Many assume that the best, and perhaps only, way to address the so-called Problem of Evil is to claim that God does not do evil, but that God merely allows evil. This assumption depends on two claims: the doing-allowing distinction exists and the doing-allowing distinction is morally significant. In this paper I try to undermine both of these claims. Against I argue that some of the most influential analyses of the doing-allowing distinction face grave difficulties and that these difficulties are (...) only exacerbated when applied to God. Against I argue that broadly Kantian considerations give a better explanation of the moral differences in paradigm cases than the doing-allowing distinction and, when applied to God, effectively dissolve the moral significance of the doing-allowing distinction. The upshot of this is that those who claim God does evil are no worse off than those who claim God allows evil regarding the PoE. (shrink)

What are scientific theories and how should they be represented? In this article, I propose a causal–structural account, according to which scientific theories are to be represented as sets of interrelated causal and credal nets. In contrast with other accounts of scientific theories (such as Sneedian structuralism, Kitcher’s unificationist view, and Darden’s theory of theoretical components), this leaves room for causality to play a substantial role. As a result, an interesting account of explanation is provided, which sheds light on explanatory (...) unification within a causalist framework. The theory of classical genetics is used as a case study. 1 Introduction2 The Theory of Classical Genetics3 Three Philosophical Accounts of the Theory of Classical Genetics3.1 The structuralist account3.2 Kitcher’s unificationism3.3 Darden and theory change in science4 A Common Lacuna: Where is Causality?5 Woodward’s Interventionist Account of Causation6 Causal Bayes Nets and Their Interrelations6.1 Causal Bayes nets6.2 Relations among causal nets6.3 Credal nets and their interrelations7 The Theory of the Gene and its Causal Graph8 A First Exemplar: Stem Length in Pea Plants8.1 Three crosses on stem length in pea plants8.2 The causal graph for stem length in pea plants8.3 Morgan’s explanatory principles and the credal net for stem length in pea plants9 Explaining Mendel’s Crosses: A Causal–Structural Account10 Monohybrid Crosses with Complete Dominance11 Exemplars,Explanatory Patterns, Generic Credal Nets, and Mechanism Schemas12 Incomplete Dominance13 Anomalies14 Multi-hybrid Crosses with Independent Assortment15 Multi-hybrid Crosses with Linkage and Crossing-Over16 Double Crossing-Over and the Linear Order of the Gene17 Causal–Structural Explanation18 Explanatory Unification19 Concluding Remarks. (shrink)

Mechanistic approaches are very common in the causal interpretation of biological and neuroscientific experimental work in today’s philosophy of science. In the mechanistic literature a strict distinction is often made between causal relations and constitutive relations, where the latter cannot be causal. One of the typical reasons for this strict distinction is that constitutive relations are supposedly synchronic whereas most if not all causal relations are diachronic. This strict distinction gives rise to a number of problems, however. Our end goal (...) in this paper is to argue that it should be given up, at least in the context of the biological and the psychological sciences. To that effect, we argue that constitutive relations in this context are diachronic, thus undermining the aforementioned reason. We offer two cases from scientific practice in which constitutive relations are regarded as both diachronic and causally efficacious, review three existing ways of dealing with the apparent diachronic nature of interlevel relations in mechanisms and propose a new account of diachronic, causal constitutive relevance. (shrink)

The paper takes a detailed look at a surprising new aspect of the dynamics of rigid bodies. Far from the usual consideration of rigid body theory as a merely technical chapter of classical physics, I demonstrate here that there are solutions to the conservation equations of mechanics that imply the spontaneous, unpredictable splitting of a rigid body in free rotation, something that has direct implications for the problem of causality. The paper also shows that the instability revealed in indeterminist splitting (...) processes does not depend solely on the bodies’ inertial properties but also on the number of dimensions of the physical space they inhabit. The paper concludes with a conjecture on the behavior of rigid bodies in four-dimensional space. (shrink)

