Non-reductive interventionist theories of causation and methodologies of causal reasoning embedded in that theoretical framework have become increasingly popular in recent years. This paper argues that one variant of an interventionist account of causation, viz. the one presented, for example, in Woodward (2003), is unsuited as a theoretical fundament of interventionist methodologies of causal reasoning, because it renders corresponding methodologies incapable of uncovering a causal structure in a finite number of steps. This finding runs counter to Woodward's own assessment and (...) to other recent studies which presume that Woodward's version of interventionism is effectively applicable to uncover causal structures, e.g. Campbell (2007) or Shapiro and Sober (2007). (shrink)

This comment discusses Kaidesoja and raises the issue whether his analysis justifies stronger conclusions than he presents in the book. My comments focus on four issues. First, I argue that his naturalistic reconstruction of critical realist transcendental arguments shows that transcendental arguments should be treated as a rare curiosity rather than a general argumentative strategy. Second, I suggest that Kaidesoja’s analysis does not really justify his optimism about the usefulness of causal powers ontology in the social sciences. Third, I raise (...) some doubts about the heuristic value of Mario Bunge’s social ontology that Kaidesoja presents as a replacement for critical realist ontology. Finally, I propose an alternative way to analyze failures of aggregativity that might better serve Kaidesoja’s purposes than the Wimsattian scheme he employs in the book. (shrink)

A certain type of counterfactual is thought to be intimately related to causation, control, and explanation. The time asymmetry of these phenomena therefore plausibly arises from a time asymmetry of counterfactual dependence. But why is counterfactual dependence time asymmetric? The most influential account of the time asymmetry of counterfactual dependence is David Albert’s account, which posits a new, time-asymmetric fundamental physical law, the so-called “past hypothesis.” Albert argues that the time asymmetry of counterfactual dependence arises from holding fixed the past (...) hypothesis when evaluating counterfactuals. In this paper, I argue that Albert’s account misconstrues the time asymmetry of counterfactual dependence. (shrink)

Evidentialism says that a subject S’s justification is entirely determined by S’s evidence. The plausibility of evidentialism depends on what kind of entities constitute a subject S’s evidence and what one takes the support relation to consist in. Conee and Feldman’s mainstream evidentialism incorporates a psychologist answer to and an explanationist answer to. ME naturally accommodates perceptual justification. However, it does not accommodate intuitive cases of inferential justification. In the second part of the paper, I consider and reject a reply (...) based on a refined explanationist theory of the support relation proposed by K McCain. (shrink)

In the recent philosophy of explanation, a growing attention to and discussion of non-causal explanations has emerged, as there seem to be compelling examples of non-causal explanations in the sciences, in pure mathematics, and in metaphysics. I defend the claim that the counterfactual theory of explanation captures the explanatory character of both non-causal scientific and metaphysical explanations. According to the CTE, scientific and metaphysical explanations are explanatory by virtue of revealing counterfactual dependencies between the explanandum and the explanans. I support (...) this claim by illustrating that CTE is applicable to Euler’s explanation and Loewer’s explanation. (shrink)

Philosophical works on actual causation make wide use of thought experiments. The principal aim of this paper is to show how thought experiments are used in the contemporary debate over actual causation and to discuss their role in relation to formal approaches in terms of causal models. I claim that a recourse to thought experiments is not something old fashioned or superseded by abstract models, but it is useful to interpret abstract models themselves and to use our intuitions to judge (...) the results of the model. Recent research on actual causation has stressed the importance of integrating formal models with some notion of normality; I suggest that thought experiments can be useful in eliciting intuitions where normality is not intended in a statistical sense. The first expository part gives a short presentation of the notion of actual causation, summarising some typical problems of counterfactual approaches and how they are treated in causal and structural models. The second part focuses on the problems of model isomorphism and criticises some radical ideas opposing the role of thought experiments, claiming that they may also be of use in evaluating formal models. (shrink)

