An influential tradition in the philosophy of causation has it that all token causal facts are, or are reducible to, facts about difference-making. Challenges to this tradition have typically focused on pre-emption cases, in which a cause apparently fails to make a difference to its effect. However, a novel challenge to the difference-making approach has recently been issued by Alyssa Ney. Ney defends causal foundationalism, which she characterizes as the thesis that facts about difference-making depend upon facts about physical causation. (...) She takes this to imply that causation is not fundamentally a matter of difference-making. In this paper, I defend the difference-making approach against Ney’s argument. I also offer some positive reasons for thinking, pace Ney, that causation is fundamentally a matter of difference-making. (shrink)

The starting point in the development of probabilistic analyses of token causation has usually been the naïve intuition that, in some relevant sense, a cause raises the probability of its effect. But there are well-known examples both of non-probability-raising causation and of probability-raising non-causation. Sophisticated extant probabilistic analyses treat many such cases correctly, but only at the cost of excluding the possibilities of direct non-probability-raising causation, failures of causal transitivity, action-at-a-distance, prevention, and causation by absence and omission. I show that (...) an examination of the structure of these problem cases suggests a different treatment, one which avoids the costs of extant probabilistic analyses. (shrink)

In this article I explore some statistical difficulties confronting going conceptions of ‘group’ as understood in accounts of group selection. Most such theories require real groups but define the reality of groups in ways that make it impossible to test for their reality. There are alternatives, but they either require or invite a nominalism about groups that many theorists abjure.

A number of developmental psychologists have argued for a theory they call the theory theory - a theory of cognitive development that suggests that infants and small children make sense of their world by constructing cognitive representations that have many of the attributes of scientific theories. In this paper I argue that there are indeed close parallels between the activities of children and scientists, but that these parallels will be better understood if one recognizes that both scientists and children are (...) not so much theorists as model builders. (shrink)

Structural models of systems of causal connections have become a common tool in the analysis of the concept of causation. In the present paper I offer a general argument to show that one of the most powerful definitions of the concept of actual cause, provided within the structural models framework, is not sufficient to grant a full account of our intuitive judgements about actual causation, so that we are still waiting for a comprehensive definition. This is done not simply by (...) focusing on a set of case studies, but by arguing that our intuitions about two different kinds of causal patterns, i.e., overdetermination and counterdetermination, cannot be addressed using that definition. (shrink)

The development of causal modelling since the 1950s has been accompanied by a number of controversies, the most striking of which concerns the Markov condition. Reichenbach's conjunctive forks did satisfy the Markov condition, while Salmon's interactive forks did not. Subsequently some experts in the field have argued that adequate causal models should always satisfy the Markov condition, while others have claimed that non-Markovian causal models are needed in some cases. This paper argues for the second position by considering the multi-causal (...) forks, which are widespread in contemporary medicine (Section 2). A non-Markovian causal model for such forks is introduced and shown to be mathematically tractable (Sections 6, 7, and 8). The paper also gives a general discussion of the controversy about the Markov condition (Section 1), and of the related controversy about probabilistic causality (Sections 3, 4, and 5). (shrink)

We suggest to define objective probabilities by similarity-weighted empirical frequencies, where more similar cases get a higher weight in the computation of frequencies. This formula is justified intuitively and axiomatically, but raises the question, which similarity function should be used? We propose to estimate the similarity function from the data, and thus obtain objective probabilities. We compare this definition to others, and attempt to delineate the scope of situations in which objective probabilities can be used.

