"How do we go about weighing evidence, testing hypotheses and making inferences? According to the model of 'inference to the Best explanation', we work out what to inter from the evidence by thinking about what would actually explain that evidence, and we take the ability of a hypothesis to explain the evidence as a sign that the hypothesis is correct. In inference to the Best Explanation, Peter Lipton gives this important and influential idea the development and assessment it deserves." "The (...) second edition has been substantially enlarged and reworked, with a new chapter on the relationship between explanation and Bayesianism, and an extension and defence of the account of contractive explanation. It also includes an expanded defence of the claims that our inferences really are guided by diverse explanatory considerations, and that this pattern of inference can take us towards the truth. This edition of Inference to the Best Explanation has also been updated throughout and incudes a new bibliography."--BOOK JACKET. (shrink)

This volume presents a selection of the most influential recent discussions of the crucial metaphysical questions: what is it for one event to cause another? The subject of causation bears on many topics, such as time, explanation, mental states, the laws of nature, and the philosphy of science.

What happens when someone acts? A familiar answer goes like this. There is something that the agent wants, and there is an action that he believes conducive to its attainment. His desire for the end, and his belief in the action as a means, justify taking the action, and they jointly cause an intention to take it, which in turn causes the corresponding movements of the agent's body. I think that the standard story is flawed in several respects. The flaw (...) that will concern me in this paper is that the story fails to include an agent-or, more precisely, fails to cast the agent in his proper role. (shrink)

In The Emergent Self, William Hasker joins one of the most heated debates in contemporary analytic philosophy, that over the nature of mind.

A scholarly edition of a work by David Hume. The edition presents an authoritative text, together with an introduction, commentary notes, and scholarly apparatus.

"This is an important book, and no one interested in issues which touch on the free will will want to ignore it."--Ethics. In this stimulating and thought-provoking book, the author defends the thesis that free will is incompatible with determinism. He disputes the view that determinism is necessary for moral responsbility. Finding no good reason for accepting determinism, but believing moral responsiblity to be indubitable, he concludes that determinism should be rejected.

This is a revised and updated version of Swinburne's controversial treatment of the eternal philosophical problem of the relation between mind and body. He argues that we can only make sense of the interaction between the mental and the physical in terms of the soul, and that there is no scientific explanation of the evolution of the soul.

The paper argues that it is possible for an incompatibilist to accept John Martin Fischer's plausible insistence that the question whether we are morally responsible agents ought not to depend on whether the laws of physics turn out to be deterministic or merely probabilistic. The incompatibilist should do so by rejecting the fundamentalism which entails that the question whether determinism is true is a question merely about the nature of the basic physical laws. It is argued that this is a (...) better option for ensuring the irrelevance of physics than the embrace of semi-compatibilism, since there are reasons for supposing that alternate possibilities are necessary for moral responsibility, despite Fischer's claims to the contrary. There are two distinct reasons for supposing that alternate possibilities might be necessary for moral responsibility—one of which is to do with fairness, the other to do with agency itself. It is suggested that if one focuses on the second of these reasons, Fischer's arguments for supposing that alternate possibilities are unnecessary for moral responsibility can be met by the incompatibilist. Some possible reasons for denying that alternate possibilities are necessary for the existence of agency are then raised and rejected. (shrink)

: To act is to be the author of an intentional bodily movement. I will show that, in order for that authorship to be assured, the agent must both amount to more than the mereological sum of her mental or neural states and events, and have an irreducible causal power over, at least, some of them. Hence, agent-causalism is the best position for any realist about action to assume. I will contend that, contrary to what many have claimed, agent-causalism is (...) not an unscientific theory, since it can ground its view of the agent on a form of emergent dualism that can account for robust forms of agency without having to challenge the natural supervenience of the mental on the physical. I claim that the conditions of possibility for a causally effective emergent self are the presence of neuronal indeterminism and the break of causal closure, both of which will be shown to be compatible with our current scientific picture of the world. Keywords: Action; Self; Emergent Dualism; Downward Causation; Indeterminism Prendere sul serio l’agente: ovvero, come un’attenta analisi dell’agentività ci conduce al dualismo emergentista Riassunto: Agire vuol dire essere l’autore di un movimento corporeo intenzionale. Intendo mostrare che, per assicurarsi il riconoscimento di essere l’autore di un’azione, l’agente debba essere più della semplice somma mereologica dei propri stati mentali ed eventi neurali e che debba anche avere un irriducibile potere causale su di essi, o al limite su parte di essi. Pertanto, una concezione causale riferita all’agente è la migliore posizione da assumere per chi voglia dirsi realista riguardo alle azioni. Intendo affermare che, diversamente a quanto da molti sostenuto, la concezione causale riferita all’agente non sia una teoria non scientifica, dal momento che può fondare la sua concezione dell’agente su una forma di dualismo emergentista in grado di legittimare forme robuste di agentività senza dover mettere in dubbio la sopravvenienza del mentale sul fisico. Indeterminismo neurale e rottura della chiusura causale saranno indicate come condizioni di possibilità per un sé causalmente ed effettivamente emergente e mostrerò come entrambi siano compatibili con l’immagine scientifica del mondo che oggi abbiamo. Parole chiave : Azione; Sé; Dualismo emergentista; Causalità rivolta verso il basso; Indeterminismo. (shrink)

