This paper provides an inferentialist account of model-based understanding by combining a counterfactual account of explanation and an inferentialist account of representation with a view of modeling as extended cognition. This account makes it understandable how the manipulation of surrogate systems like models can provide genuinely new empirical understanding about the world. Similarly, the account provides an answer to the question how models, that always incorporate assumptions that are literally untrue of the model target, can still provide factive explanations. Finally, (...) the paper shows how the contrastive counterfactual theory of explanation can provide tools for assessing the explanatory power of models. (shrink)

Once upon a time, coherentism was the dominant response to the regress problem in epistemology, but in recent decades the view has fallen into disrepute: now almost everyone is a foundationalist (with a few infinitists sprinkled here and there). In this paper, I sketch a new way of thinking about coherentism, and show how it avoids many of the problems often thought fatal for the view, including the isolation objection, worries over circularity, and concerns that the concept of coherence is (...) too vague or metaphorical for serious theoretical use. The key to my approach is to take a familiar tool from discussions of the regress problem -- namely, directed graphs depicting the support relations between beliefs -- and to use that tool in a more sophisticated manner than it is standardly employed. (shrink)

Väitöskirjassani Understanding Interests and Causal Explanation (2001) hahmottelin teoriaa yksittäisten tapahtumien kausaalisesta selittämisestä. Tässä kirjoituksessa tarkastelen niitä haasteita tai vaatimuksia, joihin teoriani yritti vastata. Alustavien huomioiden jälkeen esittelen ensiksi erityisesti selittämisen teoriaan liittyviä haasteita ja sen jälkeen yleisempiä filosofisia vaatimuksia hyväksyttävälle selittämisen teorialle.

BACKGROUND: Representing is about theories and theory formation. Philosophy of science has a long-standing interest in representing. At least since Ian Hacking's modern classic Representing and Intervening analytical philosophers have struggled to combine that interest with a study of the roles of intervention studies. With few exceptions this focus of philosophy of science has been on physics and other natural sciences. In particular, there have been few attempts to analyse the use of the notion of intervention in other disciplines where (...) intervention studies are important, such as in nursing research. One unintended consequence of this is that the relations between representing and intervening tend to be less understood outside the natural sciences. OBJECTIVES AND DESIGN: This article highlights a number of possible topics on which nursing science and analytic philosophy of science can fruitfully interact. The basic idea is simple. Building on a characterisation of interventions in terms of what is intervened on and with respect to what, we suggest that interventions in nursing research typically are a blend of varieties belonging to the three dimensions of agency, epistemology, and ontology. The details of the blend determine the relation of the particular intervention study to traditional representational categories such as inductivism and hypothetico-deductive method, and have a bearing on its explanatory power and other more theory independent features of research as well. The framework we suggest should be relevant for nurse researchers who want to adopt a more general and analytically entrenched perspective on representing and intervening than the methodological boundaries in nursing research typically allow. (shrink)

SummaryThis paper addresses the question of what scientific realism implies and what it does not when it is articulated so as to provide the best defense against plausible philosophical alternatives. A summary is presented of “abductive” arguments for scientific realism, and of the epistemological and semantic conceptions upon which they depend. Taking these arguments to be the best current defense of realism, it is inquired what, in the sense just mentioned, realism implies and what it does not. It is concluded (...) that realism implies the strong rejection of epistemological foundationalism, a non‐Humean conception of causation and of explanation, and a causal rather than conceptual account of the unity of natural definitions. It is denied that realism implies bivalence or the existence of one true theory, one preferred vocabulary or one distinctly privileged science. It is further denied that realism implies that there are no unrecognized conventional aspects to scientific theorizing and it is denied that realism implies that scientists routinely do good experimental metaphysics. (shrink)

Counterfactual conditionals have been appealed to in various ways to show how the mind can be causally efficacious. However, it has often been overestimated what the truth of certain counterfactuals actually indicates about causation. The paper first identifies four approaches that seem to commit precisely this mistake. The arguments discussed involve erroneous assumptions about the connection of counterfactual dependence and genuine causation, as well as a disregard of the requisite evaluation conditions of counterfactuals. In a second step, the paper uses (...) the insights of the foregoing analyses to formulate a set of counterfactuals-based conditions that are characterized as sufficient to establish singular causal claims. The paper concludes that there are ample reasons to believe that some mental events satisfy all these conditions with respect to certain further events and, hence, that mental events sometimes are causes. (shrink)

