There are many putative counterexamples to the view that all scientific explanations are causal explanations. Using a new theory of what it is to be a causal explanation, Bradford Skow has recently argued that several of the putative counterexamples fail to be non-causal. This paper defends some of the counterexamples by showing how Skow’s argument relies on an overly permissive theory of causal explanations.

Leibniz is often cited as an authority when it comes to the formulation and answer strategy of the question “Why is there something rather than nothing?” Yet much current research assumes that Leibniz advocates an unambiguous question and strategy for the answer. In this respect, one repeatedly finds the argument in the literature that alternative explanatory approaches to this question violate Leibniz’s intention, since he derives the question from the principle of sufficient reason and also demands a causal explanation (...) to the question. In particular, the new research on non-causal explanatory strategies to the Leibniz question seems to concern this counter-argument. In this paper, however, I will argue that while Leibniz raises the question by means of the principle of sufficient reason, he even favours a non-causal explanatory strategy to the question. Thus, a more accurate Leibniz interpretation seems not only to legitimise but also to support non-causal explanations to the Leibniz question. (shrink)

In non-causal explanations, some non-causal facts (such as mathematical, modal or metaphysical) are used to explain some physical facts. However, precisely because these explanations abstract away from causal facts, they face two challenges: 1) it is not clear why would one rather than the other non-causal explanantia be relevant for the explanandum; and 2) why would standing in a particular explanatory relation (e.g., “counterfactual dependence”, “constraint”, “entailment”, “constitution”, “grounding”, and so on), and not in some other, (...) be explanatory. I develop an explanatory relevance account which is based on erotetic constraints and show how it addresses these two challenges. (shrink)

Can an identity be the proper subject of an explanation? A popular stance, albeit not one often argued for, gives a negative answer to this question. Building from a contentious passage from Jaegwon Kim in this direction, we reconstruct an argument to the conclusion that identities, to the extent in which they are necessary, cannot be explained. The notion of contrastive explanation, characterized as difference-seeking, will be crucial for this argument; however, we will eventually find the argument to be unsatisfactory. (...) On the contrary, the discussion provides enough resource to sketch a very simple framework for a non-causal contrastive explanation of identities. Many instances will be provided, with different varieties of explanans, ultimately suggesting that certain entailment or biconditional principles involving identities (first and foremost, so-called two-level identity criteria) may indeed be taken to have an inherent explanatory value. (shrink)

In the last couple of years, a few seemingly independent debates on scientific explanation have emerged, with several key questions that take different forms in different areas. For example, the questions what makes an explanation distinctly mathematical and are there any non-causal explanations in sciences (i.e., explanations that don’t cite causes in the explanans) sometimes take a form of the question of what makes mathematical models explanatory, especially whether highly idealized models in science can be explanatory and in virtue (...) of what they are explanatory. These questions raise further issues about counterfactuals, modality, and explanatory asymmetries: i.e., do mathematical and non-causal explanations support counterfactuals, and how ought we to understand explanatory asymmetries in non-causal explanations? Even though these are very common issues in the philosophy of physics and mathematics, they can be found in different guises in the philosophy of biology where there is the statistical interpretation of the Modern Synthesis theory of evolution, according to which the post-Darwinian theory of natural selection explains evolutionary change by citing statistical properties of populations and not the causes of changes. These questions also arise in philosophy of ecology or neuroscience in regard to the nature of topological explanations. The question here is can the mathematical (or more precisely topological) properties in network models in biology, ecology, neuroscience, and computer science be explanatory of physical phenomena, or are they just different ways to represent causal structures. The aim of this special issue is to unify all these debates around several overlapping questions. These questions are: are there genuinely or distinctively mathematical and non-causal explanations?; are all distinctively mathematical explanations also non-causal; in virtue of what they are explanatory; does the instantiation, implementation, or in general, applicability of mathematical structures to a variety of phenomena and systems play any explanatory role? This special issue provides a platform for unifying the debates around several key issues and thus opens up avenues for better understanding of mathematical and non-causal explanations in general, but also, it will enable even better understanding of key issues within each of the debates. (shrink)

Implicit contextual factors mean that the boundary between causal and noncausal explanation is not as neat as one might hope: as the phenomenon to be explained is given descriptions with varying degrees of granularity, the nature of the favored explanation alternates between causal and non-causal. While it is not surprising that different descriptions of the same phenomenon should favor different explanations, it is puzzling why re-describing the phenomenon should make any difference for the causal nature of (...) the favored explanation. I argue that this is a problem for the ontic framework of causal and noncausal explanation, and instead propose a pragmatic-modal account of causal and non-causal explanation. This account has the added advantage of dissolving several important disagreements concerning the status of non-causal explanation. (shrink)

