A probabilistic and counterfactual theory of causality is developed within the framework of branching time. The theory combines ideas developed by James Fetzer, Donald Nute, Patrick Suppes, Ming Xu, John Pollock, David Lewis and Mellor among others.

It is widely agreed that Hume’s description of human inductive reasoning is inadequate. But many philosophers think that this inadequacy in no way affects the force of Hume’s argument for the unjustifiability of inductive reasoning. I argue that this constellation of opinions contains a serious tension, given that Hume was not merely pointing out that induction is fallible. I then explore a recent diagnosis of where Hume’s sceptical argument goes wrong, due to Elliott Sober. Sober argues that Hume committed a (...) quantifier-shift fallacy, i.e. inferred a statement of ∃∀ form from one of V∀∃ form. The implications of this diagnosis for the traditional problem of induction are briefly examined. The author suggests that, were his philosophy received, we must alter from the foundation the greatest part of the sciences. Hume 1740. (shrink)

Verificationism has often seemed attractive to philosophers because of its apparent abilityto deliver us from scepticism. However, I argue that purely epistemological considerationsprovide insufficient reason for embracing verificationism over realism. I distinguish twotypes of sceptical problem: those that stem from underdetermination by the actual data,and those that stem from underdetermination by all possible data. Verificationismevades problems of the second sort, but is powerless in the face of problems of the firstsort. But problems of the first sort are equally pressing. Furthermore, (...) there is some reasonto think that the two types of problem have a common origin. Thus the desire to avoidscepticism provides insufficient reason for adopting verificationism. (shrink)

Many philosophers agree that Hume was not simply objecting to inductive inferences on the grounds of their logical invalidity and that his description of our inductive behaviour was inadequate, but none the less regard his argument against induction as irrefutable. I argue that this constellation of opinions contains a serious tension. In the light of the tension, I re-examine Hume’s actual sceptical argument and show that the argument as it stands is valid but unsound. I argue that it can only (...) be converted into a sound one if our inductive behaviour can be characterized as a process of rule-governed ampliation. Drawing on some Bayesian ideas, I argue that our inductive behaviour probably cannot be characterized in that way, so our immunity from Hume is secure. Finally, I compare my response to Hume’s argument with some other well known responses. (shrink)

This article offers a critical review of various ontological-relativist arguments, mostly deriving from the work of W. V. Quine and Thomas K hn. I maintain that these arguments are (1) internally contradictory, (2) incapable of accounting for our knowledge of the growth of scientific knowledge, and (3) shown up as fallacious from the standpoint of a causal-realist approach to issues of truth, meaning, and interpretation. Moreover, they have often been viewed as lending support to such programmes as the 'strong' sociology (...) of knowledge and the turn towards wholesale cultural-relativist doctrines, whether of the Wittgensteinian ('language-games') or Heideggerian (depth-hermeneutic) varieties. Thus Richard Rorty recommends that we should henceforth drop all that old-fashioned talk of 'truth', 'knowledge', or 'reality', since whatever warrants those descriptions from time to time is just the product of some currently favoured language-game or range of elective metaphors. Such ideas have little in common with Quine's general outlook of robust physicalism, nor again - though the case is less clear - with Kuhn's reconsidered approach to these issues as set forth in his 1969 Postscript to The Structure of Scientific Revolutions . However, both thinkers have left themselves open to misconstrual by adopting a sceptical-relativist fallback position in response to the well-known problems with logical empiricism. This essay therefore reviews those problems with reference to alternative, more adequate accounts of what is involved in the process of scientific discovery and theory-change. (shrink)

This paper examines the relationship between physical theories of causation and theories of difference-making. It is plausible to think that such theories are compatible with one another as they are aimed at different targets: the former, an empirical account of actual causal relations; the latter, an account that will capture the truth of most of our ordinary causal claims. The question then becomes: what is the relationship between physical causation and difference-making? Is one kind of causal fact more fundamental than (...) the other? This paper defends causal foundationalism: the view that facts about difference-making are dependent on the obtaining of facts about physical causation. However, the paper's main goal is to clarify the structure of the debate. At the end of the paper, it is shown how settling the issue about the relationship between physical theories of causation and theories of difference-making has more than mere intrinsic interest in unifying the very different pursuits that have been undertaken in the philosophy of causation. It can help to break a stalemate that has arisen in the current debate about mental causation. (shrink)

