation. This is demonstrated by the presentation of a counterexample to the counterfactual analyses of causation. I then present a unified theory of causation that is based upon probability and counterfactuals. This theory accounts for both deterministic and indeterministic causation, and (...) is not subject to many of the traditional problems facing theories of causation. (shrink)

This paper describes the development of theories of scientific explanation since Hempel's earliest models in the 1940ies. It focuses on deductive and probabilistic whyexplanations and their main problems: lawlikeness, explanation-prediction asymmetries, causality, deductive and probabilistic relevance, maximal specifity and homogenity, the height of the probability value. For all of these topic the paper explains the most important approaches as well as their criticism, including the author's own accounts. Three main theses of this paper are: (1) Both deductive and probabilistic explanations (...) are important in science, not reducible to each other. (2) One must distinguish between (cause giving) explanations and (reason giving) justifications and predictions. (3) The adequacy of deductive as well as probabilistic explanations is relative to a pragmatically given background knowledge-which does not exclude, however, the possibility of purely semantic models. (shrink)

A probabilistic and counterfactual theory of causality is developed within the framework of branching time. The theory combines ideas developed by James Fetzer, Donald Nute, Patrick Suppes, Ming Xu, John Pollock, David Lewis and Mellor among others.

Darwinians are realists about the force of selection, but there has been surprisingly little discussion about what form this realism should take. Arguments about the units of selection in general and genic selectionism in particular reveal two realist assumptions: (1) for any selection process, there is a uniquely correct identification of the operative selective forces and the level at which each impinges; and (2) selective forces must satisfy the Pareto-style requirement of probabilistic causation. I argue that both assumptions are false; (...) we must temper realism about the force of selection and revise the way we think about probabilistic causation. (shrink)

There have been two principal paradigms for the formulation of the causal relation--logical implication and functional relationship. In this paper, I present a case for preferring the functional relationship formulation and then discuss how the functional relationship formulation may be implemented in the probabilistic case in a manner analogous to the way others have implemented the logical implication formulation in the probabilistic case. I show how the "local independence" assumption found in many models used in the behavioral and social sciences (...) is a consequence of the functional relation concept of probabilistic causality. I also discuss why causal relations are underdetermined in experience and are to be dealt with pragmatically in a hypothesis-testing framework. (shrink)

I shall argue in this paper that extant definitions of causality are incorrect since they do not cohere in the proper way with the concept of a cause of the universe. This lack of coherence is twofold. For some extant definitions of a cause, there are possible instances of the concept of a cause of the universe that do not satisfy the definitions. For these or other extant definitions, there are entities or occurrences that are not instances of the concept (...) of a cause of the universe but that count as causes of the universe according to the definitions. This failure of coherence between extant definitions of causality and the concept of a cause of the universe entails either that there is some other definition of a cause that is correct or that causality is indefinable. I shall suggest in the conclusion of this paper that it is more plausible to believe that causality is indefinable. (shrink)

How can a present or future event causally influence one in the past? Even though the case of such a relationship is often quickly dismissed as impossible, this paper suggests that this reaction is hasty and omits interesting as well as substantive arguments and considerations. In an introductory synopsis of the metaphysics of causality, we first present and discuss arguments for and against the possibility of backwards causation. On the other hand, we suggest that a probabilistic notion of causality according (...) to Suppes need not exclude that causes occur temporally after their effects. (shrink)

The unanimity theory is an account of property-level causation requiring that causes raise the probability of their effects in specified test situations. Richard Otte (1981) and others have presented counterexamples in which one property is probabilistically sufficient for at least one other property. Given the continuing discussion (e.g., Cartwright 1989; Cartwright and Dupre 1988; Eells 1988a,b), many apparently think that these problems are minor. By considering the impact of Otte's cases on recent versions of the theory, by raising several new (...) examples, and by criticizing natural replies, I argue that the problems for the unanimity theory are severe. (shrink)