This thesis provides an account of acquaintance with abstract objects. The notion of acquaintance is integral to theorising on reference and singular thought, since it is generally taken to be the relation that must exist between a subject and an object, in order for the subject to refer to, and entertain singular thoughts about the object. The most common way of understanding acquaintance is as a form of causal connection. However, this implies a problem. We seem to be able (...) to refer and have singular thoughts about abstract objects. But given that abstract objects are causally inert, this would mean that we are unable to become acquainted with them. This problem shall be the focus of this thesis. I first argue that these traditional causal interpretations of acquaintance are lacking. Instead, I show that acquaintance is dependent to some degree on factors internal to the subject, namely the skills that they possess. From doctors to sommeliers to mathematicians (and possibly even philosophers!) – these subjects seem to succeed in becoming acquainted with certain objects precisely in virtue of their respective skills. Thus, building off from Evans’ The Varieties of Reference, I present a novel account of acquaintance, which I term as Skill-based Acquaintance (SBA). On SBA, a subject is said to be acquainted with an object when they possess discriminating knowledge of that object, gained through the use of their capacities and skills. The SBA account is applied to virtual (Ch.3), fictional (Ch.4), and mathematical objects (Ch.5), as well as God (Ch.6). SBA is successful in explaining how subjects can indeed become acquainted with these problematic categories of objects - some of which are abstract – thus being able to refer and entertain singular thoughts about them. Overall, then, SBA is shown to have greater explanatory power than competing accounts and should thus be preferred. (shrink)

Lange’s collection of expanded, mostly previously published essays, packed with numerous, beautiful examples of putatively non-causal explanations from biology, physics, and mathematics, challenges the increasingly ossified causal consensus about scientific explanation, and, in so doing, launches a new field of philosophic investigation. However, those who embraced causal monism about explanation have done so because appeal to causal factors sorts good from bad scientific explanations and because the explanatory force of good explanations seems to derive from revealing (...) the relevant causal (or ontic) structures. The taxonomic project of collecting examples and sorting their types is an essential starting place for a theory of non-causal explanation. But the title of Lange’s book requires something further: showing that the putative explanations are, in fact, explanatory and revealing the non-causal source of their explanatory power. This project is incomplete if there are examples of putative non-causal explanations that fit the form but that nobody would accept as explanatory (absent a radical revision of intuitions). Here we provide some reasons for thinking that there are such examples. (shrink)

In this BA dissertation, I deploy examples of non-causal explanations of physical phenomena as evidence against the view that causal models of explanation can fully account for explanatory practices in science. I begin by discussing the problems faced by Hempel’s models and the causal models built to replace them. I then offer three everyday examples of non-causal explanation, citing sticks, pilots and apples. I suggest a general form for such explanations, under which they can be phrased (...) as inductive-statistical arguments incorporating plausible assumptions. I then show the applicability of this form to explanatory practices in thermal physics. I explore the possibility that population genetics provides a similar form of explanation, and offer a novel defence of the statistical interpretation of population genetics proposed by Matthen and Ariew (2002). I close with remarks concerning how, when faced with competing causal and non-causal explanations of the same phenomenon, we can perform an Inference to the Best Explanation. (shrink)

This dissertation focuses on teleology and functions in biology. More precisely, it focuses on the scientific legitimacy of teleofunctional attributions and explanations in biology. It belongs to a multi-faceted debate that can be traced back to at least the 1970s. One aspect of the debate concerns the naturalization of functions. Most authors try to reduce, translate or explain functions and teleology in terms of efficient causes so that they find their place in the framework of the natural sciences. Our approach (...) here is radically different, as we question the premise that teleological explanations are disguised causal explanations. On the contrary, we defend that they are acceptable in natural sciences and in biology in particular. We challenge the idea that teleological explanations are immature causal explanations, as well as the idea that teleology and functions are some sort of intentional explanations. We therefore reject the accusations of anthropomorphism, vitalism and finalism. On the contrary, we defend that teleology, causality and intentionality correspond to different, autonomous and complementary modes of representation of the outside world. -/- ———[FRANÇAIS]——— -/- Ce travail de thèse porte sur la téléologie et les fonctions en biologie. Plus précisément, il porte sur la légitimité scientifique des attributions et des explications téléofonctionnelles en biologie. Il s’inscrit dans le cadre d’un débat à plusieurs facettes que l’on peut faire remonter au moins jusqu’aux années 1970 et qui est encore très actif aujourd’hui. L’un des aspects du débat porte sur la naturalisation des fonctions, c’est-à-dire sur la manière de les réduire, de les traduire ou de les expliciter en termes de causes efficientes de sorte qu’elles trouvent leur place dans le cadre des sciences de la nature. Notre approche ici est radicalement différente, car nous remettons en question la prémisse selon laquelle les explications téléologiques seraient des explications causales déguisées. Nous défendons au contraire qu’elles sont acceptables en tant que telles aussi bien de façon générale que dans le domaine scientifique et en particulier en biologie. De façon générale, nous remettons en question l’idée selon laquelle la pensée téléologique serait une pensée causale immature, ainsi que sa dépendance présumée vis-à-vis de la psychologie, c’est-à-dire de l’intentionnalité. Nous rejetons donc les accusations d’anthropomorphisme, de vitalisme et de finalisme formulées contre elle. Nous défendons au contraire que la téléologie, la causalité et l’intentionnalité correspondent à des modes différents, autonomes et complémentaires de représentation du monde extérieur. (shrink)