This paper begins by developing a causal theory of mechanisms in medicine, and illustrates the theory with the example of the mechanism of the disease anthrax as elucidated by Koch. The causal approach to mechanisms is then compared to the Machamer, Darden, Craver approach. At first sight the two approaches appear to be very different, but it is argued that the divergence is less than it initially seems. There are some differences, however, and it is argued that, where these differences (...) occur, the causal approach to mechanisms is superior. (shrink)

Kerr and Godfrey-Smith argue that two mathematically equivalent, alternative formal representations drawn from population genetics, the contextualist and collectivist formalisms, may be equally good for quantifying the dynamics of some natural systems, despite important differences between the formalisms. I draw on constraints on causal representation from Woodward (Making things happen, Oxford University Press, New York, 2003) and Eberhardt and Scheines (Philos Sci 74(5):981–995, 2006) to argue that one or the other formalism will be superior for arbitrary natural systems in which (...) individuals form different types of groups. (shrink)

This paper investigates the relations between causality and propensity. Aparticular version of the propensity theory of probability is introduced, and it is argued that propensities in this sense are not causes. Some conclusions regarding propensities can, however, be inferred from causal statements, but these hold only under restrictive conditions which prevent cause being defined in terms of propensity. The notion of a Bayesian propensity network is introduced, and the relations between such networks and causal networks is investigated. It is argued (...) that causal networks cannot be identified with Bayesian propensity networks, but that causal networks can be a valuable heuristic guide for the construction of Bayesian propensity networks. (shrink)

Some writers have urged that evolutionary theory produces generalizations that hold only ceteris paribus, that is, provided “everything else is equal.” Others have claimed that all laws in the special sciences, or even all laws in science generally, hold only ceteris paribus. However, if we lack a way to determine when everything else really is equal, hedging generalizations with the phrase ceteris paribus renders those generalizations vacuous. I propose a solution to this problem for the case of causal equations from (...) classical population genetics. When coupled with the right proviso, equations in classical population genetics function as strict laws. (shrink)

The paper begins with a discussion of Russell's view that the notion of cause is unnecessary for science and can therefore be eliminated. It is argued that this is true for theoretical physics but untrue for medicine, where the notion of cause plays a central role. Medical theories are closely connected with practical action (attempts to cure and prevent disease), whereas theoretical physics is more remote from applications. This suggests the view that causal laws are appropriate in a context where (...) there is a close connection to action. This leads to a development of an action-related theory of causality which is similar to the agency theory of Menzies and Price, but differs from it in a number of respects, one of which is the following. Menzies and Price connect ‘A causes B’ with an action to produce B by instantiating A, but, particularly in the case of medicine, the law can also be linked to the action of trying to avoid B by ensuring that A is not instantiated. The action-related theory has in common with the agency theory of Menzies and Price the ability to explain causal asymmetry in a simple fashion, but the introduction of avoidance actions together with some ideas taken from Russell enable some of the objections to agency accounts of causality to be met. Introduction Russell on causality Preliminary exposition of the action-related theory Differences between the action-related theory and the agency theory of Menzies and Price Explanation of causal asymmetry Objections to the action-related theory Extension of the theory to the indeterminate case. (shrink)

In this paper I argue that constitutive relevance relations in mechanisms behave like a special kind of causal relation in at least one important respect: Under suitable circumstances constitutive relevance relations produce the Markov factorization. Based on this observation one may wonder whether standard methods for causal discovery could be fruitfully applied to uncover constitutive relevance relations. This paper is intended as a first step into this new area of philosophical research. I investigate to what extent the PC algorithm, originally (...) developed for causal search, can be used for constitutive relevance discovery. I also discuss possible objections and certain limitations of a constitutive relevance discovery procedure based on PC. (shrink)

Schurz (Synthese 164:201–234, 2008) proposed a justification of creative abduction on the basis of the Reichenbachian principle of the common cause. In this paper we take up the idea of combining creative abduction with causal principles and model instances of successful creative abduction within a Bayes net framework. We identify necessary conditions for such inferences and investigate their unificatory power. We also sketch several interesting applications of modeling creative abduction Bayesian style. In particular, we discuss use-novel predictions, confirmation, and the (...) problem of underdetermination in the context of abductive inferences. (shrink)

In this paper we develop an analysis of dispositions by means of causal Bayes nets. In particular, we analyze dispositions as cause-effect structures that increase the probability of the manifestation when the stimulus is brought about by intervention in certain circumstances. We then highlight several advantages of our analysis and how it can handle problems arising for classical analyses of dispositions such as masks, mimickers, and finks.

