In a recent paper (1994) Wesley Salmon has replied to criticisms (e.g., Dowe 1992c, Kitcher 1989) of his (1984) theory of causality, and has offered a revised theory which, he argues, is not open to those criticisms. The key change concerns the characterization of causal processes, where Salmon has traded "the capacity for mark transmission" for "the transmission of an invariant quantity." Salmon argues against the view presented in Dowe (1992c), namely that the concept of "possession of a conserved quantity" (...) is sufficient to account for the difference between causal and pseudo processes. Here that view is defended, and important questions are raised about the notion of transmission and about gerrymandered aggregates. (shrink)

In a recent paper Wesley Salmon has replied to criticisms of his theory of causality, and has offered a revised theory which, he argues, is not open to those criticisms. The key change concerns the characterization of causal processes, where Salmon has traded “the capacity for mark transmission” for “the transmission of an invariant quantity.” Salmon argues against the view presented in Dowe, namely that the concept of “possession of a conserved quantity” is sufficient to account for the difference between (...) causal and pseudo processes. Here that view is defended, and important questions are raised about the notion of transmission and about gerrymandered aggregates. (shrink)

This paper presents a drastically revised version of the theory of causality, based on analyses of causal processes and causal interactions, advocated in Salmon (1984). Relying heavily on modified versions of proposals by P. Dowe, this article answers penetrating objections by Dowe and P. Kitcher to the earlier theory. It shows how the new theory circumvents a host of difficulties that have been raised in the literature. The result is, I hope, a more satisfactory analysis of physical causality.

This paper discusses several distinct process theories of causality offered in recent years by Phil Dowe and me. It addresses problems concerning the explication of causal process, causal interaction, and causal transmission, whether given in terms of transmission of marks, transmission of invariant or conserved quantities, or mere possession of conserved quantities. Renouncing the mark-transmission and invariant quantity criteria, I accept a conserved quantity theory similar to Dowe's--differing basically with respect to causal transmission. This paper also responds to several fundamental (...) constructive criticisms contained in Christopher Hitchcock's discussion of both the mark-transmission and the conserved quantity theories. (shrink)

The need to find an intrinsic characterization of what makes a relation between events causal arises not only in local theories of causation like Salmon's process theory but also in global approaches like Lewis' counterfactual theory. According to the localist intuition, whether a process connecting two events is causal should depend only on what goes on between the events, not on conditions that hold elsewhere in the world. If such intrinsic characterizations could be found, an identification of the causal relation (...) in the actual world (though not in other possible worlds) with physical processes may be feasible (the a posteriori identification). I consider recent proposals made for intrinsic characterizations of causality and conclude that none of them is able to deliver the intended result. (shrink)

Probabilistic accounts of causality have long had trouble with ‘spurious’ evidential correlations. Such correlations are also central to the case for causal decision theory—the argument that evidential decision theory is inadequate to cope with certain sorts of decision problem. However, there are now several strong defences of the evidential theory. Here I present what I regard as the best defence, and apply it to the probabilistic approach to causality. I argue that provided a probabilistic theory appeals to the notions of (...) agency and effective strategy, it can avoid the problem of spurious causes. I show that such an appeal has other advantages; and argue that it is not illegitimate, even for a causal realist. (shrink)

There are three main kinds of causal connections between two events: the transfer of energy, the transfer of information and the triggering of the transfer of one of them or both. There are also other kinds of causal connections.

What are the conceptually necessary and sufficient conditions for a person, or organism, to perceive a given object? More precisely, what is the nature of our ordinary thought about perception that gives rise to our willingness or unwillingness to say that S perceives O? Some form of causal theory of perception is now, I think, widely accepted. Such a theory maintains that it is part of our concept of perception that S perceives O only if O causes a percept, or (...) perceptual state, of S. I accept this causal requirement, though with some qualification. The crucial problem for the theory of perception is what must be added to the causal requirement. Not all causes of a percept are said to be perceived. Smith looks at a tree illuminated by the sun, when the sun is not itself in view. His visual percept is caused by the tree, but not only the tree. The percept's causal ancestry includes the sun and the array of light two inches from Smith's eyes. Why do we say that he sees the tree, but not the sun or the array of light? What principles underlie this invidious choice among causes? Similarly, we think of a bat as perceiving objects in his path, but not as perceiving himself. Yet, since he emits the sonar signals these objects reflect, he is himself a cause of his percepts. Why don't we say he perceives himself? (shrink)

Causation has traditionally been analyzed either as a relation of nomic dependence or as a relation of counterfactual dependence. I argue for a third program, a physicalistic reduction of the causal relation to one of energy-momentum transference in the technical sense of physics. This physicalistic analysis is argued to have the virtues of easily handling the standard counterexamples to the nomic and counterfactual analyses, offering a plausible epistemology for our knowledge of causes, and elucidating the nature of the relation between (...) causation and physical science. (shrink)

This paper examines Wesley Salmon's "process" theory of causality, arguing in particular that there are four areas of inadequacy. These are that the theory is circular, that it is too vague at a crucial point, that statistical forks do not serve their intended purpose, and that Salmon has not adequately demonstrated that the theory avoids Hume's strictures about "hidden powers". A new theory is suggested, based on "conserved quantities", which fulfills Salmon's broad objectives, and which avoids the problems discussed.

The leading accounts of the nature of causation divide into probability-raising and process-linkage views. On the probability-raising view, causation is rooted in the comparative probability of the effect with the cause versus without. On the process-linkage view, causation is rooted in the existence of a connecting line from cause to effect. I propose a third alternative which synthesizes these views while solving their problem cases. On this alternative, causation is rooted in the comparative probability of the connecting line to the (...) effect with the cause versus without: causes as probability raisers of processes. In the first two sections, I (briefly) outline the probability-raising and process-linkage views and identify a space of problems, and then I focus on developing the alternative probability-raisers-of-processes solution. I identify problems for this view as well, though I conclude that it is at least in important respects a step forward. (shrink)