There is a major debate as to whether there are non-causal mathematical explanations of physical facts that show how the facts under question arise from a degree of mathematical necessity considered stronger than that of contingent causal laws. We focus on Marc Lange’s account of distinctively mathematical explanations to argue that purported mathematical explanations are essentially causal explanations in disguise and are no different from ordinary applications of mathematics. This is because these explanations work not by appealing (...) to what the world must be like as a matter of mathematical necessity but by appealing to various contingent causal facts. (shrink)

Several authors have used the notion of causal specificity in order to defend non-parity about genetic causes (Waters 2007, Woodward 2010, Weber 2017, forthcoming). Non-parity in this context is the idea that DNA and some other biomolecules that are often described as information-bearers by biologists play a unique role in life processes, an idea that has been challenged by Developmental Systems Theory (e.g., Oyama 2000). Indeed, it has proven to be quite difficult to state clearly what the alleged special (...) role of genetic causes consists in. In this paper, I show that the set of biomolecules that are normally considered to be information-bearers (DNA, mRNA) can be shown to be the most specific causes of protein primary structure, provided that causal specificity is measured over a relevant space of biological possibilities, disregarding physical as well as logically possible states of the causal variables. (shrink)

A finer-grained delineation of a given explanandum reveals a nexus of closely related causal and non- causal explanations, complementing one another in ways that yield further explanatory traction on the phenomenon in question. By taking a narrower construal of what counts as a causal explanation, a new class of distinctively mathematical explanations pops into focus; Lange’s characterization of distinctively mathematical explanations can be extended to cover these. This new class of distinctively mathematical explanations is illustrated with the (...) Lotka-Volterra equations. There are at least two distinct ways those equations might hold of a system, one of which yields straightforwardly causal explanations, but the other of which yields explanations that are distinctively mathematical in terms of nomological strength. In the first, one first picks out a system or class of systems, finds that the equations hold in a causal -explanatory way; in the second, one starts with the equations and explanations that must apply to any system of which the equations hold, and only then turns to the world to see of what, if any, systems it does in fact hold. Using this new way in which a model might hold of a system, I highlight four specific avenues by which causal and non- causal explanations can complement one another. (shrink)

Can the persistence of a human being's soul at death and prior to the bodily resurrection be sufficient to guarantee that the resurrected human being is numerically identical to the human being who died? According to Thomas Aquinas, it can. Yet, given that Aquinas holds that the human being is identical to the composite of soul and body and ceases to exist at death, it's difficult to see how he can maintain this view. In this paper, I address Aquinas's response (...) to this objection . After making a crucial clarification concerning the nature of the non-repeatability principle on which the objection relies, I argue that the contemporary notion of immanent causal relations provides us with a way of understanding Aquinas's defence that renders it both highly interesting and philosophically plausible. (shrink)

We plead for a fluid margin, or mixed/indeterminate buffer zone, between Physical and Non-Physical Causal Closures, and for a Neutrosophic Causal Closure Principle claiming that the chances of all physical effects are determined by their prior partially physical and partially non-physical causes.

A key challenge for probabilistic causal models is to distinguish non-causal probabilistic dependencies from true causal relations. To accomplish this task, causal models are usually required to satisfy several constraints. Two prominent constraints are the causal Markov condition and the faithfulness condition. However, other constraints are also needed. One of these additional constraints is the causal sufficiency condition, which states that models must not omit any direct common causes of the variables they contain. In (...) this paper, I argue that the causal sufficiency condition is problematic: (1) it is incompatible with the requirement that the variables in a model must not stand in non-causal necessary dependence relations, such as mathematical or conceptual relations, or relations described in terms of supervenience or grounding, (2) it presupposes more causal knowledge as primitive than is actually needed to create adequate causal models, and (3) if models are only required to be causally sufficient, they cannot deal with cases where variables are probabilistically related by accident, such as Sober’s example of the relationship between bread prices in England and the sea level in Venice. I show that these problems can be avoided if causal models are required to be monotonic in the following sense: the causal relations occurring in a model M would not disappear if further variables were added to M. I give a definition of this monotonicity condition and conclude that causal models should be required to be monotonic rather than causally sufficient. (shrink)

The principle of causality is one of the most fundamental principles that has been discovered in the history of philosophy and science. Several foundations revolve around this concept. The importance of this principle in classical physics lies in giving physicists the ability to predict phenomena. Furthermore, due to causality is recognized as a fundamental principle in classical physics. With the introduction of the principle of uncertainty, the principle of causality is empirically called into question. Because the claim of the principle (...) of uncertainty in quantum mechanics is that the relationships between fundamental particles are not causally related to each other, and even the behavior of an electron or a subatomic particle is not based on the principle of causality. If we want to identify the speed of particles, we will not be able to identify their state, and if we want to determine their state, we will not be able to identify their speed. The best way to resolve this conflict is to bring the discussion into philosophy, which is exactly what has been done in Islamic philosophy. The concept of causality in Western philosophy seems to be based on Newtonian concepts. But what has been stated in Islamic philosophy is based on metaphysical concepts, and therefore the principle of uncertainty cannot contradict the concept of causality in Islamic philosophy, especially what has been discussed in the philosophy of Avicenna and Allameh Tabataba'i. (shrink)

