In response to counterexamples involving late preemption, David Lewis (1986) revised his original (1973) counterfactual analysis of causation to include the notion of quasi-dependence. Jonardon Ganeri, Paul Noordhof and Murali Ramachandran (1998) argue that their ‘PCA*-analysis’ of causation solves the problem of late preemption and is superior to Lewis’s analysis. I show that neither quasi-dependence nor the PCA*-analysis solves the problem of late preemption.

Critics of contemporary metaphysics argue that it attempts to do the hard work of science from the ease of the armchair. Physics, not metaphysics, tells us about the fundamental facts of the world, and empirical psychology is best placed to reveal the content of our concepts about the world. Exploring and understanding the world through metaphysical reflection is obsolete. In this paper, I will show why this critique of metaphysics fails, arguing that metaphysical methods used to make claims about the (...) world are similar to scientific methods used to make claims about the world, but that the subjects of metaphysics are not the subjects of science. Those who argue that metaphysics uses a problematic methodology to make claims about subjects better covered by natural science get the situation exactly the wrong way around: metaphysics has a distinctive subject matter, not a distinctive methodology. The questions metaphysicians address are different from those of scientists, but the methods employed to develop and select theories are similar. In the first section of the paper, I will describe the sort of subject matter that metaphysics tends to engage with. In the second section of the paper, I will show how metaphysical theories are classes of models and discuss the roles of experience, common sense and thought experiments in the construction and evaluation of such models. Finally, in the last section I will discuss the way these methodological points help us to understand the metaphysical project. Getting the right account of the metaphysical method allows us to better understand the relationship between science and metaphysics, to explain why doing metaphysics successfully involves having a range of different theories, to understand the role of thought experiments involving fictional worlds, and to situate metaphysical realism in a scientifically realist context. (shrink)

Counterfactual analyses of causation can provide elegant analyses of many cases of causation. However, they fail to give intuitively correct analyses of cases involving a commonplace variety of late preemptive causation. I argue that a small emendation can solve the problem.

A theory of the causal relate as aspects or property instances is developed. A supposed problem for transitivity is assessed and then resolved with aspects as the causal relata.

A probability distribution is regular if it does not assign probability zero to any possible event. While some hold that probabilities should always be regular, three counter-arguments have been posed based on examples where, if regularity holds, then perfectly similar events must have different probabilities. Howson and Benci et al. have raised technical objections to these symmetry arguments, but we see here that their objections fail. Howson says that Williamson’s “isomorphic” events are not in fact isomorphic, but Howson is speaking (...) of set-theoretic representations of events in a probability model. While those sets are not isomorphic, Williamson’s physical events are, in the relevant sense. Benci et al. claim that all three arguments rest on a conflation of different models, but they do not. They are founded on the premise that similar events should have the same probability in the same model, or in one case, on the assumption that a single rotation-invariant distribution is possible. Having failed to refute the symmetry arguments on such technical grounds, one could deny their implicit premises, which is a heavy cost, or adopt varying degrees of instrumentalism or pluralism about regularity, but that would not serve the project of accurately modelling chances. (shrink)

According to the noncausal libertarian view of free will, in order for a person’s action to be free, it must be uncaused. A standard criticism of this view—the control objection—is that a person cannot have control over whether an uncaused action occurs and, so, such an action cannot be free. The background to this criticism is the claim that control over action is plausibly a causal rather than noncausal matter. In this paper, I defend noncausal libertarianism against the control objection (...) by developing a new noncausal theory of control. What emerges is not simply a defense of noncausal libertarianism against the control objection but a new theory of control more generally. (shrink)