Mechanisms play an important role in many sciences when it comes to questions concerning explanation, prediction, and control. Answering such questions in a quantitative way requires a formal represention of mechanisms. Gebharter (2014) suggests to represent mechanisms by means of one or more causal arrows of an acyclic causal net. In this paper we show how this approach can be extended in such a way that it can also be fruitfully applied to mechanisms featuring causal feedback.

Causal Bayes nets (CBNs) provide one of the most powerful tools for modelling coarse-grained type-level causal structure. As in other fields (e.g., thermodynamics) the question arises how such coarse-grained characterisations are related to the characterisation of their underlying structure (in this case: token-level causal relations). Answering this question meets what is called a “coherence-requirement” in the reduction debate: How are different accounts of one and the same system (or kind of system) related to each other. We argue that CBNs as (...) tools for type-level causal inference are abstract enough to roughly fit any current token-level theory of causation as long as certain modelling assumptions are satisfied, but accounts of actual causation, i.e. accounts that attempt to infer token-causation based on CBNs, for the very same reason, face certain limitations. (shrink)

The aim of the paper is to relate Bell's notion of local causality to the Causal Markov Condition. To this end, first a framework, called local physical theory, will be introduced integrating spatiotemporal and probabilistic entities and the notions of local causality and Markovity will be defined. Then, illustrated in a simple stochastic model, it will be shown how a discrete local physical theory transforms into a Bayesian network and how the Causal Markov Condition arises as a special case of (...) Bell's local causality and Markovity. (shrink)

According to the theory developed here, we may trace out the processes emanating from a cause in such a way that any consequence lying along one of these processes counts as an effect of the cause. This theory gives intuitive verdicts in a diverse range of problem cases from the literature. Its claims about causation will never be retracted when we include additional variables in our model. And it validates some plausible principles about causation, including Sartorio's ‘Causes as Difference Makers’ (...) principle and Hitchcock's ‘Principle of Sufficient Reason’. (shrink)

I provide a theory of causation within the causal modeling framework. In contrast to most of its predecessors, this theory is model-invariant in the following sense: if the theory says that C caused (didn't cause) E in a causal model, M, then it will continue to say that C caused (didn't cause) E once we've removed an inessential variable from M. I suggest that, if this theory is true, then we should understand a cause as something which transmits deviant or (...) non-inertial behavior to its effect. (shrink)

While structural equations modeling is increasingly used in philosophical theorizing about causation, it remains unclear what it takes for a particular structural equations model to be correct. To the extent that this issue has been addressed, the consensus appears to be that it takes a certain family of causal counterfactuals being true. I argue that this account faces difficulties in securing the independent manipulability of the structural determination relations represented in a correct structural equations model. I then offer an alternate (...) understanding of structural determination, and I demonstrate that this theory guarantees that structural determination relations are independently manipulable. The account provides a straightforward way of understanding hypothetical interventions, as well as a criterion for distinguishing hypothetical changes in the values of variables which constitute interventions from those which do not. It additionally affords a semantics for causal counterfactual conditionals which is able to yield a clean solution to a problem case for the standard ‘closest possible world’ semantics. (shrink)

According to the predictive coding theory of cognition , brains are predictive machines that use perception and action to minimize prediction error, i.e. the discrepancy between bottom–up, externally-generated sensory signals and top–down, internally-generated sensory predictions. Many consider PCT to have an explanatory scope that is unparalleled in contemporary cognitive science and see in it a framework that could potentially provide us with a unified account of cognition. It is also commonly assumed that PCT is a representational theory of sorts, in (...) the sense that it postulates that our cognitive contact with the world is mediated by internal representations. However, the exact sense in which PCT is representational remains unclear; neither is it clear that it deserves such status—that is, whether it really invokes structures that are truly and nontrivially representational in nature. In the present article, I argue that the representational pretensions of PCT are completely justified. This is because the theory postulates cognitive structures—namely action-guiding, detachable, structural models that afford representational error detection—that play genuinely representational functions within the cognitive system. (shrink)