Causal selection is the task of picking out, from a field of known causally relevant factors, some factors as elements of an explanation. The Causal Parity Thesis in the philosophy of biology challenges the usual ways of making such selections among different causes operating in a developing organism. The main target of this thesis is usually gene centrism, the doctrine that genes play some special role in ontogeny, which is often described in terms of information-bearing or programming. This paper is (...) concerned with the attempt of confronting the challenge coming from the Causal Parity Thesis by offering principles of causal selection that are spelled out in terms of an explicit philosophical account of causation, namely an interventionist account. I show that two such accounts that have been developed, although they contain important insights about causation in biology, nonetheless fail to provide an adequate reply to the Causal Parity challenge: Ken Waters's account of actual-difference making and Jim Woodward's account of causal specificity. A combination of the two also doesn't do the trick, nor does Laura Franklin-Hall's account of explanation (in this volume). We need additional conceptual resources. I argue that the resources we need consist in a special class of counterfactual conditionals, namely counterfactuals the antecedents of which describe biologically normal interventions. (shrink)

After a brief survey of the literature on ceteris paribus clauses and ceteris paribus laws (1), the problem of exceptions, which creates the need for cp laws, is discussed (2). It emerges that the so-called skeptical view of laws of nature does not apply to laws of any kind whatever. Only some laws of physics are plagued with exceptions, not THE laws (3). Cp clauses promise a remedy, which has to be located among the further reactions to the skeptical view (...) (4). After inspecting various translations of the Latin term 'ceteris paribus' (5), the paper arrives at the conclusion that, on the most reasonable translation, there are no such things as cp laws, for reasons of logical form. Cp clauses have an indexical content, so that they need singular propositions as their habitat, not general ones. Cp clauses and the universal generalizations they are supposed to modify are not fit for each other (6). (shrink)

