In this paper, I draw on a recent account of perceptual knowledge according to which knowledge is contrastive. I extend the contrastive account of perceptual knowledge to yield a contrastive account of self-knowledge. Along the way, I develop a contrastive account of the propositional attitudes (beliefs, desires, regrets and so on) and suggest that a contrastive account of the propositional attitudes implies an anti-individualist account of propositional attitude concepts (the concepts of belief, desire, regret, and so on).

Jonathan Schaffer introduced a new type of causal structure called 'trumping'. According to Schaffer, trumping is a species of causal preemption. Both Schaffer and I have argued that causation has a contrastive structure. In this paper, I analyze the structure of trumping cases from the perspective of contrastive causation, and argue that the case is much more complex than it first appears. Nonetheless, there is little reason to regard trumping as a species of causal preemption.

I present and develop the view that omissions are de re possibilities of actual events. Omissions do not literally fail to occur; rather, they possibly occur. An omission is a tripartite metaphysical entity composed of an actual event, a possible event, and a contextually specified counterpart relation between them. This view resolves ontological, causal, and semantic puzzles about omissions, and also accounts for important data about moral responsibility for outcomes resulting from omissions.

In this paper, I discuss Michael Moore’s and Jonathan Schaffer’s views on the ontology of omissions in context of their stances on the problem of omissive causation. First, I consider, from a general point of view, the question of the ontology of omissions, and how it relates to the problem of omissive causation. Then I describe Moore’s and Schaffer’s particular views on omissions and how they combine with their stances on the problem of omissive causation. I charge Moore and Schaffer (...) with inconsistencies and insufficiencies within their overall theories, and consider their replies. Finally, I propose my own view on the ontology of omissions and solution to the problem of omissive causation. (shrink)

This article compares causal and constitutive explanation. While scientific inquiry usually addresses both causal and constitutive questions, making the distinction is crucial for a detailed understanding of scientific questions and their interrelations. These explanations have different kinds of explananda and they track different sorts of dependencies. Constitutive explanations do not address events or behaviors, but causal capacities. While there are some interesting relations between building and causal manipulation, causation and constitution are not to be confused. Constitution is a synchronous and (...) asymmetric relation between relata that cannot be conceived as independent existences. However, despite their metaphysical differences, the same key ideas about explanation largely apply to both. Causal and constitutive explanations face similar challenges (such as the problems of relevance and explanatory regress) and both are in the business of mapping networks of counterfactual dependence—i.e. mechanisms—although the relevant counterfactuals are of a different sort. In the final section the issue of developmental explanation is discussed. It is argued that developmental explanations deserve their own place in taxonomy of explanations, although ultimately developmental dependencies can be analyzed as combinations of causal and constitutive dependencies. Hence, causal and constitutive explanation are distinct, but not always completely separate forms of explanation. (shrink)

Recent years have seen renewed interest in the emergence issue. The contemporary debate, in contrast with that of past times, has to do not so much with the mind–body problem as with the relationship between the physical and other domains; mostly with the biological domain. One of the main sources of this renewed interest is the study of complex and, in general, far-from-equilibrium self-preserving systems, which seem to fulfil one of the necessary conditions for an entity to be emergent; namely, (...) that its causal powers are not predictable from the causal powers of basic physical properties. However, I argue that much of the current emergentism debate has misfired by focusing on the interpretation of self-maintaining systems. In contrast, I claim that if we want to find emergent properties, we should look not at complex systems, but at selection (natural selection, in particular). I argue that selection processes make the causal world ‘exuberant’ by making non-physical functional and relational properties enter the causal web of the world. (shrink)