If well-designed, the results of a Randomised Clinical Trial can justify a causal claim between treatment and effect in the study population; however, additional information might be needed to carry over this result to another population. RCTs have been criticized exactly on grounds of failing to provide this sort of information Evidence, inference and enquiry. Oxford University Press, New York, 2011), as well as to black-box important details regarding the mechanisms underpinning the causal law instantiated by the RCT result. On (...) the other side, so-called In Silico Clinical Trials face the same criticisms addressed against standard modelling and simulation techniques, and cannot be equated to experiments Philosophy of molecular medicine: foundational issues in research and practice, Routledge, New York, 2017; Parker in Synthese 169:483–496, 2009; Parke in Philos Sci 81:516–536, 2014; Diez Roux in Am J Epidemiol 181:100–102, 2015 and related discussions in Frigg and Reiss in Synthese 169:593–613, 2009; Winsberg in Synthese 169:575–592, 2009; Beisbart and Norton in Int Stud Philos Sci 26:403–422, 2012). We undertake a formal analysis of both methods in order to identify their distinct contribution to causal inference in the clinical setting. Britton et al.’s study :E2098–E2105, 2013) on the impact of ion current variability on cardiac electrophysiology is used for illustrative purposes. We deduce that, by predicting variability through interpolation, ISCTs aid with problems regarding extrapolation of RCTs results, and therefore in assessing their external validity. Furthermore, ISCTs can be said to encode “thick” causal knowledge —as opposed to “thin” difference-making information inferred from RCTs. Hence, ISCTs and RCTs cannot replace one another but rather, they are complementary in that the former provide information about the determinants of variability of causal effects, while the latter can, under certain conditions, establish causality in the first place. (shrink)

Traditionally, such a statement as ‘I was misled’ is intended to be taken seriously by the addressee and considered an expression of the true state of affairs. Unfortunately, the statement has many logical implications that often go unnoticed by both the one who utters it and the addressee. The paper critically explores the instrumentality of logical and epistemological analyses in its attempt to lay bare the implications of the statement being true and of entertaining the underlying belief in the first (...) place that ‘I was misled’. By so doing, the paper attempts to show that even if the function of philosophy is merely to analyse language, it would still have contributed indispensably and significantly to human society by clarifying ambiguous concepts. It also attempts to demonstrate the relevance of critical thinking in human society. (shrink)

A prominent Thomistic cosmological argument maintains that an infinite regress of causes, which exhibits a certain pattern of ontological dependence among its members, would be vicious and so must terminate in a first member. Interestingly, Jonathan Schaffer offers a similar argument in the contemporary grounding literature for the view called metaphysical foundationalism. I consider the striking similarities between both arguments and conclude that both are unsuccessful for the same reason. I argue this negative result gives us indirect reason to consider (...) metaphysical infinitism as a genuine possibility, the view that chains of ontological dependence or ground can descend indefinitely. (shrink)

, whereby some causes are deemed more important than others, are ubiquitous in historical studies. Drawing from influential recent work on causation, I develop a definition of causal-explanatory strength. This makes clear exactly which aspects of explanatory weighting are subjective and which objective. It also sheds new light on several traditional issues, showing for instance that: underlying causes need not be more important than proximate ones; several different causes can each be responsible for most of an effect; small causes need (...) not be less important than big ones; and non-additive interactive effects between causes present no particular difficulty. Key Words: causation • explanation • history • interaction • proximate • underlying. (shrink)

Pre-emption cases have been taken by almost everyone to imply the unviability of the simple counterfactual theory of causation. Yet there is ample motivation from scientific practice to endorse a simple version of the theory if we can. There is a way in which a simple counterfactual theory, at least if understood contrastively, can be supported even while acknowledging that intuition goes firmly against it in pre-emption cases—or rather, only in some of those cases. For I present several new pre-emption (...) cases in which causal intuition does not go against the counterfactual theory, a fact that has been verified experimentally. I suggest an account of framing effects that can square the circle. Crucially, this account offers hope of theoretical salvation—but only to the counterfactual theory of causation, not to others. Again, there is (admittedly only preliminary) experimental support for this account. (shrink)

The causal impacts of genes and environment on any one biological trait are inextricably entangled, and consequently it is widely accepted that it makes no sense in singleton cases to privilege either factor for particular credit. On the other hand, at a population level it may well be the case that one of the factors is responsible for more variation than the other. Standard methodological practice in biology uses the statistical technique of analysis of variance to measure this latter kind (...) of causal efficacy. In this paper, I argue that: 1) analysis of variance is in fact badly suited to this role; and. (shrink)

I argue that causation is a contrastive relation: c-rather-than-C* causes e-rather-than-E*, where C* and E* are contrast classes associated respectively with actual events c and e. I explain why this is an improvement on the traditional binary view, and develop a detailed definition. It turns out that causation is only well defined in ‘uniform’ cases, where either all or none of the members of C* are related appropriately to members of E*.

