This paper is about the metaphysical debate whether objects persist over time by the selfsame object existing at different times (nowadays called “endurance” by metaphysicians), or by different temporal parts, or stages, existing at different times (called “perdurance”). I aim to illuminate the debate by using some elementary kinematics and real analysis: resources which metaphysicians have, surprisingly, not availed themselves of. There are two main results, which are of interest to both endurantists and perdurantists. (1) I describe a precise formal (...) equivalence between the way that the two metaphysical positions represent the motion of the objects of classical mechanics (both point-particles and continua). (2) I make precise, and prove a result about, the idea that the persistence of objects moving in a void is to be analysed in terms of tracking the continuous curves in spacetime that connect points occupied by matter. The result is entirely elementary: it is a corollary of the Heine–Borel theorem. (shrink)

Some recent philosophical debate about persistence has focussed on an argument against perdurantism that discusses rotating perfectly homogeneous discs. The argument has been mostly discussed by metaphysicians, though it appeals to ideas from classical mechanics, especially about rotation. In contrast, I assess the RDA from the perspective of the philosophy of physics. After introducing the argument and emphasizing the relevance of physics, I review some metaphysicians' replies to the argument, especially those by Callender, Lewis, Robinson and Sider. Thereafter, I argue (...) for three main conclusions. They all arise from the fact, emphasized in Section 2, that classical mechanics is both more subtle, and more problematic, than philosophers generally realize. The first conclusion is that the RDA can be formulated more strongly than is usually recognized: it is not necessary to ``imagine away'' the dynamical effects of rotation. The second is that in general relativity, the RDA fails because of frame-dragging. The third is that even setting aside general relativity, the strong formulation of the RDA can after all be defeated. Namely, by the perdurantist taking objects in classical mechanics to have only temporally extended, i.e. non-instantaneous, temporal parts: which immediately blocks the RDA. Admittedly, this version of perdurantism defines persistence in a weaker sense of `definition' than pointilliste versions that aim to define persistence assuming only instantaneous temporal parts. But I argue that temporally extended temporal parts: can do the jobs within the endurantism- perdurantism debate that the perdurantist wants temporal parts to do; and are supported by both classical and quantum mechanics. (shrink)

Causation is widely assumed to be a binary relation: c causes e. I will argue that causation is a quaternary, contrastive relation: c rather than C* causes e rather than E*, where C* and E* are nonempty sets of contrast events. Or at least, I will argue that treating causation as contrastive helps resolve some paradoxes.

Russell famously argued that causation should be dispensed with. He gave two explicit arguments for this conclusion, both of which can be defused if we loosen the ties between causation and determinism. I show that we can define a concept of causation which meets Russell’s conditions but does not reduce to triviality. Unfortunately, a further serious problem is implicit beneath the details of Russell’s arguments, which I call the causal exclusion problem. Meeting this problem involves deploying a minimalist pragmatic account (...) of the nature and function of modal language. Russell’s scruples about causation can be accommodated, even as we partially legitimise the pervasive causal explanations in folk and scientific practice. (shrink)

Is the common cause principle merely one of a set of useful heuristics for discovering causal relations, or is it rather a piece of heavy duty metaphysics, capable of grounding the direction of causation itself? Since the principle was introduced in Reichenbach’s groundbreaking work The Direction of Time (1956), there have been a series of attempts to pursue the latter program—to take the probabilistic relationships constitutive of the principle of the common cause and use them to ground the direction of (...) causation. These attempts have not all explicitly appealed to the principle as originally formulated; it has also appeared in the guise of independence conditions, counterfactual overdetermination, and, in the causal modelling literature, as the causal markov condition. In this paper, I identify a set of difficulties for grounding the asymmetry of causation on the principle and its descendents. The first difficulty, concerning what I call the vertical placement of causation, consists of a tension between considerations that drive towards the macroscopic scale, and considerations that drive towards the microscopic scale—the worry is that these considerations cannot both be comfortably accommodated. The second difficulty consists of a novel potential counterexample to the principle based on the familiar Einstein Podolsky Rosen (EPR) correlations in quantum mechanics. (shrink)

We argue that the health sciences make causal claims on the basis of evidence both of physical mechanisms, and of probabilistic dependencies. Consequently, an analysis of causality solely in terms of physical mechanisms or solely in terms of probabilistic relationships, does not do justice to the causal claims of these sciences. Yet there seems to be a single relation of cause in these sciences - pluralism about causality will not do either. Instead, we maintain, the health sciences require a theory (...) of causality that unifies its mechanistic and probabilistic aspects. We argue that the epistemic theory of causality provides the required unification. (shrink)