Diagnoses in medicine are often taken to serve as explanations of patients’ symptoms and signs. This article examines how they do so. I begin by arguing that although some instances of diagnostic explanation can be formulated as covering law arguments, they are explanatory neither in virtue of their argumentative structures nor in virtue of general regularities between diagnoses and clinical presentations. I then consider the theory that medical diagnoses explain symptoms and signs by identifying their actual causes in particular cases. (...) While I take this to be largely correct, I argue that for a diagnosis to function as a satisfactory causal explanation of a patient’s symptoms and signs, it also needs to be supplemented by understanding the mechanisms by which the identified cause produces the symptoms and signs. This mechanistic understanding comes not from the diagnosis itself, but rather from the theoretical framework within which the physician operates. (shrink)

Philosophical debates about free will have focused on determinism—a potential ‘threat from behind’ because determinism entails that there are conditions in the distant past that, in accord with the laws of nature, are sufficient for all of our decisions. Neuroscience is consistent with indeterminism, so it is better understood as posing a ‘threat from below’: If our decision-making processes are carried out by neural processes, then it might seem that our decisions are not based on our prior conscious deliberations or (...) reasoning. The response to this threat will require a neurophilosophical theory of mind that makes sense of the causal role of our conscious reasons and reasoning. Without such a theory, our conscious self seems bypassed by the neural processes in our brains, and this view seems to explain why many scientists assume that neuroscience challenges free will. However, I argue that most people are amenable to the possibility of a future theory of mind that is physicalist (if not reductionist), yet preserves much of our ordinary experience and understanding of conscious decision-making and self-control. I outline such a theory using the resources of causal interventionism. I argue that this view is best understood as a minimal revision to our understanding of free will, rather than an elimination of it. And I argue that this view has more reasonable and effective implications for our moral and legal practices than an eliminativist or skeptical theory of free will. (shrink)

Structural models of systems of causal connections have become a common tool in the analysis of the concept of causation. In the present paper I offer a general argument to show that one of the most powerful definitions of the concept of actual cause, provided within the structural models framework, is not sufficient to grant a full account of our intuitive judgements about actual causation, so that we are still waiting for a comprehensive definition. This is done not simply by (...) focusing on a set of case studies, but by arguing that our intuitions about two different kinds of causal patterns, i.e., overdetermination and counterdetermination, cannot be addressed using that definition. (shrink)

It is argued that a popular way of accounting for the distinctive value of knowledge by appeal to the distinctive value of cognitive achievements fails because it is a mistake to identify knowledge with cognitive achievements. Nevertheless, it is claimed that understanding, properly conceived, is a type of cognitive achievement, and thus that the distinctive value of cognitive achievements can explain why understanding is of special value.

This paper considers the applicability of standard accounts of causation to living systems. In particular it examines critically the increasing tendency to equate causal explanation with the identification of a mechanism. A range of differences between living systems and paradigm mechanisms are identified and discussed. While in principle it might be possible to accommodate an account of mechanism to these features, the attempt to do so risks reducing the idea of a mechanism to vacuity. It is proposed that the solution (...) to this problem requires the development of a philosophical account of process adequate to apply to living systems. (shrink)

Contributing to the recent debate on whether or not explanations ought to be differentiated from arguments, this article argues that the distinction matters to science education. I articulate the distinction in terms of explanations and arguments having to meet different standards of adequacy. Standards of explanatory adequacy are important because they correspond to what counts as a good explanation in a science classroom, whereas a focus on evidence-based argumentation can obscure such standards of what makes an explanation explanatory. I provide (...) further reasons for the relevance of not conflating explanations with arguments (and having standards of explanatory adequacy in view). First, what guides the adoption of the particular standards of explanatory adequacy that are relevant in a scientific case is the explanatory aim pursued in this context. Apart from explanatory aims being an important aspect of the nature of science, including explanatory aims in classroom instruction also promotes students seeing explanations as more than facts, and engages them in developing explanations as responses to interesting explanatory problems. Second, it is of relevance to science curricula that science aims at intervening in natural processes, not only for technological applications, but also as part of experimental discovery. Not any argument enables intervention in nature, as successful intervention specifically presupposes causal explanations. Students can fruitfully explore in the classroom how an explanatory account suggests different options for intervention. (shrink)

