Absences pose a dilemma for theories of causation. Allowing them to be causes seems to make theories too permissive (Lewis, 2000). Banning them from being causes seems to make theories too restrictive (Schaffer, 2000, 2004). An increasingly popular approach to this dilemma is to acknowledge that norms can affect which absences count as causes (e.g., Thomson, 2003; McGrath, 2005; Henne et al., 2017; Willemsen, 2018). In this article, I distinguish between two influential implementations of such ‘abnormality’ approaches and argue that (...) so-called ‘double-prevention mechanisms’ provide counterexamples against both. (shrink)

It is widely thought that there is an important argument to be made that starts with premises taken from the science of physics and ends with the conclusion of physicalism. The standard view is that this argument takes the form of a causal argument for physicalism. Roughly, physics tells us that the physical realm is causally complete, and so minds (among other entities) must be physical if they are to interact with the world as we think they do. In what (...) follows, I raise problems for this view. After an initial review of the causal argument, I begin my case by showing that the totality of physical truths do not deductively entail the causal completeness of the physical realm, using a double-prevention scenario and causation by omission to show that nonphysical causes of physical effects would not need to violate physical conservation laws. I then move on to raise problems for an inductive argument for causal completeness by drawing on the neo-Russellian view that there is no causation in fundamental physics, and so causation must itself be a realized or derived entity. I conclude by suggesting that the underlying problem is that the causal argument has fallen out of touch with the sophisticated understanding that philosophers have developed of the role of causation within physics. (shrink)

“Perception is controlled hallucination,” according to proponents of predictive processing accounts of vision. I say they are right that something like this is a consequence of their view but wrong in how they have pursued the idea. The focus of my counterproposal is the causal theory of perception, which I develop in terms of a productive concept of causation. Cases of what otherwise seem like successful perception are instead mere veridical hallucination if predictive processing accounts are correct, I argue, because (...) of the role played within such accounts by absences, which cannot enter into productive causal relations. -/- I offer two arguments in support of the productive theory of perception. The first is loosely Kantian, focusing on the role that receptivity and spontaneity play in perception. The second focuses on what perception and hallucination have in common. I conclude with a series of objections and replies. (shrink)

One way of assessing the philosophical literature on causation is to consider views on the nature of the causal relation. Early theorists were 'monists', taking there to be one causal relation. More recent theorists, however, have turned to pluralism, which holds that the causal relation is only accurately captured by two (or more) relations. I argue that one way of being a pluralist – the way which takes there to be exactly two types of causation – is self defeating, if (...) it promises to handle intuitions about all causal situations. I illustrate the point via neuron diagrams. (shrink)

Kaplan and Craver :601–627, 2011) and Piccinini and Craver :283–311, 2011) argue that only mechanistic explanations of cognition are genuine causal explanations, because only evidence of mechanisms reveals the causal structure of cognition. I first argue that this claim is grounded in a commitment to the mechanistic account of causality, which cannot be endorsed by a defender of causal-nonmechanistic explanations. Then, I defend the epistemic theory of causality, which holds that causal explanations are not genuine to the extent that they (...) reveal mechanistic causal structure, but, rather, to the extent that they have evidential support and yield successful prediction, explanation, and control inferences. Finally, I enact an epistemic unification of causal explanation in cognitive science, according to which both mechanistic and nonmechanistic explanations of cognition can be genuine causal explanations. (shrink)

A central argument for non-reductive accounts of group agency is that complex social entities are capable of exerting causal influence independently of and superseding the causal efficacy of the individuals constituting them. A prominent counter is that non-reductionists run into an insuperable dilemma between identity and redundancy – with identity undermining independent higher-level efficacy and redundancy leading to overdetermination or exclusion. This paper argues that critics of non-reductionism can manage with a simpler and more persuasive reductio strategy called mapping: allow (...) that group agents are causally efficacious in their own right and chart how their causal efficacy is carried out; how it relates to the causal efficacy of individual determiners; how it connects to the causal relevance of background structural factors. The focus exclusively on whether groups are or are not causally efficacious black-boxes implementation, while close attention to how causation is wired increases the visibility of individualist arguments and countenances structure-oriented explanations. (shrink)

I argue that there are some causal relata for which it is indeterminate whether one caused the other. Positing indeterminacy in causation helps us defend contested principles in the logic of causation and makes possible new ways of thinking about the theoretical impact of symmetric causal overdetermination. I close by discussing amendments of current theories of causation that would help explain causal indeterminacy.