Biological and epidemiological phenomena are often measured with error or imperfectly captured in data. When the true state of this imperfect measure is a confounder of an outcome exposure relationship of interest, it was previously widely believed that adjustment for the mismeasured observed variables provides a less biased estimate of the true average causal effect than not adjusting. However, this is not always the case and depends on both the nature of the measurement and confounding. We describe two sets of (...) conditions under which adjusting for a non-deferentially mismeasured proxy comes closer to the unidentifiable true average causal effect than the unadjusted or crude estimate. The first set of conditions apply when the exposure is discrete or continuous and the confounder is ordinal, and the expectation of the outcome is monotonic in the confounder for both treatment levels contrasted. The second set of conditions apply when the exposure and the confounder are categorical. In all settings, the mismeasurement must be non-differential, as differential mismeasurement, particularly an unknown pattern, can cause unpredictable results. (shrink)

In ‘The logic of partial supposition’ (Analysis vol. 81), Benjamin Eva and Stephan Hartmann investigate partial imaging, a credence-revision method which combines the partiality familiar from Jeffrey Conditioning(The Logic of Decision, 1983) with the formal notion of imaging familiar from Lewis’s ‘Causal decision theory’ (1981). They argue that because partial imaging is non-monotonic, it ‘fail[s] to provide a plausible account of the norms of partial subjunctive suppositions’.In this note, I present a notion of partial imaging that does satisfy monotonicity, and (...) discuss some of the applications and ramifications. The account frames conditioning as a form of imaging, and rejects Gärdenfors’s principle of linearity in ‘Imaging and conditionalization’ (1982). (shrink)

I respond to a dilemma for Causal Decision Theory (CDT) under determinism, posed in Adam Elga's paper “Confessions of a Causal Decision Theorist”. The treatment I present highlights (i) the status of laws as predictors, and (ii) the consequences of decision dependence, which arises natively out of Jeffrey Conditioning and CDT's characteristic equation.My argument leverages decision dependence to work around a key assumption of Elga's proof: to wit, that in the two problems he presents, the CDTer must employ subjunctive-suppositional (rather (...) than evidential) transformations of a shared prior. (shrink)

Do accounts of scientific theory formation and revision have implications for theories of everyday cognition? We maintain that failing to distinguish between importantly different types of theories of scientific inference has led to fundamental misunderstandings of the relationship between science and everyday cognition. In this article, we focus on one influential manifestation of this phenomenon which is found in Fodor's well-known critique of theories of cognitive architecture. We argue that in developing his critique, Fodor confounds a variety of distinct claims (...) about the holistic nature of scientific inference. Having done so, we outline more promising relations that hold between theories of scientific inference and ordinary cognition. (shrink)

Two long-standing arguments in cognitive science invoke the assumption that holistic inference is computationally infeasible. The first is Fodor’s skeptical argument toward computational modeling of ordinary inductive reasoning. The second advocates modular computational mechanisms of the kind posited by Cosmides, Tooby and Sperber. Based on advances in machine learning related to Bayes nets, as well as investigations into the structure of scientific and ordinary information, I maintain neither argument establishes its architectural conclusion. Similar considerations also undermine Fodor’s decades-long diagnosis of (...) artificial intelligence research as confounded by an inability to circumscribe the amount of information relevant to inferential processes. This diagnosis is particularly inapposite with respect to Bayes nets, since one of their strengths as machine learning systems has been their capacity to reason probabilistically about large data sets whose size overwhelms the capacities of individual human reasoners. A general moral follows from these criticisms: Insights into artificial and human cognitive systems are likely to be cultivated by focusing greater attention on the structure and density of connections among items of information that are available to them. (shrink)