How do people attribute responsibility in situations where the contributions of multiple agents combine to produce a joint outcome? The prevalence of over-determination in such cases makes this a difficult problem for counterfactual theories of causal responsibility. In this article, we explore a general framework for assigning responsibility in multiple agent contexts. We draw on the structural model account of actual causation (e.g., Halpern & Pearl, 2005) and its extension to responsibility judgments (Chockler & Halpern, 2004). We review the main (...) theoretical and empirical issues that arise from this literature and propose a novel model of intuitive judgments of responsibility. This model is a function of both pivotality (whether an agent made a difference to the outcome) and criticality (how important the agent is perceived to be for the outcome, before any actions are taken). The model explains empirical results from previous studies and is supported by a new experiment that manipulates both pivotality and criticality. We also discuss possible extensions of this model to deal with a broader range of causal situations. Overall, our approach emphasizes the close interrelations between causality, counterfactuals, and responsibility attributions. (shrink)

Backtracking influence is influence that zigzags in time. For example, backtracking influence exists when an event E_1 makes an event E_2 more likely by way of a nomic connection that goes from E_1 back in time to an event C and then forward in time to E_2. I contend that in our local region of spacetime, at least, backtracking influence is redundant in the sense that any existing backtracking influence exerted by E_1 on E_2 is equivalent to E_1's temporally direct (...) influence on E_2. I prove the redundancy of backtracking influence using several plausible physical principles without assuming any fundamental temporal or causal asymmetry. This explanation can play a prominent role in an account of why causation appears to be objectively asymmetric regardless of whether the fundamental laws are symmetric. (shrink)

Bayesian networks are computer programs which represent probabilitistic relationships graphically as directed acyclic graphs, and which can use those graphs to reason probabilistically , often at relatively low computational cost. Almost every expert system in the past tried to support probabilistic reasoning, but because of the computational difficulties they took approximating short-cuts, such as those afforded by MYCIN's certainty factors. That all changed with the publication of Judea Pearl's Probabilistic Reasoning in Intelligent Systems, in 1988, which synthesized a decade of (...) research making accurate graphical probabilistic reasoning computationally achievable.Bayesian network technology is now one of the fastest growing fields of research in artificial intelligence. That it has become a publication industry in its own right is shown by a search on Google scholar :This development, together with a parallel related growth in the use of causal discovery algorithms which automate the learning of Bayesian networks from sample data, has generated considerable interest, and controversy, within the philosophy-of-science community.Three central questions bringing together AI researchers and philosophers of science are: Are Bayesian networks Bayesian? What is the relation between probability and causality? Are the assumptions behind causal discovery of Bayesian networks realistic or fantastical?Jon Williamson, as a philosopher of science with a keen interest in the technology, asks and answers these questions in his new book. Although it is self-contained, his book is not very likely as an introduction to the technology , nor is it optimal even as an introduction to the philosophical problems in interpreting Bayesian networks . Rather Williamson's book is an attempt to move the debate forward by solving the central problems of the …. (shrink)

I argue against Barbara Montero's claim that Conservation of Energy has nothing to do with physicalism. I reject her reconstruction of the argument for physicalism from CoE, and offer an alternative reconstruction that better captures the intuitions of those who believe that there is a conflict between interactionist dualism and CoE.

Most philosophers of physics are eliminativists about causation. Following Bertrand Russell’s lead, they think that causation is a folk concept that cannot be rationally reconstructed within a worldview informed by contemporary physics. Against this thesis, I argue that physics contributes to shaping the concept of causation, in two ways. 1. Special Relativity is a physical theory that expresses causal constraints. 2. The physical concept of a conserved quantity can be used in the functional reduction of the notion of causation. The (...) empirical part of this reduction makes the hypothesis that the transference of an amount of a conserved quantity is a necessary and sufficient condition for causation. This hypothesis is defended against several objections from physics: that amounts of energy do not possess the appropriate identity conditions required for being able to be transmitted, that there is no universal principle of the conservation of energy in General Relativity, and that there are at least two types of physical systems in which causation does not involve any transference: entangled systems in quantum mechanics and the Aharanov-Bohm effect. In order to show that physics provides means to elaborate the concept of causation it is important to avoid certain misunderstandings. In particular, the claim that there is causation in a physical world does not mean that causation is an additional ingredient of the "furniture" of the world, over and above the ingredients identified by physics. (shrink)