When agents violate norms, they are typically judged to be more of a cause of resulting outcomes. In this paper, we suggest that norm violations also affect the causality attributed to other agents, a phenomenon we refer to as "causal superseding." We propose and test a counterfactual reasoning model of this phenomenon in four experiments. Experiments 1 and 2 provide an initial demonstration of the causal superseding effect and distinguish it from previously studied effects. Experiment 3 shows that (...) this causal superseding effect is dependent on a particular event structure, following a prediction of our counterfactual model. Experiment 4 demonstrates that causal superseding can occur with violations of non-moral norms. We propose a model of the superseding effect based on the idea of counterfactual sufficiency. (shrink)

Some non-reductionists claim that so-called ‘exclusion arguments’ against their position rely on a notion of causal sufficiency that is particularly problematic. I argue that such concerns about the role of causal sufficiency in exclusion arguments are relatively superficial since exclusionists can address them by reformulating exclusion arguments in terms of physical sufficiency. The resulting exclusion arguments still face familiar problems, but these are not related to the choice between causal sufficiency and physical sufficiency. The upshot is that (...) objections to the notion of causal sufficiency can be answered in a straightforward fashion and that such objections therefore do not pose a serious threat to exclusion arguments. (shrink)

Philippe Huneman has recently questioned the widespread application of mechanistic models of scientific explanation based on the existence of structural explanations, i.e. explanations that account for the phenomenon to be explained in virtue of the mathematical properties of the system where the phenomenon obtains, rather than in terms of the mechanisms that causally produce the phenomenon. Structural explanations are very diverse, including cases like explanations in terms of bowtie structures, in terms of the topological properties of the system, or in (...) terms of equilibrium. The role of mathematics in bowtie structured systems and in topologically constrained systems has recently been examined in different papers. However, the specific role that mathematical properties play in equilibrium explanations requires further examination, as different authors defend different interpretations, some of them closer to the new-mechanistic approach than to the structural model advocated by Huneman. In this paper, we cover this gap by investigating the explanatory role that mathematics play in Blaser and Kirschner’s nested equilibrium model of the stability of persistent long-term human-microbe associations. We argue that their model is explanatory because: i) it provides a mathematical structure in the form of a set of differential equations that together satisfy an ESS; ii) that the nested nature of the ESSs makes the explanation of host-microbe persistent associations robust to any perturbation; iii) that this is so because the properties of the ESS directly mirror the properties of the biological system in a non-causal way. The combination of these three theses make equilibrium explanations look more similar to structural explanations than to causal-mechanistic explanation. (shrink)

It has seemed, to many, that there is an important connection between the ways in which some theoretical posits explain our observations, and our reasons for being ontologically committed to those posits. One way to spell out this connection is in terms of what has become known as the explanatory criterion of ontological commitment. This is, roughly, the view that we ought to posit only those entities that are indispensable to our best explanations. Our primary aim is to argue that (...) the moral nonnaturalist places herself in an invidious position if she simply accepts that the nonnatural moral facts that she posits are not explanatory. Instead, we offer the nonnaturalist an alternative strategy for dealing with moral explanations. The strategy is to retain the explanatory criterion of ontological commitment and maintain that moral properties are, in fact, explanatory. The explanations they provide are not causal explanations; instead, moral properties make a non-causal difference to the physical facts. (shrink)

Metaethical realists disagree about the nature of normative properties. Naturalists think that they are ordinary natural properties: causally efficacious, a posteriori knowable, and usable in the best explanations of natural and social sciences. Non-naturalist realists, in contrast, argue that they are sui generis: causally inert, a priori knowable and not a part of the subject matter of sciences. It has been assumed so far that naturalists can explain causally how the normative predicates manage to refer to normative properties, whereas non-naturalists (...) are unable to provide equally satisfactory metasemantic explanations. This article first describes how the previous non-naturalist accounts of reference fail to tell us how the normative predicates could have come to refer to the non-natural properties rather than to the natural ones. I will then use the so-called qua-problem to show how the causal theories of reference of naturalists also fail to fix the reference of normative predicates to unique natural properties. Finally, I will suggest that, just as naturalists need to rely on the non-causal mechanism of reference magnetism to solve the previous problem, non-naturalists, too, can rely on the very same idea to respond to the pressing metasemantic challenges that they face concerning reference. (shrink)

We develop a new version of the causal theory of spacetime. Whereas traditional versions of the theory seek to identify spatiotemporal relations with causal relations, the version we develop takes causal relations to be the grounds for spatiotemporal relations. Causation is thus distinct from, and more basic than, spacetime. We argue that this non-identity theory, suitably developed, avoids the challenges facing the traditional identity theory.