Concerning any object of philosophical analysis, we can ask several questions, including the two posed in the title of this paper. Despite difficulties in formulating a precise criterion to distinguish causal processes from pseudoprocesses, and causal interactions from mere spatiotemporal intersections, I argue that Salmon answered the first of these questions with extraordinary clarity. The second question, by contrast, has received very little attention. I will present two problems: in the first, it seems that Salmon has provided exactly the conceptual (...) resources needed to solve the problem; in the second, it is difficult to see how causal processes and interactions may be used to shed any light. In general, the way to carry Salmon's program forward will be to demonstrate that these resources can be made to do real philosophical work. (shrink)

This paper presents an attempt to integrate theories of causal processes—of the kind developed by Wesley Salmon and Phil Dowe—into a theory of causal models using Bayesian networks. We suggest that arcs in causal models must correspond to possible causal processes. Moreover, we suggest that when processes are rendered physically impossible by what occurs on distinct paths, the original model must be restricted by removing the relevant arc. These two techniques suffice to explain cases of late preëmption and other cases (...) that have proved problematic for causal models. (shrink)

In this paper I show how the conserved quantity theory, or more generally the process theory of Wesley Salmon and myself, provides a sufficient condition in an analysis of causation. To do so I will show how it handles the problem of alleged 'misconnections'. I show what the conserved quantity theory says about such cases, and why intuitions are not to be taken as sacrosanct.

We argue that intelligible appeals to interlevel causes (top-down and bottom-up) can be understood, without remainder, as appeals to mechanistically mediated effects. Mechanistically mediated effects are hybrids of causal and constitutive relations, where the causal relations are exclusively intralevel. The idea of causation would have to stretch to the breaking point to accommodate interlevel causes. The notion of a mechanistically mediated effect is preferable because it can do all of the required work without appealing to mysterious interlevel causes. When interlevel (...) causes can be translated into mechanistically mediated effects, the posited relationship is intelligible and should raise no special philosophical objections. When they cannot, they are suspect. (shrink)

I discuss the idea of relativistic causality, i.e., the requirement that causal processes or signals can propagate only within the light-cone. After briefly locating this requirement in the philosophy of causation, my main aim is to draw philosophers' attention to the fact that it is subtle, indeed problematic, in relativistic quantum physics: there are scenarios in which it seems to fail. I set aside two such scenarios, which are familiar to philosophers of physics: the pilot-wave approach, and the Newton-Wigner representation. (...) I instead stress two unfamiliar scenarios: the Drummond-Hathrell and Scharnhorst effects. These effects also illustrate a general moral in the philosophy of geometry: that the mathematical structures, especially the metric tensor, that represent geometry get their geometric significance by dint of detailed physical arguments. (shrink)

The paper argues for four claims: (1) The problem of mental causation and the argument for its solution in terms of the identity of mental with physical causes are independent of the theory of causation one favours. (2) If one considers our experience of agency as described by folk psychology to be veridical, one is committed to an anti-Humean metaphysics of causation in terms of powers that establish necessary connections. The same goes for functional properties in general. (3) A metaphysics (...) of causation in terms of powers is compatible with physics. (4) If combined with the argument for mental causes being identical with physical causes, that metaphysics leads to a conservative reductionism. (shrink)

I think that there is an awful lot wrong with the exclusion problem. So, it seems, does just about everybody else. But of course everyone disagrees about exactly _what_ is wrong with it, and I think there is more to be said about that. So I propose to say a few more words about why the exclusion problem is not really a problem after all—at least, not for the nonreductive physicalist. The genuine _dualist_ is still in trouble. Indeed, one of (...) my main points will be that the nonreductive physicalist is in a rather different position vis à vis the exclusion problem than the dualist is. Properly understanding nonreductive physicalism—and clearly recognizing that it is, after all, a form of _physicalism_—goes a long way toward solving the exclusion problem. (shrink)

According to an increasing number of authors, the best, if not the only, argument in favour of physicalism is the so-called 'overdetermination argument'. This argument, if sound, establishes that all the entities that enter into causal interactions with the physical world are physical. One key premise in the overdetermination argument is the principle of the causal closure of the physical world, said to be supported by contemporary physics. In this paper, I examine various ways in which physics may support the (...) principle, either as a methodological guide or as depending on some other laws and principles of physics. (shrink)

In this paper, we critically analyze the mechanicist approach to scientific explanation and propose to complement this view with a conception of level as interval of quasi-decompositionality. This conception allows us to metaphysically base the mechanicist proposals on characteristic systemic features. By means of concrete examples, we claim that this proposal is able to overcome some of the alleged limitations of predominant models of mechanistic explanation.