This paper focuses on an influential line of response to the exclusion problem for nonreductive physicalism, one defended with the most subtlety by Karen Bennett. According to this line of thought, a successful nonreductive response to the exclusion problem, a response that allows one to maintain each of the core components of nonreductive physicalism, may consist in showing that the manner in which the effects of mental causes also have distinct and sufficient physical causes is disanalogous to other types of (...) cases in which an effect may have distinct sufficient causes. After laying out the formulation of the problem that Bennett endorses, along with her response to the problem, I offer an initial critique of this response insofar as it is couched in terms of her preferred formulation. I then present a general critique of disanalogy-style responses to the exclusion problem. I argue that extant implementations of this strategy are at best underdeveloped, and suggest that lack of clarity in the use of “overdetermination” may function to mask the shortcomings of this strategy. While others have questioned the details of such responses, the worries that I raise concern the very logic of the exclusion problem and how such responses fit into this logic. (shrink)

Causal contextualism holds that sentences of the form ‘c causes e’ have context-sensitive truth-conditions. We consider four arguments invoked by Jonathan Schaffer in favor of this view. First, he argues that his brand of contextualism helps solve puzzles about transitivity. Second, he contends that how one describes the relata of the causal relation sometimes affects the truth of one’s claim. Third, Schaffer invokes the phenomenon of contrastive focus to conclude that causal statements implicitly designate salient alternatives to the cause and (...) effect. Fourth, he claims that the appropriateness of a causal statement depends on what is contextually taken for granted or made salient. We show that causal invariantism can explain these linguistic data at least as well as contextualism. We then argue that pace Schaffer, some causal sentences are always correct and can never be plausibly denied, regardless of the context. (shrink)

Causal analysis in the social sciences takes advantage of a variety of methods and of a multi-fold source of information and evidence. This pluralistic methodology and source of information raises the question of whether we should accordingly have a pluralistic metaphysics and epistemology. This paper focuses on epistemology and argues that a pluralistic methodology and evidence don’t entail a pluralistic epistemology. It will be shown that causal models employ a single rationale of testing, based on the notion of variation. Further, (...) I shall argue that this monistic epistemology is also involved in alternative philosophical theories of causation. (shrink)

(No abstract is available for this citation).

Research on human causal induction has shown that people have general prior assumptions about causal strength and about how causes interact with the background. We propose that these prior assumptions about the parameters of causal systems do not only manifest themselves in estimations of causal strength or the selection of causes but also when deciding between alternative causal structures. In three experiments, we requested subjects to choose which of two observable variables was the cause and which the effect. We found (...) strong evidence that learners have interindividually variable but intraindividually stable priors about causal parameters that express a preference for causal determinism. These priors predict which structure subjects preferentially select. The priors can be manipulated experimentally and appear to be domain-general. Heuristic strategies of structure induction are suggested that can be viewed as simplified implementations of the priors. (shrink)

This paper argues for a conditional claim concerning a famous argument—developed by Church in elucidation of some remarks by Frege to the effect that the bedeutung of a sentence is the sentence’s truth-value—the Frege–Gödel–Church argument, or FGC for short. The point we make is this :if, and just to the extent that, Arthur Smullyan’s argument against Quine's use of FGC is sound, then essentially the same rejoinder disposes also of Davidson's use of FGC against ‘correspondence’ theories of truth. We thus (...) dispute a contention by Professor Davidson that it is coherent to accept that Smullyan’s rejoinder takes away the force of Quine’s version of FGC, while still consistently using FGC to establish that if true sentences (or utterances) correspond to anything, they all correspond to the same thing. We show that the differences between the cases discussed by Smullyan and Davidson’s version of FGC on which Davidson relies for his contention are irrelevant to the point under dispute. (shrink)

One of the most recent controversies to arise in the field of bioethics concerns the ethics for the Groningen Protocol: the guidelines proposed by the Groningen Academic Hospital in The Netherlands, which would permit doctors to actively euthanise terminally ill infants who are suffering. The Groningen Protocol has been met with an intense amount of criticism, some even calling it a relapse into a Hitleresque style of eugenics, where people with disabilities are killed solely because of their handicaps. The purpose (...) of this paper is threefold. First, the paper will attempt to disabuse readers of this erroneous understanding of the Groningen Protocol by showing how such a policy does not aim at making quality-of-life judgements, given that it restricts euthanasia to suffering and terminally ill infants. Second, the paper illustrates that what the Groningen Protocol proposes to do is both ethical and also the most humane alternative for these suffering and dying infants. Lastly, responses are given to some of the worries expressed by ethicists on the practice of any type of non-voluntary active euthanasia. (shrink)