Bertrand Russell once pointed out that modern science doesn’t deal with causal laws and that assuming otherwise is not only wrong but such thinking is erroneously thought to do no harm. However, looking into the scientific practice of simulation or experimentation reveals a general causal comprehension of physical processes. In this paper I trace causal experiences to the existence of innate causal capacity by which we organize sensory information. This capacity, I argue, is something we have (...) got in virtue of natural selection as can be seen from experiments with intelligent animals like crows and chimpanzees. So understanding the empirical world is impossible without the use of causal categories. The reason why Russell believed that modern science does not refer to causal laws is, I think, because he argued that the laws of mathematical physics give us a non-causal description of reality. In contrast to such a claim I hold that theoretical laws are prescriptive rules of description rather than descriptions themselves. (shrink)

The chapter explores whether, or to what extent, recent work in experimental philosophy puts pressure on the idea that the concept of causation is ‘egalitarian’. Causal selection – where experimental subjects tend to rate the causal strength of (for example) a norm-violator more strongly than a non-norm-violator – is a well established phenomenon, and is in prima facie tension with an egalitarian conception of causation; it also, indirectly, puts prima facie pressure on the idea that causation is a (...) worldly phenomenon whose obtaining is independent of facts about norms. The chapter explores both the various psychological mechanisms and the broadly pragmatic approaches to explaining causal selection. It argues that the answer to the question whether or not the concept of causation is egalitarian is currently significantly empirically underdetermined, and suggests some avenues for further investigation. (shrink)

Patient-centered care (PCC) is an approach to healthcare that values patients’ preference, need, and autonomy. The estimation of healthcare partly depends on how well PCC is implemented. In addition, the result of clinical research can inform the assessment of the implementation of PCC. In clinical research, health-related quality of life (HRQL) theoretical models offer a conceptual toolbox that informs clinical research and guides the hypotheses generation. Wilson and Cleary (1995) developed the most widely used HRQL theoretical model (Bakas et al., (...) 2012). Ontological assumptions about causation in Wilson and Cleary’s model will influence which kind of hypotheses will be generated. I will argue that Wilson and Cleary’s model instilled a kind of causal bias into hypothesis generation in clinical research on HRQL. Causation from biomedical factors to non-biomedical factors is frequently hypothesized while causation from non-biomedical factors to biomedical factors is rarely hypothesized. It leads to that the interdependence and interaction between constituent parts of patients are ignored, which is an obstacle to the implementation of PCC. In addition, I will propose a revised HRQL theoretical model which avoids the causal bias brought by Wilson and Cleary’s model. By doing so, I leave room for the improvement of the practice of healthcare by analyzing the ontological assumptions about causation. (shrink)

Michael Devitt develops a hybrid causal theory of non-empty proper names and certain singular terms that semantically resemble them. He considers that the use of a name designates an object not by virtue of the different information we know about it, but by a causal network that starts from the first uses of the name to designate the object, through a "reference borrowing" from the previous uses. DOI: 10.13140/RG.2.2.35828.50564.

Non-reductive physicalists hold that mental properties are realized by physical properties. The realization relation is typically taken to be a metaphysical necessitation relation. Here, I explore how the metaphysical necessitation feature of realization can be explained by what is known as ‘the subset view’ of realization. The subset view holds that the causal powers that are associated with a realized property are a proper subset of the causal powers that are associated with the realizer property. I argue that (...) the said explanation of the metaphysical necessitation feature requires a careful treatment of the relationship between properties and causal powers. (shrink)

I discuss an important feature of the notion of cause in Post. An. 1. 13, 78b13–28, which has been either neglected or misunderstood. Some have treated it as if Aristotle were introducing a false principle about explanation; others have understood the point in terms of coextensiveness of cause and effect. However, none offers a full exegesis of Aristotle's tangled argument or accounts for all of the text's peculiarities. My aim is to disentangle Aristotle's steps to show that he is arguing (...) in favour of a logical requirement for a middle term's being the appropriate cause of its explanandum. Coextensiveness between the middle term and the attribute it explains is advanced as a sine qua non condition of a middle term's being an appropriate or primary cause. This condition is not restricted either to negative causes or to middle terms in second‐figure syllogisms, but ranges over all primary causes qua primary. (shrink)

I examine the meaning and merits of a premise in the Exclusion Argument, the causal closure principle that all physical effects have physical causes. I do so by addressing two questions. First, if we grant the other premises, exactly what kind of closure principle is required to make the Exclusion Argument valid? Second, what are the merits of the requisite closure principle? Concerning the first, I argue that the Exclusion Argument requires a strong, “stringently pure” version of closure. The (...) latter employs two qualifications concerning the physical sufficiency and relative proximity of the physical cause required for every physical effect. The second question is addressed in two steps. I begin by challenging the adequacy of the empirical support offered by David Papineau for closure. Then I assess the merits of “level” and “domain” versions of stringently pure closure. I argue that a domain version lacks adequate and non-question-begging support within the context of the Exclusion Argument. And I argue that the level version leads to a puzzling metaphysics of the physical domain. Thus, we have grounds for rejecting the version of closure required for the Exclusion Argument. This means we can resist the Exclusion Argument while avoiding the implausible implications that come with rejecting one of its other premises. That is, because there are grounds to reject causal closure, one can reasonably affirm the non-overdeterminative causal efficacy of conscious mental states while denying that the latter are identical with physical states. (shrink)

Causal selection has to do with the distinction we make between background conditions and “the” true cause or causes of some outcome of interest. A longstanding consensus in philosophy views causal selection as lacking any objective rationale and as guided, instead, by arbitrary, pragmatic, and non-scientific considerations. I argue against this position in the context of causal selection for disease traits. In this domain, causes are selected on the basis of the type of causal control they (...) exhibit over a disease of interest. My analysis clarifies the principled rationale that guides this selection and how it involves both pragmatic and objective considerations, which have been overlooked in the extant literature. (shrink)

