According to Woodward’s influential account of explanation, explanations have a counterfactual structure, and explanatory counterfactuals are analysed in terms of causal relations and interventions. In this paper, we provide a formal semantics of explanatory counterfactuals based on a Ramsey Test semantics of conditionals. Like Woodward’s account, our account is guided by causal considerations. Unlike Woodward’s account, it makes no reference to causal graphs and it also covers cases of explanation where interventions are impossible.

We propose an analysis of backward causation in terms of interventionism that can avoid several problems typically associated with backward causation. Its main advantage over other accounts is that it allows for reducing the problematic task of supporting backward causal claims to the unproblematic task of finding evidence for several ordinary forward directed causal hypotheses.

The essence of an accident consists in its relationship to a substance. For we should not imagine that an accident is a thing in its own right to which gets attached a relationship or a link to a substance in which that accident exists. For if so, an accident would be something in its own right, dependent on substance only as extrinsic, and on this view, an accident could be cognized apart from the substance. These outcomes are impossible, however. Hence, (...) what an accident is to something of the substance: either a measure, or a state, and so on. Thus, the Philosopher says that an accident has being only because it belongs to something that has being. (shrink)

State of art paper on the topic causation, around the problem of the nature of causation. Central theories of contemporary philosophical literature are discussed and analysed, namely, regularity theories of Hume and Mackie, counterfactual theories of Lewis, probabilistic theories of Reichenbach, Lewis and Menzies and causal processes theories of Salmon and Dowe.

Achieving understanding of nature is one of the aims of science. In this paper we offer an analysis of the nature of scientific understanding that accords with actual scientific practice and accommodates the historical diversity of conceptions of understanding. Its core idea is a general criterion for the intelligibility of scientific theories that is essentially contextual: which theories conform to this criterion depends on contextual factors, and can change in the course of time. Our analysis provides a general account of (...) how understanding is provided by scientific explanations of diverse types. In this way, it reconciles conflicting views of explanatory understanding, such as the causal-mechanical and the unificationist conceptions. (shrink)

The Doctrine of Double Effect [DDE] states roughly that it is harder to justify causing or allowing harm as a means to an end than it is to justify conduct that results in harm as a side effect. This chapter argues that a theory of deontological constraints on harming needs something like the DDE in order to avoid the charge that it reflects a narcissistic obsession with the cleanliness of our own hands. Unfortunately, the DDE is often interpreted as maintaining (...) that we must avoid acting with certain intentions, which, this chapter contends, embodies an equally narcissistic obsession with the purity of our own hearts. The chapter argues that the DDE is better interpreted as a denial of the Machiavellian idea that beneficial ends justify harmful means. On this view, the objective fact that our conduct will secure benefits for some individuals at the expense of other individuals weakens the extent to which those benefits count as reasons to engage in that conduct. The chapter argues that this version of the DDE provides a plausible, non-narcissistic foundation for deontological constraints. (shrink)

We have two main aims: to construct mathematical models for analysing determinism, causality and supervenience; and then to use these to demonstrate the possibility of constructing an ontic construal of the operation of free will - one requiring both the presentation of genuine alternatives to an agent and their selecting between them in a manner that permits the attribution of responsibility. Determinism is modelled using trans-temporal ontic links between discrete juxtaposed universe states and shown to be distinct from predictability. Causality (...) is defined on a temporal sequence of δ-algebras and quantified using a measure. The measure leads to definitions of causal overdetermination and epiphenomena. Proofs are constructed to demonstrate deterministic universes must carry their properties essentially but not necessarily locally. We argue determinism and causality are separate doctrines. These models and results are marshalled to put the case that a counterfactual construal of ontic choice cannot work. In response we propose ‘immanence’ - a modified form of indeterminism whereby a universe can present choices to its denizens. We prove that beings subsumed within a universe cannot pilot their own actions. We then argue these beings can exercise free will only when selecting between choices inhering within immanent relata. A being is responsible for its selections if and only if it is constituted of a temporally evolving deterministic substructure. Our proposal is novel: it avoids injecting indeterminism into the decision process. Topological models for property supervenience are developed and used to reconstruct standard definitions from the literature. These are then used to demonstrate considerations of supervenience do not affect our arguments. We have demonstrated that a model of the exercise of free will involving both genuine choices and responsibility is possible but can only operate within a non-deterministic universe possessing specific traits. (shrink)