We often explain by citing an absence or an omission. Apart from the problem of assigning a causal role to such apparently negative factors as absences and omissions, there is a puzzle as to why only some absences and omissions, out of indefinitely many, should figure in explanations. In this paper we solve this ’many absences problem’ by using the contrastive model of explanation. The contrastive model of explanation is developed by adapting Peter Lipton’s account. What initially appears to be (...) only a trivial amendment to Lipton’s Difference Condition enables us both to offer a much more satisfactory solution to the ’many absences problem’ than David Lewis did, and also to explain why explanation in terms of absences and omissions should be so common. (shrink)

I focus on token, deterministic causal claims as they feature in causal explanations. Adequately handling absences is difficult for most causal theories, including theories of causal explanation. Yet so is adequately handling cases of late preemption. The best account of absence-causal claims as they appear in causal explanations is Jonathan Schaffer's quaternary, contrastive account. Yet Schaffer's account cannot handle preemption. The account that best handles late preemption is James Woodward's interventionist account. Yet Woodward's account is inadequate when it comes to (...) absences. I propose an account that handles both absences and preemption by transposing Schaffer's account into an interventionist framework. (shrink)

The claim of this paper is that the everyday functions of knowledge make most sense if we see knowledge as contrastive. That is, we can best understand how the concept does what it does by thinking in terms of a relation “a knows that p rather than q.” There is always a contrast with an alternative. Contrastive interpretations of knowledge, and objections to them, have become fairly common in recent philosophy. The version defended here is fairly mild in that there (...) is no suggestion that we cannot think in terms of a simpler not explicitly contrastive relation “a knows that p.” Some, for instance Schaffer (2005) and Karjalainen and Morton (2003), have hinted that this stronger possibility may be right. But all that I am arguing now is that facts that are easily expressed in contrastive terms are vital to understanding why we need the concept of knowledge. In a piece that is in some ways a companion to this one ("Contrastivism" in Duncan Pritchard and Sven Bernecker, eds. The Routledge Companion to Epistemology. Routledge 2010, 513-522), I give a general survey of theories of contrastive knowledge and the differences between them. (shrink)

If counterfactual dependence is sufficient for causation and if omissions can be causes, then all events have many more causes than common sense tends to recognize. This problem is standardly addressed by appeal to pragmatics. However, Carolina Sartorio [2010] has recently raised what I shall argue is a more interesting problem concerning omissions for counterfactual theories of causation—more interesting because it demands a more subtle pragmatic solution. I discuss the relationship between the idea that causes are proportional to their effects, (...) the idea that causation is contrastive, and the question of the dimensions along which causal explanations should be evaluated with respect to one another. (shrink)

Instances of negative causation—preventions, omissions, and the like—have long created philosophical worries. In this paper, I argue that concerns about negative causation can be addressed in the context of causal explanation generally, and mechanistic explanation specifically. The gravest concern about negative causation is that it exacerbates the problem of causal promiscuity—that is, the problem that arises when a particular account of causation identifies too many causes for a particular effect. In the explanatory context, the problem of promiscuity can be solved (...) by characterizing the phenomenon to be explained as a contrast between two or more events or non-events. This contrastive strategy also can solve other problems that negative causation presents for the leading accounts of mechanistic explanation. Along the way, I argue that to be effective, accounts of causal explanation must incorporate negative causation. I also develop a taxonomy of negative causation and incorporate each variety of negative causation into the leading accounts of mechanistic explanation. (shrink)

This paper explains what it is to believe something for a reason. My thesis is that you believe something for a reason just in case the reason non-deviantly causes your belief. In the course of arguing for my thesis, I present a new argument that reasons are causes, and offer an informative account of causal non-deviance.

A critical survey of the main philosophical theories about events and event talk, organized in three main sections: (i) Events and Other Categories (Events vs. Objects; Events vs. Facts; Events vs. Properties; Events vs. Times); (ii) Types of Events (Activities, Accomplishments, Achievements, and States; Static and Dynamic Events; Actions and Bodily Movements; Mental and Physical Events; Negative Events); (iii) Existence, Identity, and Indeterminacy.