The question I raise is whether Mark Balaguer’s event-causal libertarianism can withstand the disappearing agent objection. The concern is that with the causal role of the events antecedent to a decision already given, nothing settles whether the decision occurs, and so the agent does not settle whether the decision occurs. Thus it would seem that in this view the agent will not have the control in making decisions required for moral responsibility. I examine whether Balaguer’s position has the resources to (...) answer this objection. (shrink)

The objective probability of every physical event is fixed by prior physical events and laws alone. (This thesis is sometimes expressed in terms of explanation: In tracing the causal history of any physical event, one need not advert to any non-physical events or laws. To the extent that there is any explanation available for a physical event, there is a complete explanation available couched entirely in physical vocabulary. We prefer the probability formulation, as it should be acceptable to any physicalist, (...) though some reject the explanation formulation.) (3) Causal Exclusion. (shrink)

The problem of freedom and determinism has vexed philosophers for several millennia, and continues to be a topic of lively debate today. One of the proposed solutions to the problem that has received a great deal of attention is the Theory of Agent Causation. While the theory has enjoyed its share of advocates, and perhaps more than its share of critics, the theory’s advocates and critics have always agreed on one thing: the Theory of Agent Causation is an incompatibilist theory. (...) That is, both believers and nonbelievers in the theory have taken it for granted that the most plausible version of the Theory of Agent Causation is one according to which freedom and determinism are incompatible. In fact, so entrenched is this assumption that no one on either side of the debate has ever questioned it. Yet it turns out that this assumption is wrong – the most plausible version of the Theory of Agent Causation is a compatibilist one. (shrink)

The Theory of Agent Causation has always been formulated as an incompatibilist view, but I think that this has been a mistake. The aim of this paper is to argue that, contrary to what agent causation theorists and their opponents have always believed, the most plausible version of the Theory of Agent Causation is actually a compatibilist version of that theory. I formulate the traditional version of the Theory of Agent Causation, and consider a series of objections to it and (...) related views. With each objection comes a corresponding revision of the theory that is motivated by that objection, and with each revision the theory becomes increasingly compatibilistic until, finally, we arrive at a completely compatibilistic version of the Theory of Agent Causation, which I take to be the most plausible version of that theory. (shrink)

In a recent paper I argued that agent causation theorists should be compatibilists. In this paper, I argue that compatibilists should be agent causation theorists. I consider six of the main problems facing compatibilism: (i) the powerful intuition that one can't be responsible for actions that were somehow determined before one was born; (ii) Peter van Inwagen's modal argument, involving the inference rule (β); (iii) the objection to compatibilism that is based on claiming that the ability to do otherwise is (...) a necessary condition for freedom; (iv) "manipulation arguments," involving cases in which an agent is manipulated by some powerful being into doing something that he or she would not normally do, but in such a way that the compatibilist's favorite conditions for a free action are satisfied; (v) the problem of constitutive luck; and (vi) the claim that it is not fair to blame someone for an action if that person was determined by forces outside of his or her control to perform that action. And in the case of each of these problems, I argue that the compatibilist has a much more plausible response to that problem if she endorses the theory of agent causation than she does otherwise. (shrink)