Many believe that because we are so small, we must be utterly insignificant on the cosmic scale. But whether this is so depends on what it takes to be important. On one view, what matters for importance is the difference to value that something makes. On this view, what determines our cosmic importance is not our size, but what else of value is out there. But a rival view also seems plausible: that importance requires sufficient causal impact on the (...) relevant scale; since we have no such impact on the grand scale, that would entail our cosmic insignificance. I argue that despite appearances, causal impact is neither necessary nor sufficient for importance. All that matters is impact on value. Since parts can have non-causal impact on the value of the wholes that contain them, this means that we might have great impact on the grandest scale without ever leaving our little planet. (shrink)

The paper argues that on three out of eight possible hypotheses about the EPR experiment we can construct novel and realistic decision problems on which (a) Causal Decision Theory and Evidential Decision Theory conflict (b) Causal Decision Theory and the EPR statistics conflict. We infer that anyone who fully accepts any of these three hypotheses has strong reasons to reject Causal Decision Theory. Finally, we extend the original construction to show that anyone who gives any of the (...) three hypotheses any non-zero credence has strong reasons to reject Causal Decision Theory. However, we concede that no version of the Many Worlds Interpretation (Vaidman, in Zalta, E.N. (ed.), Stanford Encyclopaedia of Philosophy 2014) gives rise to the conflicts that we point out. (shrink)

Many common arguments for physicalism begin with the principle that the cosmos is “causally closed.” But how good are the arguments for causal closure itself? I argue that the deductive, a priori arguments on behalf of causal closure tend to beg the question. The extant inductive arguments fare no better. They commit a sampling error or a non-sequitur, or else offer conclusions that remain compatible with causal openness. In short, we have no good arguments that the physical (...) world is causally closed. (shrink)

I defend the view, hardly original with me, that there is no evidence, deductive or non-deductive, for any of our causal beliefs, that does not already assume that there are some causal connections, and hence that there is no way in which experience on its own, or with causalität-free principles, can support the structure of out causal knowledge. The deductive case is perhaps obvious. In the case of non-deductive arguments, I consider how experience of constant conjunctions, together (...) with the employment of an inference to the best explanation, might lead to a causal conclusion. But I argue that such an inference would need as a premiss that the causal explanation of the constant conjunction is the best explanation of that evidence, and ‘best’ in this context requires the thought that the causal hypothesis best fits with our existing causal beliefs. (shrink)

The development of causal modelling since the 1950s has been accompanied by a number of controversies, the most striking of which concerns the Markov condition. Reichenbach's conjunctive forks did satisfy the Markov condition, while Salmon's interactive forks did not. Subsequently some experts in the field have argued that adequate causal models should always satisfy the Markov condition, while others have claimed that non-Markovian causal models are needed in some cases. This paper argues for the second position by (...) considering the multi-causal forks, which are widespread in contemporary medicine (Section 2). A non-Markovian causal model for such forks is introduced and shown to be mathematically tractable (Sections 6, 7, and 8). The paper also gives a general discussion of the controversy about the Markov condition (Section 1), and of the related controversy about probabilistic causality (Sections 3, 4, and 5). (shrink)

For over 20 years, Jaegwon Kim’s Causal Exclusion Argument has stood as the major hurdle for non-reductive physicalism. If successful, Kim’s argument would show that the high-level properties posited by non-reductive physicalists must either be identical with lower-level physical properties, or else must be causally inert. The most prominent objection to the Causal Exclusion Argument—the so-called Overdetermination Objection—points out that there are some notions of causation that are left untouched by the argument. If causation is simply counterfactual dependence, (...) for example, then the Causal Exclusion Argument fails. Thus, much of the existing debate turns on the issue of which account of causation is appropriate. In this paper, however, I take a bolder approach and argue that Kim’s preferred version of the Causal Exclusion Argument fails no matter what account one gives of causation. Any notion of causation that is strong enough to support the premises of the argument is too strong to play the role required in the logic of the argument. I also consider a second version of the Causal Exclusion Argument, and suggest that although it may avoid the problems of the first version, it begs the question against a particular form of non-reductive physicalism, namely emergentism. (shrink)

Current debates surrounding the virtues and shortcomings of randomization are symptomatic of a lack of appreciation of the fact that causation can be inferred by two distinct inference methods, each requiring its own, specific experimental design. There is a non-statistical type of inference associated with controlled experiments in basic biomedical research; and a statistical variety associated with randomized controlled trials in clinical research. I argue that the main difference between the two hinges on the satisfaction of the comparability requirement, which (...) is in turn dictated by the nature of the objects of study, namely homogeneous or heterogeneous populations of biological systems. Among other things, this entails that the objection according to which randomized experiments fail to provide better evidence for causation because randomization cannot guarantee comparability is mistaken. (shrink)