Most philosophers of physics are eliminativists about causation. Following Bertrand Russell’s lead, they think that causation is a folk concept that cannot be rationally reconstructed within a worldview informed by contemporary physics. Against this thesis, I argue that physics contributes to shaping the concept of causation, in two ways. (1) Special Relativity is a physical theory that expresses causal constraints. (2) The physical concept of a conserved quantity can be used in the functional reduction of the notion of causation. The (...) empirical part of this reduction makes the hypothesis that the transference of an amount of a conserved quantity is a necessary and sufficient condition for causation. This hypothesis is defended against several objections from physics: that amounts of energy do not possess the appropriate identity conditions required for being able to be transmitted, that there is no universal principle of the conservation of energy in General Relativity, and that there are at least two types of physical systems in which causation does not involve any transference: entangled systems in quantum mechanics and the Aharonov–Bohm effect. In order to show that physics provides means to elaborate the concept of causation it is important to avoid certain misunderstandings. In particular, the claim that there is causation in a physical world does not mean that causation is an additional ingredient of the “furniture” of the world, over and above the ingredients identified by physics. (shrink)

In Search Of Mechanisms is a book about the methodology of biology. It is a work by Carl Craver and Lindley Darden, both of whom are well-known individually for their advocacy of mechanistic explanation—in the neurosciences and in the fields of genetics, cytology and molecular biology . Here, the two join forces to give a unified model of biological explanation, not limited to a particular area of biological enquiry, as rooted in the search for mechanisms.The objectives of the book are (...) threefold. First, it sets out to describe what biologists do. This first objective can be described as the goal of making biology more comprehensible. If biology involves the search for mechanisms, then one cannot understand the history of biology and its current practice without understanding mechanisms. Secondly, the book seeks to give a proscriptive account of explanation and the process of explaining some phenomena. This second objective relates to an interest in contributing to the .. (shrink)

Abstract Recent criticisms of intuition from experimental philosophy and elsewhere have helped undermine the authority of traditional conceptual analysis. As the product of more empirically informed philosophical methodology, this result is compelling and philosophically salutary. But the negative critiques rarely suggest a positive alternative. In particular, a normative account of concept determination—how concepts should be characterized—is strikingly absent from such work. Carnap's underappreciated theory of explication provides such a theory. Analyses of complex concepts in empirical sciences illustrates and supports this (...) claim, and counteracts the charge explication is only suitable for highly mathematical, axiomatic contexts. Explication is also defended against the influential criticism it is “philosophically unilluminating”. Content Type Journal Article Category Original paper in Philosophy of Science Pages 1-19 DOI 10.1007/s13194-011-0027-5 Authors James Justus, Philosophy Department, Florida State University and University of Sydney, Tallahassee, FL 32306, USA Journal European Journal for Philosophy of Science Online ISSN 1879-4920 Print ISSN 1879-4912. (shrink)