I argue that there are some causal relata for which it is indeterminate whether one caused the other. Positing indeterminacy in causation helps us defend contested principles in the logic of causation and makes possible new ways of thinking about the theoretical impact of symmetric causal overdetermination. I close by discussing amendments of current theories of causation that would help explain causal indeterminacy.

Essay Review of Tim Maudlin's "The Metaphysics within Physics", Oxford University Press, 2007.

Causal selection and priority are at the heart of discussions of the causal parity thesis, which says that all causes of a given effect are on a par, and that any justified priority assigned to a given cause results from causal explanatory interests. In theories of causation that provide necessary and sufficient conditions for the truth of causal claims, status as cause is an either/or issue: either a given cause satisfies the conditions or it does not. Consequently, assessments of causal (...) parity and priority require more resources, which can either be additional or part of the causal analysis itself. While adding resources in terms of a theory of causal explanation has been standard, here we develop a unified conceptual analysis that includes a range of different precise causal concepts that allows for assessments of causal priority in terms of different kinds of causal relevance. (shrink)

Hall has recently argued that there are two concepts of causality, picking out two different kinds of causal relation. McGrath, and Hitchcock and Knobe, have recently argued that the facts about causality depend on what counts as a “default” or “normal” state, or even on the moral facts. In the light of these claims you might be tempted to agree with Skyrms that causal relations constitute, metaphysically speaking, an “amiable jumble”, or with Cartwright that ‘causation’, though a single word, encompasses (...) many different kinds of things. This paper argues, drawing on the author’s recent work on explanation, that the evidence adduced in support of causal pluralism can be accommodated easily by a unified theory of causality—a theory according to which all singular causal claims concern the same fundamental causal network. (shrink)

The exclusion argument is widely thought to put considerable pressure on dualism if not to refute it outright. We argue to the contrary that, whether or not their position is ultimately true, dualists have a plausible response. The response focuses on the notion of ‘distinctness’ as it occurs in the argument: if 'distinctness' is understood one way, the exclusion principle on which the argument is founded can be denied by the dualist; if it is understood another way, the argument is (...) not persuasive. (shrink)

Philosophers discussing causation take on, as one of their responsibilities, the task of specifying an ontology of causation. Both standard and non-standard accounts of that ontology make two assumptions: that the ontological category of causal relata admits a unique specification, and that cause and effect are of the same ontological type. These assumptions are rarely made explicit, but there is in fact little reason to think them true. It is argued here that, if the question has any interest, there are (...) some considerations in favor of rejecting Uniqueness and Uniformity, no good reasons in favor of them, but good reason to think that we are not in a position to make a decisive pronouncement, since some of the information needed to make a judgement on the matter is empirical in character. Philosophers have therefore made two mistakes about the relata of causation: one is to have accepted unwarranted restrictions on the categories pertinent to causation; the other is to have wrongly assumed that we are in a position to name them. (shrink)

Debates in animal cognition are frequently polarized between the romantic view that some species have human-like causal understanding and the killjoy view that human causal reasoning is unique. These apparently endless debates are often characterized by conceptual confusions and accusations of straw-men positions. What is needed is an account of causal understanding that enables researchers to investigate both similarities and differences in cognitive abilities in an incremental evolutionary framework. Here we outline the ways in which a three-dimensional model of causal (...) understanding fulfills these criteria. We describe how this approach clarifies what is at stake, illuminates recent experiments on both physical and social cognition, and plots a path for productive future research that avoids the romantic/killjoy dichotomy. (shrink)

This paper develops axiomatic foundations for a probabilistic-interventionist theory of causal strength. Transferring methods from Bayesian confirmation theory, I proceed in three steps: I develop a framework for defining and comparing measures of causal strength; I argue that no single measure can satisfy all natural constraints; I prove two representation theorems for popular measures of causal strength: Pearl's causal effect measure and Eells' difference measure. In other words, I demonstrate these two measures can be derived from a set of plausible (...) adequacy conditions. The paper concludes by sketching future research avenues. (shrink)