Przedmowa Problematyka związana z zależnościami przyczynowymi, ich modelowaniem i odkrywa¬niem, po długiej nieobecności w filozofii i metodologii nauk, budzi współcześnie duże zainteresowanie. Wiąże się to przede wszystkim z dynamicznym rozwojem, zwłaszcza od lat 1990., technik obli¬czeniowych. Wypracowane w tym czasie sieci bayesowskie uznaje się za matematyczny język przyczynowości. Pozwalają one na daleko idącą auto¬matyzację wnioskowań, co jest także zachętą do podjęcia prób algorytmiza¬cji odkrywania przyczyn. Na potrzeby badań naukowych, które pozwalają na przeprowadzenie eksperymentu z randomizacją, standardowe metody ustalania zależności przyczynowych (...) opracowano na początku XX wieku. Zupełnie inaczej sprawa przedstawia się w przypadku badań nieeksperymentalnych, gdzie podobne rozwiązania pozostają kwestią przyszłości. Zadaniem tej książki jest podanie warunków, które powinny być spełnione przez te rozwiązania, oraz sformułowanie proceduralnego kryterium zależności przy¬czynowych jako szczegółowej realizacji tych warunków. Pociąga ono waż¬kie konsekwencje dla filozofii i metodologii nauk, które ujawnia – podany w Części II – zarys me-todolo¬gii proceduralnej. W literaturze przedmiotu brakuje w miarę wszechstronnego i systema¬tycznego omówie¬nia najnowszych filozoficznych i metodologicznych dys¬kusji na temat przy¬czynowości, co niech będzie wytłumaczeniem, dlaczego w niektórych punktach obecnej książki szczegółowo referuję trudno dos¬tępne teksty źró¬dłowe. Przymiotnik „proceduralny” używam tu w znaczeniu węższym niż Huw Price (w którego pracach właściwszy byłby termin „kryterialny”) dla podkre¬ślenia – zgodnie z łacińskim źródłosłowem procedo – że dla ustalenia przy¬czyny niezbędne jest podjęcie przez uczonych określonych interakcji z ba¬daną rzeczywistością. Zalążki zamysłu prezentowanego w tej książce przedstawiłem podczas warsztatów filozoficznych „Philosophy and Probability” w roku 2002, zor¬ganizowanych przez Instytut Filozofii Uniwersytetu w Konstancji. Wdzięczny jestem uczestnikom tych warsztatów za uwagi, a przede wszyst¬kim następującym osobom: Luc Bovens, Brandon Fitelson, Alan Hájek, Stephan Hartmann oraz Jon Williamson. Podczas międzynarodowej konferencji „Analytical Pragmatism”, zorgani¬zowanej w Lublinie w roku 2003 przez Wydział Filo¬zofii Katolickiego Uniwersytetu Lubelskiego, odniosłem swoją koncepcję do prac Nancy Cartwright. Szczególnie inspirujący okazał się komentarz Huw Price’a do mojego referatu i przeprowadzona z nim dysku¬sja. Ujęcie koncepcji metodologii proceduralnej na szerszym tle współ-czes¬nego nurtu empirystycznego w filozofii nauki przedstawiłem w roku 2004 podczas konferencji „5th Quadrennial Fellows Conference”, zorgani¬zowanej przez Instytut Filozofii Uniwersytetu Jagiellońskiego oraz Centrum Filozofii Nauki w Pittsburghu. Szczególnie pomocne w dalszych moich pra¬cach były uwagi Jamesa Bogena, Janet Kourany, Jamesa Lennoxa, Johna Nortona, Thomasa Bonka, Jana Woleńskiego i Johna Worralla, za które wyra¬żam swoją wdzięczność. Korpus książki powstał podczas mojego stażu w Centrum Filozofii Nauki w Pittsburghu, który odbyłem jako stypendysta Fundacji na Rzecz Nauki Polskiej w roku akademickim 2004-2005. Uczestniczyłem w tym czasie w życiu naukowym Centrum i w pracach badawczych zespołu z Instytutu Filozofii Uniwersytetu Carnegie-Mellon w Pittsburghu kierowanego przez Clarka Glymoura. Na jego ręce składam podziękowanie za wiele po¬mocnych uwag do moich wy¬stąpień oraz tekstów i za dyskusje przede wszystkim z nim samym i z jego najbliższymi współpracownikami: Peterem Spirtesem oraz Richardem Scheinesem, a także pozostałymi członkami tego zes¬połu, doktorantami i uczestnikami seminarium badawczego „Causality in the Social Sciences”. Za wieloletnie wsparcie, wielopłaszczyznowe inspiracje towarzyszące pi¬saniu tej książki, a także liczne pomocne uwagi do jej wcześniejszych wersji dziękuję przede wszystkim Księdzu Profesorowi Andrzejowi Bronkowi oraz Księdzu Profesorowi Józefowi Herbutowi, współprowadzącemu seminarium doktorskie w Katedrze Metodologii Nauk Katolickiego Uniwersytetu Lubel¬skiego im. Jana Pawła II, jak również pozostałym uczestnikom tego semina¬rium. Dziękuję mojej Żonie, dr Annie Kawalec za wiele wysiłku włożonego w ulepszenie redakcji – językowej i merytorycznej – obecnej książki. Książkę tę można czytać na kilka sposobów. Czytelnikom zainteresowa¬nym przede wszystkim prowadzeniem badań empirycznych polecałbym rozpoczęcie od Rozdziału 2. i kontynuację pozostałych rozdziałów Części I, a następnie Dodatków. Czytelnikom zainteresowanym problemami filozo¬fii i metodologii nauk polecałbym rozpoczęcie lektury książki od Części II i uzupełniającą lekturę Rozdziału 2., a następnie Wprowadzenia i Zakończenia. Czytelnikom mniej zainteresowanym zagadnieniami teoretycznymi pole¬całbym zapoznanie się z fascynującymi dziejami odkrycia przyczyn cholery przez Johna Snowa, które rekonstruuję w Rozdziale 1. W dalszej części nato¬miast polecałbym przejście do Wprowadzenia i Zakończenia, które w mniej specjalistyczny sposób przybliżają proponowane tu rozstrzygnięcia. Tekst książki nie był dotąd publikowany. Wyjątkiem są pewne fragmenty Rozdziału 8. oraz 9., które w zmienionej postaci ukazały się w Rocznikach Filozoficznych (Kawalec 2004). -/- Lublin, luty 2006 r. (shrink)