Modal arguments like the Knowledge Argument, the Conceivability Argument and the Inverted Spectrum Argument could be used to argue for experiential primitivism; the view that experiential truths aren’t entailed from nonexperiential truths. A way to resist these arguments is to follow Stoljar (Ignorance and imagination. Oxford University Press, Oxford, 2006) and plead ignorance of a type of experience-relevant nonexperiential truth. If we are ignorant of such a truth, we can’t imagine or conceive of the various sorts of scenarios that are (...) required to make these arguments sound. While I am sympathetic to this response, in this article I will argue that we have good reason to believe that this particular ignorance hypothesis is false. (shrink)

This paper offers an analysis of the logical form of plural action sentences that shows that collective actions so ascribed are a matter of all members of a group contributing to bringing some event about. It then uses this as the basis for a reductive account of the content of we-intentions according to which what distinguishes we-intentions from I-intentions is that we-intentions are directed about bringing it about that members of a group act in accordance with a shared plan.

Two key research issues in the field of causal learning are how people acquire causal knowledge when observing data that are presented sequentially, and the level of abstraction at which learning takes place. Does sequential causal learning solely involve the acquisition of specific cause-effect links, or do learners also acquire knowledge about abstract causal constraints? Recent empirical studies have revealed that experience with one set of causal cues can dramatically alter subsequent learning and performance with entirely different cues, suggesting that (...) learning involves abstract transfer, and such transfer effects involve sequential presentation of distinct sets of causal cues. It has been demonstrated that pre-training can modulate classic causal learning phenomena such as forward and backward blocking. To account for these effects, we propose a Bayesian theory of sequential causal learning. The theory assumes that humans are able to consider and use several alternative causal generative models, each instantiating a different causal integration rule. Model selection is used to decide which integration rule to use in a given learning environment in order to infer causal knowledge from sequential data. Detailed computer simulations demonstrate that humans rely on the abstract characteristics of outcome variables to select a causal integration rule, which in turn alters causal learning in a variety of blocking and overshadowing paradigms. When the nature of the outcome variable is ambiguous, humans select the model that yields the best fit with the recent environment, and then apply it to subsequent learning tasks. Based on sequential patterns of cue-outcome co-occurrence, the theory can account for a range of phenomena in sequential causal learning, including various blocking effects, primacy effects in some experimental conditions, and apparently abstract transfer of causal knowledge. (shrink)

In all three versions of the “Jian’ai” 兼愛 Chapter in the Mozi 墨子, variations of a central argument may be found. This argument proceeds by advancing a diagnosis for what causes the various evils that beset the world, and it is on this basis that the Mohists propose jian’ai as the solution. The study examines this main argument in some detail, with the aim of improving both our understanding of the Mohist ethical doctrine and also our appreciation of their argumentative (...) practices. The study shows that distinct ethical injunctions of varying degrees of stringency can be derived from the argument, though they all embody an underlying notion of impartiality. This impartiality—while in many ways recognizably attractive to us—puts Mohist jian’ai in tension with certain notions regarding the ethical significance of special relations. In addition, the paper argues that the Mohists main argument for jian’ai contains a critical flaw. (shrink)

With regard to intrinsically morally relevant factors it is natural to suppose that if a variation in a given factor makes a moral difference anywhere, then it makes the same moral difference everywhere (henceforth: the constancy assumption). Jonathan Dancy (and other moral particularists) reject the constancy assumption. Partly on the basis thereof, they infer that ethical decisions should be made “case by case, without the comforting support of moral principles”. In this article, I challenge Dancy's defence and use of a (...) denial of the constancy assumption on three points. First, Dancy's appeal to moral intuitions do not justify denying a significant version of the constancy assumption. Second, if we reject the constancy assumption, surprisingly, it may in one crucial respect be very hard to defend moral particularism as commonly articulated. Third, rejecting the constancy assumption does not motivate seeing moral reasoning as essentially a case‐by‐case matter. (shrink)

What makes killing morally wrong? And what makes killing morally worse than letting die? Standard answers to these two questions presuppose that killing someone involves shortening that person's life. Yet, as I argue in the first two sections of this article, this presupposition is false: Life-prolonging killings are conceivable. In the last two sections of the article, I explore the significance of the conceivability of such killings for various discussions of the two questions just mentioned. In particular, I show why (...) the conceivability of life-prolonging killings renders Frances M. Kamm's attempt to provide an answer to the second question problematic. (shrink)