How can a model that stops short of representing the whole truth about the causal production of a phenomenon help us to understand the phenomenon? I answer this question from the perspective of what I call the simple view of understanding, on which to understand a phenomenon is to grasp a correct explanation of the phenomenon. Idealizations, I have argued in previous work, flag factors that are casually relevant but explanatorily irrelevant to the phenomena to be explained. Though useful to (...) the would-be understander, such flagging is only a first step. Are there any further and more advanced ways that idealized models aid understanding? Yes, I propose: the manipulation of idealized models can provide considerable insight into the reasons that some causal factors are difference-makers and others are not, which helps the understander to grasp the nature of explanatory connections and so to better grasp the explanation itself. (shrink)

I examine the adequacy of the causal graph-structural equations approach to causation for modeling biological mechanisms. I focus in particular on mechanisms with complex dynamics such as the PER biological clock mechanism in Drosophila. I show that a quantitative model of this mechanism that uses coupled differential equations – the well-known Goldbeter model – cannot be adequately represented in the standard causal graph framework, even though this framework does permit causal cycles. The reason is that the model contains dynamical information (...) about the mechanism that concerns causal properties but that does not correspond to variables that could be subject to independent interventions. Thus, a representation of the mechanisms as a causal structural model necessarily suppresses causally relevant information. (shrink)

Spirtes, Glymour and Scheines [Causation, Prediction, and Search Springer] described a pointwise consistent estimator of the Markov equivalence class of any causal structure that can be represented by a directed acyclic graph for any parametric family with a uniformly consistent test of conditional independence, under the Causal Markov and Causal Faithfulness assumptions. Robins et al. [Biometrika 90 491–515], however, proved that there are no uniformly consistent estimators of Markov equivalence classes of causal structures under those assumptions. Subsequently, Kalisch and B¨uhlmann (...) [J. Mach. Learn. Res. 8 613–636] described a uniformly consistent estimator of the Markov equivalence class of a linear Gaussian causal structure under the Causal Markov and Strong Causal Faithfulness assumptions. However, the Strong Faithfulness assumption may be false with high probability in many domains. We describe a uniformly consistent estimator of both the Markov equivalence class of a linear Gaussian causal structure and the identifiable structural coefficients in the Markov equivalence class under the Causal Markov assumption and the considerably weaker k-Triangle-Faithfulness assumption. (shrink)

The paper explores a deductive-nomological account of metaphysical explanation: some truths metaphysically explain, or ground, another truth just in case the laws of metaphysics determine the latter truth on the basis of the former. I develop and motivate a specific conception of metaphysical laws, on which they are general rules that regulate the existence and features of derivative entities. I propose an analysis of the notion of ‘determination via the laws’, based on a restricted form of logical entailment. I argue (...) that the DN-account of ground can be defended against the well-known objections to the DN-approach to scientific explanation. The goal of the paper is to show that the DN-account of metaphysical explanation is a well-motivated and defensible theory. (shrink)

Humans often attribute the things that happen to one or another actual cause. In this chapter, we survey some recent philosophical and psychological research on causal attribution. We pay special attention to the relation between graphical causal modeling and theories of causal attribution. We think that the study of causal attribution is one place where formal and experimental techniques nicely complement one another.

The statistical technique of analysis of variance is often used by biologists as a measure of causal factors’ relative strength or importance. I argue that it is a tool ill suited to this purpose, on several grounds. I suggest a superior alternative, and outline some implications. I finish with a diagnosis of the source of error – an unwitting inheritance of bad philosophy that now requires the remedy of better philosophy.

The functionalist approach to kinds has suffered recently due to its association with law-based approaches to induction and explanation. Philosophers of science increasingly view nomological approaches as inappropriate for the special sciences like psychology and biology, which has led to a surge of interest in approaches to natural kinds that are more obviously compatible with mechanistic and model-based methods, especially homeostatic property cluster theory. But can the functionalist approach to kinds be weaned off its dependency on laws? Dan Weiskopf has (...) recently offered a reboot of the functionalist program by replacing its nomological commitments with a model-based approach more closely derived from practice in psychology. Roughly, Weiskopf holds that the natural kinds of psychology will be the functional properties that feature in many empirically successful cognitive models, and that those properties need not be localized to parts of an underlying mechanism. I here skeptically examine the three modeling practices that Weiskopf thinks introduce such non-localizable properties: fictionalization, reification, and functional abstraction. In each case, I argue that recognizing functional properties introduced by these practices as autonomous kinds comes at clear cost to those explanations’ counterfactual explanatory power. At each step, a tempting functionalist response is parochialism: to hold that the false or omitted counterfactuals fall outside the modeler’s explanatory aims, and so should not be counted against functional kinds. I conclude by noting the dangers this attitude poses to scientific disagreement, inviting functionalists to better articulate how the individuation conditions for functional kinds might outstrip the perspective of a single modeler. (shrink)