Mental causation is a problem and not just a problem for the nonphysicalist. One of the many lessons learned from Jaegwon Kim’s writings in the philosophy of mind is that mental causation is a problem for the nonreductive physicalist as well. A central component of the common sense picture we have of ourselves as persons is that our beliefs and desires causally explain our actions. But the completeness of the “brain sciences” threatens this picture. If all of our actions are (...) causally explained by neurophysiological events occurring in our brains, what causal role is left for our reasons and motives? It would seem that these brain events do all the causal work there is to do, thus robbing the mental of its efficacy altogether or else making it a merely superfluous or redundant causal factor. This essay presents a systematic treatment of this exclusion dilemma from the perspective of a nonreductive physicalist. I argue that both horns of this dilemma can be avoided if we ground mental causation in counterfactual dependence between distinct events and understand the mind-body relation as event realization. Although in the final analysis our actions are overdetermined by their mental and neurophysiological antecedents, this overdetermination is entirely unproblematic. (shrink)

The goal of this paper is to present and defend an empiricist, neo-Hempelian account of scientific explanation as ampliative, specialized embedding. The proposal aims to preserve what I take to be the core of Hempel’s empiricist account, by weakening it in some respects and strengthening it in others, introducing two new conditions that solve most of Hempel’s problems without abandoning his empiricist strictures. According to this proposal, to explain a phenomenon is to make it expectable by introducing new conceptual/ontological machinery (...) and using special, and non-ad hoc, non-accidental regularities. The new conditions are elaborated making essential use of two central structuralist ideas, namely T-theoreticity and specialization. I first introduce and qualify the project, then present the new account in detail and assess it vis-à-vis its rivals, and finally discuss some possible objections, concluding that the account fares better than its monistic rivals and well enough to qualify as a promising neo-Hempelian account. Even for those unpersuaded by its monistic goals, it has the merit of calling attention to two new necessary conditions not explicitly emphasized thus far and showing how they serve to answer many of the criticisms addressed against Hempel’s account. (shrink)

I argue that, on a dispositionalist account of causation and indeed on any other view of causation according to which causation is a real relation, general relativity does not give causal principles a role in explaining phenomena. In doing so, I bring out a surprisingly substantial constraint on adequate views about the explanations and ontology of GR, namely the requirement that such views show how GR can explain motion that is free of disturbing influences.

In this thesis, I give a metascientific account of causality in medicine. I begin with two historical cases of causal discovery. These are the discovery of the causation of Burkitt’s lymphoma by the Epstein-Barr virus, and of the various viral causes suggested for cervical cancer. These historical cases then support a philosophical discussion of causality in medicine. This begins with an introduction to the Russo- Williamson thesis (RWT), and discussion of a range of counter-arguments against it. Despite these, I argue (...) that the RWT is historically workable, given a small number of modifications. I then expand Russo and Williamson’s account. I first develop their suggestion that causal relationships in medicine require some kind of evidence of mechanism. I begin with a number of accounts of mechanisms and produce a range of consensus features of them. I then develop this consensus position by reference to the two historical case studies with an eye to their operational competence. In particular, I suggest that it is mechanistic models and their representations which we are concerned with in medicine, rather than the mechanism as it exists in the world. -/- I then employ these mechanistic models to give an account of the sorts of evidence used in formulating and evaluating causal claims. Again, I use the two human viral oncogenesis cases to give this account. I characterise and distinguish evidence of mechanism from evidence of difference-making, and relate this to mechanistic models. I then suggest the relationship between types of evidence presents us with a means of tackling the reference-class problem. This sets the scene for the final chapter. Here, I suggest the manner in which these two different classes of evidence become integrated is also reflected in the way that developing research programmes change as their associated causal claims develop. (shrink)