Science depends on judgments of the bearing of evidence on theory. Scientists must judge whether an observation or the result of an experiment supports, disconfirms, or is simply irrelevant to a given hypothesis. Similarly, scientists may judge that, given all the available evidence, a hypothesis ought to be accepted as correct or nearly so, rejected as false, or neither. Occasionally, these evidential judgments can be made on deductive grounds. If an experimental result strictly contradicts a hypothesis, then the truth of (...) the data deductively entails the falsity of the hypothesis. In the great majority of cases, however, the connection between evidence and hypothesis is non-demonstrative, or inductive. In particular, this is so whenever a general hypothesis is inferred to be correct on the basis of the available data, since the truth of the data will not deductively entail the truth of the hypothesis. It always remains possible that the hypothesis is false even though the data are correct. (shrink)

This paper argues that emergent conscious properties can't bestow emergent causal powers. It supports this conclusion by way of a dilemma. Necessarily, an emergent conscious property brings about its effects actively or other than actively. If actively, then, the paper argues, the emergent conscious property can't have causal powers at all. And if other than actively, then, the paper argues, the emergentist finds himself committed to incompatible accounts of causation.

Reasoning about causation in fact is an essential element of attributing legal responsibility. Therefore, the automation of the attribution of legal responsibility requires a modelling effort aimed at the following: a thorough understanding of the relation between the legal concepts of responsibility and of causation in fact; a thorough understanding of the relation between causation in fact and the common sense concept of causation; and, finally, the specification of an ontology of the concepts that are minimally required for (automatic) common (...) sense reasoning about causation. This article offers a worked-out example of the indicated analysis. Such example consists of: a definition of the legal concept of responsibility (in terms of liability and accountability); a definition of the legal concept of causation in fact (in terms of the initiation of physical processes by an agent and of the provision of reasons and/or opportunities to other agents); CausatiOnt, an AI-like ontology of the common sense (causal) concepts that are minimally needed for reasoning about the legal concept of causation in fact (in particular, the concepts of category, dimension, object, agent, process, event and act). (shrink)

How do people attribute responsibility in situations where the contributions of multiple agents combine to produce a joint outcome? The prevalence of over-determination in such cases makes this a difficult problem for counterfactual theories of causal responsibility. In this article, we explore a general framework for assigning responsibility in multiple agent contexts. We draw on the structural model account of actual causation (e.g., Halpern & Pearl, 2005) and its extension to responsibility judgments (Chockler & Halpern, 2004). We review the main (...) theoretical and empirical issues that arise from this literature and propose a novel model of intuitive judgments of responsibility. This model is a function of both pivotality (whether an agent made a difference to the outcome) and criticality (how important the agent is perceived to be for the outcome, before any actions are taken). The model explains empirical results from previous studies and is supported by a new experiment that manipulates both pivotality and criticality. We also discuss possible extensions of this model to deal with a broader range of causal situations. Overall, our approach emphasizes the close interrelations between causality, counterfactuals, and responsibility attributions. (shrink)

Backtracking influence is influence that zigzags in time. For example, backtracking influence exists when an event E_1 makes an event E_2 more likely by way of a nomic connection that goes from E_1 back in time to an event C and then forward in time to E_2. I contend that in our local region of spacetime, at least, backtracking influence is redundant in the sense that any existing backtracking influence exerted by E_1 on E_2 is equivalent to E_1's temporally direct (...) influence on E_2. I prove the redundancy of backtracking influence using several plausible physical principles without assuming any fundamental temporal or causal asymmetry. This explanation can play a prominent role in an account of why causation appears to be objectively asymmetric regardless of whether the fundamental laws are symmetric. (shrink)

Probabilistic phenomena are often perceived as being problematic targets for contrastive explanation. It is usually thought that the possibility of contrastive explanation hinges on whether or not the probabilistic behaviour is irreducibly indeterministic, and that the possible remaining contrastive explananda are token event probabilities or complete probability distributions over such token outcomes. This paper uses the invariance-under-interventions account of contrastive explanation to argue against both ideas. First, the problem of contrastive explanation also arises in cases in which the probabilistic behaviour (...) of the explanandum is due to unobserved causal heterogeneity. Second, it turns out that, in contrast to the case of pure indeterminism, the plausible contrastive explananda under causal heterogeneity are not token event probabilities, but population-level statistical facts. (shrink)