Why does the problem of free will seem so intractable? I surmise that in large measure it does so because the free will debate, at least in its modern form, is conducted in terms of a mistaken approach to causality in general. At the heart of this approach is the assumption that all causation is fundamentally event causation. Of course, it is well-known that some philosophers of action want to invoke in addition an irreducible notion of agent causation, applicable only (...) in the sphere of intelligent agency. But such a view is generally dismissed as incompatible with the naturalism that has now become orthodoxy amongst mainstream analytical philosophers of mind. What I want to argue is that substances, not events, are the primary relata of causal relations and that agent causation should properly be conceived of as a species of substance causation. I shall try to show that by thus reconceiving the nature of causation and of agency, the problem of free will can be made more tractable. I shall also argue for a contention that may seem even less plausible at first sight, namely, that such a view of agency is perfectly compatible with a volitionist theory of action. (shrink)

Why does the problem of free will seem so intractable? I surmise that in large measure it does so because the free will debate, at least in its modern form, is conducted in terms of a mistaken approach to causality in general. At the heart of this approach is the assumption that all causation is fundamentally event causation. Of course, it is well-known that some philosophers of action want to invoke in addition an irreducible notion of agent causation, applicable only (...) in the sphere of intelligent agency. But such a view is generally dismissed as incompatible with the naturalism that has now become orthodoxy amongst mainstream analytical philosophers of mind. What I want to argue is that substances, not events, are the primary relata of causal relations and that agent causation should properly be conceived of as a species of substance causation. I shall try to show that by thus reconceiving the nature of causation and of agency, the problem of free will can be made more tractable. I shall also argue for a contention that may seem even less plausible at first sight, namely, that such a view of agency is perfectly compatible with a volitionist theory of action. (shrink)

How do we go about weighing evidence, testing hypotheses, and making inferences? The model of " inference to the best explanation " -- that we infer the hypothesis that would, if correct, provide the best explanation of the available evidence--offers a compelling account of inferences both in science and in ordinary life. Widely cited by epistemologists and philosophers of science, IBE has nonetheless remained little more than a slogan. Now this influential work has been thoroughly revised and updated, and features (...) a new introduction and two new chapters. Inference to the Best Explanation is an unrivaled exposition of a theory of particular interest in the fields both of epistemology and the philosophy of science. (shrink)

Science depends on judgments of the bearing of evidence on theory. Scientists must judge whether an observation or the result of an experiment supports, disconfirms, or is simply irrelevant to a given hypothesis. Similarly, scientists may judge that, given all the available evidence, a hypothesis ought to be accepted as correct or nearly so, rejected as false, or neither. Occasionally, these evidential judgments can be made on deductive grounds. If an experimental result strictly contradicts a hypothesis, then the truth of (...) the data deductively entails the falsity of the hypothesis. In the great majority of cases, however, the connection between evidence and hypothesis is non-demonstrative, or inductive. In particular, this is so whenever a general hypothesis is inferred to be correct on the basis of the available data, since the truth of the data will not deductively entail the truth of the hypothesis. It always remains possible that the hypothesis is false even though the data are correct. (shrink)

In this paper I will revisit an argument that I have called “the supervenience argument”; it is sometimes called “the exclusion argument” in the literature. I want to reconsider several aspects of this argument in light of some of the criticisms and comments it has elicited, clarifying some points and offering a slightly reformulated—and improved—version of the argument. My primary aim, however, is to discuss and respond to Ned Block’s edifying and challenging critique of the argument in his “Do Causal (...) Powers Drain Away?”—in particular, his claim that the argument has the consequence that if there is no bottom microphysical level, causal powers will “drain away”, leaving us with no causation anywhere. The supervenience argument was designed to show that on a certain popular and influential view of mentality and its relationship to the physical, mental properties turn out to be epiphenomenal, that is, without causal powers of their own. (shrink)

Action theory has given rise to some perplexing puzzles in the past half century. The most prominent one can be summarized as follows: What distinguishes intentional from unintentional acts? Thanks to the ingenuity of philosophers and their thought experiments, we know better than to assume that the difference lies in the mere presence of an intention, or in its causal efficacy in generating the action. The intention might be present and may also cause the intended behavior, yet the behavior may (...) not be an intentional action; it may not be an action at all. The classic example is that of the nervous nephew who intends to kill his uncle to inherit his estate, and whose intention makes him so nervous as to drive recklessly, thereby running over a pedestrian... who happens to be his very uncle. The intention to kill is present and it causes the killing, yet the killing is not an intentional action. Rather, what appears to distinguish intentional from non-intentional action is voluntary control of the proper sort, and what distinguishes action from non-action is behavior caused in a particular manner. But spelling out the sort and specifying the manner have proven vexing tasks. (shrink)

A framework for representing a specific kind of emergent property instance is given. A solution to a generalized version of the exclusion argument is then provided and it is shown that upwards and downwards causation is unproblematical for that kind of emergence. One real example of this kind of emergence is briefly described and the suggestion made that emergence may be more common than current opinions allow.