Causal assessment is the problem of establishing whether a relation between (variable) X and (variable) Y is causal. This problem, to be sure, is widespread across the sciences. According to accredited positions in the philosophy of causality and in social science methodology, invariance under intervention provides the most reliable test to decide whether X causes Y. This account of invariance (under intervention) has been criticised, among other reasons, because it makes manipulations on the putative causal factor fundamental for the causal (...) methodology; consequently, the argument goes, the account is ill-suited to those contexts where manipulations are not performed, for instance, the social sciences. The article aims to extend the account of invariance (under intervention), in a way that manipulations on the putative causal factors are not methodologically fundamental, and yet invariance remains key for causal assessment both in experimental and non-experimental contexts. (shrink)

A long-running dispute concerns which adaptation-related explananda natural selection can be said to explain. At issue are explananda of the form: why a given individual organism has a given adaptation rather than that same individual having another trait. It is broadly agreed that one must be ready to back up a “no” answer with an appropriate theory of trans-world identity for individuals. I argue, against the conventional wisdom, that the same is true for a “yes” answer. My conclusion recasts the (...) landscape and opens the door to a potential resolution. (shrink)

Several authors have recently claimed that the notorious causal exclusion problem, according to which higher-level causes are threatened with causal pre-emption by lower-level causes, can be avoided if causal relevance is understood in terms of Woodward's interventionist account of causation. They argue that if causal relevance is defined in interventionist terms, there are cases where only higher-level properties, but not the lower-level properties underlying them, qualify as causes of a certain effect. In this article, I show that the line of (...) reasoning supposed to establish this claim does not succeed and that interventionism is not better capable of dealing with higher-level causal claims than other accounts of causation. According to Woodward, higher-level causal claims are nonetheless more adequate than lower-level ones if they describe a realization-independent dependency relationship and, hence, meet the requirement that causes should be proportional to their effects. I argue, however, that combining interventionism with proportionality considerations raises difficulties and that, therefore, Woodward's account does not vindicate higher-level causation. (shrink)

My aim in this paper is to make a case for the singularist view from the perspective of a mechanical theory of causation, and to explain what, from this perspective, causal generalizations mean, and what role they play within the mechanical theory.

Dimensional analysis can offer us explanations by allowing us to answer What-if–things-had-been-different? questions rather than in virtue of, say, unifying diverse phenomena, important as that is. Additionally, it is argued that dimensional analysis is a form of modelling as it involves several of the aspects crucial in modelling, such as misrepresenting aspects of a target system. By highlighting the continuities dimensional analysis has with forms of modelling we are able to describe more precisely what makes dimensional analysis explanatory and understand (...) otherwise puzzling aspects of dimensional reasoning, such as introducing fictitious dimensions and excluding dimensionally relevant information to characterise some systems. Finally, thinking of dimensional arguments as a form of modelling allows an explication of the role abstraction and multiple realisability; not as compatibility with other possible worlds but as compatibility with different fictional descriptions of our own world. (shrink)

Comparing different causes’ importance, and apportioning responsibility between them, requires making good sense of the notion of partial explanation, that is, of degree of explanation. How much is this subjective, how much objective? If the causes in question are probabilistic, how much is the outcome due to them and how much to simple chance? I formulate the notion of degree of causation, or effect size, relating it to influential recent work in the literature on causation. I examine to what extent (...) mainstream social science methods--both quantitative and qualitative--succeed in establishing effect sizes so understood. The answer turns out to be, roughly: only to some extent. Next, the standard understanding of effect size, even though widespread, still has several underappreciated consequences. I detail some of those. Finally, I discuss the separate issue of explanandum-dependence, which is essential to assessing any cause’s explanatory importance and yet which has been comparatively neglected. (shrink)