Is double prevention causation? Some say yes and some say no, but the answer is yes and no. Interrupting double prevention, where A prevents B from continuing to prevent something, is causation, while blocking double prevention, where A intervenes before B has begun preventing anything, is not. I present two arguments for this thesis. First, it sorts canonical examples of double prevention correctly. Second, well-known theoretical arguments that double prevention is not causation only show that blocking double prevention is not (...) causation. (shrink)

Any theory of mind needs to explain mental causation. Kim’s exclusion argument concludes that non-reductive physicalism cannot meet this challenge. One classic reply is that mental properties capture the causally relevant level of generality, because they are insensitive to physical realization. However, this reply suggests downward exclusion, contrary to physicalism’s assumption of closure. This paper shows how non-reductive physicalists can solve this problem by introducing a contrastive account of causation with non-exhaustive contrasts. That view has independent justification, because it is (...) also needed to solve other puzzles. On this theory, both a mental property and its physical realizer can cause the same physical effect without lapsing into any problematic overdetermination when they cause that effect in contrast with distinct foils. This contrastive solution has advantages over previous accounts of mental causation and is defended against objections. (shrink)

The Exclusion Problem for mental causation suggests that there is a tension between the claim that the mental causes physical effects, and the claim that the mental does not overdetermine its physical effects. In response, Karen Bennett puts forward an extra necessary condition for overdetermination : if one candidate cause were to occur but the other were not to occur, the effect would still occur. She thus denies one of the assumptions of EP, the assumption that if an effect has (...) two sufficient causes, it is overdetermined. If sound, her argument does two things: it solves EP, and it shows how to use counterfactuals in order to make the notion of overdetermination precise. However, the argument is not sound. (shrink)

David Lewis championed a counterfactual account of causation, but counterfactual accounts have a notoriously difficult time handling cases of late preemption. These are cases in which we still count one event as the cause of another although the effect does not depend on the cause in the way taken to be necessary by the account. Lewis recognized these cases, but they have been shown to be problematic even for his final analysis of causation, the Influence Account. In this paper, I (...) show how these cases led to and remain a problem for Lewis’s Influence Account, and I give a new counterfactual account available to a Lewisian that can handle late preemption cases. What is crucial to my account is that a cause not merely has counterfactual influence over its effects, but that the cause has influence of the right kind. I conclude by arguing that even if problems remain with this account, they will not be problems of late preemption, and the problems stemming from other kinds of cases will be no worse for my account than for other counterfactual accounts. (shrink)

This paper argues that the semantic facts about ‘because’ are best explained via a metaphorical treatment of metaphysical explanation that treats causal explanation as explanation par excellence. Along the way, it defends a commitment to a unified causal sense of ‘because’ and offers a proprietary explanation of grounding skepticism. With the causal metaphor account of metaphysical explanation on the table, an extended discussion of the relationship between conceptual structure and metaphysics ends with a suggestion that the semantic facts about ‘because’ (...) tell against grounding-causation unity. (shrink)

This paper presents evidence that ‘because’ is importantly ambiguous between two closely related senses covering what are usually called causal explanations, on the one hand, and grounding or metaphysical explanations, on the other hand. To this end, it introduces the lexical categories of monosemy, polysemy and homonymy; describes a test for polysemy; and discusses the results of the test when applied to ‘because’. It also shows how to understand so-called hybrid explanations in light of the semantic facts established by the (...) analysis. (shrink)

One of the most plausible and widely discussed examples of strong emergence is molecular structure. The only detailed account of it, which has been very influential, is due to Robin Hendry and is formulated in terms of downward causation. This paper explains Hendry’s account of the strong emergence of molecular structure and argues that it is coherent only if one assumes a diachronic reflexive notion of downward causation. However, in the context of this notion of downward causation, the strong emergence (...) of molecular structure faces three challenges that have not been met and which have so far remained unnoticed. First, the putative empirical evidence presented for the strong emergence of molecular structure equally undermines supervenience, which is one of the main tenets of strong emergence. Secondly, it is ambiguous how the assumption of determinate nuclear positions is invoked for the support of strong emergence, as the role of this assumption in Hendry’s argument can be interpreted in more than one way. Lastly, there are understandings of causation which render the postulation of a downward causal relation between a molecule’s structure and its quantum mechanical entities, untenable. (shrink)

The leading regularity, counterfactual, and agential accounts of causation converge on the idea that causation is probability-raising. While the necessity of probability-raising for causation remains in dispute, the sufficiency of probability-raising for causation is generally assumed, at least in the direct (no intermediaries involved) and precisely described case. I offer a class of counterexamples: overlappings.