Here we enrich Paul Weirich’s thesis holding that a simulation model can create knowledge in the form of causal explanations. We sustain the validity of exporting results from the model to the modelized world in virtue of the similarity between model and world, which is analyzable in terms of partial identity of structure, eliminating the superficial similarity that repeats empirical results by adjusting data via calibration. The structure of relations rescues from the world critical results to analyze such similarity, as (...) so certain properties which condition the kind of relations held between the elements of the model representing entities of the world. (shrink)

Epistemic theories of objective chance hold that chances are idealised epistemic probabilities of some sort. After giving a brief history of this approach to objective chance, I argue for a particular version of this view, that the chance of an event E is its epistemic probability, given maximal knowledge of the possible causes of E. The main argument for this view is the demonstration that it entails all of the commonly-accepted properties of chance. For example, this analysis entails that chances (...) supervene on the physical facts, that the chance function is an expert probability, that the existence of stable frequencies results from invariant chances in repeated experiments, and that chances are probably close to long-run relative frequencies in repeated experiments. Despite these virtues, epistemic approaches to chance have been neglected in recent decades, on account of their conflict with accepted views about closely related topics such as causation, laws of nature, and epistemic probability. However, existing views on these topics are also very problematic, and I believe that the epistemic view of chance is a key piece of this puzzle that, once in place, allows all the other pieces to fit together into a new and coherent way. I also respond to some criticisms of the epistemic theory that I favour. (shrink)

Much of the recent philosophical debate on causation and causal explanation in the biological and biomedical sciences has focused on the notion of mechanism. Mechanisms, their nature and epistemic roles have been tackled by a range of so-called neo-mechanistic theories, and widely discussed. Without denying the merits of this approach, our paper aims to show how lately it has failed to give proper credit to processes, which are central to the field, especially of contemporary molecular biology. Processes can be summed (...) up in the notion of ‘pathway’, which is far from being just equivalent to that of ‘mechanism’ and has a profound epistemological and explanatory relevance. It is argued that an adequate consideration of pathways impels some rethinking of scientific explanation in molecular biology, namely its functional and contextual features. A number of examples are given to suggest that the focus of philosophical attention in this disciplinary field should shift from the notion of mechanism to the notion of pathway. (shrink)

What does it mean to say that mind-body dualism is causally problematic in a way that other mind-body theories, such as the psychophysical type identity theory, are not? After considering and rejecting various proposals, I advance my own, which focuses on what grounds the causal closure of the physical realm. A metametaphysical implication of my proposal is that philosophers working without the notion of grounding in their toolkit are metaphysically impoverished. They cannot do justice to the thought, encountered in every (...) introductory class in the philosophy of mind, that dualism has a special problem accounting for mental causation. (shrink)

The folk picture of agency suggests that human beings have basic agency over some of their omissions. For example, someone may follow through on a decision never to support a political party without doing anything in order to make themselves omit. A number of features appear to signal their agency: the omission is not just called intentional, it is also seen as an achievement and explained in terms of the reasons for the decision. Some philosophers have tried to debunk the (...) appearance and argue that agency over omissions always proceeds from agency over actions (on the model of Ulysses ordering his crew to tie him to the mast). Others have tried to uphold the appearance by understanding basic agency over omissions on the model of their theories of action (and most often on the model of causal theories). Against both groups, I argue for a vindication of the appearance in terms of a theory of sui generis basic agency over omissions. Basic intentional omissions are the behavioral outcomes of fluent control by their agents’ intentions. When an intention to omit fluently controls for the right omission, the omission is an intentional achievement performed because of the intention. The intention appropriately regulates the agent’s behavior so as to ensure the intended omission. We can therefore see basic agency over omissions as regulative and distinct from the agency exercised over actions. (shrink)

The emphasis on the organization of entities and their activities and interactions has been labeled one of the most distinct contributions of mechanistic philosophy. In this paper I discuss the manner in which the organization of entities and their activities and interactions participates in bringing about phenomena. I present a well-known example from molecular biology—the functioning of the genetic switch in phage lambda—and discuss Marco J. Nathan’s notion of causation by concentration. Nathan introduces causation by concentration to account for the (...) irreducible causal role that the concentration ratio between two kinds of proteins possesses in the genetic switch mechanism in phage lambda. I discuss what the irreducibility of this causal role amounts to and provide a mechanistic interpretation of Nathan’s causation by concentration; that is, I explain this irreducible causal role as one organizational feature of this mechanism. The paper concludes that biological mechanisms need a causal pluralist framework [similar to Glennan’s account in (2009), (2010) and (2017) but slightly modified] where organizational features such as the concentration ratio have a causally relevant role, yet all the causally productive relations occur at the level of entities or individuals. (shrink)