This paper sets out to analyze how causation works by focusing on biology, as represented by epidemiology and by scientific information on how the body works (“physiology”). It starts by exploring the specificity of evolved physiological systems, in which evolutionary, developmental and proximal causes all fit together, and the concept of function is meaningful; in contrast, this structure does not apply in epidemiology (or outside biology). Using these two contrasting branches of biology, I examine the role both of mechanism and (...) of difference making in causation. I find that causation necessarily involves both mechanism and difference making, and that they play complementary roles. Both are seen as ontologically necessary, even if the evidence is not always available for both. Influential monist accounts that focus on one of these, at the expense of ignoring the other, are found to be inadequate on these and on other grounds. Recent attempts to combine them are reviewed, notably that of Russo and Williamson (Int Stud Philos Sci 21:157–170, 2007), and it is argued that their epistemic view requires there to be a source of the different types of evidence that a rational agent would consider, and that this source must be ontic. I then analyze how causal relationships work in evolved physiological systems and in those studied by epidemiology, with a particular focus on how mechanism interacts with input. Finally, I consider this concept of causation from the perspective of everyday language, and of its possible generalisability outside biology. (shrink)

Negative facts get a bad press. One reason for this is that it is not clear what negative facts are. We provide a theory of negative facts on which they are no stranger than positive atomic facts. We show that none of the usual arguments hold water against this account. Negative facts exist in the usual sense of existence and conform to an acceptable Eleatic principle. Furthermore, there are good reasons to want them around, including their roles in causation, chance-making (...) and truth-making, and in constituting holes and edges. (shrink)

This paper advances the thesis of methodological mechanism, the claim that to be committed to mechanism is to adopt a certain methodological postulate, i.e. to look for causal pathways for the phenomena of interest. We argue that methodological mechanism incorporates a minimal account of understanding mechanisms, according to which a mechanism just is a causal pathway described in the language of theory. In order to argue for this position we discuss a central example of a biological mechanism, the mechanism of (...) cell death, known as apoptosis. We argue that this example shows that our philosophically deflationary account is sufficient in order to have an illuminating account of mechanisms as the concept is used in biology. (shrink)

In this paper, I examine the comparatively neglected intuition of production regarding causality. I begin by examining the weaknesses of current production accounts of causality. I then distinguish between giving a good production account of causality and a good account of production. I argue that an account of production is needed to make sense of vital practices in causal inference. Finally, I offer an information transmission account of production based on John Collier’s work that solves the primary weaknesses of current (...) production accounts: applicability and absences. (shrink)

In this paper I will argue that what makes our ordinary judgements about token causation true can be explicated in terms of interferences into quasi-inertial processes. These interferences and quasi-inertial processes can in turn be fully explicated in scientific terms. In this sense the account presented here is reductive. I will furthermore argue that this version of a process-theory of causation can deal with the traditional problems that process theories have to face, such as the problem of misconnection and the (...) problem of disconnection as well as with a problem concerning the mis-classification of pre-emption cases. (shrink)

In this paper, I discuss Michael Moore’s and Jonathan Schaffer’s views on the ontology of omissions in context of their stances on the problem of omissive causation. First, I consider, from a general point of view, the question of the ontology of omissions, and how it relates to the problem of omissive causation. Then I describe Moore’s and Schaffer’s particular views on omissions and how they combine with their stances on the problem of omissive causation. I charge Moore and Schaffer (...) with inconsistencies and insufficiencies within their overall theories, and consider their replies. Finally, I propose my own view on the ontology of omissions and solution to the problem of omissive causation. (shrink)

Few philosophers believe in the existence of so-called negative properties. Indeed, many find it mind-boggling just to imagine such entities. By contrast, I believe not only that negative properties are quite conceivable, but also that there are good reasons for thinking that some such properties actually exist. In this paper, I would like to explicate a concept of negative properties which I think avoids the logical absurdities commonly believed to frustrate theories of negative existences. To do this, I shall deploy (...) a conceptual approach to ontology: I first evaluate the ontological commitments of ordinary negative statements, before I argue that, from the perspective of conceptual analysis, negative properties are no more suspicious than positive properties. In the second part of this paper, I probe the extent to which one can explain the distinction between positive and negative properties in terms of facts about logico-semantical entailment. (shrink)