Nineteenth-century medical advances were entwined with a conceptual innovation: the idea that many cases of disease which were previously thought to have diverse causes could be explained by the action of a single kind of cause, for example a certain bacterial or parasitic infestation. The focus of modern epidemiology, however, is on chronic non-communicable diseases, which frequently do not seem to be attributable to any single causal factor. This paper is an effort to resolve the resulting tension. The paper criticises (...) the monocausal model of disease, so successful in the nineteenth century. It also argues that a multifactorial model of disease can only be satisfactory if it amounts to more than a mere rejection of the monocausal model. A third alternative, the contrastive model, is proposed and defended on the grounds that it links the notions of disease and of general explanation, while avoiding the philosophical naiveties and practical difficulties of the monocausal model. (shrink)

Denis Walsh has written a striking new defense in this journal of the statisticalist (i.e., noncausalist) position regarding the forces of evolution. I defend the causalist view against his new objections. I argue that the heart of the issue lies in the nature of nonadditive causation. Detailed consideration of that turns out to defuse Walsh’s ‘description‐dependence’ critique of causalism. Nevertheless, the critique does suggest a basis for reconciliation between the two competing views. *Received December 2009; revised December 2009. †To contact (...) the author, please write to: Department of Philosophy, 599 Lucas Hall, One University Boulevard, University of Missouri, St. Louis, MO 63121; e‐mail: [email protected]. (shrink)

What conception of causation is at work in the law? I argue that the law implicitly relies on a contrastive conception. In a liability case where the defendant's breach of duty must be shown to have caused the plaintiff's damages, it is not enough to consider what would have happened if the cause had not occurredthe law requires us to look to a specific replacement for the effect, which in this case is the hypothetical outcome in which the plaintiff came (...) off better. In place of I suggest the more explicit An explicitly contrastive approach can thus potentially help the lawyer phrase her causal question in a more explicit way, while shedding light on our conception of causation. (shrink)

An argument proposed by Steglich-Petersen (2012) establishes that while contrastive causation can be applied to general causation and causal explanation, it is a mistake to consider it in cases of singular causation. I attempt to show that there is no mistake. Steglich-Petersen’s argument does not seem to be strong enough and is actually circular. Furthermore, I briefly argue that even if we take his argument to be valid, there is still a response from the side of contrastive causation.

If a person's head moves, she may or may not have moved her head, and, if she did move it, she may have actively performed the movement of her head or merely, by doing something else, caused a passive movement. And, if she performed the movement, she might have done so intentionally or not. This short array of contrasts (and others like them) has motivated questions about the nature, variety, and identity of action. Beyond the matter of her moving, when (...) the person moves her head, she may be indicating agreement or shaking an insect off her ear. Should we think of the consequences, conventional or causal, of physical behavior as constituents of an action distinct from but ‘generated by’ the movement? Or should we think that there is a single action describable in a host of ways? Also, actions, in even the most minimal sense, seem to be essentially ‘active’. But, how can we explain what this property amounts to and defend our wavering intuitions about which events fall in the category of the ‘active’ and which do not? (shrink)

Questions about the metaphysics of causation may be usefully divided as follows. First, there are questions about the nature of the causal relata, including (1.1) whether they are in spacetime immanence), (1.2) how fine grained they are individuation), and (1.3) how many there are adicity). Second, there are questions about the metaphysics of the causal relation, including (2.1) what is the difference between causally related and causally unrelated sequences connection), (2.2) what is the difference between sequences related as cause to (...) effect, and those related as effect to cause or as joint effects of a common cause direction), and (2.3) what is the difference between sequences involving the cause, and those involving mere conditions selection). (shrink)