This paper critiques the new mechanistic explanatory program on grounds that, even when applied to the kinds of examples that it was originally designed to treat, it does not distinguish correct explanations from those that blunder. First, I offer a systematization of the explanatory account, one according to which explanations are mechanistic models that satisfy three desiderata: they must 1) represent causal relations, 2) describe the proper parts, and 3) depict the system at the right ‘level.’ Second, I argue that (...) even the most developed attempts to fulfill these desiderata fall short by failing to appropriately constrain explanatorily apt mechanistic models. -/- *This paper used to be called "The Emperor's New Mechanisms". (shrink)

Negative causation occurs when an absence serves as cause, effect, or causal intermediary. Negative causation is genuine causation, or so I shall argue. It involves no physical connection between cause and effect. Thus causes need not be physically connected to their effects.

Standard models of statistical explanation face two intractable difficulties. In his 1984 Salmon argues that because statistical explanations are essentially probabilistic we can make sense of statistical explanation only by rejecting the intuition that scientific explanations are contrastive. Further, frequently the point of a statistical explanation is to identify the etiology of its explanandum, but on standard models probabilistic explanations often fail to do so. This paper offers an alternative conception of statistical explanations on which explanations of the frequency of (...) a property consist in the derivation of that frequency from a statistical specification of the mechanism by which instances of the relevant property are produced. Such explanations are contrastive precisely because they identify the determinate causal etiologies of their explananda. (shrink)

There is, no doubt, a temptation to treat preventions, such as ‘the father’s grabbing the child prevented the accident’, and cases of ‘causation’ by omission, such as ‘the father’s inattention was the cause of the child’s accident’, as cases of genuine causation. I think they are not, and in this paper I defend a theory of what they are. More specifically, the counterfactual theory defended here is that a claim about prevention or ‘causation’ by omission should be understood not as (...) being directly about actual genuine causation but primarily as a counterfactual claim about genuine causation.1 The relation between actual causation and the mere possibility of causation allows my theory to explain both the difference and the similarity between the two notions (causation and prevention/omission). Further, the difference explains certain intuitions we have and the similarity justifies and explains the fact that for practical purposes we usually treat preventions and omissions as if they were genuine causation. Finally, the fact that this counterfactual theory of prevention and omission takes causation as primitive suggests that it is consistent with any theory of causation. This allows us to construct two arguments against what I will call genuinism, the view that cases of prevention and ‘causation’ by omission really are cases of genuine causation. In section II I show that genuinism does not account for the so-called intuition of difference. In section III I outline a number of problems that various theories of causation have with preventions and ‘causation’ by omission. These problems are ipso facto problems for genuinism, whereas I show in section V how the counterfactual theory solves those problems. Further, I answer a genuinist argument based on another type of intuition—the genuinist intuition—by showing why we have that intuition and how it should be handled (section VI). (shrink)

A tension exists between those who do—e.g. Meyer (The British Journal for the Philosophy of Science 71:959–985, 2020 ) and Chemero ( 2011 )—and those who do not—e.g. Kaplan and Craver (Philosophy of Science 78:601–627, 2011 ) Piccinini and Craver (Synthese 183:283–311, 2011 )—afford nonmechanistic explanations a role in cognitive science. Here, I argue that one’s perspective on this matter will cohere with one’s interpretation of the tasks of cognitive science; that is, of the actions for which cognitive scientists are (...) specially fitted. To make this point concrete, I consider two tasks of cognitive science—establishing causal claims and unifying—and show how these tasks are interpreted differently by mechanists and nonmechanists respectively. I then argue that, as a result of these different interpretations, we cannot expect to resolve the tension between mechanists and nonmechanists by appeal to cognitive scientific practice. I conclude that we should give up on the debate between mechanists and nonmechanists, and search for progress elsewhere. (shrink)