Many contemporary philosophers of physics (and philosophers of science more generally) follow Bertrand Russell in arguing that there is no room for causal notions in physics. Causation, as James Woodward has put it, has a ‘human face’, which makes causal notions sit ill with fundamental theories of physics. In this paper I examine a range of anti-causal arguments and show that the human face of causation is the face of scientific representations much more generally. Physics, like other sciences, is deeply (...) permeated with causal reasoning. (shrink)

Classical dispersion relations are derived from a time-asymmetric constraint. I argue that the standard causal interpretation of this constraint plays a scientifically legitimate role in dispersion theory, and hence provides a counterexample to the causal skepticism advanced by John Norton and others. Norton ([2009]) argues that the causal interpretation of the time-asymmetric constraint is an empty honorific and that the constraint can be motivated by purely non-causal considerations. In this paper I respond to Norton's criticisms and argue that Norton's skepticism (...) derives its force partly by holding causal principles to a standard too high to be met by other scientifically legitimate constraints. (shrink)

The idea that the brain is an information processing system raises some challenging questions about whether information exists independently of brains. Answering these questions is relevant for clarifying the theoretical foundations of the sciences of mind and brain, but also for appropriately interpreting and evaluating the evidence about how brains—and other biological systems—work. This article claims that (1) informational descriptions in the sciences of mind and brain can be genuinely explanatory, despite assuming a mind-dependent notion of information; and (2) that (...) either Popperian objectivity or response-dependence in normal receivers is sufficient for grounding the explanatory role that information often plays in these sciences. Hierarchical predictive processing, which has become a central theoretical framework for neurocognitive research, is used as a case study for supporting these two claims. (shrink)

Recent approaches to causal modelling rely upon the causal Markov condition, which specifies which probability distributions are compatible with a directed acyclic graph. Further principles are required in order to choose among the large number of DAGs compatible with a given probability distribution. Here we present a principle that we call frugality. This principle tells one to choose the DAG with the fewest causal arrows. We argue that frugality has several desirable properties compared to the other principles that have been (...) suggested, including the well-known causal faithfulness condition. _1_ Introduction _2_ The Causal Markov Condition _3_ Faithfulness _4_ Frugality _4.1_ Basic independences and frugality _4.2_ General properties of directed acyclic graphs satisfying frugality _4.3_ Connection to minimality assumptions _5_ Frugality as a Parsimony Principle _6_ Conclusion Appendix. (shrink)

Do evolutionary processes such as selection and random drift cause evolutionary change, or are they merely convenient ways of describing or summarizing it? Philosophers have lined up on both sides of this question. One recent defense (Reisman and Forber 2005) of the causal status of selection and drift appeals to a manipulability theory of causation. Yet, even if one accepts manipulability, there are still reasons to doubt that genetic drift, in particular, is genuinely causal. We will address two challenges to (...) treating drift as causal within a manipulation framework. We will argue that both challenges ultimately fail, but that they raise interesting and subtle issues about the nature of causation and the differences between selection and drift. ‡Thanks to audiences at the ‘PBDB1' Conference and the 2006 PSA Meeting for valuable feedback. †To contact the authors, please write to: Patrick Forber, Tufts University, Philosophy Department, Miner Hall, Medford, MA 02155; e-mail: [email protected]. (shrink)

The likelihood theory of evidence (LTE) says, roughly, that all the information relevant to the bearing of data on hypotheses (or models) is contained in the likelihoods. There exist counterexamples in which one can tell which of two hypotheses is true from the full data, but not from the likelihoods alone. These examples suggest that some forms of scientific reasoning, such as the consilience of inductions (Whewell, 1858. In Novum organon renovatum (Part II of the 3rd ed.). The philosophy of (...) the inductive sciences. London: Cass, 1967), cannot be represented within Bayesian and Likelihoodist philosophies of science. (shrink)