The basic idea of counterfactual theories of causation is that the meaning of causal claims can be explained in terms of counterfactual conditionals of the form “If A had not occurred, C would not have occurred”. While counterfactual analyses have been given of type-causal concepts, most counterfactual analyses have focused on singular causal or token-causal claims of the form “event c caused event e”. Analyses of token-causation have become popular in the last thirty years, especially since the development in the (...) 1970's of possible world semantics for counterfactuals. The best known counterfactual analysis of causation is David Lewis's (1973b) theory. However, intense discussion over thirty years has cast doubt on the adequacy of any simple analysis of singular causation in terms of counterfactuals. Recent years have seen a proliferation of different refinements of the basic idea to achieve a closer match with commonsense judgements about causation. (shrink)

I put forward several desiderata that a philosophical theory of causality should satisfy: it should account for the objectivity of causality, it should underpin formalisms for causal reasoning, it should admit a viable epistemology, it should be able to cope with the great variety of causal claims that are made, and it should be ontologically parsimonious. I argue that Nancy Cartwright’s dispositional account of causality goes part way towards meeting these criteria but is lacking in important respects. I go on (...) to argue that my epistemic account, which ties causal relationships to an agent’s knowledge and ignorance, performs well in the light of the desiderata. Such an account, I claim, is all we require from a theory of causality. (shrink)

Comparisons of rival explanations or theories often involve vague appeals to explanatory power. In this paper, we dissect this metaphor by distinguishing between different dimensions of the goodness of an explanation: non-sensitivity, cognitive salience, precision, factual accuracy and degree of integration. These dimensions are partially independent and often come into conflict. Our main contribution is to go beyond simple stipulation or description by explicating why these factors are taken to be explanatory virtues. We accomplish this by using the contrastive-counterfactual approach (...) to explanation and the view of understanding as an inferential ability. By combining these perspectives, we show how the explanatory power of an explanation in a given dimension can be assessed by showing the range of answers it provides to what-if-things-had-been-different questions and the theoretical and pragmatic importance of these questions. Our account also explains intuitions linking explanation to unification or to exhibition of a mechanism. (shrink)

A much-discussed idea in the causation literature is that it is preferable to invoke causes which are proportional to—neither too general nor too specific for—the effect. This article presents various ways of understanding this idea. In what sense are such causal claims ‘preferable’? And what is it for one event to be ‘proportional’ to another? In a companion article, ‘Proportionality in Causation, Part II: Applications and Challenges’, I discuss the principal applications of the resulting theories of proportionality, and the challenges (...) they face. (shrink)

In this paper, we develop a non-reductive variant of the regularity theory of causation proposed in Andreas and Günther (Pacific Philosophical Quarterly 105: 2–32, 2024). The variant is motivated as a refinement of Lewis’s (Journal of Philosophy 70:556–567, 1973) regularity theory. We do not pursue a reductive theory here because we found a challenge for Baumgartner's (Erkenntnis 78:85–109, 2013) regularity theory which applies to our previous theory as well. The challenge is sidestepped by a framework of law-like propositions resembling structural (...) equations. We furthermore improve the deviancy condition of our previous theory. Finally, we show that the present theory can compete with the most advanced regularity and counterfactual accounts. (shrink)

The goal of this paper is to argue that there is indeterminacy in causation. I present three types of cases in which it is indeterminate whether an event c caused another event e: (1) cases of absence causation recently discussed by Bernstein and by Swanson, (2) cases leading to Sorites paradoxes for causation, and (3) cases where c and e occur in certain indeterministic causal structures and it is therefore indeterminate whether there is a causal relation between them. These cases, (...) I argue, provide strong evidence that indeterminacy is an important general feature of the causal relation and that philosophical theorizing about causation should take this observation into account. (shrink)

This paper addresses the question of what constitutes an apt interpreted model for the purpose of analyzing causation. I first collect universally adopted aptness principles into a basic account, flagging open questions and choice points along the way. I then explore various additional aptness principles that have been proposed in the literature but have not been widely adopted, the motivations behind their proposals, and the concerns with each that stand in the way of universal adoption. I conclude that the remaining (...) work of articulating aptness for a SEM analysis of causation is tied up with issues to do with modality, ontology, and mereology. Continuing this work is therefore likely to shed light on the relationship between these areas and causation more generally. (shrink)