Mechanistic approaches are very common in the causal interpretation of biological and neuroscientific experimental work in today’s philosophy of science. In the mechanistic literature a strict distinction is often made between causal relations and constitutive relations, where the latter cannot be causal. One of the typical reasons for this strict distinction is that constitutive relations are supposedly synchronic whereas most if not all causal relations are diachronic. This strict distinction gives rise to a number of problems, however. Our end goal (...) in this paper is to argue that it should be given up, at least in the context of the biological and the psychological sciences. To that effect, we argue that constitutive relations in this context are diachronic, thus undermining the aforementioned reason. We offer two cases from scientific practice in which constitutive relations are regarded as both diachronic and causally efficacious, review three existing ways of dealing with the apparent diachronic nature of interlevel relations in mechanisms and propose a new account of diachronic, causal constitutive relevance. (shrink)

The concept of explanation is central to scientific practice. However, scientists explain phenomena in very different ways. That is, there are many different kinds of explanation; e.g. causal, mechanistic, statistical, or equilibrium explanations. In light of the myriad kinds of explanation identified in the literature, most philosophers of science have adopted some kind of explanatory pluralism. While pluralism about explanation seems plausible, it faces a dilemma Explanation beyond causation, Oxford University Press, Oxford, pp 39–56, 2018). Either there is nothing that (...) unifies all instances of scientific explanation that makes them count as explanations, or there is some set of unifying features, which seems incompatible with explanatory pluralism. Different philosophers have adopted different horns of this dilemma. Some argue that no unified account of explanation is possible. Others suggest that there is a set of necessary features that can unify all explanations under a single account Explanation beyond causation, Oxford University Press, Oxford, pp 74–95, 2018; Strevens in Depth: an account of scientific explanation, Harvard University Press, Cambridge, 2008). In this paper, we argue that none of the features identified by existing accounts of explanation are necessary for all explanations. However, we argue that a unified account can still be provided that accommodates pluralism. This can be accomplished, we argue, by reconceiving of scientific explanation as a cluster concept: there are multiple subsets of features that are sufficient for providing an explanation, but no single feature is necessary for all explanations. Reconceiving of explanation as a cluster concept not only accounts for the diversity of kinds of explanations, but also accounts for the widespread disagreement in the explanation literature and enables explanatory pluralism to avoid Pincock’s dilemma. (shrink)

Causation is important when considering: how an organism maintains health; why disease arises in a healthy person; and, how one may intervene to change the course of a disease. This paper explores the form of causative relationships in health, disease and intervention, with particular regard to the pathological and biopsychosocial models. Consistent with the philosophical view of dispositionalism, we believe that objects are the fundamental relata of causation. By accepting the broad scope of the biopsychosocial model, we argue that psychological (...) and social constructs be considered objects. We think that this ‘biopsychosocial dispositionalism’ offers the flexibility required to describe causation throughout health, disease and intervention pathways. When constructing mechanistic chains to describe causative pathways, we argue that an object will causally connect with others through actions; transfers of energy from one object to another, initiated by the manifestation of one or more dispositional property. Finally, our analysis of causative interactions utilises the concept that a common form of interaction exists between disease and intervention pathways. This common form will always be an object, but the mode of interaction will vary with each disease. We describe how intervention may act through objects being shared between converging mechanistic chains, or through the removal and/or insertion of objects in such chains. We believe that this analysis provides novel insight to the forms of causative transactions that can occur. In addition, we hope that the findings of this analysis represent the first step towards developing a framework for appraising the composition of mechanistic theories. (shrink)

In this paper, I argue that absences pose a challenge to our understanding of physicalism that has not been properly appreciated. I do this by setting out a thought experiment involving a being in whom absence properties occupy the causal roles that functionalists take to define mental properties, in which case these absence properties realize the being’s mental properties. Such a being should be compatible with the truth of physicalism, I argue, even though its mental properties are neither themselves physical (...) properties nor are they realized/grounded in physical properties—instead, they are realized/grounded in (nonphysical) absence properties. I argue that the case serves as a counterexample to leading formulations of physicalism that appeal to realization or grounding, but not to standard supervenience-based formulations of physicalism, because the latter typically include negative truths in their supervenience base in addition to physical truths. I show how realization- and grounding-based formulations of physicalism might be revised to handle the problem, but also recommend that we rethink how we understand physicalism and the role that negative entities can play for physicalists. (shrink)