The basic kinds of physical causality that are foundational for other kinds of causality involve objects and the causal relations between them. These interactions do not involve events. If events were ontologically significant entities for causality in general, then they would play a role in simple mechanical interactions. But arguments about simple collisions looked at from different frames of reference show that events cannot play a role in simple mechanical interactions, and neither can the entirely hypothetical causal relations between events. (...) These arguments show that physics, which should be authoritative when it comes to the metaphysics of causality, gives no reasons to believe that events are causal agents. Force relations and some cases of energy-momentum transfer are examples of causal relations, with forces being paradigmatic in the macroscopic world, though it is conceivable that there are other kinds of causal relation. A relation between two objects is a causal relation if and only if when it is instantiated by the two objects there is a possibility that the objects that are the terms of the relation could change. The basic metaphysics of causality is about objects, causal relations, changes in objects, and a causal primitive. The paper also includes a discussion of the metaphysics of forces and a discussion of the metaphysics of energy and momentum exchanges. (shrink)

The emphasis on the organization of entities and their activities and interactions has been labeled one of the most distinct contributions of mechanistic philosophy. In this paper I discuss the manner in which the organization of entities and their activities and interactions participates in bringing about phenomena. I present a well-known example from molecular biology—the functioning of the genetic switch in phage lambda—and discuss Marco J. Nathan’s notion of causation by concentration. Nathan introduces causation by concentration to account for the (...) irreducible causal role that the concentration ratio between two kinds of proteins possesses in the genetic switch mechanism in phage lambda. I discuss what the irreducibility of this causal role amounts to and provide a mechanistic interpretation of Nathan’s causation by concentration; that is, I explain this irreducible causal role as one organizational feature of this mechanism. The paper concludes that biological mechanisms need a causal pluralist framework [similar to Glennan’s account in (2009), (2010) and (2017) but slightly modified] where organizational features such as the concentration ratio have a causally relevant role, yet all the causally productive relations occur at the level of entities or individuals. (shrink)

Identifying causal relations from correlational data is a fundamental challenge in personality psychology. In most cases, random assignment is not feasible, leaving observational studies as the primary methodological tool. Here, we document several techniques from behavior genetics that attempt to demonstrate causality. Although no one method is conclusive at ruling out all possible confounds, combining techniques can triangulate on causal relations. Behavior genetic tools leverage information gained by sampling pairs of individuals with assumed genetic and environmental relatedness or by measuring (...) genetic variants in unrelated individuals. These designs can find evidence consistent with causality, while simultaneously providing strong controls against common confounds. We conclude by discussing several potential problems that may limit the utility of these techniques when applied to personality. Ultimately, genetically informative designs can aid in drawing causal conclusions from correlational studies. (shrink)

This paper argues that the semantic facts about ‘because’ are best explained via a metaphorical treatment of metaphysical explanation that treats causal explanation as explanation par excellence. Along the way, it defends a commitment to a unified causal sense of ‘because’ and offers a proprietary explanation of grounding skepticism. With the causal metaphor account of metaphysical explanation on the table, an extended discussion of the relationship between conceptual structure and metaphysics ends with a suggestion that the semantic facts about ‘because’ (...) tell against grounding-causation unity. (shrink)

Sydney Shoemaker's "Subset Account" offers a new take on determinable properties and the realization relation as well as a defense of non-reductive physicalism from the problem of mental causation. At the heart of this account are the claims that (1) mental properties are determinable properties and (2) the causal powers that individuate a determinable property are a proper subset of the causal powers that individuate the determinates of that property. The second claim, however, has led to the accusation that the (...) effects caused by the instantiation of a determinable property will also be caused by the instantiation of the determinates of that property—so instead of solving the problem of mental causation, the Subset Account ends up guaranteeing that the effects of mental properties (and all other types of determinable property) will be causally overdetermined! In this paper, I explore this objection. I argue that both sides in this debate have failed to engage the question at the heart of the objection: Given that both a determinable property and its determinates have the power to cause some effect (E), does it follow that both will actually cause E when the relevant conditions obtain? To make genuine progress towards answering this question, we need to take a serious look at the metaphysics of causation. With the debate properly reframed and issues about the metaphysics of causation front and center, I explore the question of whether the Subset Account is doomed to result in problematic causal overdetermination. (shrink)