We often hear how scientists have discovered that a certain human trait – or a trait of another type of organism – is innate, genetic, heritable, inherited, naturally selected etc. All these claims have something in common: they all declare a trait to have significant organism internal (for instance genetic) causes that are present in the organism at its birth. I call claims like these “nativist claims”. Nativist claims are important. They shape our overall understanding of what we are, what (...) we can do and what is good for us. They also impact our practical decision making. For instance, we know that certain types of genes increase the risk of certain diseases, and increasingly this has an effect on healthcare practices. Many people believe that in the matter of children’s education we should consider the fact that many cognitive abilities are highly heritable. Associations between genetics and aggressive behavior have been taken to be alleviating circumstances in criminal sentencing. It is therefore important to correctly understand the content and implications of nativist claims. In this doctoral thesis I analyze, by philosophical means, the content of different kinds of nativist claim. What does it mean that some psychological tendency is innate? What does it mean that intelligence is 50% heritable? How (if at all) do genes come to carry information about the traits of an organism – they are nothing like the information carriers familiar to us, such as newspapers, e-mails, secret codes, and audio files. This thesis emphasizes two things. Firstly, I demonstrate, that in different scientific contexts these words ('innate', 'genetically caused' etc.) can mean very different things. Therefore, the implications of a given nativist claim in one scientific context can be remarkably different from another nativist claim’s implications in some other scientific context. Secondly, I explain how the development of innate, heritable and genetically caused traits can be determined in different and often unexpected ways by our environment and experiences. I demonstrate that the content of a concept that is considered innate by cognitive scientists can be determined by what the organism that possesses the concept has experienced. I also show that even a trait which is 100% heritable can at the same time be a socially constructed trait. (shrink)

According to orthodoxy, the most fundamental kind of causation involves one event causing another event. I argue against this event‐causal view. Instead, the most fundamental kind of causation is thing causation, which involves a thing causing a thing to do something. Event causation is reducible to thing causation, but thing causation is not reducible to event causation, because event causation cannot accommodate cases of fine‐grained causation. I defend my view from objections, including C. D. Broad's influential “timing” argument, and I (...) conclude with implications for agent‐causal theories of free will. (shrink)

Proportionality—the idea that causes are neither too general nor too specific for their effects—seems to recommend implausibly disjunctive causes (McGrath, 1998 ; Shapiro & Sober, 2012 ; Franklin-Hall, 2016 ). I argue that this problem should be avoided by appeal to the notion of cohesion. I propose an account of cohesion in terms of the similarity structure of property-spaces, argue that it is not objectionably mysterious, and that alternative approaches—based on naturalness, interventionism, and contrastivism—are inadequate without appeal to it. In (...) an appendix, I show how my proposal can be perspicuously formalized by adapting structural equation models. (shrink)

This dissertation begins by addressing the question of when a causal model is apt for deciding questions of actual causation with respect to some target situation. I first provide relevant background about causal models, explain what makes them promising as a tool for analyzing actual causation, and motivate the need for a theory of aptness as part of such an analysis (Chapter 1). I then define what it is for a model on a given interpretation to be accurate of, that (...) is, say only true things about, some target situation. This involves a systematization of various representational principles mentioned and/or discussed throughout the literature into a method of interpretation, which I propose be taken as standard (Chapter 2). Next, I explain and address two reasons for which accuracy as I’ve defined it is insufficient for aptness. The first reason – already discussed in the literature – is the problem of structural isomorphs. In response, I propose the aptness condition of Explicit Partial Mediation (Chapter 3). The second reason – which has yet to be noticed – is the problem of the indeterminacy of accuracy. As I demonstrate, a model is accurate of a target situation only relative to a set of background possibilities – what I call a modal profile. It follows that a model represents a situation only relative to some modal profile or other. I go on to discuss the ramifications of this observation for a theory of actual causation in terms of models. I argue that the relativity be taken at face value and built into our metaphysical account of causation, resulting in a view that I call causal relativism (Chapter 4). I explore one advantage of this view in detail: that the resulting account can defend the principle of strong proportionality against several objections (Chapter 5). Finally, I apply the earlier discussion of aptness to attempts to provide a semantics of counterfactuals in terms of causal models – an interventionist semantics. I show how just as a similarity semantics relies on an opaque notion of similarity, an interventionist semantics relies on an analogous notion of aptness. The challenge of articulating aptness thus undermines the claim that an interventionist semantics avoids representational problems inherent in a similarity semantics (Chapter 6). I close with a recap and suggestions for future research (Chapter 7). -/- . (shrink)