This is a review of William Hasker's 'The Emergent Self' (Ithaca: Cornell University Press, 2001).

Nearly all defences of the agent-causal theory of free will portray the theory as a distinctively libertarian one — a theory that only libertarians have reason to accept. According to what I call ‘the standard argument for the agent-causal theory of free will’, the reason to embrace agent-causal libertarianism is that libertarians can solve the problem of enhanced control only if they furnish agents with the agent-causal power. In this way it is assumed that there is only reason to accept (...) the agent-causal theory if there is reason to accept libertarianism. I aim to refute this claim. I will argue that the reasons we have for endorsing the agent-causal theory of free will are nonpartisan. The real reason for going agent-causal has nothing to do with determinism or indeterminism, but rather with avoiding reductionism about agency and the self. As we will see, if there is reason for libertarians to accept the agent-causal theory, there is just as much reason for compatibilists to accept it. It is in this sense that I contend that if anyone should be an agent-causalist, then everyone should be an agent-causalist. (shrink)

In this paper I seek to defend libertarianism about free will and moral responsibility against two well-known arguments: the luck argument and the Mind argument. Both of these arguments purport to show that indeterminism is incompatible with the degree of control necessary for free will and moral responsibility. I begin the discussion by elaborating these arguments, clarifying important features of my preferred version of libertarianism—features that will be central to an adequate response to the arguments—and showing why a strategy of (...) reconciliation (often referred to as “deliberative libertarianism”) will not work. I then consider four formulations of the luck argument and find them all wanting. This discussion will place us in a favorable position to understand why the Mind argument also fails. (shrink)

Event-causal libertarians maintain that an agent’s freely bringing about a choice is reducible to states and events involving him bringing about the choice. Agent-causal libertarians demur, arguing that free will requires that the agent be irreducibly causally involved. Derk Pereboom and Meghan Griffith have defended agent-causal libertarianism on this score, arguing that since on event-causal libertarianism an agent’s contribution to his choice is exhausted by the causal role of states and events involving him, and since these states and events leave (...) it open which decision he will make, he does not settle which decision occurs, and thus “disappears.” My aim is to explain why this argument fails. In particular, I demonstrate that event-causal libertarians can dismantle the argument by enriching the reductive base in their analysis of free will to include a state that plays the functional role of the self-determining agent and with which the agent is identified. (shrink)

I offer a brief review of, and critical response to, Neil Levy’s fascinating recent book Hard Luck, where he argues that no one is ever free or morally responsible not because of determinism or indeterminism, but because of luck. Two of Levy’s central arguments in defending his free will nihilism concern the nature and role of explanation in a theory of moral responsibility and the nature of akrasia. With respect to explanation, Levy argues that an adequate theory of moral responsibility (...) must be able provide contrastive explanations of why an agent performs one action rather than another, and that libertarians lack the resources to provide such explanations. With respect to akrasia, Levy argues that it is impossible to be directly morally responsible for akratic actions. In response I argue that any sense of contrastive explanation that can reasonably be thought to be a requirement on an adequate theory of moral responsibility is a sense that agent-causal libertarians can secure. I then argue that the agent-causal theory of free will offers an alternative and attractive understanding of motivation and self-control that makes it plausible to think that we can be morally responsible for akratic actions. (shrink)

First published in 2002. Routledge is an imprint of Taylor & Francis, an informa company.

First published in 2002. Routledge is an imprint of Taylor & Francis, an informa company.

First published in 2002. Routledge is an imprint of Taylor & Francis, an informa company.