It is a commonplace that in philosophy, intuitions supply evidence for and against philosophical theories. Recent work in experimental philosophy has brought to bear the intuitions of philosophically naïve subjects in a number of different ways. One line of response to this work has been to claim that philosophers have expertise that privileges their intuitive judgments, and allows them to disregard the judgments of non-experts. This expertise is supposed to be analogous to the expertise of the mathematician or the physicist. (...) This paper critically evaluates this appeal to philosophical expertise. (shrink)

Much of the philosophical literature on causation has focused on the concept of actual causation, sometimes called token causation. In particular, it is this notion of actual causation that many philosophical theories of causation have attempted to capture.2 In this paper, we address the question: what purpose does this concept serve? As we shall see in the next section, one does not need this concept for purposes of prediction or rational deliberation. What then could the purpose be? We will argue (...) that one can gain an important clue here by looking at the ways in which causal judgments are shaped by people‘s understanding of norms. (shrink)

Concerning any object of philosophical analysis, we can ask several questions, including the two posed in the title of this paper. Despite difficulties in formulating a precise criterion to distinguish causal processes from pseudoprocesses, and causal interactions from mere spatiotemporal intersections, I argue that Salmon answered the first of these questions with extraordinary clarity. The second question, by contrast, has received very little attention. I will present two problems: in the first, it seems that Salmon has provided exactly the conceptual (...) resources needed to solve the problem; in the second, it is difficult to see how causal processes and interactions may be used to shed any light. In general, the way to carry Salmon's program forward will be to demonstrate that these resources can be made to do real philosophical work. (shrink)

I offer an account of how the quantum theory we have helps us explain so much. The account depends on a pragmatist interpretation of the theory: this takes a quantum state to serve as a source of sound advice to physically situated agents on the content and appropriate degree of belief about matters concerning which they are currently inevitably ignorant. The general account of how to use quantum states and probabilities to explain otherwise puzzling regularities is then illustrated by showing (...) how we can explain single-particle interference phenomena, the stability of matter, and interference of Bose–Einstein condensates. Finally, I note some open problems and relate this account to alternative approaches to explanation that emphasize the importance of causation, of unification, and of structure. 1 Introduction2 Two Requirements on Explanations in Physics3 What We Can use Quantum Theory to Explain4 The Function of Quantum States and Born Probabilities5 How These Functions Contribute to the Explanatory Task6 Example One: Single-Particle Interference7 Example Two: Explanation of the Stability of Matter8 Example Three: Bose Condensation9 Conclusion. (shrink)

I offer an account of how the quantum theory we have helps us explain the enormous variety of phenomena it is generally taken to explain. The account depends on what I have elsewhere called a pragmatist interpretation of the theory. This rejects views according to which a quantum state describes or represents a physical system, holding instead that it functions as a source of sound advice to physically situated agents like us on the content and appropriate degree of belief about (...) matters concerning which they are currently inevitably ignorant. So while the account given here is incompatible with some views of structural explanation in quantum theory it is nevertheless able to incorporate what I take to be their valuable insights. (shrink)

George Molnar’s contention that some dispositional properties are displayed without the aid of any activating conditions poses a challenge to the conditional analysis of dispositions. Since the invocation of activating conditions is regarded as a crucial feature of the analysis, spontaneous dispositions are believed to expose its inadequacy by eluding its scope. The challenge goes to the very heart of the conditional approach to dispositions, allegedly revealing a deep flaw in all its incarnations. Granting that there may be spontaneously manifestable (...) dispositions, I argue that the conditional analysis can successfully accommodate them by elevating the potential causal relation between a disposition and its manifestation. (shrink)

This paper presents an attempt to integrate theories of causal processes—of the kind developed by Wesley Salmon and Phil Dowe—into a theory of causal models using Bayesian networks. We suggest that arcs in causal models must correspond to possible causal processes. Moreover, we suggest that when processes are rendered physically impossible by what occurs on distinct paths, the original model must be restricted by removing the relevant arc. These two techniques suffice to explain cases of late preëmption and other cases (...) that have proved problematic for causal models. (shrink)