The last several decades of research on counterfactual conditionals in the fields of philosophy and linguistics have yielded a predominant paradigm according to which the notion of similarity plays the starring role. Roughly, a counterfactual of the form A > C is true iff the closest A-worlds are all C-worlds, where the closeness of a world is a function of its similarity, in a certain sense, to the actual world. I argue that this is deeply misguided. In some cases we (...) may only care about the closest A-worlds, but quite often we care about some broader variety of A-worlds varying in closeness. After presenting several problem cases for the similarity-based paradigm, some new and some known, I propose an alternative paradigm for the semantics of counterfactuals, which introduces the notion of A-scenarios, understood roughly as different ways of making A true. According to this view, A > C is true iff all the contextually relevant ways of making A true also make C true. I then more carefully spell out a working view that formalizes this idea with greater precision, and explore the consequences of that version of the view. (shrink)

Can we hold citizens causally responsible for the outcomes of their voting decisions? They could stand in the causal relationship required for such responsibility either collectively or individually. Recent accounts ascribing responsibility to citizens have primarily taken the collective route because of a major obstacle to using an individualistic approach, namely, the problem of overdetermination: the actions of each citizen do not make an individual difference to, and therefore cannot be a cause of, the overall political outcome. I suggest, drawing (...) on Parfit (1984) and Wright (1985), that we should allow for the idea that individuals can be causally responsible in virtue of making a difference to an outcome not only as an individual, but also as part of a set of agents. I conclude that we therefore can overcome the problem of overdetermination for the individualistic approach and that it merits further investigation. (shrink)

This paper examines the relationship between physical theories of causation and theories of difference-making. It is plausible to think that such theories are compatible with one another as they are aimed at different targets: the former, an empirical account of actual causal relations; the latter, an account that will capture the truth of most of our ordinary causal claims. The question then becomes: what is the relationship between physical causation and difference-making? Is one kind of causal fact more fundamental than (...) the other? This paper defends causal foundationalism: the view that facts about difference-making are dependent on the obtaining of facts about physical causation. However, the paper's main goal is to clarify the structure of the debate. At the end of the paper, it is shown how settling the issue about the relationship between physical theories of causation and theories of difference-making has more than mere intrinsic interest in unifying the very different pursuits that have been undertaken in the philosophy of causation. It can help to break a stalemate that has arisen in the current debate about mental causation. (shrink)

In this paper, I provide an account of epistemic anxiety as an emotional response to epistemic risk: the risk of believing in error. The motivation for this account is threefold. First, it makes epistemic anxiety a species of anxiety, thus rendering psychologically respectable a notion that has heretofore been taken seriously only by epistemologists. Second, it illuminates the relationship between anxiety and risk. It is standard in psychology to conceive of anxiety as a response to risk, but psychologists – very (...) reasonably – have little to say about risk itself, as opposed to risk judgement. In this paper, I specify what risk must be like to be the kind of thing to which anxiety can be a response. Third, my account improves on extant accounts of epistemic anxiety in the literature. It is more fleshed out than Jennifer Nagel’s, which is largely agnostic about the nature of epistemic anxiety, focusing instead on what work it does in our epistemic lives. In offering an account of epistemic anxiety as an emotion, my account explains how it is able to do the epistemological work to which Nagel puts it. My account is also more plausible than Juliette Vazard’s, on which epistemic anxiety is an emotional response to potential threat to one’s practical interests. Vazard’s account cannot distinguish epistemic anxiety from anxiety in general, and also fails to capture all instances of what we want to call epistemic anxiety. My account does better on both counts. (shrink)

This chapter contains sections titled: Highlights of Past Literature Current Work Future Work.