Teleological explanations (TEs) account for the existence or properties of an entity in terms of a function: we have hearts because they pump blood, and telephones for communication. While many teleological explanations seem appropriate, others are clearly not warranted-for example, that rain exists for plants to grow. Five experiments explore the theoretical commitments that underlie teleological explanations. With the analysis of [Wright, L. (1976). Teleological Explanations. Berkeley, CA: University of California Press] from philosophy as a point of departure, we examine (...) in Experiment 1 whether teleological explanations are interpreted causally, and confirm that TEs are only accepted when the function invoked in the explanation played a causal role in bringing about what is being explained. However, we also find that playing a causal role is not sufficient for all participants to accept TEs. Experiment 2 shows that this is not because participants fail to appreciate the causal structure of the scenarios used as stimuli. In Experiments 3-5 we show that the additional requirement for TE acceptance is that the process by which the function played a causal role must be general in the sense of conforming to a predictable pattern. These findings motivate a proposal, Explanation for Export, which suggests that a psychological function of explanation is to highlight information likely to subserve future prediction and intervention. We relate our proposal to normative accounts of explanation from philosophy of science, as well as to claims from psychology and artificial intelligence. (shrink)

Constant conjunction theory of causation had been the dominant theory in philosophy for a long time and regained attention recently. This paper gives a logical framework of causation based on the theory. The basic idea is that causal statements are empirical, and are derived from our past experience by observing constant conjunction between objects. The logic is defined on linear time structures. A causal statement is evaluated at time points, such that its value depends on what has been in the (...) past. We first give a semantics that contains basic conditions that, we think, must hold for a concept of causation, on which we define the minimal causal logic. Then we discuss its possible extensions for various concepts of causation. Complete deductive systems are given. (shrink)

A theory of conceptual development must specify the innate representational primitives, must characterize the ways in which the initial state differs from the adult state, and must characterize the processes through which one is transformed into the other. The Origin of Concepts (henceforth TOOC) defends three theses. With respect to the initial state, the innate stock of primitives is not limited to sensory, perceptual, or sensorimotor representations; rather, there are also innate conceptual representations. With respect to developmental change, conceptual development (...) consists of episodes of qualitative change, resulting in systems of representation that are more powerful than, and sometimes incommensurable with, those from which they are built. With respect to a learning mechanism that achieves conceptual discontinuity, I offer Quinian bootstrapping. TOOC concludes with a discussion of how an understanding of conceptual development constrains a theory of concepts. (shrink)

This article discusses the role of simplicity and the notion of a best balance of simplicity and strength within the best systems account (BSA) of laws of nature. The article explores whether there is anything in scientific practice that corresponds to the notion of simplicity or to the trade-off between simplicity and strength to which the BSA appeals. Various theoretical rationales for simplicity preferences and their bearing on the identification of laws are also explored. It is concluded that there are (...) a number of issues about the role of simplicity within the BSA and its relation to strength that need to be addressed before the BSA can be regarded as an adequate account of laws. 1 Introduction2 The Best Systems Account3 The Trade-Off between Simplicity and Strength: Preliminary Considerations4 Alternative Conceptions of the Relationship between Simplicity and Strength5 Two Roles for Simplicity6 Simplicity in the Best Systems Account: Curve-Fitting7 Simplicity as a Corrective for Overfitting8 Descriptive Simplicity in the Best Systems Account?9 Simplicity as Due to Human Intellectual Limitations10 Summary11 Concluding Remarks. (shrink)

Epistemic relativists often appeal to an epistemic incommensurability thesis. One notable example is the position advanced by Wittgenstein in On certainty (1969). However, Ian Hacking’s radical denial of the possibility of objective epistemic reasons for belief poses, we suggest, an even more forceful challenge to mainstream meta-epistemology. Our central objective will be to develop a novel strategy for defusing Hacking’s line of argument. Specifically, we show that the epistemic incommensurability thesis can be resisted even if we grant the very insights (...) that lead Hacking to claim that epistemic reasons are always relative to a style of reasoning. Surprisingly, the key to defusing the argument is to be found in recent mainstream work on the epistemic state of objectual understanding. (shrink)

