Minimally, causal realism (as understood here) is the view that accounts of causation in terms of mere, regular or probabilistic conjunction are unsatisfactory, and that causal phenomena are correctly associated with some form of de re necessity. Classic arguments, however, some of which date back to Sextus Empiricus and have appeared many times since, including famously in Russell, suggest that the very notion of causal realism is incoherent. In this paper I argue that if such objections seem compelling, it is (...) only because everyday expressions concerning causal phenomena are misleading with respect to certain metaphysical details. These expressions generally make reference to the relations of events or states of affairs, but ignore or obscure the role played by causal properties. I argue that on a proposed alternative, an analysis in terms of causal processes, more refined descriptions of causal phenomena escape the charge of incoherence. Causal necessity is here located in the relations of causal properties. I distinguish this view from the recent process theories of Salmon and Dowe, which are disinterested in causal realism. (shrink)

1. Causal knowledge is an indispensable element in science. Causal assertions are embedded in both the results and the procedures of scientific investigation. 2. It is therefore worthwhile to investigate the meaning of causal statements and the ways in which we can arrive at causal knowledge.

Ehring shows the inadequacy of received theories of causation, and, introducing conceptual devices of his own, provides a wholly new account of causation as the persistence over time of individual properties, or "tropes.".

When two causally independent processes each have a quantity that increases monotonically (either deterministically or in probabilistic expectation), the two quantities will be correlated, thus providing a counterexample to Reichenbach's principle of the common cause. Several philosophers have denied this, but I argue that their efforts to save the principle are unsuccessful. Still, one salvage attempt does suggest a weaker principle that avoids the initial counterexample. However, even this weakened principle is mistaken, as can be seen by exploring the concepts (...) of homology and homoplasy used in evolutionary biology. I argue that the kernel of truth in the principle of the common cause is to be found by separating metaphysical and epistemological issues; as far as the epistemology is concerned, the Likelihood Principle is central. (shrink)

Causation is widely assumed to be a binary relation: c causes e. I will argue that causation is a quaternary, contrastive relation: c rather than C* causes e rather than E*, where C* and E* are nonempty sets of contrast events. Or at least, I will argue that treating causation as contrastive helps resolve some paradoxes.

This paper examines a promising probabilistic theory of singular causation developed by David Lewis. I argue that Lewis' theory must be made more sophisticated to deal with certain counterexamples involving pre-emption. These counterexamples appear to show that in the usual case singular causation requires an unbroken causal process to link cause with effect. I propose a new probabilistic account of singular causation, within the framework developed by Lewis, which captures this intuition.

This paper examines Wesley Salmon's "process" theory of causality, arguing in particular that there are four areas of inadequacy. These are that the theory is circular, that it is too vague at a crucial point, that statistical forks do not serve their intended purpose, and that Salmon has not adequately demonstrated that the theory avoids Hume's strictures about "hidden powers". A new theory is suggested, based on "conserved quantities", which fulfills Salmon's broad objectives, and which avoids the problems discussed.

There is, no doubt, a temptation to treat preventions, such as ‘the father’s grabbing the child prevented the accident’, and cases of ‘causation’ by omission, such as ‘the father’s inattention was the cause of the child’s accident’, as cases of genuine causation. I think they are not, and in this paper I defend a theory of what they are. More specifically, the counterfactual theory defended here is that a claim about prevention or ‘causation’ by omission should be understood not as (...) being directly about actual genuine causation but primarily as a counterfactual claim about genuine causation.1 The relation between actual causation and the mere possibility of causation allows my theory to explain both the difference and the similarity between the two notions (causation and prevention/omission). Further, the difference explains certain intuitions we have and the similarity justifies and explains the fact that for practical purposes we usually treat preventions and omissions as if they were genuine causation. Finally, the fact that this counterfactual theory of prevention and omission takes causation as primitive suggests that it is consistent with any theory of causation. This allows us to construct two arguments against what I will call genuinism, the view that cases of prevention and ‘causation’ by omission really are cases of genuine causation. In section II I show that genuinism does not account for the so-called intuition of difference. In section III I outline a number of problems that various theories of causation have with preventions and ‘causation’ by omission. These problems are ipso facto problems for genuinism, whereas I show in section V how the counterfactual theory solves those problems. Further, I answer a genuinist argument based on another type of intuition—the genuinist intuition—by showing why we have that intuition and how it should be handled (section VI). (shrink)

La autora presenta algunas criticas generales al proyecto de reducir las leyes causales a probabilidades. Además, muestra que las leyes causales son imprescindibles para poder diferenciar las strategias efectivas de las que no lo son y da un criterio para considerar cuando podemos deducir causalidad a través de datos estadísticos.