In this paper I present a limitation to what may be called strictly-interventionistic causal-model semantic theories for subjunctive conditionals. And I offer a line of response to Briggs’ counterexample to Modus Ponens—given within a strictly-interventionistic framework—for the subjunctive conditional. The paper also contains some discussion of backtracking counterfactuals and backtracking interpretations. The limitation inherent to strict interventionism is brought out via a class of counterexamples. A causal-model semantics is strictly interventionistic just in case the procedure it gives for evaluating a (...) subjunctive conditional requires making the values of the variables implicated in the antecedent independent from the values of the parents of these antecedent variables. Most causal-model semantic theories that have gained attention are strictly interventionistic. (shrink)

This paper explores the prospects of employing a functional approach in order to improve our concept of actual causation. Claims of actual causation play an important role for a variety of purposes. In particular, they are relevant for identifying suitable targets for intervention, and they are relevant for our practices of ascribing responsibility. I argue that this gives rise to the challenge of purpose. The challenge of purpose arises when different goals demand adjustments of the concept that pull in opposing (...) directions. More specifically, I argue that a common distinction between certain kinds of preempted and preempting factors is difficult to motivate from an interventionist viewpoint. This indicates that an appropriately revised concept of actual causation would not distinguish between these two kinds of factors. From the viewpoint of retributivist responsibility, however, the distinction between preempted and preempting factors sometimes is important, which indicates that the distinction should be retained. (shrink)

When we deliberate about what to do, we appear to be free to decide on different options. Three accounts use ordinary beliefs to explain this apparent freedom—appealing to different types of ‘epistemic freedom’. When an agent has epistemic freedom, her evidence while deliberating does not determine what decision she makes. This ‘epistemic gap’ between her evidence and decision explains why her decision appears free. The varieties of epistemic freedom appealed to might look similar. But there is an important difference. Two (...) rely on an agent's ability to justifiably form beliefs unconstrained by evidence, and identify decisions as beliefs—either beliefs about acts or about decisions. But, when used to explain apparent freedom, these accounts face serious problems: they imply that agents have epistemic freedom over evidence-based beliefs, and rely on a faulty notion of justification. Underlying these troubles, it turns out that these accounts presuppose an unexplained apparent ability to form different beliefs. A third variety of epistemic freedom uses ignorance conditions instead (Levi and Kapitan). We appear free partly because we’re ignorant of what we’ll decide. Ignorance-based accounts avoid the above problems, and remain a promising alternative. (shrink)

We standardly evaluate counterfactuals and abilities in temporally asymmetric terms—by keeping the past fixed and holding the future open. Only future events depend counterfactually on what happens now. Past events do not. Conversely, past events are relevant to what abilities one has now in a way that future events are not. Lewis, Sider and others continue to evaluate counterfactuals and abilities in temporally asymmetric terms, even in cases of backwards time travel. I’ll argue that we need more temporally neutral methods. (...) The past shouldn’t always be held fixed, because backwards time travel requires backwards counterfactual dependence. Future events should sometimes be held fixed, because they’re in the causal history of the past, and agents have evidence of them independently of their decisions now. We need temporally neutral methods to maintain connections between causation, counterfactuals and evidence, and if counterfactuals are used to explain the temporal asymmetry of causation. (shrink)

Do time travellers retain their normal freedom and abilities when they travel back in time? Lewis, Horwich and Sider argue that they do. Time-travelling Tim can kill his young grandfather, his younger self, or whomever else he pleases—and so, it seems can reasonably deliberate about whether to do these things. He might not succeed. But he is still just as free as a non-time traveller. I’ll disagree. The freedom of time travellers is limited by a rational constraint. Tim can’t reasonably (...) deliberate on killing his grandfather, certain that he’ll fail. If Tim follows his evidence, and appropriately self-predicts, he will be certain he won’t kill his grandfather. So if Tim is both evidentially and deliberatively rational, he can’t deliberate on killing his grandfather. This result has consequences. Firstly, it shows how evidential limits in the actual world contribute to our conception of the future as open. Secondly, it undercuts arguments against the possibility of time travel. Thirdly, it affects how we evaluate counterfactuals in time travel worlds, as well as our own. I’ll use the constraint to motivate an evidential and temporally neutral method of evaluating counterfactuals that holds fixed what a relevant deliberating agent has evidence of, independently of her decision. Using this method, an agent’s local abilities may be affected by what happens globally at other times, including the future. (shrink)