As physical entities that translate symbols into physical actions, computers offer insights into the nature of meaning and agency. • Physical symbol systems, generically known as agents, link abstractions to material actions. The meaning of a symbol is defined as the physical actions an agent takes when the symbol is encountered. • An agent has autonomy when it has the power to select actions based on internal decision processes. Autonomy offers a partial escape from constraints imposed by direct physical influences (...) such as gravity and the transfer of momentum. Swimming upstream is an example. • Symbols are names that can designate other entities. It appears difficult to explain the use of names and symbols in terms of more primitive functionality. The ability to use names and symbols, i.e., symbol grounding, may be a fundamental cognitive building block. • The standard understanding of causality—wiggling X results in Y wiggling—applies to both physical causes (e.g., one billiard ball hitting another) and symbolic causes (e.g., a traffic light changing color). Because symbols are abstract, they cannot produce direct physical effects. For a symbol to be a cause requires that the affected entity determine its own response. This is called autonomous causality. • This analysis of meaning and autonomy offers new perspectives on free will. (shrink)

Since its inception, quantum mechanics has faced a series of “mysteries” that emerge from it if we consider this scientific theory from a realistic point of view. In the early development of the theory, scientists like Albert Einstein noticed the consequences of accepting a theory like this, which allow phenomena such as non-locality. This led a part of the scientific community to believe that quantum mechanics was an incomplete theory, since there should be variables that might explain those “disturbing” phenomena (...) such as the correlation between entangled particles.Different interpretations of quantum mechanics have been proposed that have tried to reveal or explain the reality underlying its formalism. One of those interpretations is the “Transactional Interpretation” of quantum mechanics, advocated mainly by physicist John G. Cramer. According to this interpretation, quantum events are understood as causal interactions between delayed waves traveling forward in time and advanced waves traveling backward in time. This interpretation opens the door to accept a model of causation to the past or retrocausation.Authors like Phil Dowe or Huw Price have shown a favorable attitude toward the model of retrocausation because it seems to be capable of effectively solve some of the most disturbing features derived from quantum mechanics. Despite the intuitive cost is rather high, retrocausation is one of the possible interpretations for the results of the correlation between entangled particles to provide us with an explanatory framework with some interesting advantages.In this paper we explore such advantages, as the retrocausation gives us a generalizable explanatory model for all cases of this type and assumes processes and entities that are not only in the field of speculation but allow certain possibilities of testing. Also, we will rescue the importance of not-predictive explanatory models as has been the case in other fields of natural sciences, provided that those model do not be ad hoc due to restrictions that prevent its application to any case. (shrink)

In this essay we show that some thesis -contended by some authors searching on physical causation- about causal pseudoprocesses, processes of which the most salient aspect seems to be travelling faster than the speed of light, must be rejected o seriously modified. Secondly, we will argue that there is just one non causal entities category and that special relativity does not provide any particular motivation to appeal to an ontological distinction based on identity over time. In addition, this conclusion we (...) will enable us to offer a more accurate definition of a pseudoprocess than that found in the relevant literature. In third place, we will show in some detail how such processes, understood the way that we suggest here, are and have been valuable methodological tools when studying –through usual measurement procedures- effective causal relations holding in the physical world. Finally, we will connect our conclusions with R. Torretti’s criticism on the relation between ordinary causation and governed-by-differential-equations physical processes and we will show that, although neutral on his anthropocentric background, the account here argued provides, against what Torretti claims, a natural explanation to the physical connection between ordinary causation and causal processes such as studied by physics. (shrink)

Recently some philosophers suggested an exclusion problem for moral non-naturalism, which is similar to the exclusion problem in philosophy of mind. In this article, the author aims to advance the discussion of exclusion in morality by investigating two influential solutions to the exclusion problem: the autonomy solution and the overdetermination solution. The author attempts to show that the moral non-naturalist can solve the exclusion problem in a way that is different from the approach to solving mental-physical exclusion. First, the author (...) argues that while the autonomy solution to mental exclusion may work, a similar autonomy approach to moral exclusion is implausible. Second, the author argues that whereas the overdetermination solution to mental exclusion fails, a similar overdetermination solution to moral exclusion is promising. (shrink)

The paper takes a detailed look at a surprising new aspect of the dynamics of rigid bodies. Far from the usual consideration of rigid body theory as a merely technical chapter of classical physics, I demonstrate here that there are solutions to the conservation equations of mechanics that imply the spontaneous, unpredictable splitting of a rigid body in free rotation, something that has direct implications for the problem of causality. The paper also shows that the instability revealed in indeterminist splitting (...) processes does not depend solely on the bodies’ inertial properties but also on the number of dimensions of the physical space they inhabit. The paper concludes with a conjecture on the behavior of rigid bodies in four-dimensional space. (shrink)