This paper sets out to analyze how causation works by focusing on biology, as represented by epidemiology and by scientific information on how the body works (“physiology”). It starts by exploring the specificity of evolved physiological systems, in which evolutionary, developmental and proximal causes all fit together, and the concept of function is meaningful; in contrast, this structure does not apply in epidemiology (or outside biology). Using these two contrasting branches of biology, I examine the role both of mechanism and (...) of difference making in causation. I find that causation necessarily involves both mechanism and difference making, and that they play complementary roles. Both are seen as ontologically necessary, even if the evidence is not always available for both. Influential monist accounts that focus on one of these, at the expense of ignoring the other, are found to be inadequate on these and on other grounds. Recent attempts to combine them are reviewed, notably that of Russo and Williamson (Int Stud Philos Sci 21:157–170, 2007), and it is argued that their epistemic view requires there to be a source of the different types of evidence that a rational agent would consider, and that this source must be ontic. I then analyze how causal relationships work in evolved physiological systems and in those studied by epidemiology, with a particular focus on how mechanism interacts with input. Finally, I consider this concept of causation from the perspective of everyday language, and of its possible generalisability outside biology. (shrink)

What is the relationship between pain and the body? I claim that pain is best explained as a type of personal experience and the bodily response during pain is best explained in terms of a type of mechanical neurophysiologic operation. I apply the radical philosophy of identity theory from philosophy of mind to the relationship between the personal experience of pain and specific neurophysiologic mechanism and argue that the relationship between them is best explained as one of type identity. Specifically, (...) pain is a specific type of personal experience identical to a specific type of allostatic stress response comprised of interdependent nervous, endocrine and immune mechanical operations. (shrink)

Recent accounts of actual causation are stated in terms of extended causal models. These extended causal models contain two elements representing two seemingly distinct modalities. The first element are structural equations which represent the or mechanisms of the model, just as ordinary causal models do. The second element are ranking functions which represent normality or typicality. The aim of this paper is to show that these two modalities can be unified. I do so by formulating two constraints under which extended (...) causal models with their two modalities can be subsumed under so called which contain just one modality. These two constraints will be formally precise versions of Lewissystem of weights or priorities” governing overall similarity between possible worlds. (shrink)

How do people attribute responsibility in situations where the contributions of multiple agents combine to produce a joint outcome? The prevalence of over-determination in such cases makes this a difficult problem for counterfactual theories of causal responsibility. In this article, we explore a general framework for assigning responsibility in multiple agent contexts. We draw on the structural model account of actual causation (e.g., Halpern & Pearl, 2005) and its extension to responsibility judgments (Chockler & Halpern, 2004). We review the main (...) theoretical and empirical issues that arise from this literature and propose a novel model of intuitive judgments of responsibility. This model is a function of both pivotality (whether an agent made a difference to the outcome) and criticality (how important the agent is perceived to be for the outcome, before any actions are taken). The model explains empirical results from previous studies and is supported by a new experiment that manipulates both pivotality and criticality. We also discuss possible extensions of this model to deal with a broader range of causal situations. Overall, our approach emphasizes the close interrelations between causality, counterfactuals, and responsibility attributions. (shrink)

Two strains of interventionist responses to the causal exclusion argument are reviewed and critically assessed. On the one hand, one can argue that manipulating supervenient mental states is an effective strategy for manipulating the subvenient physical states, and hence should count as genuine causes to the subvenient physical states. But unless the supervenient and subvenient states manifest some difference in their manipulability conditions, there is no reason to treat them as distinct, which in turn goes against the basic assumption of (...) nonreductive physicalism. On the other hand, one can preserve the distinction between the two by introducing asymmetric manipulability conditions that the supervenience thesis entails. But this response can be used to argue that mental causes never have physical effects. However, this argumentation can also be used to show that mental causes can have mental effects. (shrink)