We argue in favor of merely disjunctive effects, namely cases in which an event or fact, C, is not a cause of an effect, E1, and is also not a cause of a distinct effect, E2, and yet C is a cause of the disjunctive effect (E1 orE2). Disjunctive effects let us retain the additivity and the distributivity of causation. According to additivity, if C is a cause of E1 and C is a cause of E2, then C is a (...) cause of E1 and E2. According to distributivity, if C is a cause of E1 and E2, then C is a cause of E1 and C is a cause of E2. We draw an analogy between causation and intensional notions like believing, wanting, and owing, which also admit of merely disjunctive cases. We argue that both the Lewisian counterfactual account of causation (including its recent emendation by Sartorio) and the contrastive account of causation fail to properly account for this phenomenon. (shrink)

Bertrand Russell famously argued that causation is not part of the fundamental physical description of the world, describing the notion of cause as “a relic of a bygone age”. This paper assesses one of Russell’s arguments for this conclusion: the ‘Directionality Argument’, which holds that the time symmetry of fundamental physics is inconsistent with the time asymmetry of causation. We claim that the coherence and success of the Directionality Argument crucially depends on the proper interpretation of the ‘ time symmetry’ (...) of fundamental physics as it appears in the argument, and offer two alternative interpretations. We argue that: if ‘ time symmetry’ is understood as the time -reversal invariance of physical theories, then the crucial premise of the Directionality Argument should be rejected; and if ‘ time symmetry’ is understood as the temporally bidirectional nomic dependence relations of physical laws, then the crucial premise of the Directionality Argument is far more plausible. We defend the second reading as continuous with Russell’s writings, and consider the consequences of the bidirectionality of nomic dependence relations in physics for the metaphysics of causation. (shrink)

The authors examined cue competition effects in young children using the blicket detector paradigm, in which objects are placed either singly or in pairs on a novel machine and children must judge which objects have the causal power to make the machine work. Cue competition effects were found in a 5- to 6-year-old group but not in a 4-year-old group. Equivalent levels of forward and backward blocking were found in the former group. Children's counterfactual judgments were subsequently examined by asking (...) whether or not the machine would have gone off in the absence of I of 2 objects that had been placed on it as a pair. Cue competition effects were demonstrated only in 5- to 6-year-olds using this mode of assessing causal reasoning. (shrink)

In this thesis, I give a metascientific account of causality in medicine. I begin with two historical cases of causal discovery. These are the discovery of the causation of Burkitt’s lymphoma by the Epstein-Barr virus, and of the various viral causes suggested for cervical cancer. These historical cases then support a philosophical discussion of causality in medicine. This begins with an introduction to the Russo- Williamson thesis (RWT), and discussion of a range of counter-arguments against it. Despite these, I argue (...) that the RWT is historically workable, given a small number of modifications. I then expand Russo and Williamson’s account. I first develop their suggestion that causal relationships in medicine require some kind of evidence of mechanism. I begin with a number of accounts of mechanisms and produce a range of consensus features of them. I then develop this consensus position by reference to the two historical case studies with an eye to their operational competence. In particular, I suggest that it is mechanistic models and their representations which we are concerned with in medicine, rather than the mechanism as it exists in the world. -/- I then employ these mechanistic models to give an account of the sorts of evidence used in formulating and evaluating causal claims. Again, I use the two human viral oncogenesis cases to give this account. I characterise and distinguish evidence of mechanism from evidence of difference-making, and relate this to mechanistic models. I then suggest the relationship between types of evidence presents us with a means of tackling the reference-class problem. This sets the scene for the final chapter. Here, I suggest the manner in which these two different classes of evidence become integrated is also reflected in the way that developing research programmes change as their associated causal claims develop. (shrink)

I focus on token, deterministic causal claims as they feature in causal explanations. Adequately handling absences is difficult for most causal theories, including theories of causal explanation. Yet so is adequately handling cases of late preemption. The best account of absence-causal claims as they appear in causal explanations is Jonathan Schaffer's quaternary, contrastive account. Yet Schaffer's account cannot handle preemption. The account that best handles late preemption is James Woodward's interventionist account. Yet Woodward's account is inadequate when it comes to (...) absences. I propose an account that handles both absences and preemption by transposing Schaffer's account into an interventionist framework. (shrink)

The Linda paradox is a key topic in current debates on the rationality of human reasoning and its limitations. We present a novel analysis of this paradox, based on the notion of verisimilitude as studied in the philosophy of science. The comparison with an alternative analysis based on probabilistic confirmation suggests how to overcome some problems of our account by introducing an adequately defined notion of verisimilitudinarian confirmation.