Michael Moore’s Causation and Responsibility offers an integrated conception of the law, morality, and metaphysics, centered on the notion of causation, grounded in a detailed knowledge of case law, and supported on every point by cogent argument. This is outstanding work. It is a worthy successor to Harte and Honoré’s classic Causation in the Law, and I expect that it will guide discussion for many years to come.

Sydney Shoemaker's "Subset Account" offers a new take on determinable properties and the realization relation as well as a defense of non-reductive physicalism from the problem of mental causation. At the heart of this account are the claims that (1) mental properties are determinable properties and (2) the causal powers that individuate a determinable property are a proper subset of the causal powers that individuate the determinates of that property. The second claim, however, has led to the accusation that the (...) effects caused by the instantiation of a determinable property will also be caused by the instantiation of the determinates of that property—so instead of solving the problem of mental causation, the Subset Account ends up guaranteeing that the effects of mental properties (and all other types of determinable property) will be causally overdetermined! In this paper, I explore this objection. I argue that both sides in this debate have failed to engage the question at the heart of the objection: Given that both a determinable property and its determinates have the power to cause some effect (E), does it follow that both will actually cause E when the relevant conditions obtain? To make genuine progress towards answering this question, we need to take a serious look at the metaphysics of causation. With the debate properly reframed and issues about the metaphysics of causation front and center, I explore the question of whether the Subset Account is doomed to result in problematic causal overdetermination. (shrink)

The physical and/or intrinsic connection approach to causation has become prominent in the recent literature, with Salmon, Dowe, Menzies, and Armstrong among its leading proponents. I show that there is a type of causation, causation by disconnection, with no physical or intrinsic connection between cause and effect. Only Hume-style conditions approaches and hybrid conditions-connections approaches appear to be able to handle causation by disconnection. Some Hume-style, extrinsic, absence-relating, necessary and/or sufficient condition component of the causal relation proves to be needed.

This is a critical discussion of Vihvelin’s recent book Causes, Laws, and Free Will. I discuss Vihvelin’s ideas on Frankfurt-style cases and the actual-sequence view of freedom that is inspired by them.

ABSTRACT Frankfurt’s ‘Alternate Possibilities and Moral Responsibility’ broke with the tradition of understanding the kind of freedom required for responsibility in terms of alternative possibilities. At the same time, it inspired and motivated a new family of views in its place: views that focus exclusively on actual sequences or the actual causes of behaviour. But, what exactly does that ‘exclusiveness’ claim amount to? At first sight, it may seem natural to interpret it as the claim that the only facts that (...) are relevant to an agent’s freedom are certain facts about actual causes. This would imply that any non-actual facts are simply irrelevant to the freedom of agents. This paper argues that this interpretation is mistaken, and proposes a better one. It also discusses the related but more general question of the type of project that we are invested in when giving a theory of freedom: Are we interested in the bottom-level grounding facts, or are we interested in some higher-level facts? (shrink)

Sixteen years after Kim’s seminal paper offering a welcomed analysis of the emergence concept, I propose in this paper a needed extension of Kim’s work that does more justice to the actual diversity of emergentism. Rather than defining emergence as a monolithic third way between reductive physicalism and substance pluralism, and this through a conjunction of supervenience and irreducibility, I develop a comprehensive taxonomy of the possible varieties of emergence in which each taxon—theoretical, explanatory and causal emergence—is properly identified and (...) defined. This taxonomy has two advantages. First, it is unificatory in the sense that the taxa it contains derive from a common unity principle, which consequently constitutes the very hallmark of emergentism. Second, it can be shown that the emergence taxa it contains are able to meet the challenges that are commonly considered as being the hot topics on the emergentists’ agenda, namely the positivity, the consistency and the triviality/liberality challenges. (shrink)

On the face of it, causal responsibility seems to be “additive” in the following sense: if I cause some effects, then it seems that I also cause the sum (aggregate, conjunction, etc.) of those effects. Let’s call the claim that causation behaves in this way, Additivity.