The idea that causation can be reduced to transmission of an amount of some conserved quantity between events is spelled out and defended against important objections. Transmission is understood as a symmetrical relation of copresence in two distinct events. The actual asymmetry of causality has its origin in the asymmetrical character of certain irreversible physical processes and then spreads through the causal net. This conception is compatible with the possibility of backwards causation and with a causal theory of time. Genidentity, (...) the persistence of concrete objects, can be given an explanation in causal terms. The transmission theory is shown to escape difficulties faced by two important alternative theories of causation: Salmon's (1984) Mark Transmission Theory and Dowe's (1992a) Conserved Quantities Theory. (shrink)

This paper offers a defense of backwards in time causation models in quantum mechanics. Particular attention is given to Cramer's transactional account, which is shown to have the threefold virtue of solving the Bell problem, explaining the complex conjugate aspect of the quantum mechanical formalism, and explaining various quantum mysteries such as Schrödinger's cat. The question is therefore asked, why has this model not received more attention from physicists and philosophers? One objection given by physicists in assessing Cramer's theory was (...) that it is not testable. This paper seeks to answer this concern by utilizing an argument that backwards causation models entail a fork theory of causal direction. From the backwards causation model together with the fork theory one can deduce empirical predictions. Finally, the objection that this strategy is questionable because of its appeal to philosophy is deflected. (shrink)

In the literature on scientific explanation two types of pluralism are very common. The first concerns the distinction between explanations of singular facts and explanations of laws: there is a consensus that they have a different structure. The second concerns the distinction between causal explanations and uni.cation explanations: most people agree that both are useful and that their structure is different. In this article we argue for pluralism within the area of causal explanations: we claim that the structure of a (...) causal explanation depends on the causal structure of the relevant fragment of the world and on the interests of the explainer. (shrink)

This paper discusses several distinct process theories of causality offered in recent years by Phil Dowe and me. It addresses problems concerning the explication of causal process, causal interaction, and causal transmission, whether given in terms of transmission of marks, transmission of invariant or conserved quantities, or mere possession of conserved quantities. Renouncing the mark-transmission and invariant quantity criteria, I accept a conserved quantity theory similar to Dowe's--differing basically with respect to causal transmission. This paper also responds to several fundamental (...) constructive criticisms contained in Christopher Hitchcock's discussion of both the mark-transmission and the conserved quantity theories. (shrink)

It is generally assumed that compatibility with special relativity is guaranteed by the invariance of the fundamental equations of quantum physics under Lorentz transformations and the impossibility of transferring energy or information faster than the speed of light. Despite this, various contradictions persist, which make us suspect the solidity of that compatibility. This paper focuses on collapse theories—although they are not the only way of interpreting quantum theory—in order to examine what seems to be insurmountable difficulties we encounter when trying (...) to construct a space–time picture of such typically quantum processes as state vector reduction or the non-separability of entangled systems. The inescapable nature of such difficulties suggests the need to go further in the search for new formulations that surpass our current conceptions of matter and space–time. (shrink)

In this paper, I have two primary goals. First, I show that the causal-based story approach in A hybrid formal theory of arguments, stories and criminal evidence is ill suited to negative causation. In the literature, the causal-based approach requires that hypothetical stories be causally linked to the explanandum. Many take these links to denote physical or psychological causation, or temporal precedence. However, understanding causality in those terms, as I will show, cannot capture cases of negative causation, which are of (...) interest to the Law. In keeping with this, I also discuss some of the difficulties Hybrid Theory invites by remaining silent on the nature of the causal links. In my second aim, I sketch a way for Hybrid Theory to overcome this problem. By replacing the original, underlying causal structure with contrastive causation in the law, Hybrid Theory can represent reasoning in which the evidence that is appealed to is causally linked via negative causation to the explananda. (shrink)