Saying so can make it so, J. L. Austin taught us long ago. Famously, John Searle has developed this Austinian insight in an account of the construction of institutional reality. Searle maintains that so-called Status Function Declarations, allegedly having a “double direction of fit”, synchronically create worldly institutional facts, corresponding to the propositional content of the declarations. I argue that Searle’s account of the making of institutional reality is in tension with the special theory of relativity—irrespective of whether the account (...) is interpreted as involving causal generation or non-causal grounding of worldly institutional facts—and should be replaced by a more modest theory which interprets the results of Status Function Declarations in terms of mere Cambridge change and institutional truth. I end the paper by indicating the import of this more modest theory for theorizing about the causal potency of institutional phenomena generated by declarations. (shrink)

I argue that Mumford and Anjum’s recent theory of simultaneous causation among powerful meso-level objects is problematic in several respects: it is based on a false dichotomy, it is incompatible with standard meso-level physics, it is explanatory deficient, and it threatens to render the powers metaphysics incoherent. Powers theorists are advised, therefore, to adopt a purely sequential conception of causation.

Contemporary philosophical work on causation is a tangled mess of disparate aims, approaches, and accounts. Best to cut through it by means of ruthless but, hopefully, sensible judgments. The ones that follow are designed to sketch the most fruitful avenues for future work.

Recent work in psychology and experimental philosophy has shown that judgments of actual causation are often influenced by consideration of defaults, typicality, and normality. A number of philosophers and computer scientists have also suggested that an appeal to such factors can help deal with problems facing existing accounts of actual causation. This article develops a flexible formal framework for incorporating defaults, typicality, and normality into an account of actual causation. The resulting account takes actual causation to be both graded and (...) comparative. We then show how our account would handle a number of standard cases. 1 Introduction2 Causal Models3 The HP Definition of Actual Causation4 The Problem of Isomorphism5 Defaults, Typicality, and Normality6 Extended Causal Models7 Examples7.1 Omissions7.2 Knobe effects7.3 Causes versus background conditions7.4 Bogus prevention7.5 Causal chains7.6 Legal doctrines of intervening causes7.7 Pre-emption and short circuits8 Conclusion. (shrink)

The starting point in the development of probabilistic analyses of token causation has usually been the naïve intuition that, in some relevant sense, a cause raises the probability of its effect. But there are well-known examples both of non-probability-raising causation and of probability-raising non-causation. Sophisticated extant probabilistic analyses treat many such cases correctly, but only at the cost of excluding the possibilities of direct non-probability-raising causation, failures of causal transitivity, action-at-a-distance, prevention, and causation by absence and omission. I show that (...) an examination of the structure of these problem cases suggests a different treatment, one which avoids the costs of extant probabilistic analyses. (shrink)

An influential tradition in the philosophy of causation has it that all token causal facts are, or are reducible to, facts about difference-making. Challenges to this tradition have typically focused on pre-emption cases, in which a cause apparently fails to make a difference to its effect. However, a novel challenge to the difference-making approach has recently been issued by Alyssa Ney. Ney defends causal foundationalism, which she characterizes as the thesis that facts about difference-making depend upon facts about physical causation. (...) She takes this to imply that causation is not fundamentally a matter of difference-making. In this paper, I defend the difference-making approach against Ney’s argument. I also offer some positive reasons for thinking, pace Ney, that causation is fundamentally a matter of difference-making. (shrink)

Recent papers by a number of philosophers have been concerned with the question of whether natural selection is a causal process, and if it is, whether the causes of selection are properties of individuals or properties of populations. I shall argue that much confusion in this debate arises because of a failure to distinguish between causal productivity and causal relevance. Causal productivity is a relation that holds between events connected via continuous causal processes, while causal relevance is a relationship that (...) can hold between a variety of different kinds of facts and the events that counterfactually depend upon them. I shall argue that the productive character of natural selection derives from the aggregation of individual processes in which organisms live, reproduce and die. At the same time, a causal explanation of the distribution of traits will necessarily appeal both to causally relevant properties of individuals and to causally relevant properties that exist only at the level of the population. (shrink)