Incorporating normality ascriptions into counterfactual theories of causation was supposed to handle isomorphs. It doesn’t—conjoining isomorphs can produce cases that such ascriptions cannot resolve.

What determines the degree to which some event harms a subject? According to the counterfactual comparative account, an event is harmful for a subject to the extent that she would have been overall better off if it had not occurred. Unlike the causation based account, this view nicely accounts for deprivational harms, including the harm of death, and for cases in which events constitute a harm rather than causing it. However, I argue, it ultimately fails, since not every intrinsically bad (...) state that is counterfactually dependent on an event contributes to its degree of harm. So while the causation based account is too restrictive, the counterfactual comparative view is not restrictive enough. In light of this, I suggest an alternative, explanation based account of overall harm, according to which the degree to which some event is harmful for a subject is determined by the degree to which (crudely) the states explained by it are overall more intrinsically bad than intrinsically good for her. (shrink)

In this article, I defend a biconditional counterfactual account of causation, which places equal emphasis on what I call “the presence condition” and “the absence condition,” whereas Lewis's classical counterfactual theory focuses only on the absence condition. I attempt to show that biconditionalism provides a promising treatment of supervenient causation, namely, causal cases involving the supervenience relationship. Although some philosophers confuse this account with the proportionality constraint on causation, I argue that biconditionalism is distinct from and superior to proportionalism in (...) accommodating our reliable causal intuitions. (shrink)

Many epidemics consist in individuals spreading infection to others. From the population perspective, they also have population characteristics important in modeling, explaining, and intervening in epidemics. I analyze epidemiology’s contemporary population perspective through the example of epidemics by examining two central principles attributed to Geoffrey Rose: a distinction between the causes of cases and the causes of incidence, and between “high-risk” and “population” strategies of prevention. Both principles require revision or clarification to capture the sense in which they describe distinct (...) perspectives on the same phenomenon, each perspective capturing a different level of contrastive analysis. (shrink)

According to the so-called 'proportionality principle', causes should be proportional to their effects: they should be both enough and not too much for the occurrence of their effects. This principle is the subject of an ongoing debate. On the one hand, many maintain that it is required to address the problem of causal exclusion and take it to capture a crucial aspect of causation. On the other hand, many object that it renders accounts of causation implausibly restrictive and often reject (...) the principle wholesale. I argue that there is exaggeration on both sides. While one half of the principle is overly demanding, the other half is unobjectionable. And while the unobjectionable half does not block exclusion arguments on its own, it provides a nuanced picture of higher-level causation, fits with recent developments in philosophy of causation, and motivates adjustments to standard difference-making accounts of causation. I conclude that at least half of the proportionality principle is worth taking seriously. (shrink)

According to the theory developed here, we may trace out the processes emanating from a cause in such a way that any consequence lying along one of these processes counts as an effect of the cause. This theory gives intuitive verdicts in a diverse range of problem cases from the literature. Its claims about causation will never be retracted when we include additional variables in our model. And it validates some plausible principles about causation, including Sartorio's ‘Causes as Difference Makers’ (...) principle and Hitchcock's ‘Principle of Sufficient Reason’. (shrink)