We present a theory of actions based on a theory of events in branching time, in which "particular" or "token" actions are taken to be sets of transitions from their initial states to the outcomes. We also present a simple theory of composition of events by which composite events can be formed out of other events. Various kinds of actions, including instantaneous group actions and sequential group actions, are introduced by way of composition, and an extended stit theory of agency (...) is proposed, in which the stit operators are combined or equipped with reified group actions. (shrink)

Defenders of pragmatic encroachment in epistemology (or what I call practicalism) need to address two main problems. First, the view seems to imply, absurdly, that knowledge can come and go quite easily—in particular, that it might come and go along with our variable practical interests. We can call this the stability problem. Second, there seems to be no fully satisfying way of explaining whose practical interests matter. We can call this the “whose stakes?” problem. I argue that both problems can (...) be addressed in roughly the same terms. More exactly, I argue that by first clarifying the whose stakes? problem an answer to the stability problem naturally falls out. (shrink)

This paper critiques the new mechanistic explanatory program on grounds that, even when applied to the kinds of examples that it was originally designed to treat, it does not distinguish correct explanations from those that blunder. First, I offer a systematization of the explanatory account, one according to which explanations are mechanistic models that satisfy three desiderata: they must 1) represent causal relations, 2) describe the proper parts, and 3) depict the system at the right ‘level.’ Second, I argue that (...) even the most developed attempts to fulfill these desiderata fall short by failing to appropriately constrain explanatorily apt mechanistic models. -/- *This paper used to be called "The Emperor's New Mechanisms". (shrink)

This article delineates some of the main issues that are debated by philosophers of law. It explores the connections between legal philosophy and other areas of philosophy, while also seeking to specify the distinctiveness of many of the concerns that have preoccupied philosophers of law. It illustrates its abstract points with examples focused on the separability of law and morality, the nature of the rule of law, the nature of rights, justifications for the imposition of punishment, and the identification of (...) basic legal entitlements. (shrink)

This paper will examine the nature of mechanisms and the distinction between the relevant and irrelevant parts involved in a mechanism’s operation. I first consider Craver’s account of this distinction in his book on the nature of mechanisms, and explain some problems. I then offer a novel account of the distinction that appeals to some resources from Mackie’s theory of causation. I end by explaining how this account enables us to better understand what mechanisms are and their various features.

In Essays on the Active Powers, Thomas Reid offers two different accounts of motives. According to the first, motives are the ends for which we act. According to the second, they are mental states, such as desires, that incite us to action. These two accounts, I claim, do not fit comfortably with Reid's agent causal account of human action. My project in this article is to explain why and then to propose a strategy for reconciling these two accounts with Reid's (...) views about action. (shrink)

This paper provides a discussion and defense of a recent formulation of the idea that moral responsibility for actions depends on the capacity to respond to reasons. This formulation appears in several publications by John Martin Fischer and Mark Ravizza, where the authors argue that moral responsibility involves a kind of control over one's actions which they call "guidance control." This kind of control does not require an agent's ability to do something different from what he actually does, but instead (...) requires only that the actual process leading to the action be responsive in some suitable way to the reasons that the agent has for acting. After summarizing this view, I offer the following two innovations to the authors' view: I argue that the level of control required for moral responsibility (which I call "regular reasons-responsiveness") is much stronger than what the author's view allows for; and 2) I give a common-sense account of the kinds of motivational mechanism relevant to moral responsibility. Given these innovations, I show that this kind of view allows us to easily answer some counterexamples that appear in the current literature on moral responsibility. (shrink)

It is argued in this paper that although much attention has been paid to causal chains and common causes within the literature on probabilistic causality, a primary virtue of that approach is its ability to deal with cases of multiple causation. In doing so some ways are indicated in which contemporary sine qua non analyses of causation are too narrow (and ways in which probabilistic causality is not) and an argument by Reichenbach designed to provide a basis for the asymmetry (...) of causation is refined. The importance of referring causal claims to an abstract model is also emphasized. (shrink)