Directed duties are those duties whose violation would wrong someone in particular. Under what conditions, and in virtue of what, is a duty directed to someone? This is the Question of Direction. In this article, I explore the possibility of providing a Contractualist answer to the Question of Direction—one where the directedness of a directed duty is explained by the way in which that duty is derived in Contractualist moral reasoning. After presenting and rejecting three attempts at such an answer, (...) I arrive at a satisfactory one: An agent has a duty to an individual to perform some action just in case and because, were the agent not to perform that action, the agent would make the individual occupy the standpoint relative to which any principle that permits the agent not to perform that action is unacceptable. (shrink)

This paper explores the status of causation in structuralist metaphysics of physics. What role (if any) does causation play in understanding ‘structure’ in ontological structural realism? I address this question by examining, in a structuralist setting, arguments for and against the idea that fundamental physics deals, perhaps exclusively, with causal properties. I will argue (against Esfeld, Dorato and others) that a structuralist interpretation of fundamental physics should diverge from ‘causal structuralism’. Nevertheless, causation outside fundamental physics, and the basic motivation for (...) causal structuralism outside fundamental physics, can be captured with an appropriate conception of causation. (shrink)

The paper examines definitions of ‘cause’ in the epidemiological literature. Those definitions all describe causes as factors that make a difference to the distribution of disease or to individual health status. In the philosophical jargon, causes in epidemiology are difference-makers. Two claims are defended. First, it is argued that those definitions underpin an epistemology and a methodology that hinge upon the notion of variation, contra the dominant Humean paradigm according to which we infer causality from regularity. Second, despite the fact (...) that causes be defined in terms of ‘difference-making’, this cannot fixes the causal metaphysics. Causality in epidemiology ought to be interpreted according to the epistemic theory. In this approach relations are deemed causal depending on the evidence and on the available methods. Indeed, evidence to establish causal claims requires difference-making considerations; furthermore, those definitions of cause reflect the ‘variational’ epistemology and methodology of epidemiology. (shrink)

Loewer (in: Physicalism and its discontents, 2001; Philos Phenomenol Res 65:655–663, 2002; in: Contemporary debates in philosophy of mind, 2007) has argued that the nonreductive physicalist should respond to the exclusion problem by endorsing the overdetermination entailed by their view. Kim’s (Physicalism, or something near enough, 2005; in: Contemporary debates in the philosophy of mind, 2007) argument against this reply is based on the premise that mental causation must be a productive relation in order to sustain human agency. In this (...) paper, I challenge the premise that mental causation is a productive relation by appealing to the underlying double prevention structure of the physiological mechanisms of human action. Since the causal pathways from an agent’s mental events to bodily movement involves an absence, mental causation is not productive. This places Kim in a troublesome dilemma in his debate with Loewer: either surrender mental causation or deny that causation is a productive relation. With the support offered for productive mental causation undermined, responses to the exclusion problem based on accepting overdetermination remain viable options for the nonreductive physicalist. (shrink)

According to Russo and Williamson (Int Stud Philos Sci 21(2):157–170, 2007, Hist Philos Life Sci 33:389–396, 2011a, Philos Sci 1(1):47–69, 2011b ), in order to establish a causal claim of the form, ‘_C_ is a cause of _E_’, one typically needs evidence that there is an underlying mechanism between _C_ and _E_ as well as evidence that _C_ makes a difference to _E_. This thesis has been used to argue that hierarchies of evidence, as championed by evidence-based movements, tend to (...) give primacy to evidence of difference making over evidence of mechanisms and are flawed because the two sorts of evidence are required and they should be treated on a par. An alternative approach gives primacy to evidence of mechanism over evidence of difference making. In this paper, we argue that this alternative approach is equally flawed, again because both sorts of evidence need to be treated on a par. As an illustration of this parity, we explain how scientists working in the ‘EnviroGenomarkers’ project constantly make use of the two evidential components in a dynamic and intertwined way. We argue that such an interplay is needed not only for causal assessment but also for policy purposes. (shrink)

Empirical research has recently emerged as a key method for understanding the nature of causation, and our concept of causation. One thread of research aims to test intuitions about the nature of causation in a variety of classic cases. These experiments have principally been used to try to resolve certain debates within analytic philosophy, most notably that between proponents of transference and dependence views of causation. The other major thread of empirical research on our concept of causation has investigated the (...) role that normative considerations play in causal judgments. These experimental results suggest that philosophical accounts of our concept of causation should take a broader view of what might be relevant. For both lines of research, we describe some of the significant experiments and outline key philosophical morals that have been drawn, all while pointing out various limitations. We conclude by considering other kinds of empirical research that should be philosophically interesting for those studying the nature of causation and our concept of causation. In particular, we point towards the need for philosophical research about causal perception, causal reasoning, and causal learning, as well as ways in which this research could play a role in prescriptive metaphysics. (shrink)