In this dissertation I develop a pluralist theory of actual causation. I argue that we need to distinguish between total, path-changing, and contributing actual causation. The pluralist theory accounts for a set of example cases that have raised problems for extant unified theories and it is supported by considerations about the various functions of causal concepts. The dissertation also analyses the context-sensitivity of actual causation. I show that principled accounts of causal reasoning in legal inquiry face limitations and I argue (...) that the context-sensitivity of actual causation is best represented by a distinction between default and deviant states in causal models. (shrink)

According to the ‘standard story’ in the philosophy of action, actions are those movements of a creature’s body that are caused and rationalized by the creature’s mental states. The attractions of the causal condition have been widely discussed. The rationalization condition is nearly ubiquitous, but it is notoriously obscure, and its motivation has rarely been made explicit. This paper presents a new argument for including the rationalization condition in the causal theory of action, and sketches a broadly Davidsonian theory of (...) what rationalization is. (shrink)

In this paper I argue that there is a significant but often overlooked metaphysical distinction to be made between contextualism and contrastivism. The orthodox view is that contrastivism is merely a form of contextualism. This is a mistake. The contextualist view is incompatible with certain naturalist claims about the metaphysical nature of concepts within whichever domain is being investigated, while the contrastivist view is compatible with these claims. So, choosing one view over the other will involve choosing to affirm or (...) deny a significant metaphysical claim. As such, a demarcation ought to be put in place between contextualism and contrastivism. (shrink)

The Doctrine of Microphysical Supervenience states that microphysical duplicates cannot differ in their intrinsic properties. According to Merricks :59–71, 1998a, Objects and persons, Oxford University Press, 2001), however, this thesis is false, since microphysical duplicates can differ with respect to the intrinsic property of consciousness. In my view, Merricks’ argument is plausible, and extant attempts to reject it are problematic. However, the argument also threatens to make consciousness appear mysterious, by implying that consciousness facts fail to be microphysically determined and (...) that there can be brute and inexplicable differences in consciousness between material things. The paper therefore develops an account that can respect the soundness of Merricks’ argument while avoiding these problematic consequences. At the heart of the proposal is the idea that consciousness can be microphysically grounded despite failing to microphysical supervene. The proposed view also has the interesting consequence that consciousness is an intrinsic property despite depending on extrinsic factors for its instantiation. (shrink)

A central argument for non-reductive accounts of group agency is that complex social entities are capable of exerting causal influence independently of and superseding the causal efficacy of the individuals constituting them. A prominent counter is that non-reductionists run into an insuperable dilemma between identity and redundancy – with identity undermining independent higher-level efficacy and redundancy leading to overdetermination or exclusion. This paper argues that critics of non-reductionism can manage with a simpler and more persuasive reductio strategy called mapping: allow (...) that group agents are causally efficacious in their own right and chart how their causal efficacy is carried out; how it relates to the causal efficacy of individual determiners; how it connects to the causal relevance of background structural factors. The focus exclusively on whether groups are or are not causally efficacious black-boxes implementation, while close attention to how causation is wired increases the visibility of individualist arguments and countenances structure-oriented explanations. (shrink)

In Counterfactual Conditionals, Daniel Dohrn discusses the standard account of counterfactuals, conditionals of the form ‘If A had been the case, then B would have been the case’. According to the standard account, a counterfactual is true if the then-sentence is true in all closest worlds in which the if-sentence is true. Closeness is spelled out in terms of an ordering of worlds by their similarity. Dohrn explores resources of defending the standard account against several challenges. In particular, he defends (...) the standard logics for counterfactuals. He discusses exemplary doubts as to whether conditionals have truth conditions. He inquires into the interaction between truth and probability of counterfactuals. He tackles problems with the similarity ordering. He address the interaction between counterfactuals and normalcy conditions. He closes with elaborating peculiarities of future-directed counterfactuals. (shrink)