The prevention, treatment and management of disease are closely linked to how the causes of a particular disease are explained. For multi-factorial conditions, the causal explanations are inevitably complex and competing models may exist to explain the same condition. Selecting one particular causal explanation over another will carry practical and ethical consequences that are acutely relevant for health policy. In this paper our focus is two-fold; the different models of causal explanation that are put forward within current scientific literature for (...) the high and rising prevalence of the common complex conditions of coronary artery disease and type 2 diabetes mellitus ; and how these explanations are taken up within national health policy guidelines. We examine the causal explanations for these two conditions through a systematic database search of current scientific literature. By identifying different causal explanations we propose a three-tier taxonomy of the most prominent models of explanations: evolutionary, lifecourse, and lifestyle and environment. We elaborate this taxonomy with a micro-level thematic analysis to illustrate how some explanations are semantically and rhetorically foregrounded over others. We then investigate the uptake of the scientific causal explanations in health policy documents with regard to the prevention and management recommendations of current National Service Frameworks for CAD and T2D. Our findings indicate a lack of congruence between the complexity and frequent overlap of causal explanations evident in the scientific literature and the predominant focus on lifestyle recommendations found in the mainstream health policy documents. (shrink)

Backtracking influence is influence that zigzags in time. For example, backtracking influence exists when an event E_1 makes an event E_2 more likely by way of a nomic connection that goes from E_1 back in time to an event C and then forward in time to E_2. I contend that in our local region of spacetime, at least, backtracking influence is redundant in the sense that any existing backtracking influence exerted by E_1 on E_2 is equivalent to E_1's temporally direct (...) influence on E_2. I prove the redundancy of backtracking influence using several plausible physical principles without assuming any fundamental temporal or causal asymmetry. This explanation can play a prominent role in an account of why causation appears to be objectively asymmetric regardless of whether the fundamental laws are symmetric. (shrink)

I conduct an empirical analysis of the temporally asymmetric character of our epistemic access to the world by providing an experimental scheme whose results represent the core empirical content of the epistemic asymmetry. I augment this empirical content by formulating a gedanken experiment inspired by a proposal from David Albert. This second experiment cannot be conducted using any technology that is likely to be developed in the foreseeable future, but the expected results help us to state an important constraint on (...) our ability to know the future. Finally, I show that a third experiment concerning precognition, described by Michael Scriven and John Mackie, does not characterize any additional empirical content but does help to illustrate why it is unlikely that any precognition exists. (shrink)

Quine argues that if sentences that are set theoretically equivalent are interchangeable salva veritate, then all transparent operators are truth-functional. Criticisms of this argument fail to take into account the conditional character of the conclusion. Quine also argues that, for any person P with minimal logical acuity, if ‘belief’ has a sense in which it is a transparent operator, then, in that sense of the word, P believes everything if P believes anything. The suggestion is made that he intends that (...) result to show us that ‘believes’ has no transparent sense. Criticisms of this argument are either based on unwarranted assertions or on definitions of key terms that depart from Quine’s usage of those terms. (shrink)

I argue, contrary to standard claims, that accomplice liability need not be a causal relation. One can be an accomplice to another’s crime without causally contributing to the criminal act of the principal. This is because the acts of aid and encouragement that constitute the basis for accomplice liability typically occur in contexts of under- and over-determination, where causal analysis is confounded. While causation is relevant to justifying accomplice liability in general, only potential causation is necessary in particular cases. I (...) develop this argument through the example of the role of U.S. legal officials in abetting the acts of unlawful interrogation that have taken place since 2001. I also suggest that there may be a limited justification for ex post ratificatory accomplice liability. (shrink)

Sydney Shoemaker's "Subset Account" offers a new take on determinable properties and the realization relation as well as a defense of non-reductive physicalism from the problem of mental causation. At the heart of this account are the claims that (1) mental properties are determinable properties and (2) the causal powers that individuate a determinable property are a proper subset of the causal powers that individuate the determinates of that property. The second claim, however, has led to the accusation that the (...) effects caused by the instantiation of a determinable property will also be caused by the instantiation of the determinates of that property—so instead of solving the problem of mental causation, the Subset Account ends up guaranteeing that the effects of mental properties (and all other types of determinable property) will be causally overdetermined! In this paper, I explore this objection. I argue that both sides in this debate have failed to engage the question at the heart of the objection: Given that both a determinable property and its determinates have the power to cause some effect (E), does it follow that both will actually cause E when the relevant conditions obtain? To make genuine progress towards answering this question, we need to take a serious look at the metaphysics of causation. With the debate properly reframed and issues about the metaphysics of causation front and center, I explore the question of whether the Subset Account is doomed to result in problematic causal overdetermination. (shrink)