Conditionalization is a norm that governs the rational reallocation of credence. I distinguish between factive and non-factive formulations of Conditionalization. Factive formulations assume that the conditioning proposition is true. Non-factive formulations allow that the conditioning proposition may be false. I argue that non-factive formulations provide a better foundation for philosophical and scientific applications of Bayesian decision theory. I furthermore argue that previous formulations of Conditionalization, factive and non-factive alike, have almost universally ignored, downplayed, or mishandled a crucial causal aspect of (...) Conditionalization. To formulate Conditionalization adequately, one must explicitly address the causal structure of the transition from old credences to new credences. I offer a formulation of Conditionalization that takes these considerations into account, and I compare my preferred formulation with some prominent formulations found in the literature. (shrink)

I argue for the epiphenomenality of functional properties by means of thought experiments and general principles. General principles suggest that an object’s causal powers nomologically supervene on its intrinsic properties and that its functional role does not. This implies that it is possible for an object to lose its functional role without undergoing any change to its intrinsic properties or causal powers. Nor is it difficult to conceive of such scenarios. Various thought experiments are introduced for just this purpose. But (...) properties that can vary independently of an object’s causal powers in this way are epiphenomenal, for their instantiation by an object at a time makes no difference to the causal processes the object is involved in at that time. The same processes would have occurred even if such properties hadn’t been instantiated. The intrinsicness thesis is controversial, but it is not controversial that it is true of productive causation, and many believe that productive causation is the only form of mental causation worth having. Nevertheless, in the paper’s final section I consider dependence-based accounts of causation, which do not presuppose the intrinsicness of causal powers, and show that they too have difficulties accounting for the efficacy of functional properties. (shrink)

I present an alternative account of causation in the biomedical and social sciences according to which the meaning of causal claims is given by their inferential relations to other claims. Specifically, I will argue that causal claims are inferentially related to certain evidential claims as well as claims about explanation, prediction, intervention and responsibility. I explain in some detail what it means for a claim to be inferentially related to another and finally derive some implication of the proposed account for (...) the epistemology, semantics and metaphysics of causation. (shrink)

Jaegwon Kim’s views on mental causation and the exclusion argument are evaluated systematically. Particular attention is paid to different theories of causation. It is argued that the exclusion argument and its premises do not cohere well with any systematic view of causation.

Cognitive scientists have been debating how the folk concept of intentional action works. We suggest a simple account: people consider that an agent did X intentionally to the extent that X was causally dependent on how much the agent wanted X to happen (or not to happen). Combined with recent models of human causal cognition, this definition provides a good account of the way people use the concept of intentional action, and offers natural explanations for puzzling phenomena such as the (...) side-effect effect. We provide empirical support for our theory, in studies where we show that people’s causation and intentionality judgments track each other closely, in everyday situations as well as in scenarios with unusual causal structures. Study 5 additionally shows that the effect of norm violations on intentionality judgments depends on the causal structure of the situation, in a way uniquely predicted by our theory. Taken together, these results suggest that the folk concept of intentional action has been difficult to define because it is made of cognitive building blocks, such as our intuitive concept of causation, whose logic cognitive scientists are just starting to understand. (shrink)

: Among the current philosophical accounts of causation two are the most prominent. The first is James Woodward's interventionist counterfactual approach; the second is the mechanistic approach advocated by Peter Machamer, Lindley Darden, Carl Craver, Jim Bogen and Stuart Glennan. Thecounterfactual approach takes it that causes make a difference to their effects, where this difference-making is cashed out in terms of actual and counterfactual interventions. The mechanistic approach takes it that two events are causally related if and only if there (...) is a mechanism that connects them. In this paper I examine them both in some detail. After pointing out some important problems that both approaches face, I argue that there is a sense in which the counterfactual approach is more basic than the mechanistic one in that a proper account of mechanisms depends on counterfactuals while counterfactuals need not be supported (or depend on) mechanisms. Nonetheless, I also argue that if both approaches work in tandem in practice, they can offer us a better understanding of aspects of Hume's secret connexion and hence a glimpse of it. (shrink)