I use recent developments in neuroscience to show how volitional mental events can be causal within a physicalist paradigm. (1) I begin by attacking the logic of Jaegwon Kim’s exclusion argument, according to which mental information cannot be causal of physical events. I argue that the exclusion argument falls apart if indeterminism is the case. If I am right, I must still build an account of how mental events are causal in the brain. To that end I take as my (...) foundation (2) a new understanding of the neural code that emphasizes rapid synaptic resetting over the traditional emphasis on neural spiking. (3) Such a neural code is an instance of ‘criterial causation,’ which requires modifying standard interventionist conceptions of causation. A synaptic reweighting neural code provides (4) a physical mechanism that accomplishes downward informational causation, (5) a middle path between determinism and randomness, and (6) a way for mind/brain events to turn out otherwise. This ‘synaptic neural code’ allows a constrained form of randomness parameterized by information realized in and set in synaptic weights, which in turn allows physical/informational criteria to be met in multiple possible ways when combined with an account of how randomness in the synapse is amplified to the level of randomness in spike timing. This new view of the neural code also provides (7) a way out of self-causation arguments against the possibility of mental causation. It leads to (8) an emphasis on deliberation and voluntary attentional manipulation as the core of volitional mental causation rather than, say, the correlates of unconscious premotor computations seen in Libet’s readiness potentials. And this new view of the neural code leads to (9) a new theory of the neural correlates of qualia as the ‘precompiled’ informational format that can be manipulated by voluntary attention, which gives qualia a causal role within a physicalist paradigm. I elaborate each of these ideas in turn below. -/- -/- (1) Countering Kim’s exclusion argument. The exclusion argument is, roughly, that the physical substrate does all the causal work that the supervenient mental state is supposed to do, so mental or informational events can play no causal role in material events. On Kim’s reductionistic view, all causation seeps away to the rootmost physical level, i.e. particles or strings. Add to that an assumption of determinism, and the laws of physics applicable at the rootmost level are sufficient to account for event outcomes at that level and every level that might supervene on that level. So informational causation, including voluntary mental causation or any type of free will that relies on it, is ruled out. -/- I argue that indeterminism undermines this sufficiency, so provides an opening whereby physically realized mental events could be downwardly causal. I argue that biological systems introduced a new kind of physical causation into the universe, one based upon triggering physical actions in response to detected spatiotemporal patterns in energy. This is a very different kind of causation than traditional Newtonian conceptions of the causal attributes of energy, such as mass, momentum, frequency or position, which seem to underlie deterministic and exclusionary intuitions. But patterns, unlike amounts of energy, lack mass and momentum and can be created and destroyed. They only become causal if there are physical detectors that respond to some pattern in energetic inputs. Basing causal chains upon successions of detected patterns in energy, rather than the transfer of energy among particles, opens the door not only to informational downward causation but to causal chains (such as mental causal chains or causal chains that might underlie a game of baseball or bridge) that are not describable by or solely explainable by the laws of physics applicable at the rootmost level. Yes, a succession of patterns must be realized in a physical causal chain that is consistent with the laws of physics, but many other possible causal chains that are also consistent with physical laws are ruled out by informational criteria imposed on indeterministic particle outcomes. Physical/informational criteria set in synaptic weights effectively sculpt informational causal chains out of the ‘substrate’ of possible physical causal chains. -/- (2) A new view of the neural code: I develop a new understanding the neural code that emphasizes rapid and dynamic synaptic weight resetting over neural firing as the core engine of information processing in the brain. The neural code is not solely a spike code, but a code where information is transmitted and transformed by flexibly and temporarily changing synaptic weights on a millisecond timescale. One metaphor is the rapid reshaping of the mouth (analogous to rapid, temporary synaptic weight resetting) that must take place just before vibrating air (analogous to spike trains) passes through, if information is to be realized and communicated. What rapid synaptic resetting allows is a moment by moment changing of the physical and informational parameters or criteria that have to be met before a neuron will fire. This dictates what information neurons will be responsive to and what they will ‘say’ to one another from moment to moment. -/- (3) Rethinking interventionist models of causation: Standard interventionist models of causation manipulate A to determine what effects, if any, there might be on B and other variables. If instead of manipulating A's output, we manipulate the criteria, parameters or conditions that B places on A's input, which must be satisfied before B changes or acts, then changes in B do not follow passively from changes in A as they would if A and B were billiard balls. Inputs from A can be identical, but in one case B changes in response to A, and in another it does not. This constant reparameterization of B is what neurons do when they change each other's synaptic weights. What I call "criterial causation" emphasizes that what can vary is either outputs from A to other nodes, or how inputs from A are decoded by receiving nodes. On this view, standard interventionist and Newtonian models of causation are a special case where B places no conditions on input from A. But the brain, if anything, emphasizes causation via reparameterization of B, by, for example, rapidly changing synaptic weights on post-synaptic neurons. -/- (4) How downward causation works: Downward causation means that events at a supervening level can influence outcomes at the rootmost level. In this context it would mean that information could influence particle paths. While it would be impossible self-causation if a supervening event changed its own present physical basis, it is not impossible that supervening events, such as mental information, could bias future particle paths. How might this work in the brain? The key pattern in the brain to which neurons respond is temporal coincidence. A neuron will only fire if it receives a certain number of coincident inputs from other neurons. Criterial causation occurs where physical criteria imposed by synaptic weights on coincident inputs in turn realize informational criteria for firing. This permits information to be downwardly causal regarding which indeterministic events at the rootmost level will be realized; Only those rootmost physical causal chains that meet physically realized informational criteria can drive a postsynaptic neuron to fire, and thus become causal at the level of information processing. Typically the only thing that the set of all possible rootmost physical causal chains that meet those criteria have in common is that they meet the informational criteria set. To try to cut information out of the causal picture here is a mistake; The only way to understand why it is that just this subset of possible physical causal chains—namely those that are also informational causal chains—can occur, is to understand that it is informational criteria that dictate that class of possible outcomes. -/- The information that will be realized when a neuron’s criteria for firing have been met is already implicit in the set of synaptic