It has long been thought that Augustine holds that corporeal objects cannot act upon incorporeal souls. However, precisely how and why Augustine imposes limitations upon the causal powers of corporeal objects remains obscure. In this paper, the author clarifies Augustine’s views about the causal and dependence relations between body and soul. He argues that, contrary to what is often thought, Augustine allows that corporeal objects do act upon souls and merely rules out that corporeal objects exercise a particular kind of (...) causal power (that of efficient or sustaining causes). He clarifies how Augustine conceives of the kind of causal influence exercised by souls and bodies. (shrink)

This essay is concerned with concentrations of entities, which play an important—albeit often overlooked—role in scientific explanation. First, I discuss an example from molecular biology to show that concentrations can play an irreducible causal role. Second, I provide a preliminary philosophical analysis of this causal role, suggesting some implications for extant theories of causation. I conclude by introducing the concept of causation by concentration, a form of statistical causation whose widespread presence throughout the sciences has been unduly neglected and which (...) deserves to be studied in more depth. 1 Introduction2 Solving Lillie's Paradox: Lysogenic Induction in Phage λ3 Repressor Concentration and the Tuning of the Switch4 Concentration and Causality5 Preemption in Concentrations: Analysis and Implications6 Causation by Concentration: General Definition, Refinements, and Further Applications. (shrink)

According to recent arguments for panpsychism, all physical properties are dispositional, dispositions require categorical grounds, and the only categorical properties we know are phenomenal properties. Therefore, phenomenal properties can be posited as the categorical grounds of all physical properties—in order to solve the mind–body problem and/or in order avoid noumenalism about the grounds of the physical world. One challenge to this case comes from dispositionalism, which agrees that all physical properties are dispositional, but denies that dispositions require categorical grounds. In (...) this paper, I propose that this challenge can be met by the claim that the only dispositional properties we know are phenomenal properties, in particular, phenomenal properties associated with agency, intention and/or motivation. Versions of this claim have been common in the history of philosophy, and have also been supported by a number of contemporary dispositionalists. I will defend a new and updated version of it. Combined with other premises from the original case for panpsychism—which are not affected by the challenge from dispositionalism—it forms an argument that dispositionalism entails panpsychism. (shrink)

Can we explain why some propositions are necessary? Blackburn (Fact, science, and value. Blackwell, Oxford, 1987) has presented a dilemma aimed at showing that the necessity of a proposition cannot be explained either in the case where the explanans is another necessary proposition (necessity horn) or in the case where the explanans is a contingent proposition (contingency horn). Blackburn’s dilemma is intended to show that necessary truth is an explanatorily irreducible kind of truth: there is nothing that explains why propositions (...) are necessary, nothing that makes necessary necessary truths. In this paper, I criticize the contingency horn of Blackburn’s dilemma. On the one hand, I show that the official reconstruction of the horn uses a principle that is incompatible with the notion of explanation plausibly needed to explain why propositions are necessary; on the other, I show that a simpler formulation of the horn, which does not make use of such a controversial principle, makes essential use of principles that are incompatible with the idea that possibilities can have explanatory roles. I then defend the view that possibilities can have explanatory roles, and that the explanatory role of possibilities is best represented, within possible worlds, as the existence of trans-world relations of explanation. (shrink)

According to proponents of the causal exclusion problem, there cannot be a sufficient physical cause and a distinct mental cause of the same piece of behaviour. Increasingly, the causal exclusion problem is circumvented via this compatibilist reasoning: a sufficient physical cause of the behavioural effect necessitates the mental cause of the behavioural effect, so the effect has a sufficient physical cause and a mental cause as well. In this paper, I argue that this compatibilist reply fails to resolve the causal (...) exclusion problem. (shrink)

In recent papers, Lei Zhong argues that the autonomy solution to the causal exclusion problem is unavailable to anyone that endorses the counterfactual model of causation. The linchpin of his argument is that the counterfactual theory entails the downward causation principle, which conflicts with the autonomy solution. In this note I argue that the counterfactual theory does not entail the downward causation principle, so it is possible to advocate for the autonomy solution to the causal exclusion problem from within the (...) counterfactual theory of causation. (shrink)

It is argued that, given certain reasonable premises, an infinite number of qualitatively identical but numerically distinct minds exist per functioning brain. The three main premises are (1) mental properties supervene on brain properties; (2) the universe is composed of particles with nonzero extension; and (3) each particle is composed of continuum-many point-sized bits of particle-stuff, and these points of particle-stuff persist through time.