Can an identity be the proper subject of an explanation? A popular stance, albeit not one often argued for, gives a negative answer to this question. Building from a contentious passage from Jaegwon Kim in this direction, we reconstruct an argument to the conclusion that identities, to the extent in which they are necessary, cannot be explained. The notion of contrastive explanation, characterized as difference-seeking, will be crucial for this argument; however, we will eventually find the argument to be unsatisfactory. (...) On the contrary, the discussion provides enough resource to sketch a very simple framework for a non-causal contrastive explanation of identities. Many instances will be provided, with different varieties of explanans, ultimately suggesting that certain entailment or biconditional principles involving identities (first and foremost, so-called two-level identity criteria) may indeed be taken to have an inherent explanatory value. (shrink)

In cases in which many causes together bring about an effect, it is common to select some as particularly important. Philosophers since Mill have been pessimistic about analyzing this reasoning because of its variability and the multifarious causal and pragmatic details of how it works. I argue Mill was right to think these details matter but wrong that they preclude philosophical analysis of causal selection. I show that analyzing the pragmatic details of scientific debates about the important causes of the (...) Bhopal Gas Tragedy can illuminate causal reasoning about disasters and shed new light on causality and causal selection. (shrink)

A well-known objection to Humean accounts of laws charges them with circularity. While the view has it that particular facts explain the natural laws, natural laws are often relied upon in order to explain particular facts. Thus, the Humean is committed to circular explanations—or so goes the argument. In this paper, I review two ways of dealing with the circularity objection against Humean views of laws. Then, by introducing a contrastive treatment of explanations, I put forward a new one, which, (...) if it does not end up dethroning its competitors, I am hoping is still worth exploring. (shrink)

Loewer (in: Physicalism and its discontents, 2001; Philos Phenomenol Res 65:655–663, 2002; in: Contemporary debates in philosophy of mind, 2007) has argued that the nonreductive physicalist should respond to the exclusion problem by endorsing the overdetermination entailed by their view. Kim’s (Physicalism, or something near enough, 2005; in: Contemporary debates in the philosophy of mind, 2007) argument against this reply is based on the premise that mental causation must be a productive relation in order to sustain human agency. In this (...) paper, I challenge the premise that mental causation is a productive relation by appealing to the underlying double prevention structure of the physiological mechanisms of human action. Since the causal pathways from an agent’s mental events to bodily movement involves an absence, mental causation is not productive. This places Kim in a troublesome dilemma in his debate with Loewer: either surrender mental causation or deny that causation is a productive relation. With the support offered for productive mental causation undermined, responses to the exclusion problem based on accepting overdetermination remain viable options for the nonreductive physicalist. (shrink)

Our approach aims at accounting for causal claims in terms of how the physical states of the underlying dynamical system evolve with time. Causal claims assert connections between two sets of physicals states—their truth depends on whether the two sets in question are genuinely connected by time evolution such that physical states from one set evolve with time into the states of the other set. We demonstrate the virtues of our approach by showing how it is able to account for (...) typical causes, causally relevant factors, being ‘the’ cause, and cases of overdetermination and causation by absences. (shrink)

In this paper, I have two primary goals. First, I show that the causal-based story approach in A hybrid formal theory of arguments, stories and criminal evidence is ill suited to negative causation. In the literature, the causal-based approach requires that hypothetical stories be causally linked to the explanandum. Many take these links to denote physical or psychological causation, or temporal precedence. However, understanding causality in those terms, as I will show, cannot capture cases of negative causation, which are of (...) interest to the Law. In keeping with this, I also discuss some of the difficulties Hybrid Theory invites by remaining silent on the nature of the causal links. In my second aim, I sketch a way for Hybrid Theory to overcome this problem. By replacing the original, underlying causal structure with contrastive causation in the law, Hybrid Theory can represent reasoning in which the evidence that is appealed to is causally linked via negative causation to the explananda. (shrink)