Fundamental physics makes no clear use of causal notions; it uses laws that operate in relevant respects in both temporal directions and that relate whole systems across times. But by relating causation to evidence, we can explain how causation fits in to a physical picture of the world and explain its temporal asymmetry. This paper takes up a deliberative approach to causation, according to which causal relations correspond to the evidential relations we need when we decide on one thing in (...) order to achieve another. Tamsin's taking her umbrella is a cause of her staying dry, for example, if and only if her deciding to take her umbrella for the sake of staying dry is adequate grounds for believing she'll stay dry. This correspondence explains why causation matters: knowledge of causal structure helps us make decisions that are evidence of outcomes we seek. The account also explains why we can control the future and not the past, and why causes come before their effects. When agents properly deliberate, their decisions can never count as evidence for any outcomes they may seek in the past. From this it follows that causal relations don't run backwards. This deliberative asymmetry is itself traced back to asymmetries of evidence and entropy, providing a new way of deriving causal asymmetry from temporally symmetric laws. (shrink)

In their article 'Causes and Explanations: A Structural-Model Approach. Part I: Causes', Joseph Halpern and Judea Pearl draw upon structural equation models to develop an attractive analysis of 'actual cause'. Their analysis is designed for the case of deterministic causation. I show that their account can be naturally extended to provide an elegant treatment of probabilistic causation.

ABSTRACT Joseph Halpern and Judea Pearl draw upon structural equation models to develop an attractive analysis of ‘actual cause’. Their analysis is designed for the case of deterministic causation. I show that their account can be naturally extended to provide an elegant treatment of probabilistic causation. 1Introduction 2Preemption 3Structural Equation Models 4The Halpern and Pearl Definition of ‘Actual Cause’ 5Preemption Again 6The Probabilistic Case 7Probabilistic Causal Models 8A Proposed Probabilistic Extension of Halpern and Pearl’s Definition 9Twardy and Korb’s Account 10Probabilistic (...) Fizzling 11Conclusion. (shrink)

A widespread assumption in the contemporary discussion of probabilistic models of cognition, often attributed to the Bayesian program, is that inference is optimal when the observer's priors match the true priors in the world—the actual “statistics of the environment.” But in fact the idea of a “true” prior plays no role in traditional Bayesian philosophy, which regards probability as a quantification of belief, not an objective characteristic of the world. In this paper I discuss the significance of the traditional Bayesian (...) epistemic view of probability and its mismatch with the more objectivist assumptions about probability that are widely held in contemporary cognitive science. I then introduce a novel mathematical framework, the observer lattice, that aims to clarify this issue while avoiding philosophically tendentious assumptions. The mathematical argument shows that even if we assume that “ground truth” probabilities actually do exist, there is no objective way to tell what they are. Different observers, conditioning on different information, will inevitably have different probability estimates, and there is no general procedure to determine which one is right. The argument sheds light on the use of probabilistic models in cognitive science, and in particular on what exactly it means for the mind to be “tuned” to its environment. (shrink)

Certain hypotheses cannot be directly confirmed for theoretical, practical, or moral reasons. For some of these hypotheses, however, there might be a workaround: confirmation based on analogical reasoning. In this paper we take up Dardashti, Hartmann, Thébault, and Winsberg’s (in press) idea of analyzing confirmation based on analogical inference Baysian style. We identify three types of confirmation by analogy and show that Dardashti et al.’s approach can cover two of them. We then highlight possible problems with their model as a (...) general approach to analogical inference and argue that these problems can be avoided by supplementing Bayesian update with Jeffrey conditionalization. (shrink)