It is an ongoing controversy whether natural selection is a cause of population change, or a mere statistical description of how individual births and deaths accumulate. In this paper I restate the problem in terms of the reference class problem, and propose how the structure of stable equilibrium can provide a solution in continuity with biological practice. Insofar natural selection can be understood as a tendency towards equilibrium, key statisticalist criticisms are avoided. Further, in a modification of the Newtonian-force analogy, (...) it can be suggested that a better metaphor for natural selection is that of an emergent force, similar in nature to entropic forces: with magnitude and direction, but lacking a spatiotemporal origin or point of application. (shrink)

How do we determine whether some candidate causal factor is an actual cause of some particular outcome? Many philosophers have wanted a view of actual causation which fits with folk intuitions of actual causation and those who wish to depart from folk intuitions of actual causation are often charged with the task of providing a plausible account of just how and where the folk have gone wrong. In this paper, I provide a range of empirical evidence aimed at showing just (...) how and where the folk go wrong in determining whether an actual causal relation obtains. The evidence suggests that folk intuitions of actual causation are generated by two epistemically defective processes. I situate the empirical evidence within a background discussion of debunking, arguing for a two-pronged debunking explanation of folk intuitions of actual causation. I conclude that those who wish to depart from folk intuitions of actual causation should not be compelled to square their account of actual causation with the verdicts of the folk. In the dispute over actual causation, folk intuitions deserve to be rejected. (shrink)

This paper considers the applicability of standard accounts of causation to living systems. In particular it examines critically the increasing tendency to equate causal explanation with the identification of a mechanism. A range of differences between living systems and paradigm mechanisms are identified and discussed. While in principle it might be possible to accommodate an account of mechanism to these features, the attempt to do so risks reducing the idea of a mechanism to vacuity. It is proposed that the solution (...) to this problem requires the development of a philosophical account of process adequate to apply to living systems. (shrink)

The debate between the dynamical and the statistical interpretations of natural selection is centred on the question of whether all explanations that employ the concepts of natural selection and drift are reducible to causal explanations. The proponents of the statistical interpretation answer negatively, but insist on the fact that selection/drift arguments are explanatory. However, they remain unclear on where the explanatory power comes from. The proponents of the dynamical interpretation answer positively and try to reduce selection/drift arguments to some of (...) the most prominent accounts of causal explanation. In turn, they face the criticism raised by statisticalists that current accounts of causation have to be violated in some of their core conditions or otherwise used in a very loose manner in order to account for selection/drift explanations. We propose a reconciliation of both interpretations by conveying evolutionary explanations within the unificationist model of scientific explanation. Therefore, we argue that the explanatory power in natural selection arguments is a result of successful unification of individual- and population-level facts. A short case study based on research on sympatric speciation will be presented as an example of how population- and individual-level facts are unified to explain the morphological mosaic of bill shape in island scrub jays. (shrink)

A combination of process and counterfactual theories of causation is proposed with the aim of preserving the strengths of each of the approaches while avoiding their shortcomings. The basis for the combination, or hybrid, view is the need, common to both accounts, of imposing a stability requirement on the causal relation.

This paper poses an original puzzle about the relationship between causation and moral responsibility called The Moral Difference Puzzle. Using the puzzle, the paper argues for three related ideas: (1) the existence of a new sort of moral luck; (2) an intractable conflict between the causal concepts used in moral assessment; and (3) inability of leading theories of causation to capture the sorts of causal differences that matter for moral evaluation of agents’ causal contributions to outcomes.

Many philosophers now regard causal approaches to explanation as highly promising, even in physics. This is due in large part to James Woodward's influential argument that a wide variety of scientific explanations are causal, based on his interventionist approach to causation. This article argues that some derivations describing causal relations and satisfying Woodward's criteria for causal explanation fail to be explanatory. Further, causal relations are unnecessary for a range of explanations, widespread in physics, involving highly idealized models. These constitute significant (...) limitations on the scope of causal explanation. We have good reason to doubt that causal explanation is as widespread or important in physics as Woodward and other proponents maintain. (shrink)

In order to defend mental explanations dualists may appeal to dispositions (powers). By accepting a powers theory of causation, a dualist can more plausibly defend mental explanations that are given independently of physical explanations. Accepting a power-based theory still comes with a price. Absences and double preventers are not causes in a powers theory, and solutions based on them can only defend their explanatory relevance in mental explanations. There is still a chance that such mental explanations can be causal explanations, (...) though they do not refer to real causes. (shrink)

