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  1. A Third Way to the Selected Effect/Causal Role Distinction in the Great Encode Debate.Ehud Lamm & Sophie Veigl - 2023 - Theoretical Biology Forum 2023 (1-2):53-74.
    Since the ENCODE project published its final results in a series of articles in 2012, there is no consensus on what its implications are. ENCODE’s central and most controversial claim was that there is essentially no junk DNA: most sections of the human genome believed to be «junk» are functional. This claim was met with many reservations. If researchers disagree about whether there is junk DNA, they have first to agree on a concept of function and how function, given a (...)
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  2. The meaning of "cause" in genetics.Kate E. Lynch - 2021 - Combining Human Genetics and Causal Inference to Understand Human Disease and Development. Cold Spring Harbor Perspectives in Medicine.
    Causation has multiple distinct meanings in genetics. One reason for this is meaning slippage between two concepts of the gene: Mendelian and molecular. Another reason is that a variety of genetic methods address different kinds of causal relationships. Some genetic studies address causes of traits in individuals, which can only be assessed when single genes follow predictable inheritance patterns that reliably cause a trait. A second sense concerns the causes of trait differences within a population. Whereas some single genes can (...)
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  3. Rezension von "Sind wir fest verdrahtet? " (Are We Hardwired?) von Clark & Grunstein (2000) (Überprüfung überarbeitet 2019).Michael Richard Starks - 2020 - In Willkommen in der Hölle auf Erden: Babys, Klimawandel, Bitcoin, Kartelle, China, Demokratie, Vielfalt, Dysgenie, Gleichheit, Hacker, Menschenrechte, Islam, Liberalismus, Wohlstand, Internet, Chaos, Hunger, Krankheit, Gewalt, Künstliche Intelligenz, Krieg. Reality Press. pp. 91-94.
    Dies ist eine ausgezeichnete Überprüfung der Gen-/Umgebungsinteraktionen auf das Verhalten und ist, obwohl sie etwas veraltet ist, eine einfache und lohnende Lektüre. Sie beginnen mit Zwillingsstudien, die den überwältigenden Einfluss der Genetik auf das Verhalten zeigen. Sie stellen die immer bekannter werdenden Studien von Judith Harris fest, die die Fakten erweitern und zusammenfassen, dass die gemeinsame häusliche Umgebung fast keinen Einfluss auf das Verhalten hat und dass adoptierte Kinder so anders wachsen als ihre Stiefbrüder und -schwestern wie zufällig ausgewählte Menschen. (...)
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  4. Mechanism schemas and the relationship between biological theories.Tudor M. Baetu - 2011 - In Phyllis McKay Illari Federica Russo (ed.), Causality in the Sciences. Oxford University Press.
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  5. Schaffner’s Model of Theory Reduction: Critique and Reconstruction.Rasmus Gr⊘Nfeldt Winther - 2009 - Philosophy of Science 76 (2):119-142.
    Schaffner’s model of theory reduction has played an important role in philosophy of science and philosophy of biology. Here, the model is found to be problematic because of an internal tension. Indeed, standard antireductionist external criticisms concerning reduction functions and laws in biology do not provide a full picture of the limits of Schaffner’s model. However, despite the internal tension, his model usefully highlights the importance of regulative ideals associated with the search for derivational, and embedding, deductive relations among mathematical (...)
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  6. Alternative Definitions of Epistasis: Dependence and Interaction.Michael J. Wade, Rasmus Grønfeldt Winther, Aneil F. Agrawal & Charles J. Goodnight - 2001 - Trends in Ecology and Evolution 16 (9):498-504.
    Although epistasis is at the center of the Fisher-Wright debate, biologists not involved in the controversy are often unaware that there are actually two different formal definitions of epistasis. We compare concepts of genetic independence in the two theoretical traditions of evolutionary genetics, population genetics and quantitative genetics, and show how independence of gene action (represented by the multiplicative model of population genetics) can be different from the absence of gene interaction (represented by the linear additive model of quantitative genetics). (...)
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