Recent discussions in the philosophy of biology have brought into question some fundamental assumptions regarding evolutionary processes, natural selection in particular. Some authors argue that natural selection is nothing but a population-level, statistical consequence of lower-level events (Matthen and Ariew [2002]; Walsh et al. [2002]). On this view, natural selection itself does not involve forces. Other authors reject this purely statistical, population-level account for an individual-level, causal account of natural selection (Bouchard and Rosenberg [2004]). I argue that each of these (...) positions is right in one way, but wrong in another; natural selection indeed takes place at the level of populations, but it is a causal process nonetheless. (shrink)

Causal analysis in the social sciences takes advantage of a variety of methods and of a multi-fold source of information and evidence. This pluralistic methodology and source of information raises the question of whether we should accordingly have a pluralistic metaphysics and epistemology. This paper focuses on epistemology and argues that a pluralistic methodology and evidence don’t entail a pluralistic epistemology. It will be shown that causal models employ a single rationale of testing, based on the notion of variation. Further, (...) I shall argue that this monistic epistemology is also involved in alternative philosophical theories of causation. (shrink)

With the emergence of modern physics a conflict became apparent between the Principle of Sufficient Cause and the Principle of Physical Causal Closure. Though these principles are not logically incompatible, they could no longer be considered to be both true; one of them had to be false. The present paper makes use of this seldom noticed conflict to argue on the basis of considerations of comparative rationality for the truth of causal statements that have at least some degree of philosophico-theological (...) relevance and can be taken to indicate ( not prove) the existence of God. The paper’s comparatively modest aim is to establish belief in the existence of God as a rational metaphysical option, not as a rational obligation. In its final section, enriched causal considerations lead to an indication ( not proof) of God as that which guarantees the unified continuance of the physical world. (shrink)

It is commonly assumed that causation is transitive and in this paper I aim to reconcile this widely-held assumption with apparent evidence to the contrary. I will discuss a familiar approach to certain well-known counterexamples, before introducing a more resistant sort of case of my own. I will then offer a novel solution, based on Yablo’s proportionality principle, that succeeds in even these more resistant cases. There is a catch, however. Either proportionality is a constraint on which causal claims are (...) true, and the solution works, or it is not and causation is not transitive after all. I will argue that the first horn has unacceptable consequences and should be rejected, but that the second horn is less costly than it might initially appear. (shrink)

This paper argues that a counterpart-theoretic treatment of events, combined with a counterfactual theory of causation, can help resolve three puzzles from the causation literature. First, CCT traces the apparent contextual shifts in our causal attributions to shifts in the counterpart relation which obtains in those contexts. Second, being sensitive to shifts in the counterpart relation can help diagnose what goes wrong in certain prominent examples where the transitivity of causation appears to fail. Third, CCT can help us resurrect the (...) much-maligned fragility response to the problems of late pre-emption by understanding fragility in counterpart-theoretic terms. Some reasons to prefer this CCT approach to rivals are discussed. (shrink)

Causal exclusion arguments are taken to threaten the autonomy of the special sciences, and the causal efficacy of mental properties. A recent line of response to these arguments has appealed to “independently plausible” and “well grounded” theories of causation to rebut key premises. In this paper I consider two papers which proceed in this vein and show that they share a common feature: they both require causes to be proportional to their effects. I argue that this feature is a bug, (...) and one that generalises: any attempt to rescue the autonomy of the special sciences, or the efficacy of the mental, from exclusion worries had better not look to proportionality for help. (shrink)

This paper considers the meaning and use of the English particle man . It is shown that the particle does quite different things when it appears in sentence-initial and sentence-final position; the first use involves expression of an emotional attitude as well as, on a particular intonation, intensification; this use is analyzed using a semantics for degree predicates along with a separate dimension for the expressive aspect. Further restrictions on modification with the sentence-initial particle involving monotonicity and evidence are introduced (...) and analyzed. The sentence-final use can be viewed as strengthening the action performed by the sentence. A formal semantics is given by making use of dynamic techniques and, in a sense, dynamically simulating the modification of certain speech acts. Some empirical and theoretical extensions of the analyses are proposed and some consequences discussed. (shrink)