Hume thought that if you believed in powers, you believed in necessary connections in nature. He was then able to argue that there were none such because anything could follow anything else. But Hume wrong-footed his opponents. A power does not necessitate its manifestations: rather, it disposes towards them in a way that is less than necessary but more than purely contingent. -/- In this paper a dispositional theory of causation is offered. Causes dispose towards their effects and often produce (...) them. But a set of causes, even though they may succeed in producing an effect, cannot necessitate it since the effect could have been counteracted by some additional power. This would require a separation of our concepts of causal production and causal necessitation. The most conspicuous cases of causation are those where powers accumulate and pass a requisite threshold for an effect to occur. -/- We develop a model for representing powers as constituent vectors within an n-dimensional quality space, where composition of causes appears as vector addition. Even our resultant vector, however, has to be understood as having dispositional force only. This model throws new light on causal modality and cases of prevention, causation by absence and probabilistic causation. (shrink)

Cases of overdetermination or preemption continue to play an important role in the debate about the proper interpretation of causal claims of the form "C was a cause of E". I argue that the best treatment of preemption cases is given by Mackie's venerable INUS account of causal claims. The Mackie account suffers, however, from problems of its own. Inspired by its ability to handle preemption, I propose a dramatic revision to the Mackie account – one that Mackie himself would (...) certainly have rejected – to solve these difficulties. The result is, I contend, a very attractive account of singular causal claims. (shrink)

Externalist theories of justification create the possibility of cases in which everything appears to one relevantly similar with respect to two propositions, yet one proposition is justified while the other is not. Internalists find this difficult to accept, because it seems irrational in such a case to affirm one proposition and not the other. The underlying internalist intuition supports a specific internalist theory, Phenomenal Conservatism, on which epistemic justification is conferred by appearances.

Philip Kitcher has argued for a causal correspondence view of truth, as against a deflationary view, on the grounds that the former is better poised than the latter to explain systematically successful patterns of action. Though Kitcher is right to focus on systematically successful action, rather than singular practical successes, he is wrong to conclude that causal correspondence theories are capable of explaining systematic success. Rather, I argue, truth bears no explanatory relation to systematic practical success. Consequently, the causal correspondence (...) view is not in a better position to explain success than the deflationary view; theories of truth are the wrong place to look for explanations of systematic practical success. (shrink)

Much of the modern philosophy of causation has been governed by two ideas: (i) causes make their effects inevitable; (ii) a cause is something that makes a difference to whether its effect occurs. I focus on explaining the origin of idea (ii) and its connection to (i). On my view, the frequent attempts to turn (ii) into an analysis of causation are wrongheaded. Patterns of difference-making aren't what makes causal claims true. They merely provide a useful test for causal claims. (...) Moreover, what justifies us in using them as a test is idea (i). That's how (i) and (ii) are connected. (shrink)

Laws in the special sciences are usually regarded to be non-universal. A theory of laws in the special sciences faces two challenges. (I) According to Lange's dilemma, laws in the special sciences are either false or trivially true. (II) They have to meet the ?requirement of relevance?, which is a way to require the non-accidentality of special science laws. I argue that both challenges can be met if one distinguishes four dimensions of (non-) universality. The upshot is that I argue (...) for the following explication of special science laws: L is a special science law just if (1) L is a system law, (2) L is quasi-Newtonian, and (3) L is minimally invariant. (shrink)

This paper examines the relation between Cartwright's concept of 'capacities' and Mill's concept of 'tendencies' and argues that they are not equivalent. Cartwright's concept of 'capacities' and her motivation to adopt it as a central notion in her philosophy of science are described. It is argued that the Millian concept of 'tendencies' is distinct because Mill restricts its use to a set of special cases. These are the cases in which causes combine 'mechanically'. Hence for Mill 'tendencies' do not merely (...) describe the operation of causes, but also and perhaps even primarily how they combine. This does not hold of Cartwrightian capacities which are meant to have unlimited applicability. Mill also explicitly denies the realism of 'capacities' in a sense which overlaps with Cartwright's. Her attempt to derive empirist credentials for her notion of capacity from Mill is therefore unsuccessful. (shrink)