The systems studied in the special sciences are often said to be causally autonomous, in the sense that their higher-level properties have causal powers that are independent of the causal powers of their more basic physical properties. This view was espoused by the British emergentists, who claimed that systems achieving a certain level of organizational complexity have distinctive causal powers that emerge from their constituent elements but do not derive from them. More recently, non-reductive physicalists have espoused (...) a similar view about the causal autonomy of special-science properties. They argue that since these properties can typically have multiple physical realizations, they are not identical to physical properties, and further they possess causal powers that differ from those of their physical realisers. Despite the orthodoxy of this view, it is hard to find a clear exposition of its meaning or a defence of it in terms of a well-motivated account of causation. In this paper, we aim to address this gap in the literature by clarifying what is implied by the doctrine of the causal autonomy of special-science properties and by defending the doctrine using a prominent theory of causation from the philosophy of science. (shrink)

I use a contrastive theory of causal explanation to analyze the notion of a genetic trait. The resulting definition is relational, an implication of which is that no trait is genetic always and everywhere. Rather, every trait may be either genetic or non-genetic, depending on explanatory context. I also outline some other advantages of connecting the debate to the wider causation literature, including how that yields us an account of the distinction between genetic traits and genetic dispositions.

Many philosophers believe that there is a causal condition on perception, and that this condition is a conceptual truth about perception. A highly influential argument for this claim is based on intuitive responses to Gricean-style thought experiments. Do the folk share the intuitions of philosophers? Roberts et al. (2016) presented participants with two kinds of cases: Blocker cases (similar to Grice’s case involving a mirror and a pillar) and Non-Blocker cases (similar to Grice’s case involving a clock and brain (...) stimulation). They found that a substantial minority agreed that seeing occurs in the Non-Blocker cases, and that in the Blocker cases significantly less agreed that seeing occurs. They thus hypothesized that folk intuitions better align with a no blocker condition than with a causal condition. This paper continues this line of enquiry with two new experiments. The paper investigates the generality and robustness of Roberts et al.’s findings by expanding the sense modalities tested from only vision to audition and olfaction as well. The paper also uses Gricean-style thought experiments as a case study for investigating the “reflection defense” against the negative project in experimental philosophy. Our results replicate and extend Roberts et al.’s study and support their hypothesis that folk intuitions better align with a no blocker condition. They also provide an empirical reason to doubt the reflection defense. (shrink)

Several of Thomas Aquinas's proofs for the existence of God rely on the claim that causal series cannot proceed in infinitum. I argue that Aquinas has good reason to hold this claim given his conception of causation. Because he holds that effects are ontologically dependent on their causes, he holds that the relevant causal series are wholly derivative: the later members of such series serve as causes only insofar as they have been caused by and are effects of (...) the earlier members. Because the intermediate causes in such series possess causal powers only by deriving them from all the preceding causes, they need a first and non-derivative cause to serve as the source of their causal powers. (shrink)

Aristotle grants perceptible qualities the power to generate sense perception in animals. But it is unclear whether, for him, these qualities can produce any effect other than perception. In this paper I address this issue through a novel approach. To show that they can produce non-perceptual effects, I explore contexts in his extant works where qualities appear to do causal work in nature without leading to perception in animals. This inquiry aims to demonstrate that Aristotle’s realism about qualities survives (...) a potential threat posed by views that dispute the qualities’ causal efficacy outside perception. (shrink)

Unlike any other monograph on legal liability, Michael S. Moore’s book CAUSATION AND RESPONSIBILITY contains a well-informed and in-depth discussion of the metaphysics of causation. Moore does not share the widespread view that legal scholars should not enter into metaphysical debates about causation. He shows respect for the subtleties of philosophical debates on causal relata, identity conditions for events, the ontological distinctions between events, states of affairs, facts and tropes, and the counterfactual analysis of event causation, and he considers (...) all these issues relevant to law. In this contribution, I defend an amended version of the COUNTERFACTUAL theory of event causation both against Moore’s criticism and against some traits of Lewis’s version of the theory. On a number of counts, I simply defend Lewis against Moore’s misdirected criticism. Moore’s unreasonable demand that all the commonplaces about causation have to FOLLOW from the counterfactual analysis rests on his mistaken claim that the counterfactual theory IDENTIFIES causation with counterfactual dependence. In other respects, I part company with Lewis in order to highlight some underrated strengths of the counterfactual approach. The common denominator of my revisions is that they make the counterfactual theory unambiguously singularist. The suggested revisions comprise: performing the counterfactual analysis with Davidsonian rather than with Lewisian events, taking seriously the ex post-character of singular causal statements, making explicit the indexical ceteris paribus clause that fixes the actual circumstances of the causing event, abandoning the alleged transitivity of causation, and, last but not least, doing everything in the correct order. The correct order is to start with bedrock intuitions about the nature of causality rather than with a general possible world semantics, and then to turn these intuitions into constraints for the class of counterfactuals that a counterfactual analysis of causation has to consider. (shrink)