This paper considers the meaning and use of the English particle man. It is shown that the particle does quite different things when it appears in sentence-initial and sentence-final position; the first use involves expression of an emotional attitude as well as, on a particular intonation, intensification; this use is analyzed using a semantics for degree predicates along with a separate dimension for the expressive aspect. Further restrictions on modification with the sentence-initial particle involving monotonicity and evidence are introduced and (...) analyzed. The sentence-final use can be viewed as strengthening the action performed by the sentence. A formal semantics is given by making use of dynamic techniques and, in a sense, dynamically simulating the modification of certain speech acts. Some empirical and theoretical extensions of the analyses are proposed and some consequences discussed. (shrink)

This paper offers a philosophical analysis of the connection between mental disorder and suicide risk. In contemporary psychiatry, it is commonly suggested that this connection is a causal connection that has been established through empirical discovery. Herein, I examine the extent to which this claim can be sustained. I argue that the connection between mental disorder and increased suicide risk is not wholly causal but is partly conceptual. This in part relates to the way suicidality is built into the definitions (...) of some psychiatric diagnoses. It also relates to the broader normative assumption that suicidal behavior is by definition mentally disordered behavior. The above has significant epistemological implications, which I explore. I propose that the claim that suicide is connected with mental disorder cannot be justified solely by appealing to empirical evidence but also warrants a justification on conceptual and normative grounds. (shrink)

It has been claimed that certain forms of individual essentialism render the Theory of Natural Selection unable to explain why any given individual has the traits it does. Here, three reasons are offered why the Theory ought to ignore these forms of essentialism. First, the trait-distributions explained by population genetics supervene on individual-level causal links, and thus selection must have individual-level effects. Second, even if there are individuals that possess thick essences, they lie outside the domain of the Theory. Finally, (...) the contingency of sexual reproduction suggests that essentialism is misguided in this arena. 1 The problem 2 A reprise of the controversy 3 Enter individual essences 4 How can selection not have individual-level effects? 5 Why can't we get rid of essences we don't like? 6 Is sex necessary? (shrink)

In this paper, I examine recent arguments for the widespread existence of simultaneous causation from Huemer & Kovitz and Mumford & Anjum, and conclude that they are mistaken. I argue that these arguments overlook two pictures of causation which are commonly assumed, which I call the Standard Modern Picture and the Contiguous Extended Picture.

In this paper I examine a recent argument that regularity approaches to causation can easily solve the problem of pre-emption. If this argument were successful it would neatly solve the problem of pre-emption—a problem that many still consider to be a central unsolved problem for accounts of causation. The argument is surprising in that the conclusion goes against the common consensus that regularity accounts of causation cannot solve the problem of pre-emption, at least without major amendments. This consensus was one (...) of the reasons for the decline in popularity of the regularity approach and the rise in popularity (for a few decades at least) of the counterfactual approach. In its fullest form the recent argument is due to Strevens (Causation and explanation, topics in contemporary philosophy. MIT Press, Cambridge, MA, 2007). He claims, "Mackie's account supplies, without any of the complex amendments now standard in counterfactual theories, a completely satisfactory treatment of the standard cases of pre-emption". This paper examines this argument and refutes it. I argue that Mackie's account really does fail to solve the problem of pre-emption; it fails to account for even the standard cases of pre-emption in the literature. (shrink)

In this paper, I defend a counterfactual approach to causation by absences from some recent criticisms due to Sartorio.

This paper presents ConR (Conditional R), a logic of conditionals based on Anderson and Belnap''s system R. A Routley-Meyer-style semantics for ConR is given for the system (the completeness of ConR over this semantics is proved in E. Mares and A. Fuhrmann, A Relevant Theory of Conditionals (unpublished MS)). Moreover, it is argued that adopting a relevant theory of conditionals will improve certain theories that utilize conditionals, i.e. Lewis'' theory of causation, Lewis'' dyadic deontic logic, and Chellas'' dyadic deontic logic.