This paper makes the case for a wider acceptance of a probabilistic approach to causation in negligence. This acceptance would help to remove much of the incoherence which has come to afflict the English law of personal injury law. This incoherence can also be found in other common law jurisdictions (notably those of the United States, Canada and Australia). Concentrating upon recent UK case law, the argument opposes the contention that ‘naked statistics’ can play no role in establishing causation. The (...) argument is controversial but it can be reduced to three unremarkable grounds: (1) With its acceptance (albeit in certain carefully prescribed circumstances) of liability for a negligently increased risk which has eventuated, the common law has already embraced a probabilistic conception of causation; (2) The English common law already employs a probabilistic (frequentist) approach to identifying coincidences; and (3) With the ‘balance of probabilities’ as the standard of proof in civil cases, the common law has long had a probabilistic (epistemic) concept at its core. Probabilistic approaches (at both the type and token level) are shown to be consistent with laypersons’ understanding of the concepts such as risk, chance, odds and likelihood. Moreover, a wider acceptance of a probabilistic perspective on causation would entail no major challenge to the fundamental aims of tort, viz. deterrence and corrective justice. (shrink)

This paper aims to discuss two realist conceptions about causation in the light of the general theory of relativity. I first consider the conserved quantity of causation, which explicitly relies on the energy conservation principle. Such principle is however problematic within GTR, mainly because of the dynamical nature of the spacetime structure itself. I then turn to the causal theory of properties, according to which properties are such that insofar as they are certain qualities, they are powers to produce certain (...) effects. In order to be compatible with GTR, such theory has to assume non-trivial global conditions on the spacetime structure; such assumptions seem to deprive the „singularist‟ non-Humean feature of this theory of causation. The question of the possible causal nature of spacetime properties is addressed in the conclusion. (shrink)

An actual cause of some token effect is itself a token event that helped to bring about that effect. The notion of an actual cause is different from that of a potential cause – for example a pre-empted backup – which had the capacity to bring about the effect, but which wasn't in fact operative on the occasion in question. Sometimes actual causes are also distinguished from mere background conditions: as when we judge that the struck match was a cause (...) of the fire, while the presence of oxygen was merely part of the relevant background against which the struck match operated. Actual causation is also to be distinguished from type causation: actual causation holds between token events in a particular, concrete scenario; type causation, by contrast, holds between event kinds in scenario kinds. (shrink)

These preprints were automatically compiled into a PDF from the collection of papers deposited in PhilSci-Archive in conjunction with the PSA 2012.

In this thesis, I give a metascientific account of causality in medicine. I begin with two historical cases of causal discovery. These are the discovery of the causation of Burkitt’s lymphoma by the Epstein-Barr virus, and of the various viral causes suggested for cervical cancer. These historical cases then support a philosophical discussion of causality in medicine. This begins with an introduction to the Russo- Williamson thesis (RWT), and discussion of a range of counter-arguments against it. Despite these, I argue (...) that the RWT is historically workable, given a small number of modifications. I then expand Russo and Williamson’s account. I first develop their suggestion that causal relationships in medicine require some kind of evidence of mechanism. I begin with a number of accounts of mechanisms and produce a range of consensus features of them. I then develop this consensus position by reference to the two historical case studies with an eye to their operational competence. In particular, I suggest that it is mechanistic models and their representations which we are concerned with in medicine, rather than the mechanism as it exists in the world. -/- I then employ these mechanistic models to give an account of the sorts of evidence used in formulating and evaluating causal claims. Again, I use the two human viral oncogenesis cases to give this account. I characterise and distinguish evidence of mechanism from evidence of difference-making, and relate this to mechanistic models. I then suggest the relationship between types of evidence presents us with a means of tackling the reference-class problem. This sets the scene for the final chapter. Here, I suggest the manner in which these two different classes of evidence become integrated is also reflected in the way that developing research programmes change as their associated causal claims develop. (shrink)

This article focuses on the assessment of mechanistic relations with specific attention to medicine, where mechanistic models are widely employed. I first survey recent contributions in the philosophical literature on mechanistic causation, and then take issue with Federica Russo and Jon Williamson’s thesis that two types of evidence, probabilistic and mechanistic, are at stake in the health sciences. I argue instead that a distinction should be drawn between previously acquired knowledge of mechanisms and yet-to-be-discovered knowledge of mechanisms and that both (...) probabilistic evidence and manipulation are essential with respect to newly discovered mechanisms. (shrink)