It is natural to think that our ordinary practices in giving explanations for our actions, for what we do, commit us to claiming that content properties are causally relevant to physical events such as the movements of our limbs and bodies, and events which these in turn cause. If you want to know why my body ambulates across the street, or why my arm went up before I set out, we suppose I have given you an answer when I say (...) that I wanted to greet a friend on the other side of the street, and thought that my arm's going up would be interpreted by him as a signal to stop for a moment. This widely held view might be disputed, but I shall not argue for it in this paper. I want to start with the view that our beliefs and desires and other propositional attitudes are causally relevant, in virtue of their modes and particular contents, to our movements, in order to investigate the consequences for analyses of thought content. For this purpose, I argue, in sec. II, for three necessary conditions on causal relevance: (a) a nomic sufficiency condition, (b) a logical independence condition, and (c) a screening-off condition. In sec. III, I apply these conditions to relational and functional theories of thought content, arguing that these theories cannot accommodate the causal relevance of content properties to our behaviour. I argue further that, on two plausible assumptions, one about the dependence of the mental on the physical, and the other about the availability in principle of causal explanations of our movements in terms of our non-relational physical properties, content properties can be causally relevant only if they are nomically type-correlated, relative to certain circumstances, with non-relational physical properties of our bodies. In sec. IV, I respond to a number of objections that might be advanced against this conclusion. (shrink)

The thesis explores and suggests a solution to a problem that I identify in John McDowell’s and Lynne Rudder Baker’s approaches to mental and intention-dependent (ID) causation in the physical world. I begin (chapter 1) with a brief discussion of McDowell’s non-reductive and anti-scientistic account of mind and world, which I believe offers, through its vision of the unbounded conceptual and the world as within the space of reasons, to liberate and renew philosophy. However, I find an inconsistency in McDowell’s (...) criticism of Davidson’s anomalous monism (chapter 2), stemming from a tension between McDowell’s naïve common sense view of mental causation and an understanding of ordinary physical causation which I think McDowell ought to embrace, which portrays it as both objective, in the sense of being recognition- independent, and as belonging within the space of reasons. The question of the relation between these two concepts of causation is an aspect of the more general question of the relation between the space of reasons and the realm of law. In chapter 3 I begin examining the possibility that Baker’s account of material and property constitution could form the basis of a bridge between the two spaces, and find it generally promising. However, I find that her defence of her version of non-reductive monism against Kim’s causal arguments also runs into problems, which I attribute to the fact that she holds a view of causation as secondary to causal explanation. 2 3 In chapter 4 I develop an account of what I call manifest physical causation – of objective causal relations in the world of Sellars’s manifest image. Based upon McDowell’s transcendental empiricism, which takes the existence of the ordinary perceived world as a condition of the possibility of our possession of conceptual capacities, I contend that it is this picture of reality, rather than that of fundamental physics, that should be the starting point of our theorizing. Causation in the manifest image, I argue, covers the behaviour of the familiar physical world as well as that of its well-understood extensions into the special sciences and engineering. Manifest physical causation, on my account, is productive, acts through mechanisms which are almost entirely mechanical, electromagnetic, and/ or chemical, and is causally closed. In my view, normative, semantic, contentful property-instances are not part of the manifest physical causal nexus. In my final chapter I suggest a modification of Baker’s constitution account, which I call Constituted Causation, whereby higher-level – mental and other ID – causal relations are constituted, in favourable circumstances, by lower-level ones. ID causal relations belong in their own causal nexus but are connected to the manifest physical world through constitution, a relation of unity without identity. Causation and constitution are, respectively, intra- and inter-level relations, and they are non-overlapping. The constituted network of rational and normative relations bears, I believe, striking parallels with McDowell’s view of reality. According to Baker’s view of constitution, the essential properties of constituted entities subsume those of their constituters; extending this to my account enables us to say that the real cause and explanation of someone’s action is that they consciously performed it 4 rather than that certain manifest causal processes occurred at the lower level. (shrink)

A well-known trilemma faces the interpretation of Kant’s theory of affection, namely whether the objects that affect us are empirical, noumenal, or both. I argue that according to Kant, the things that affect us and cause representations in us are not empirical objects. I articulate what I call the Causal Power Argument, according to which empirical objects cannot affect us because they do not have the right kind of power to cause representations. All the causal powers that empirical (...) objects have are moving powers, and such powers can only have spatial effects. According to Kant, however, the representations that arise in us as a result of the affection of our sensibility are non-spatial. I show that this argument is put forward by Kant in a number of passages, and figures as a decisive reason for rejecting empirical affection and instead endorsing affection by the things in themselves. (shrink)

In a previous article on cosmological arguments, I have put forward a few examples of complete infinite and circular explanations, and argued that complete non-wellfounded explanations such as these might explain the present state of the world better than their well-founded theistic counterparts (Billon, 2021). Although my aim was broader, the examples I gave there implied merely causal explanations. In this article, I would like to do three things: • Specify some general informative conditions for complete and incomplete non-wellfounded (...) causal explanations that can be used to assess candidate explanations and to generate new examples of complete non-wellfounded explanations. • Show that these conditions, which concern chains of causal explanations, easily generalize to chains of metaphysical, grounding explanations and even to chains involving other “determination relations” such as supervenience. • Apply these general conditions to the recent debates against the existence of nonwellfounded chains of grounds and show, with a couple of precise examples, that the latter can be complete, and that just like in the case of causal explanations, non-wellfoundedness can in fact be an aset rather than a liability. (shrink)