Most philosophical accounts of causation take causal relations to obtain between individuals and events in virtue of nomological relations between properties of these individuals and events. Such views fail to take into account the consequences of the fact that in general the properties of individuals and events will depend upon mechanisms that realize those properties. In this paper I attempt to rectify this failure, and in so doing to provide an account of the causal relevance of higher-level properties. I do (...) this by critiquing one prominent model of higher-level properties—Kim’s functional model of reduction—and contrasting it with a mechanistic approach to higher-level properties and causation. (shrink)

Philosophical theories of causation are commonly judged by their ability to correctly determine whether there is a causal relation present in intuitively clear example scenarios. If the theories survive this test, they are then used to answer big philosophical questions about causation. This Method of Examples is attractive because it seems to allow us to determine the quality of a theory of causation independently of answering the big philosophical questions; which is good, since it means that we can then non-circularly (...) use the theories judged to be best to answer those questions. However, the current article argues that this virtue of the Method of Examples is an illusion. In particular, I argue that the necessary step of judging whether a proposed analysis of causation is reductive can only be taken after many of the most vexing philosophical questions about causation have already been answered. It is then shown that a rejection of the methodological supremacy of the Method of Examples leads to a more pluralistic method of judging theories of causation, a pluralism that benefits non-standard approaches like interventionism and agency theories. (shrink)

Dispositionalism is the theory of modality according to which all (metaphysical and natural) modal truths are made true by some actual irreducibly dispositional property. The relationship between Dispositionalism and time is yet to be satisfactorily explored. In this paper we contribute to this task by examining how Dispositionalism deals with ‘dated truths’: propositions involving a specific time, e.g. “It might rain at 12.30”. We examine two possible accounts: the first, 'Dated Manifestations Strategy', is the idea that powers are very fine-grained, (...) and tend towards temporally very specific manifestations. We argue that such strategy should not be adopted, for it leads to unnecessary violations of ontological parsimony; it is unable to accommodate an ontology of platonic universals; and it is incapable of offering a principled explanation of the forward-looking nature of powers. We offer an alternative account, the "Duration Strategy", which relies on an independently determined arrow of time and the existence of some “duration facts” that specify how long a power takes to bring about its manifestation. We argue that the Duration Strategy is to be preferred. We then flesh out the account by exploring the connection between powers, duration facts, and processes. (shrink)

Causal Bayes nets (CBNs) provide one of the most powerful tools for modelling coarse-grained type-level causal structure. As in other fields (e.g., thermodynamics) the question arises how such coarse-grained characterisations are related to the characterisation of their underlying structure (in this case: token-level causal relations). Answering this question meets what is called a “coherence-requirement” in the reduction debate: How are different accounts of one and the same system (or kind of system) related to each other. We argue that CBNs as (...) tools for type-level causal inference are abstract enough to roughly fit any current token-level theory of causation as long as certain modelling assumptions are satisfied, but accounts of actual causation, i.e. accounts that attempt to infer token-causation based on CBNs, for the very same reason, face certain limitations. (shrink)

Several philosophers have embraced the view that high-level events—events like Zimbabwe's monetary policy and its hyper-inflation—are causally related if their corresponding low-level, fundamental physical events are causally related. I dub the view which denies this without denying that high-level events are ever causally related causal emergentism. Several extant philosophical theories of causality entail causal emergentism, while others are inconsistent with the thesis. I illustrate this with David Lewis's two theories of causation, one of which entails causal emergentism, the other of (...) which entails its negation. I then argue for causal emergentism on the grounds that it provides the only adequate means of squaring the apparent plenitude of causal relations between low-level events with the apparent scarcity of causal relations between high-level events. This tension between the apparent abundance of low-level causation and the apparent scarcity of high-level causation has been noted before. However, it has been thought that various theses about the semantics or the pragmatics of causal claims could be used to ameliorate the tension without going in for causal emergentism. I argue that none of the suggested semantic or pragmatic strategies meet with success, and recommend emergentist theories of causality in their stead. As Lewis's 1973 account illustrates, causal emergentism is consistent with the thesis that all facts reduce to microphysical facts. (shrink)