One of the most recent controversies to arise in the field of bioethics concerns the ethics for the Groningen Protocol: the guidelines proposed by the Groningen Academic Hospital in The Netherlands, which would permit doctors to actively euthanise terminally ill infants who are suffering. The Groningen Protocol has been met with an intense amount of criticism, some even calling it a relapse into a Hitleresque style of eugenics, where people with disabilities are killed solely because of their handicaps. The purpose (...) of this paper is threefold. First, the paper will attempt to disabuse readers of this erroneous understanding of the Groningen Protocol by showing how such a policy does not aim at making quality-of-life judgements, given that it restricts euthanasia to suffering and terminally ill infants. Second, the paper illustrates that what the Groningen Protocol proposes to do is both ethical and also the most humane alternative for these suffering and dying infants. Lastly, responses are given to some of the worries expressed by ethicists on the practice of any type of non-voluntary active euthanasia. (shrink)

Recent research on “causal cognition” in adults and infants shows that we can perceive singular causal relations not previously experienced. In particular, infants that are able to perceive causality seem to rely on innate beliefs and principles that allow a priori inference of a connection between cause and effect. Can causal cognition in infants justify the thesis of causal realism? On the one hand, it weakens the central pillar of the Humean arguments: the impossibility of a synthetic a priori causal (...) inference. On the other hand, if perception is the privileged way of justifying the reality of objects of the external world, that is valid in the case of causal relations as well. Moreover, the perception of causal relations, based on innate principles and beliefs, reflects the selective results of the interaction between the real constraints of the physical structure of the world and the evolution of the human mind. (shrink)

Causal claims are context sensitive. According to the old orthodoxy (Mackie 1974, Lewis 1986, inter alia), the context sensitivity of causal claims is all due to conversational pragmatics. According to the new contextualists (Hitchcock 1996, Woodward 2003, Maslen 2004, Menzies 2004, Schaffer 2005, and Hall ms), at least some of the context sensitivity of causal claims is semantic in nature. I want to discuss the prospects for causal contextualism, by asking why causal claims are context sensitive, what they are sensitive (...) to, and where they are sensitive to it. (shrink)

Negative causation occurs when an absence serves as cause, effect, or causal intermediary. Negative causation is genuine causation, or so I shall argue. It involves no physical connection between cause and effect. Thus causes need not be physically connected to their effects.

Causation and the laws of nature are nothing over and above the pattern of events, just like a movie is nothing over and above the sequence of frames. Or so I will argue. The position I will argue for is broadly inspired by Hume and Lewis, and may be expressed in the slogan: what must be, must be grounded in what is.

Essence and causation are fundamental in metaphysics, but little is said about their relations. Some essential properties are of course causal, as it is essential to footprints to have been caused by feet. But I am interested less in causation's role in essence than the reverse: the bearing a thing's essence has on its causal powers. That essencemight make a causal contribution is hinted already by the counterfactual element in causation; and the hint is confirmed by the explanation essence offers (...) of something otherwise mysterious, namely, how events exactly alike in every ordinary respect, like the bolt'ssuddenly snapping and its snapping per se, manage to disagree in what they cause. Some prior difference must exist between these events to make their causal powers unlike. Paradoxically, though, it can only be in point of a property, suddenness, which both events possess in common. Only by postulating a difference in themanner — essential or accidental — of the property's possession is the paradox resolved. Next we need an account of causation in which essence plays an explicit determinative role. That account, based on the idea that causes should becommensurate with their effects, is thatx causesy only if nothing essentially poorer would have done, and nothing essentially richer was needed. (shrink)

This article deals with a type of functional explanation, viability explanation, that has been overlooked in recent philosophy of science. Viability explanations relate traits of organisms and their environments in terms of what an individual needs to survive and reproduce. I show that viability explanations are neither causal nor historical and that, therefore, they should be accounted for as a distinct type of explanation.

Causation is widely assumed to be a binary relation: c causes e. I will argue that causation is a quaternary, contrastive relation: c rather than C* causes e rather than E*, where C* and E* are nonempty sets of contrast events. Or at least, I will argue that treating causation as contrastive helps resolve some paradoxes.