weights that impose physical criteria for firing that in turn realize informational criteria for firing. That is, the information is already implicit in these weights before any inputs arrive, just as what sound your mouth will make is implicit in its shape before vibrating air is passed through. Assuming indeterminism, many combinations of possible particle paths can satisfy given physical criteria, and many more cannot. The subset that can satisfy the physical criteria needed to make a neuron fire is also the subset that can satisfy the informational criteria for firing (such as ‘is a face’) that those synaptic weights realize. So sets of possible paths that are open to indeterministic elementary particles which do not also realize an informational causal chain are in essence “deselected” by synaptic settings by virtue of the failure of those sets of paths to meet physical/informational criteria for the release of a spike. -/- (5) Between determinism and randomness: Hume (1739) wrote “’tis impossible to admit of any medium betwixt chance and an absolute necessity.” Many other philosophers have seen no middle path to free will between the equally ‘unfree’ extremes of determinism and randomness. They have either concluded that free will does not exist, or tried to argue that a weak version of free will, namely, ‘freedom from coercion,’ is compatible with determinism. -/- A strong conception of free will, however, is not compatible with either determined or random choices, because in the determined case there are no alternative outcomes and things cannot turn out otherwise, while in the random case what happens does not happen because it was willed. A strong free will requires meeting some high demands: Beings with free will (a) must have information processing circuits that have multiple courses of physical or mental activity open to them; (b) they must really be able to choose among them; (c) they must be or must have been able to have chosen otherwise once they have chosen; and (d) the choice must not be dictated by randomness alone, but by the informational parameters realized in those circuits. This is a tough bill to fill, since it seems to require that acts of free will involve acts of self-causation. -/- Criterial causation offers a middle path between the two extremes of determinism and randomness that Hume was not in a position to see, namely, that physically realized informational criteria parameterize what class of neural activity can be causal of subsequent neural events. The information that meets preset physical/informational criteria may be random to a degree, but it must meet those criteria if it is to lead to neural firing, so is not utterly random. Preceding brain activity specifies the range of possible random outcomes to include only those that meet preset informational criteria for firing. -/- (6) How brain/mind events can turn out otherwise: The key mechanism, I argue, whereby atomic level indeterminism has its effects on macroscopic neural behavior is that it introduces randomness in spike timing. There is no need for bizarre notions such as consciousness collapsing wave packets or any other strange quantum effects beyond this. For example, quantum level noise expressed at the level of individual atoms, such as single magnesium atoms that block NMDA receptors, is amplified to the level of randomness and near chaos in neural and neural circuit spiking behavior. A single photon can even trigger neural firing in a stunning example of amplification from the quantum to macroscopic domains. The brain evolved to harness such ‘noise’ for information processing ends. Since the system is organized around coincidence detection, where spike coincidences (simultaneous arrival of spikes) are key triggers of informational realization (i.e. making neurons fire that are tuned to particular informational criteria), randomizing which incoming spike coincidences might meet a neuron's criteria for firing means informational parameters can be met in multiple ways just by chance. -/- (7) Skirting self-causation: A synaptic account of the neural code also gets around some thorny problems of self-causation that have been used to argue against the possibility of mental causation. The traditional argument is that a mental event realized in neural event x cannot change x because this would entail impossible self-causation. Criterial causation gets around this by granting that present self-causation is impossible. But it allows neurons to alter the physical realization of possible future mental events in a way that escapes the problem of self-causation of the mental upon the physical. Mental causation is crucially about setting synaptic weights. These serve as the physical grounds for the informational parameters that must be met by unpredictable future mental events. -/- (8) Voluntary attention and free will: I argue that the core circuits underlying free choice involve frontoparietal circuits that facilitate deliberation among options that are represented and manipulated in executive working memory areas. Playing out scenarios internally as virtual experience allows a superthreshold option to be chosen before specific motoric actions are planned. The chosen option can best meet criteria held in working memory, constrained by conditions of various evaluative circuits, including reward, emotional and cognitive circuits. This process also harnesses synaptic and ultimately atomic level randomness to foster the generation of novel and unforeseeable satisfactions of those criteria. Once criteria are met, executive circuits can alter synaptic weights on other circuits that will implement a planned operation or action. -/- (9) A new theory of qualia: The paradigmatic case of volitional mental control of behavior is voluntary attentional manipulation of representations in working memory such as the voluntary attentional tracking of one or a few objects among numerous otherwise identical objects. If there is a flock of indistinguishable birds, there is nothing about any individual bird that makes it more salient. But with volitional attention, any bird can be marked and kept track of. This salience is not driven by anything in the stimulus. It is voluntarily imposed on bottom-up information, and can lead to eventual motoric acts, such as shooting or pointing at the tracked bird. This leads to viewing the neural basis of attention and consciousness as not only realized in part in rapid synaptic reweighting, but also in particular patterns of spikes that serve as higher level units that traverse neural circuits and open what I call the ‘NMDA channel of communication.’ Qualia are necessary for volitional mental causation because they are the only informational format available to volitional attentional operations. Actions that follow volitional attentional operations, such as volitional tracking, cannot happen without consciousness. Qualia on this account are a ‘precompiled’ informational format made available to attentional selection and operations by earlier, unconscious information processing. -/- Conclusion: Assuming indeterminism, it is possible to be a physicalist who adheres to a strong conception of free will. On this view, mental and brain events really can turn out otherwise, yet are not utterly random. Prior neuronally realized information parameterizes what subsequent neuronally realized informational states will pass presently set physical/informational criteria for firing. This does not mean that we are utterly free to choose what we want to want. Some wants and criteria are innate, such as what smells good or bad. However, given a set of such innate parameters, the brain can generate and play out options, then select an option that adequately meets criteria, or generate further options. This process is closely tied to voluntary attentional manipulation in working memory, more commonly thought of as deliberation or imagination. Imagination is where the action is in free will. -/- - See more at: http://philosophycommons.typepad.com/flickers_of_freedom/2013/12/peter-tses-the-neural-basis-of-free -will-an-overview.html#sthash.Y5NvKvYd.dpuf. (shrink)