Ever since Wesley Salmon’s theory, the mechanical approach to causality has found an increasing number of supporters who have developed it in different directions. Mechanical views such as those advanced by Stuart Glennan, Jim Bogen and Peter Machamer, Lindley Darden and Carl Craver have met with broad consensus in recent years. This paper analyses the main features of these mechanical positions and some of the major problems they still face, referring to the latest debate on mechanisms, causal explanation and the (...) relationship between mechanisms and counterfactuals. I shall claim that the mechanical approach can be recognised as having a fundamental explanatory power, whereas the counterfactual approach, recently developed mainly by Jim Woodward and essentially linked to the notion of intervention, has an important heuristic role. Claiming that mechanisms are by no means to be seen as parasitic on counterfactuals or less fundamental than them – as it has been recently suggested –, and that yet counterfactuals can play a part in a conceptual analysis of causation, I shall look for hints in support of the peaceful coexistence of the two. (shrink)

In this paper I offer an 'integrating account' of singular causation, where the term 'integrating' refers to the following program for analysing causation. There are two intuitions about causation, both of which face serious counterexamples when used as the basis for an analysis of causation. The 'process' intuition, which says that causes and effects are linked by concrete processes, runs into trouble with cases of 'misconnections', where an event which serves to prevent another fails to do so on a particular (...) occasion and yet the two events are linked by causal processes. The chance raising intuition, according to which causes raise the chance of their effects, easily accounts for misconnections but faces the problem of chance lowering causes, a problem easily accounted for by the process approach. The integrating program attempts to provide an analysis of singular causation by synthesising the two insights, so as to solve both problems. In this paper I show that extant versions of the integrating program due to Eells, Lewis, and Menzies fail to account for the chance-lowering counterexample. I offer a new diagnosis of the chance lowering case, and use that as a basis for an integrating account of causation which does solve both cases. In doing so, I accept various assumptions of the integrating program, in particular that there are no other problems with these two approaches. As an example of the process account, I focus on the recent CQ theory of Wesley Salmon (1997). (shrink)

I argue that there are some causal relata for which it is indeterminate whether one caused the other. Positing indeterminacy in causation helps us defend contested principles in the logic of causation and makes possible new ways of thinking about the theoretical impact of symmetric causal overdetermination. I close by discussing amendments of current theories of causation that would help explain causal indeterminacy.

Hans Reichenbach’s posthumous book The Direction of Time ends somewhere between Socratic aporia and historical irony. Prompted by Feynman’s diagrammatic formulation of quantum electrodynamics, Reichenbach eventually abandoned the delicate balancing between the macroscopic foundation of the direction of time and microscopic descriptions of time order undertaken throughout the previous chapters in favor of an exclusively macroscopic theory that he had vehemently rejected in the 1920s. I analyze Reichenbach’s reasoning against the backdrop of the history of Feynman diagrams and the current (...) practice of particle physics. Building upon the debates about processes and conserved quantities between Wesley Salmon and Phil Dowe in the 1990s, and the exchange between Reichenbach and the gestalt theorist Kurt Lewin during the 1920s about topology and genidentity, I investigate whether the aporia about local time order could be avoided by following a strategy that Reichenbach adopted elsewhere in the book and develop a notion of functional genidentity that captures the practice of present-day elementary particle physics and the fact that Feynman diagrams have to be understood in the context of a complex mathematical description of scattering processes. (shrink)