This paper provides an inferentialist account of model-based understanding by combining a counterfactual account of explanation and an inferentialist account of representation with a view of modeling as extended cognition. This account makes it understandable how the manipulation of surrogate systems like models can provide genuinely new empirical understanding about the world. Similarly, the account provides an answer to the question how models, that always incorporate assumptions that are literally untrue of the model target, can still provide factive explanations. Finally, (...) the paper shows how the contrastive counterfactual theory of explanation can provide tools for assessing the explanatory power of models. (shrink)

This article delineates some of the main issues that are debated by philosophers of law. It explores the connections between legal philosophy and other areas of philosophy, while also seeking to specify the distinctiveness of many of the concerns that have preoccupied philosophers of law. It illustrates its abstract points with examples focused on the separability of law and morality, the nature of the rule of law, the nature of rights, justifications for the imposition of punishment, and the identification of (...) basic legal entitlements. (shrink)

This paper surveys three controversial new directions in research about the nature of science and briefly summarizes both the intellectual and sociological impact of this work. A bibliographic introduction to the major literature is provided and some fruitful directions for future research are proposed. Philosophers of science are also exhorted to perform 'community service' by correcting misunderstandings of the methods of science fostered by these new approaches.

Much of the modern philosophy of causation has been governed by two ideas: (i) causes make their effects inevitable; (ii) a cause is something that makes a difference to whether its effect occurs. I focus on explaining the origin of idea (ii) and its connection to (i). On my view, the frequent attempts to turn (ii) into an analysis of causation are wrongheaded. Patterns of difference-making aren't what makes causal claims true. They merely provide a useful test for causal claims. (...) Moreover, what justifies us in using them as a test is idea (i). That's how (i) and (ii) are connected. (shrink)

Much of our ordinary thought and talk about responsibility exhibits what I call the ‘pie fallacy’—the fallacy of thinking that there is a fixed amount of responsibility for every outcome, to be distributed among all those, if any, who are responsible for it. The pie fallacy is a fallacy, I argue, because how responsible an agent is for some outcome is fully grounded in facts about the agent, the outcome and the relationships between them; it does not depend, in particular, (...) on how responsible anyone else is for that same outcome. In this paper, I explore how the pie fallacy can arise by considering several different kinds of case in which two or more agents are responsible for the same outcome. I’ll end with some brief remarks on the potential consequences of my arguments for how to think about responsibility in war. (shrink)

Regularity theories of causation are guided by the idea that causes are collectively sufficient for their effects. Following Mackie [1974], that idea is typically refined to distinguish collections that include redundant members from those that do not. Causes must be collectively sufficient for their effects without redundancy. While Mackie was surely right that the regularity theory must distinguish collections that are in some sense minimally sufficient for an effect from those that include unnecessary hangers-on, I believe that redundancy is the (...) wrong mark of that distinction. I propose a way to develop the regularity theory without it. Instead of distinguishing minimal from mere sufficiency in terms of redundancy, we should look to the influence that events have in the world. Causes, so construed, must have just enough collective influence to ensure that an effect occurs. I argue that such an account provides a uniform solution to the problem of epiphenomena and to a pair of related problems that aris.. (shrink)

Hitchcock (2001a) argues that the distinction between singular and general causation conflates the two distinctions ‘actual causation vs. causal tendencies’ and ‘wide vs. narrow causation’. Based on a recent regularity account of causation I will show that Hitchcock’s introduction of the two distinctions is an unnecessary multiplication of causal concepts.

Hume's "other words" which follow his first definition of causality in the "enquiry" are standardly read as giving us a counterfactual conditional. I argue that a more accurate reading reveals them to constitute a factual conditional, One reflecting a temporal restriction implicit in the first definition. The other words, So understood, Tell us merely that a component of the relation defined in the first definition is symmetrical.

This paper argues against Papineau's claim that causal relations can be reduced to correlations and defends Cartwright's thesis that they can be nevertheless boot-strapped from them, given sufficiently rich causal background knowledge.

This paper advances the thesis of methodological mechanism, the claim that to be committed to mechanism is to adopt a certain methodological postulate, i.e. to look for causal pathways for the phenomena of interest. We argue that methodological mechanism incorporates a minimal account of understanding mechanisms, according to which a mechanism just is a causal pathway described in the language of theory. In order to argue for this position we discuss a central example of a biological mechanism, the mechanism of (...) cell death, known as apoptosis. We argue that this example shows that our philosophically deflationary account is sufficient in order to have an illuminating account of mechanisms as the concept is used in biology. (shrink)