According to the theory developed here, we may trace out the processes emanating from a cause in such a way that any consequence lying along one of these processes counts as an effect of the cause. This theory gives intuitive verdicts in a diverse range of problem cases from the literature. Its claims about causation will never be retracted when we include additional variables in our model. And it validates some plausible principles about causation, including Sartorio's ‘Causes as Difference Makers’ (...) principle and Hitchcock's ‘Principle of Sufficient Reason’. (shrink)

I provide a theory of causation within the causal modeling framework. In contrast to most of its predecessors, this theory is model-invariant in the following sense: if the theory says that C caused (didn't cause) E in a causal model, M, then it will continue to say that C caused (didn't cause) E once we've removed an inessential variable from M. I suggest that, if this theory is true, then we should understand a cause as something which transmits deviant or (...) non-inertial behavior to its effect. (shrink)

Medical interventions that cure or prevent medical conditions are central to medicine; and thus, understanding them is central to our understanding of medicine. My purpose in this paper is to explore the conceptual foundations of medicine by providing a singular analysis of the concept of a ‘preventive or curative medical intervention’. Borrowing a general account of prevention from Phil Dowe, I provide an analysis of prevention, cure, risk reduction, and a preventive or curative intervention, before turning to preventive and curative (...) medical interventions. The resulting counterfactual-mechanistic account holds that preventive and curative medical interventions reduce the probability of a medical condition in an actual population compared to their counterfactual omission, commonly by disrupting an etiological or constitutive mechanism for the condition. (shrink)

There is a widespread belief that the so-called process theories of causation developed by Wesley Salmon and Phil Dowe have given us an original account of what causation really is. In this paper, I show that this is a misconception. The notion of “causal process” does not offer us a new ontological account of causation. I make this argument by explicating the implicit ontological commitments in Salmon and Dowe’s theories. From this, it is clear that Salmon’s Mark Transmission Theory collapses (...) to a counterfactual theory of causation, while the Conserved Quantity Theory collapses to David Fair’s phsyicalist reduction of causation. (shrink)

Our approach aims at accounting for causal claims in terms of how the physical states of the underlying dynamical system evolve with time. Causal claims assert connections between two sets of physicals states—their truth depends on whether the two sets in question are genuinely connected by time evolution such that physical states from one set evolve with time into the states of the other set. We demonstrate the virtues of our approach by showing how it is able to account for (...) typical causes, causally relevant factors, being ‘the’ cause, and cases of overdetermination and causation by absences. (shrink)

Bertrand Russell famously argued that causation is not part of the fundamental physical description of the world, describing the notion of cause as “a relic of a bygone age”. This paper assesses one of Russell’s arguments for this conclusion: the ‘Directionality Argument’, which holds that the time symmetry of fundamental physics is inconsistent with the time asymmetry of causation. We claim that the coherence and success of the Directionality Argument crucially depends on the proper interpretation of the ‘ time symmetry’ (...) of fundamental physics as it appears in the argument, and offer two alternative interpretations. We argue that: if ‘ time symmetry’ is understood as the time -reversal invariance of physical theories, then the crucial premise of the Directionality Argument should be rejected; and if ‘ time symmetry’ is understood as the temporally bidirectional nomic dependence relations of physical laws, then the crucial premise of the Directionality Argument is far more plausible. We defend the second reading as continuous with Russell’s writings, and consider the consequences of the bidirectionality of nomic dependence relations in physics for the metaphysics of causation. (shrink)