J’analyse dans cet article la valeur explicative que peuvent avoir les simulations numériques. On rencontre en effet souvent l’affirmation selon laquelle les simulations permettent de prédire, de reproduire ou d’imiter des phénomènes, mais guère de les expliquer. Les simulations rendraient aussi possible l’étude du comportement d’un système par la force brute du calcul mais n’apporteraient pas une compréhension réelle de ce système et de son comportement. Dans tous les cas, il semble que, à tort ou à raison, les simulations posent, (...) du point de vue de leur valeur explicative, des problèmes spécifiques qu’il convient de démêler et de décrire précisément. J’essaie dans cet article d’analyser systématiquement ces problèmes en utilisant comme guide les théories existantes de l’explication. J’analyse d’abord le rapport des simulations à la vérité (section 2). J’examine ensuite en quoi les simulations satisfont ou non les exigences de déductivité et de nomicité, qui jouent un rôle central dans le modèle de l’explication de Hempel (section 3). J’étudie dans quelle mesure les simulations sont aptes à véhiculer l’information causale pertinente qu’on attend d’une bonne explication (section 4). Je poursuis en analysant en quoi l’abondance informationnelle et la lourdeur computationnelle des simulations peut sembler problématique par rapport au développement de nos connaissances explicatives et de notre compréhension des phénomènes (section 5). J’analyse enfin en quoi les simulations ont un rôle unificateur comme cela est attendu des bonnes explications (section 6). Au final, cette étude permet de comprendre plus précisément pourquoi les simulations, alors même qu’elles semblent pouvoir satisfaire les conditions que doivent remplir les bonnes explications, semblent spécifiquement problématiques au regard de l’activité explicative. Je suggère que les raisons sont notamment à chercher dans l’épistémologie de l’activité explicative, dans les attentes méthodologiques envers les bonnes explications et dans l’usage spécifique qu’on fait des simulations pour l’étude des cas difficiles – en plus du fait que les simulations constituent une activité qui n’est plus à taille humaine. (shrink)

Causal pluralism is currently a hot topic in philosophy. However, the consequences of this view on causation for scientific knowledge and scientific methodology are heavily underexposed in the present debate. My aim in this paper is to argue that an epistemological-methodological point of view should be valued as a line of approach on its own and to demonstrate how epistemological- methodological causal pluralism differs in its scope from conceptual and metaphysical causal pluralism. Further, I defend epistemological-methodological causal pluralism and try (...) to illustrate that scientific practice needs diverse causal concepts in diverse domains, and even diverse causal concepts within singular domains. (shrink)

The physical and/or intrinsic connection approach to causation has become prominent in the recent literature, with Salmon, Dowe, Menzies, and Armstrong among its leading proponents. I show that there is a type of causation, causation by disconnection, with no physical or intrinsic connection between cause and effect. Only Hume-style conditions approaches and hybrid conditions-connections approaches appear to be able to handle causation by disconnection. Some Hume-style, extrinsic, absence-relating, necessary and/or sufficient condition component of the causal relation proves to be needed.

In this paper I examine Salmon's response to two counterexamples to his conserved quantity (CQ) theory of causation. The first counterexample that I examine involves a time‐wise gerrymandered world line of a series of patches of wall that is absorbing energy as a result of being illuminated in an astrodome. Salmon says that since the gerrymandered world line does not fulfill his “no‐interaction requirement,” his CQ theory does not suffer from the counterexample. But I will argue that his response fails (...) both at a theoretical level and at a practical level. In so doing I point out a problem for CQ theorists' definition of a causal interaction. The second counterexample is concerned with a time‐wise gerrymandered world line of a series of patches that are in shadow, in Hitchcock's well‐known example. Salmon's response is based on a principle that Salmon thinks is derivable from the concept of a conserved quantity. However, I argue that the principle has a counterexample. (shrink)

People’s causal judgments are susceptible to the action effect, whereby they judge actions to be more causal than inactions. We offer a new explanation for this effect, the counterfactual explanation: people judge actions to be more causal than inactions because they are more inclined to consider the counterfactual alternatives to actions than to consider counterfactual alternatives to inactions. Experiment 1a conceptually replicates the original action effect for causal judgments. Experiment 1b confirms a novel prediction of the new explanation, the reverse (...) action effect, in which people judge inactions to be more causal than actions in overdetermination cases. Experiment 2 directly compares the two effects in joint-causation and overdetermination scenarios and conceptually replicates them with new scenarios. Taken together, these studies provide support for the new counterfactual explanation for the action effect in causal judgment. (shrink)