Helen Steward argues that determinism is incompatible with agency itself--not only the special human variety of agency, but also powers which can be accorded to animal agents. She offers a distinctive, non-dualistic version of libertarianism, rooted in a conception of what biological forms of organisation might make possible in the way of freedom.

Causation is everywhere in the world: it features in every science and technology. But how much do we understand it? Mumford and Anjum develop a new theory of causation based on an ontology of real powers or dispositions. They provide the first detailed outline of a thoroughly dispositional approach, and explore its surprising features.

Nelkin presents a simple and natural account of freedom and moral responsibility which responds to the great variety of challenges to the idea that we are free and responsible, before ultimately reaffirming our conception of ourselves as agents. Making Sense of Freedom and Responsibility begins with a defense of the rational abilities view, according to which one is responsible for an action if and only if one acts with the ability to recognize and act for good reasons. The view is (...) compatibilist -- that is, on the view defended, responsibility is compatible with determinism -- and one of its striking features is a certain asymmetry: it requires the ability to do otherwise for responsibility when actions are praiseworthy, but not when they are blameworthy. In defending and elaborating the view, Nelkin questions long-held assumptions such as those concerning the relation between fairness and blame and the nature of so-called reactive attitudes such as resentment and forgiveness. Her argument not only fits with a metaphysical picture of causation -- agent-causation -- often assumed to be available only to incompatibilist accounts, but receives positive support from the intuitively appealing Ought Implies Can Principle, and establishes a new interpretation of freedom and moral responsibility that dovetails with a compelling account of our inescapable commitments as rational agents. (shrink)