Rani Lill Anjum and Stephen Mumford have recently defended a new kind of modality, which they call ‘dispositional modality’. The key reason to adopt dispositional modality, according to them, is that causes never necessitate their effects. Anjum and Mumford’s chief argument against causal necessitation makes use of what they call the ‘antecedent-strengthening test’ : C causally necessitates E iff C & φ causes E, for any possible φ. This test, they claim, fails in all cases of alleged causal necessitation. In (...) this paper we argue that the AS-test is not the mark of causal necessitation. First, we show that if the AS-test is taken as the mark of causal necessitation it leads to either an absurdity or to circularity. Second, we argue that, given the Mill/Mackie framework of causes as INUS conditions, apparent counterexamples to causal necessitation fail. (shrink)

Current research in molecular epidemiology uses biomarkers to model the different disease phases from environmental exposure, to early clinical changes, to development of disease. The hope is to get a better understanding of the causal impact of a number of pollutants and chemicals on several diseases, including cancer and allergies. In a recent paper Russo and Williamson address the question of what evidential elements enter the conceptualisation and modelling stages of this type of biomarkers research. Recent research in causality has (...) examined Ned Hall’s distinction between two concepts of causality: production and dependence. In another recent paper, Illari examined the relatively under-explored production approach to causality, arguing that at least one job of an account of causal production is to illuminate our inferential practices concerning causal linking. Illari argued that an informational account solves existing problems with traditional accounts. This paper follows up this previous work by investigating the nature of the causal links established in biomarkers research. We argue that traditional accounts of productive causality are unable to provide a sensible account of the nature of the causal link in biomarkers research, while an informational account is very promising. (shrink)

This paper argues against dismissing the Principle of Alternative Possibilities merely on the ground of so-called Frankfurt-style cases. Its main claims are that the interpretation of such cases depends on which substantive theory of responsibility one endorses and that Frankfurt-style cases all involve some form of causal overdetermination which can be interpreted either as being compatible with the potentially manipulated agent’s ability to act otherwise or as a responsibility undermining constraint. The paper also argues that the possibility of such scenarios (...) can support the truth of classical compatibilism as much as the truth of semicompatibilism. (shrink)

Debates about the ethics and effects of placebos and whether ‘placebos’ in clinical trials of complex treatments such as acupuncture are adequate rage. Yet there is currently no widely accepted definition of the ‘placebo’. A definition of the placebo is likely to inform these controversies. Grünbaum’s characterization of placebos and placebo effects has been touted by some authors as the best attempt thus far, but has not won widespread acceptance largely because Grünbaum failed to specify what he means by a (...) therapeutic theory and because he does not stipulate a special role for expectation effects. Grünbaum claims that placebos are treatments whose ‘characteristic features’ do not have therapeutic effects on the target disorder. I show that with four modifications, Grünbaum’s definition provides a defensible account of placebos for the purpose of constructing placebo controls within clinical trials. The modifications I introduce are: adding a special role for expectations, insisting that placebo controls control for all and only the effects of the incidental treatment features, relativizing the definition of placebos to patients, and introducing harmful interventions and nocebos to the definitional scheme. I also provide guidance for classifying treatment features as characteristic or incidental. (shrink)

In this paper I analyse who should be able to control the use of human embryonic stem cell lines. I distinguish between different kinds of control and analyse a set of arguments that purport to show that the donors of gametes and embryos should not be able to control the use of stem cell lines derived from their embryos. I show these arguments to be either deficient or of so general a scope that they apply not only to donors but (...) also to those who derived the stem cell lines. Since we normally think that stem cell derivers have the right to control the use of the cell lines they derived, we have no justification for denying donors similar rights of control. In the final section I briefly discuss why informed consent is not an appropriate vehicle for transferring or alienating control rights. (shrink)

Abstract The thesis that teleological explanations are best understood as causal explanations is defended (contra Valentine). I shift the focus of debate from behavior simpliciter to allegedly rational behavior. Teleological explanation, in the case of rational agents, involves reason?giving; and the reasons agents give for acting must be causative of that action if those agents are to be rational in practice. I argue initially that to abandon the claim that reasons are causes of action is to abandon that which renders (...) many generalisations of systematic human behavior intelligible. I then adduce and defend against two major objections to the secondary thesis that reasons can be causes of action: (1) Experiments in attribution research suggest that agents are characteristically mistaken when reporting on the causes of their behavior; (2) Causal explanation must refer to some law, and reasons cannot enter into such laws as causes of action. In responding to these objections, I note that strict predictability is probably not a reliable indicator of genuine causal explanation; some explanations will afford predictions, others will not. (shrink)