The paper discusses how systems biology is working toward complex accounts that integrate explanation in terms of mechanisms and explanation by mathematical models—which some philosophers have viewed as rival models of explanation. Systems biology is an integrative approach, and it strongly relies on mathematical modeling. Philosophical accounts of mechanisms capture integrative in the sense of multilevel and multifield explanations, yet accounts of mechanistic explanation have failed to address how a mathematical model could contribute to such explanations. I discuss how mathematical (...) equations can be explanatorily relevant. Several cases from systems biology are discussed to illustrate the interplay between mechanistic research and mathematical modeling, and I point to questions about qualitative phenomena, where quantitative models are still indispensable to the explanation. Systems biology shows that a broader philosophical conception of mechanisms is needed, which takes into account functional-dynamical aspects, interaction in complex networks with feedback loops, system-wide functional properties such as distributed functionality and robustness, and a mechanism’s ability to respond to perturbations. I offer general conclusions for philosophical accounts of explanation. (shrink)

Recent years have seen renewed interest in the emergence issue. The contemporary debate, in contrast with that of past times, has to do not so much with the mind–body problem as with the relationship between the physical and other domains; mostly with the biological domain. One of the main sources of this renewed interest is the study of complex and, in general, far-from-equilibrium self-preserving systems, which seem to fulfil one of the necessary conditions for an entity to be emergent; namely, (...) that its causal powers are not predictable from the causal powers of basic physical properties. However, I argue that much of the current emergentism debate has misfired by focusing on the interpretation of self-maintaining systems. In contrast, I claim that if we want to find emergent properties, we should look not at complex systems, but at selection (natural selection, in particular). I argue that selection processes make the causal world ‘exuberant’ by making non-physical functional and relational properties enter the causal web of the world. (shrink)

Evolutionary developmental biology (evo-devo) is considered a ‘mechanistic science,’ in that it causally explains morphological evolution in terms of changes in developmental mechanisms. Evo-devo is also an interdisciplinary and integrative approach, as its explanations use contributions from many fields and pertain to different levels of organismal organization. Philosophical accounts of mechanistic explanation are currently highly prominent, and have been particularly able to capture the integrative nature of multifield and multilevel explanations. However, I argue that evo-devo demonstrates the need for a (...) broadened philosophical conception of mechanisms and mechanistic explanation. Mechanistic explanation (in terms of the qualitative interactions of the structural parts of a whole) has been developed as an alternative to the traditional idea of explanation as derivation from laws or quantitative principles. Against the picture promoted by Carl Craver, that mathematical models describe but usually do not explain, my discussion of cases from the strand of evo-devo which is concerned with developmental processes points to qualitative phenomena where quantitative mathematical models are an indispensable part of the explanation. While philosophical accounts have focused on the actual organization and operation of mechanisms, properties of developmental mechanisms that are about how a mechanism reacts to modifications are of major evolutionary significance, including robustness, phenotypic plasticity, and modularity. A philosophical conception of mechanisms is needed that takes into account quantitative changes, transient entities and the generation of novel types of entities, feedback loops and complex interaction networks, emergent properties, and, in particular, functional-dynamical aspects of mechanisms, including functional (as opposed to structural) organization and distributed, system-wide phenomena. I conclude with general remarks on philosophical accounts of explanation. (shrink)

The authors examined cue competition effects in young children using the blicket detector paradigm, in which objects are placed either singly or in pairs on a novel machine and children must judge which objects have the causal power to make the machine work. Cue competition effects were found in a 5- to 6-year-old group but not in a 4-year-old group. Equivalent levels of forward and backward blocking were found in the former group. Children's counterfactual judgments were subsequently examined by asking (...) whether or not the machine would have gone off in the absence of I of 2 objects that had been placed on it as a pair. Cue competition effects were demonstrated only in 5- to 6-year-olds using this mode of assessing causal reasoning. (shrink)

Both philosophers and psychologists have argued for the existence of distinct kinds of explanations, including teleological explanations that cite functions or goals, and mechanistic explanations that cite causal mechanisms. Theories of causation, in contrast, have generally been unitary, with dominant theories focusing either on counterfactual dependence or on physical connections. This paper argues that both approaches to causation are psychologically real, with different modes of explanation promoting judgments more or less consistent with each approach. Two sets of experiments isolate the (...) contributions of counterfactual dependence and physical connections in causal ascriptions involving events with people, artifacts, or biological traits, and manipulate whether the events are construed teleologically or mechanistically. The findings suggest that when events are construed teleologically, causal ascriptions are sensitive to counterfactual dependence and relatively insensitive to the presence of physical connections, but when events are construed mechanistically, causal ascriptions are sensitive to both counterfactual dependence and physical connections. The conclusion introduces an account of causation, an "exportable dependence theory," that provides a way to understand the contributions of physical connections and teleology in terms of the functions of causal ascriptions. (shrink)

(2013). Hierarchical Bayesian models as formal models of causal reasoning. Argument & Computation: Vol. 4, Formal Models of Reasoning in Cognitive Psychology, pp. 36-45. doi: 10.1080/19462166.2012.700321.