Recently some philosophers have emphasized a potentially irreconcilable conceptual antagonism between the statistical characterization of natural selection and the standard scientific discussion of natural selection in terms of forces and causes. Other philosophers have developed an account of the causal character of selectionist statements represented in terms of counterfactuals. I examine the compatibility between such statisticalism and counterfactually based causal accounts of natural selection by distinguishing two distinct statisticalist claims: firstly the suggested impossibility for natural selection to be a cause (...) acting upon populations and secondly the conceptualization that all evolutionary causes occur at the level of interactions between individual organisms. I argue that deriving the latter from the former involves supplementary assumptions concerning precisely what causation is. I critically examine two of these assumptions purportedly preventing natural selection being regarded as a cause: the locality claim and the modularity claim. I conclude that justifying the strongest version of statisticalism—i.e. evolutionary causation only occurs at the level of individual interactions between organisms—would require further metaphysical arguments that are likely to be deemed highly problematic. Additionally, I argue that such a metaphysical position would be considered incongruous with both our scientific and ordinary use of the concepts of causality and explanation as employed within our everyday epistemological framework. (shrink)

Selection explanations explain some non-accidental generalizations in virtue of a selection process. Such explanations are not particulaizable - they do not transfer as explanations of the instances of such generalizations. This is unlike many explanations in the physical sciences, where the explanation of the general fact also provides an explanation of its instances (i.e. standard D-N explanations). Are selection explanations (e.g. in biology) therefore a different kind of explanation? I argue that to understand this issue, we need to see that (...) a standard D-N explanation of some non-accidental generalization (al Fs are Gs) may also ipso facto explain its contrapositive (all non-Gs are non-Fs), but the explanation is particularizable with respect to the former but not to the latter. This can be seen by noting that the Raven Paradox counterexample to the H-D model of confirmation also generates a counterexample to the D-N model of explanation (all ravens are black does not explain why the non-black shoe is a non-raven). In such cases it is natural to take the generalization with the positive predicates to have a particularizable explanation. However, this need not be the case, and in selection explanations it is the generalization with the positive predicates whose explanation is no particularizable. Thus there is no need to suppose that selection explanations are fundamentally different. (shrink)

Reasoning about causation in fact is an essential element of attributing legal responsibility. Therefore, the automation of the attribution of legal responsibility requires a modelling effort aimed at the following: a thorough understanding of the relation between the legal concepts of responsibility and of causation in fact; a thorough understanding of the relation between causation in fact and the common sense concept of causation; and, finally, the specification of an ontology of the concepts that are minimally required for (automatic) common (...) sense reasoning about causation. This article offers a worked-out example of the indicated analysis. Such example consists of: a definition of the legal concept of responsibility (in terms of liability and accountability); a definition of the legal concept of causation in fact (in terms of the initiation of physical processes by an agent and of the provision of reasons and/or opportunities to other agents); CausatiOnt, an AI-like ontology of the common sense (causal) concepts that are minimally needed for reasoning about the legal concept of causation in fact (in particular, the concepts of category, dimension, object, agent, process, event and act). (shrink)

There is a widespread belief that the so-called process theories of causation developed by Wesley Salmon and Phil Dowe have given us an original account of what causation really is. In this paper, I show that this is a misconception. The notion of “causal process” does not offer us a new ontological account of causation. I make this argument by explicating the implicit ontological commitments in Salmon and Dowe’s theories. From this, it is clear that Salmon’s Mark Transmission Theory collapses (...) to a counterfactual theory of causation, while the Conserved Quantity Theory collapses to David Fair’s phsyicalist reduction of causation. (shrink)

Many theories of actual causation implicitly endorse the claim that if c is an actual cause of e, then either c causes e directly or every intermediary by which c indirectly causes e is itself both an actual cause of e and also an actual effect of c. We think this compositionality constraint is plausible. However, as we show, it is not always satisfied by the causal attributions ordinary people make. We conclude by considering what philosophers working on causation should (...) do when the deliverances of their theories diverge from what ordinary people say. (shrink)