I argue that four-dimensionalism and the desire satisfaction account of well-being are incompatible. For every person whose desires are satisfied, there will be many shorter-lived individuals (‘person-stages’ or ‘subpersons’) who share the person’s desires but who do not exist long enough to see those desires satisfied; not only this, but in many cases their desires are frustrated so that the desires of the beings in whom they are embedded as proper temporal parts may be fulfilled. I call this the frustrating (...) problem for four-dimensionalism. In the first half of the paper I lay the groundwork for understanding the frustrating problem, and then in the second half, I will examine six possible responses to the frustrating problem on behalf of the four-dimensionalist, (i) the Parfit (1984) inspired claim that identity is not what matters, (ii) the personal pronoun revisionism of Noonan (2010), (iii) the indirect concern account of Hudson (2001), (iv) the sensible stages account of Lewis (1986), (v) a multiple-concepts account of desire satisfaction, and (vi) a No Desire View according to which subpersons have no mental states and thus no desires to frustrate. I argue that none of these solutions will help the four-dimensionalist; she does better to reject the desire satisfaction theory, while the defender of the desire satisfaction theory does better to reject four-dimensionalism. (shrink)

In this paper I offer an anti-Humean critique to Williamson and Russo’s approach to medical mechanisms. I focus on one of the specific claims made by Williamson and Russo, namely the claim that micro-structural ‘mechanisms’ provide evidence for the stability across populations of causal relationships ascertained at the (macro-) level of (test) populations. This claim is grounded in the epistemic account of causality developed by Williamson, an account which—while not relying exclusively on mechanistic evidence for justifying causal judgements—appeals nevertheless to (...) mechanisms, and rejects their anti-Humean interpretation in terms of capacities, powers, potencies, etc. By using (and expanding on) Cartwright’s basic critique against Humean mechanisms, I suggest that, in order to move beyond the level of plausibility, Williamson and Russo’s position is in need of a clarification as to the occurent reading of the components, functioning and interferences of mechanisms. Relatedly, as concerns Williamson’s epistemic account of causation, I argue that this account is in need of a more straightforward answer as to what truth-makers its causal claims should have. (shrink)

Abstract Recent criticisms of intuition from experimental philosophy and elsewhere have helped undermine the authority of traditional conceptual analysis. As the product of more empirically informed philosophical methodology, this result is compelling and philosophically salutary. But the negative critiques rarely suggest a positive alternative. In particular, a normative account of concept determination—how concepts should be characterized—is strikingly absent from such work. Carnap's underappreciated theory of explication provides such a theory. Analyses of complex concepts in empirical sciences illustrates and supports this (...) claim, and counteracts the charge explication is only suitable for highly mathematical, axiomatic contexts. Explication is also defended against the influential criticism it is “philosophically unilluminating”. Content Type Journal Article Category Original paper in Philosophy of Science Pages 1-19 DOI 10.1007/s13194-011-0027-5 Authors James Justus, Philosophy Department, Florida State University and University of Sydney, Tallahassee, FL 32306, USA Journal European Journal for Philosophy of Science Online ISSN 1879-4920 Print ISSN 1879-4912. (shrink)

Conceptual analyses can be subdivided into two classes, good and evil. Em- pirical analysis is the good kind, routinely practiced in the sciences. Orthodox analysis is the malevolent version that plagues philosophical discourse. In this paper, I will clarify the difference between them, provide some reasons to prefer good over evil, and illustrate their consequences for the metaphysics of causation. By conducting an empirical analysis of causation rather than an orthodox analysis, one can segregate the genuine metaphysical problems that need (...) to be addressed from the many pseudo-problems that have long dogged traditional accounts of causation. (shrink)

How do people understand questions about cause and prevent? Some theories propose that people affirm that A causes B if A's occurrence makes a difference to B's occurrence in one way or another. Other theories propose that A causes B if some quantity or symbol gets passed in some way from A to B. The aim of our studies is to compare these theories' ability to explain judgements of causation and prevention. We describe six experiments that compare judgements for causal (...) paths that involve a mechanism, i.e. a continuous process of transmission or exchange from cause to effect, against paths that involve no mechanism yet a change in the cause nevertheless brings about a change in the effect. Our results show that people prefer to attribute cause when a mechanism links cause to effect. In contrast, prevention is sensitive both to the presence of an interruption to a causal mechanism and to a change in the outcome in the absence of a mechanism. In this sense, ‘prevent’ means something different than ‘cause not'. We discuss the implications of our results for existing theories of causation. (shrink)