This contribution claims that the two fundamental notions of causation at work in the health sciences are manipulative and mechanistic, and investigates what kinds of evidence matter for the assessment of causal relations. This article is a development of our 2007 article, ‘Plurality of Causality’, where we argue for a pluralistic account of causation with an eye to econometrics and a single medical example. The present contribution has a wider focus, and considers the notion of evidence within a whole range (...) of disciplines belonging to the health sciences. Section 1 addresses the relations between kinds of evidence and causal accounts, and it is shown how different notions of causation can be employed in various medical cases. Section 2 calls attention to issues crucial for any adequate epistemological theory of causation, such as the distinctions between types and tokens, observational and experimental regimes, explanation and prediction. Lastly, the notion of context is articulated, highlighted in its role in the assessment of causal links. All these issues are tackled in the framework of what we label a ‘bottom–up’ epistemology. (shrink)

This paper argues that, notwithstanding the remarkable popularity of Woodward's (2003) interventionist analysis of causation, the exact definitional details of that theory are surprisingly little understood. There exists a discrepancy in the literature between the clarity about the logical details of interventionism, on the one hand, and the enormous work interventionism is expected to do, on the other. The first part of the paper distinguishes three significantly different readings of the logical form of Woodward's (2003) interventionist theory and identifies the (...) reading that best captures the basic intuitions behind interventionism. In the second part, I show that this preferable reading is far from doing all the work that friends of interventionism would like it to do. (shrink)

Using structural equations and directed graphs, Christopher Hitchcock (2007a) proposes a theory specifying the circumstances in which counterfactual dependence of one event e on another event c is necessary and sufficient for c to count as an actual cause of e. In this paper, we argue that Hitchcock is committed to a widely-endorsed folk attribution desideratum (FAD) for theories of actual causation. We then show experimentally that Hitchcock’s theory does not satisfy the FAD, and hence, it is in need of (...) revision. (shrink)

This paper defends Pragmatic Conceptual Analysis , a proposed empirical methodology for explicating philosophical concepts. This methodology attributes to our shared concepts whatever application conditions they would need to have in order best to continue delivering benefits in the ways they have regularly delivered benefits in the past. In the first stage of my argument I argue that Pragmatic Conceptual Analysis has what I call normative authority : we have practical and epistemic reason to adopt the explications that it delivers (...) even if we think doing so requires stipulative revisions in the meanings of our concepts. I then use this normative authority to argue that Pragmatic Conceptual Analysis also has what I call descriptive authority : when we understand concept-meaning in the way we ought to understand it (in the way licensed by the normative authority of Pragmatic Conceptual Analysis) we see that, rather than being revisionary, Pragmatic Conceptual Analysis is a semantically conservative tool that uncovers (what we should think of as being) the meanings our concepts already have. (shrink)

This article discusses an epistemological problem faced by causal explanations of action and a proposed solution. The problem is to justify why one particular reason rather than another is specified as causally efficacious. It is argued that the problem arises independently of one’s preferred conception of singular causal claims, psychological and psychophysical generalizations, and our folk-psychological competence. The proposed fallibilist solution involves the supplementation of the reason given by narratives that contextualize it and provide additional criteria for justifying the causal (...) claim. It is argued that narratives have a distinctive structure that can afford the justification of causal attributions without sui generis powers of narrative explanation having to be invoked. (shrink)

The notion of reduction continues to play an important role in contemporary analytic philosophy. Being Reduced is a collection of essays that not only presents novel contributions to our understanding of reduction, but also aims at finding connections between the debates in philosophy of mind and philosophy of science, which have surprisingly remained rather detached from each other. Being reduced succeeds in this difficult task, and is a very welcome addition to the growing philosophical literature on reduction.