What is the nature of causation? How is causation linked with explanation? And can there be an adequate theory of explanation? These questions and many others are addressed in this unified and rigorous examination of the philosophical problems surrounding causation, laws and explanation. Part 1 of this book explores Hume's views on causation, theories of singular causation, and counterfactual and mechanistic approaches. Part 2 considers the regularity view of laws and laws as relations among universals, as well as recent alternative (...) approaches to laws. Part 3 examines the issues arising from deductive-nomological explanation, statistical explanation, the explanation of laws and the metaphysics of explanation. Accessible to readers of all levels, this book provides an excellent introduction to one of the most enduring problems of philosophy. (shrink)

Scottish philosopher Thomas Brown held the chair of moral philosophy at the University of Edinburgh. He was distinguished for his work in the philosophy of mind and causation, and was a founder member of the Edinburgh Review. At the beginning of the nineteenth century, controversy arose over John Leslie being appointed to the chair of mathematics at the university. City ministers opposed him because he defended Hume's view of causation, which was seen as being incompatible with the existence of God. (...) In 1805 Brown wrote a pamphlet, Observations on the Nature and Tendency of the Doctrine of Mr. Hume Concerning the Relation of Cause and Effect, which among other things aimed to show that Hume's theory was compatible with belief in God. This book, first published in 1818, is the third edition of that original pamphlet, which grew to become a thorough examination of the philosophy of causation. (shrink)

showing what makes causal facts both true and accessible enough for us to have the knowledge of them that we ordinarily take ourselves to have. Some current approaches to analyzing causation were once resisted. First, analyses that use the counterfactual conditional were viewed with suspicion because philosophers also sought (and still do seek) similar understanding of counterfactual facts. Since the same can be said for the other nomic concepts--causation, lawhood, explanation, chance, dispositions, and their conceptual kin--philosophy demonstrated a preference for (...) non-nomic definitions of causation, analytic completions of (S) with no nomic terms in the analysans. Recently, however, philosophers have been less demanding regarding what terms may be used. Attention has been given to analyzing causation in terms of chance, the counterfactual conditional, and lawhood. If we reserve the term ‘causal’ for the terms and concepts that have extremely obvious connections.. (shrink)

In this paper I examine a recent argument that regularity approaches to causation can easily solve the problem of pre-emption. If this argument were successful it would neatly solve the problem of pre-emption—a problem that many still consider to be a central unsolved problem for accounts of causation. The argument is surprising in that the conclusion goes against the common consensus that regularity accounts of causation cannot solve the problem of pre-emption, at least without major amendments. This consensus was one (...) of the reasons for the decline in popularity of the regularity approach and the rise in popularity (for a few decades at least) of the counterfactual approach. In its fullest form the recent argument is due to Strevens (Causation and explanation, topics in contemporary philosophy. MIT Press, Cambridge, MA, 2007). He claims, "Mackie's account supplies, without any of the complex amendments now standard in counterfactual theories, a completely satisfactory treatment of the standard cases of pre-emption". This paper examines this argument and refutes it. I argue that Mackie's account really does fail to solve the problem of pre-emption; it fails to account for even the standard cases of pre-emption in the literature. (shrink)

A distinction is drawn between property causation and token causation. According to the former, a positive causal factor in a population raises the probability of its effects within "background contexts". The latter, which concerns "actual physical connections" between token events, is not explicated here although its distinctness from the first concept and its importance are discussed. The applicability of both is illustrated by two currently controversial issues in evolutionary theory -- the units of selection controversy and the use of parsimony (...) as an inference rule in phylogenetic reconstruction. (shrink)

In this paper I reconsider a standard counterexample to the chance-raising theory of singular causation. Extant versions of this theory are so different that it is difficult to formulate the core thesis that they all share, despite the guiding idea that causes raise the chance of their effects. At one extreme, ‘Humean’ theories – which can be traced to Reichenbach – say that a particular event of type C is the cause of a particular event of type E only if (...) P(E|C & K) > P(E|~C & K) where K is a set of background conditions and where the probabilities are interpreted as relative frequencies. At the other extreme, explicitly non-Humean theories take chance to be a physical, particular, local feature of the world. (shrink)

Causation, says David Lewis now, is to be understood as the ancestral of counterfactual influence, where C influences E (roughly) iff little changes in C map onto big changes in E. I argue that the influence account provides neither necessary nor sufficient conditions for causation, and suggest that what is missing is the notion of effluence, or physical connection.