I argue that manipulationist theories of causation fail as accounts of causal structure, and thereby as theories of “actual causation” and causal explanation. I focus on two kinds of problem cases, which I call “Perceived Abnormality Cases” and “Ontological Dependence Cases.” The cases illustrate that basic facts about social systems—that individuals are sensitive to perceived abnormal conditions and that certain actions metaphysically depend on institutional rules—pose a challenge for manipulationist theories and for counterfactual theories more generally. I then show how (...) law-based accounts of causal structure can answer such challenges. The moral of the story is that the basic manipulationist idea that our interest in causal structure is driven by our interest in manipulating our environment faces decisive problems in a central domain of application, the social sciences. (shrink)

This article provides a conceptual account of causal understanding by connecting current psychological research on time and causality with philosophical debates on the causal asymmetry. I argue that causal relations are viewed as asymmetric because they are understood in temporal terms. I investigate evidence from causal learning and reasoning in both children and adults: causal perception, the temporal priority principle, and the use of temporal cues for causal inference. While this account does not suffice for correct inferences of causal structure, (...) I show it to serve as a preliminary understanding of causal concepts as asymmetric, that later incorporates other types of evidence. This approach supplies causal models with an asymmetric concept of causation that underlies hypotheses about causal structure, as I will illustrate from the framework of the knowledge-based causal induction model. I further argue for an integrating perspective, showing how the understanding of causes as preceding their effects underlies both psychological models and philosophical debates over time and the causal asymmetry, particularly regarding problem cases such as simultaneous causation or backwards causation, and the conceptual connection between causation and action. (shrink)

A temporal levels structure for temporal metaphysics is outlined and employed to convey a dilemma threatening the temporal collapse of Growing-Block Theories to their meta-temporal level. The outline further explains how Presentism occupies a privileged position in that temporal levels structure. Moreover, that dilemma relies crucially on the acceptance of productive causation as explaining additions to the growing block, for which it is argued any reasonable growing-block theory should incorporate. The dilemma’s first horn considers growing-block theories where productive causes are (...) only so when present; the second, growing-block theories where productive causes continue being so when past. Either way, growing-block theory collapses into Accruing-Present Theory: whereby all entities survive into succeeding present “blocks” of existents. It is argued, the presentness of surviving entities undermines their intended theoretical role, undercutting motivations for believing accruing-present theses. Ultimately, I recommend rejection of both growing-block and accruing-present theses based on these explanatory inadequacies. (shrink)

Many photographs seem to be images of absences: for instance, a photograph of a shadow seems to be an image of an absence, as shadows are plausibly thought of as being absences of light. Absence photography is puzzling, however, as, first, it is a common idea that photographs can only be images of things that have caused them, and, second, it is unclear whether absences can cause anything. In this paper, I look at various ways to unravel the puzzle. Along (...) the way, I also hope to cast some light on the idea that photography is a causal medium. (shrink)

Many photographs seem to be images of absences: for instance, a photograph of a shadow seems to be an image of an absence, as shadows are plausibly thought of as being absences of light. Absence photography is puzzling, however, as, first, it is a common idea that photographs can only be images of things that have caused them, and, second, it is unclear whether absences can cause anything. In this paper, I look at various ways to unravel the puzzle. Along (...) the way, I also hope to cast some light on the idea that photography is a causal medium. (shrink)

Understanding probabilities as something other than point values has often been motivated by the need to find more realistic models for degree of belief, and in particular the idea that degree of belief should have an objective basis in “statistical knowledge of the world.” I offer here another motivation growing out of efforts to understand how chance evolves as a function of time. If the world is “chancy” in that there are non-trivial, objective, physical probabilities at the macro-level, then the (...) chance of an event e that happens at a given time is \ until it happens. But whether the chance of e goes to one continuously or not is left open. Discontinuities in such chance trajectories can have surprising and troubling consequences for probabilistic analyses of causation and accounts of how events occur in time. This, coupled with the compelling evidence for quantum discontinuities in chance’s evolution, gives rise to a “continuity bind” with respect to chance probability trajectories. I argue that a viable option for circumventing the continuity bind is to understand the probabilities “imprecisely,” that is, as intervals rather than point values. I then develop and motivate an alternative kind of continuity appropriate for interval-valued chance probability trajectories. (shrink)