I argue that time travel to the past is impossible, given a certain metaphysical theory, namely, The Dynamic Theory of Time. I first spell out my particular way of capturing the difference between The Dynamic Theory of Time and its rival, The Static Theory of Time. Next I offer four different arguments for the conclusion that The Dynamic Theory is inconsistent with the possibility of time travel to the past. Then I argue that, even if I am wrong about this, (...) it will still be true that The Dynamic Theory entails that you should not want to travel back to the past. Finally, I conclude by considering a puzzle that arises for those who believe that time travel to the past is metaphysically impossible: What exactly are we thinking about when we seem to be thinking about traveling back in time? For it certainly does not feel like we are thinking about something that is metaphysically impossible. (shrink)

An important divide in the free will literature—one that is arguably almost as common as the distinction between compatibilism and incompatibilism—concerns the distinction between event and substance causation. As the story typically goes, event-causalists maintain that an action is free only if it is caused by appropriate mental events, and agent-causalists maintain that an action is free only if it is caused directly by a substance (the agent). This paper argues that this dichotomy is a false one. It does this (...) by introducing a new view called Causal Pluralism, which maintains that free will is compatible with both event and substance causation. Furthermore, it is argued that agent-causalists have good reason to adopt Causal Pluralism, and also that that the view has interesting implications for the free will dialectic. (shrink)

According to the modal theory of facts and states of affairs, two facts or states of affairs are identical iff they are necessarily equivalent. One important argument against the modal theory is the causal argument of John Perry, which can also be applied with equal strength to a number of more moderate-grain theories of facts and states of affairs. I argue that, at least in its original form, the causal argument is unsound. I also argue that, while the argument can (...) be modified so that it avoids the problems of the original version of the argument, such modifications are at best only successful if they appeal to additional considerations involving either grounding or aboutness. Moreover, I argue that incorporating such considerations into the causal argument allows it to refute its targeted theories only if such considerations by themselves refute these theories. If this is correct, then the causal argument is at best superfluous and we should focus on these other considerations when evaluating these theories. The broader lesson from this is arguably that, in order to best evaluate these theories of facts and states of affairs, we shouldn’t focus on arguments involving causation, but we should instead focus on arguments that involve other notions, such as grounding and aboutness. (shrink)

In a recent paper I argued that agent causation theorists should be compatibilists. In this paper, I argue that compatibilists should be agent causation theorists. I consider six of the main problems facing compatibilism: (i) the powerful intuition that one can't be responsible for actions that were somehow determined before one was born; (ii) Peter van Inwagen's modal argument, involving the inference rule (β); (iii) the objection to compatibilism that is based on claiming that the ability to do otherwise is (...) a necessary condition for freedom; (iv) "manipulation arguments," involving cases in which an agent is manipulated by some powerful being into doing something that he or she would not normally do, but in such a way that the compatibilist's favorite conditions for a free action are satisfied; (v) the problem of constitutive luck; and (vi) the claim that it is not fair to blame someone for an action if that person was determined by forces outside of his or her control to perform that action. And in the case of each of these problems, I argue that the compatibilist has a much more plausible response to that problem if she endorses the theory of agent causation than she does otherwise. (shrink)

Causation is generally conceived of as a relation that holds between events. Apart from a few cursory remarks, the case of stative causation has been widely neglected. The paper aims at contributing to a more balanced perspective by arguing for a stative variant of causation, on a par with eventive causation. The stative variant is analyzed in terms of Moltmann’s ontological notion of tropes. German causal von-modifiers are taken as a linguistic window into our understanding of causation. The study of (...) von is particularly suited to this endeavor, because von-modifiers are confined to expressing the core notion of “direct causation” :1–48, 2003). The paper develops a compositional semantics of causal von-modifiers that derives their eventive and stative readings from a single lexical entry and allows for coercive adaptions to account for the observed range of interpretive adjustments. Characteristic features of the interpretation such as the inferential behavior of causal von-modifiers and the holistic effect of the stative reading are traced back to independently motivated conceptual assumptions concerning the spatiotemporal grounding of direct causation. The formal analysis is couched in terms of Asher’s type composition logic. (shrink)