Process theories of causality seek to explicate causality as a property of individual causal processes. This paper examines the capacity of such theories to account for the asymmetry of causation. Three types of theories of asymmetry are discussed; the subjective, the temporal, and the physical, the third of these being the preferred approach. Asymmetric features of the world, namely the entropic and Kaon arrows, are considered as possible sources of causal asymmetry and a physical theory of asymmetry is subsequently developed (...) with special reference to the questions of objectivity and backwards causation. (shrink)

Germund Hesslow has argued recently [2] that a probabilistic theory of causality as advocated by Patrick Suppes [4] has two problems that a deterministic theory avoids. In this paper, I argue that Suppes' probabilistic causal calculus is free of each of these problems and, moreover, that several broader issues raised by Hesslow's discussion tend to support a probabilistic conception of causes.

For a long time, regularity accounts of causation have virtually vanished from the scene. Problems encountered within other theoretical frameworks have recently induced authors working on causation, laws of nature, or methodologies of causal reasoning – as e.g. May (Kausales Schliessen. Eine Untersuchung über kausale Erklärungen und Theorienbildung. Ph.D. thesis, Universität Hamburg, Hamburg, 1999), Ragin (Fuzzy-set social science. Chicago: University of Chicago Press, 2000), Graßhoff and May (Causal regularities. In W. Spohn, M. Ledwig, & M. Esfeld (Eds.), Current issues in (...) causation (pp. 85–114). Paderborn: Mentis, 2001), Swartz (The concept of physical law (2nd ed.). http://www.sfu.ca/philosophy/physical-law/, 2003), Halpin (Erkenntnis, 58, 137–168, 2003) – to direct their attention back to regularity theoretic analyses. In light of the latest proposals of regularity theories, the paper at hand therefore reassesses the criticism raised against regularity accounts since the INUS theory of causation of Mackie (The cement of the universe. A study of causation. Oxford: Clarendon Press, 1974). It is shown that most of these objections target strikingly over-simplified regularity theoretic sketches. By outlining ways to refute these objections it is argued that the prevalent conviction as to the overall failure of regularity theories has been hasty. (shrink)

The authors demonstrate that Hume's views can stand up to contemporary criticism and are relevant to current debates on causality.

This paper discusses several distinct process theories of causality offered in recent years by Phil Dowe and me. It addresses problems concerning the explication of causal process, causal interaction, and causal transmission, whether given in terms of transmission of marks, transmission of invariant or conserved quantities, or mere possession of conserved quantities. Renouncing the mark-transmission and invariant quantity criteria, I accept a conserved quantity theory similar to Dowe's--differing basically with respect to causal transmission. This paper also responds to several fundamental (...) constructive criticisms contained in Christopher Hitchcock's discussion of both the mark-transmission and the conserved quantity theories. (shrink)

This paper presents a drastically revised version of the theory of causality, based on analyses of causal processes and causal interactions, advocated in Salmon (1984). Relying heavily on modified versions of proposals by P. Dowe, this article answers penetrating objections by Dowe and P. Kitcher to the earlier theory. It shows how the new theory circumvents a host of difficulties that have been raised in the literature. The result is, I hope, a more satisfactory analysis of physical causality.

Causation has traditionally been analyzed either as a relation of nomic dependence or as a relation of counterfactual dependence. I argue for a third program, a physicalistic reduction of the causal relation to one of energy-momentum transference in the technical sense of physics. This physicalistic analysis is argued to have the virtues of easily handling the standard counterexamples to the nomic and counterfactual analyses, offering a plausible epistemology for our knowledge of causes, and elucidating the nature of the relation between (...) causation and physical science. (shrink)

In this important book, Ellery Eells explores and refines philosophical conceptions of probabilistic causality. In a probabilistic theory of causation, causes increase the probability of their effects rather than necessitate their effects in the ways traditional deterministic theories have specified. Philosophical interest in this subject arises from attempts to understand population sciences as well as indeterminism in physics. Taking into account issues involving spurious correlation, probabilistic causal interaction, disjunctive causal factors, and temporal ideas, Professor Eells advances the analysis of what (...) it is for one factor to be a positive causal factor for another. A salient feature of the book is a theory of token level probabilistic causation in which the evolution of the probability of a later event from an earlier event is central. This will be a book of crucial significance to philosophers of science and metaphysicians; it will also prove stimulating to many economists, psychologists, and physicists. (shrink)

Cases of overdetermination or preemption continue to play an important role in the debate about the proper interpretation of causal claims of the form "C was a cause of E". I argue that the best treatment of preemption cases is given by Mackie's venerable INUS account of causal claims. The Mackie account suffers, however, from problems of its own. Inspired by its ability to handle preemption, I propose a dramatic revision to the Mackie account – one that Mackie himself would (...) certainly have rejected – to solve these difficulties. The result is, I contend, a very attractive account of singular causal claims. (shrink)